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1 y a ring of adhesion molecules (a peripheral supramolecular activation cluster).
2 aling ceases when they fuse with the central supramolecular activation cluster.
3 ules in membrane microdomains defined as the supramolecular activation cluster.
4 t is lacking other components of the central supramolecular activation cluster.
5 forms actomyosin arcs within the peripheral supramolecular activation cluster, a region correspondin
6 preferentially' transported into the central supramolecular activation cluster after activation, whic
7 lls, only effector cells developed a central supramolecular activation cluster as defined by punctate
8 PKCtheta participates in the assembly of the supramolecular activation clusters at the contact site o
10 cruit protein kinase C-theta; to the central supramolecular activation cluster (c-SMAC) region of the
11 tivation cluster, surrounded by a peripheral supramolecular activation cluster containing F-actin.
12 ested that the inability to form the central supramolecular activation cluster (cSMAC) could underlie
13 lived protein kinase Ctheta-focusing central supramolecular activation cluster (cSMAC) have been defi
14 in kinase Ctheta focusing within the central supramolecular activation cluster (cSMAC) of the immunol
15 CD28 is colocalized with TCR in the central supramolecular activation cluster (cSMAC) region of the
16 ot required for the formation of the central supramolecular activation cluster (cSMAC) region, but do
17 CRs) clustered centrally to form the central supramolecular activation cluster (cSMAC), and centrosom
18 15-30 minutes, KIR2DL2 moved to the central supramolecular activation cluster (cSMAC), colocalizing
19 immunological synapses containing a central supramolecular activation cluster (cSMAC), suggesting th
20 ocalizes in the T cell IS within the central supramolecular activation cluster (cSMAC), where it asso
21 en CD28 clusters are segregated from central supramolecular activation cluster (cSMAC)-localized CD3
27 1, Jagged1) cluster in their apposed central-supramolecular-activation-clusters (cSMAC), whereas DC-N
28 showing that PD-L1 localizes to the central supramolecular activation cluster, decreases antiviral C
29 s with characteristic central and peripheral supramolecular activation cluster domains that were indi
30 now provide direct evidence that the distal supramolecular activation cluster (dSMAC) and peripheral
31 , a bull's-eye pattern composed of a central supramolecular activation cluster enriched with T cell r
32 this response includes integrin activation, supramolecular activation cluster formation, Ca(2+) flux
34 potentiate T cell activation cSMAC: central supramolecular activation cluster Immunoreceptor tyrosin
35 hanced and prolonged CD4 accumulation at the supramolecular activation cluster in the contact area ha
36 tion of signal transduction molecules in the supramolecular activation clusters in the MHC class I-re
37 ults suggest that the formation of a central supramolecular activation cluster is not required for ac
38 mic rearrangement of proteins into organized supramolecular activation clusters is driven primarily b
39 unologic synapse, in contrast to the central supramolecular activation cluster localization of PKC th
40 formation of these new, spatially segregated supramolecular activation clusters may generate appropri
41 contacts the plasma membrane at the central supramolecular activation cluster of the immunological s
42 colocalizes with CD3 and CD81 at the central supramolecular activation cluster of the T cell immune s
43 found in both the central and the peripheral supramolecular activation clusters of the anergic synaps
44 lecules are further sorted to the peripheral supramolecular activation cluster or central supramolecu
45 er of the immunological synapse (the central supramolecular activation cluster or cSMAC) is controver
46 ar activation cluster (dSMAC) and peripheral supramolecular activation cluster (pSMAC) correspond to
47 LFA-1-ICAM-1 interactions in the peripheral supramolecular activation cluster (pSMAC) of the immunol
48 composed of an adhesion ring, the peripheral supramolecular activation cluster (pSMAC), and a central
51 lity complex (MHC) complex pSMAC: peripheral supramolecular activation cluster PTK: protein tyrosine
52 nd localization of talin into the peripheral supramolecular activation cluster region and exclusion o
53 supramolecular activation cluster or central supramolecular activation cluster regions of the immunol
54 M)-1 binding is followed by formation of the supramolecular activation cluster (SMAC) at the immunolo
55 ating NKIS contains a central and peripheral supramolecular activation cluster (SMAC), and includes p
56 kinases, assemble into spatially segregated supramolecular activation clusters (SMACs) at the area o
57 tablished by T cells is the formation of the supramolecular activation clusters (SMACs), in which adh
60 e induced clustering of NKG2D at the central supramolecular activation cluster, surrounded by a perip
61 ule that colocalizes with the TCR within the supramolecular activation cluster, thus indicating effic
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