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1  of myelinated and unmyelinated axons in the sural and medial plantar nerves that immunostain for sub
2 hat appeared similar to myelinated fibres in sural and sciatic nerve.
3  by random regeneration in direct suture and sural cable graft groups but not in nerve guide repairs
4              Punches removed from tibial- or sural-innervated planter paw skin were immunolabeled to
5                               Contralesional sural-innervated skin had neither neurite loss nor sprou
6                         Adjacent ipsilateral sural-innervated skin had persistent hyperalgesia withou
7  staining of lateral plantar nerve (LPN) and sural nerve (SN) motor terminals, using the activity-dep
8 nd the F-wave latency (p=0.03) decreased and sural nerve action potential amplitude increased (p=0.04
9  Changes in excitability of the terminals of sural nerve afferents were used to confirm that both loc
10 ation of myelinated axons in both the distal sural nerve and nerves of the toe.
11 ical stimulation of afferent C fibers in the sural nerve and recorded from single neurons in the vent
12                           Examination of the sural nerve and the auditory nerve adjacent to the brain
13 ibres in the skin or myelinated axons in the sural nerve and toe after vincristine.
14  was evoked by electrical stimulation of the sural nerve and was recorded in the ipsilateral hamstrin
15               Stimulation of the ipsilateral sural nerve at the malleolus, just before stimulation of
16 ibres was a uniform feature in a total of 21 sural nerve biopsies and 'onion bulb' formations and/or
17 ith diabetic neuropathy progression in human sural nerve biopsies and describe their potential utilit
18                                              Sural nerve biopsies from 7 patients with diabetic neuro
19                                              Sural nerve biopsies from six affected individuals and t
20 -4 (CD152) at the protein and mRNA levels in sural nerve biopsies of patients with chronic inflammato
21 eased PMP22 messenger RNA levels in skin and sural nerve biopsies of patients with CMT1A compared wit
22 and nuclear imaging, electroencephalography, sural nerve biopsies, sleep evaluation and neuropsychome
23 y, we analysed PMP22 messenger RNA levels in sural nerve biopsies.
24 3-3 proteins measurement, skin, muscular and sural nerve biopsies.
25                                            A sural nerve biopsy from an affected patient showed marke
26 lectrophysiologic data were evaluated, and a sural nerve biopsy from one affected child was examined
27 tain circumstances replace the more invasive sural nerve biopsy in the morphological and molecular ev
28              Electrodiagnostic studies and a sural nerve biopsy showed features of a dystrophic axona
29                                              Sural nerve biopsy showed mild to moderate selective los
30                       Of the 6 who underwent sural nerve biopsy, 4 had selective loss of small myelin
31 decrease in the size of myelinated fibres on sural nerve biopsy.
32  infected and 2 uninfected animals underwent sural nerve biopsy.
33  area of the antidromic volley evoked in the sural nerve by intraspinal microstimulation in the L4/5
34 unmyelinated fiber function in the hind paw, sural nerve C-fiber morphometry, sciatic nerve neurotrop
35     Nociceptive nerve function, unmyelinated sural nerve fiber and dorsal root ganglion (DRG) cell mo
36          Nerve gaps were bridged by either a sural nerve graft or a biodegradable collagen nerve guid
37                  Enthusiasm has declined for sural nerve grafting because of the associated complexit
38 can be achieved with minimal morbidity using sural nerve grafts, which surgeons commonly use to recon
39 n of a single unit occurred naturally in the sural nerve in some cases.
40 n axonal diameter of myelinated axons in the sural nerve in untreated diabetic rats.
41                                  Because the sural nerve innervates hairy skin, these data suggest th
42             Schwann cells derived from human sural nerve may provide a valuable source of tissue for
43          A loss of > or =500 fibers/mm(2) in sural nerve MFD over 52 weeks was defined as progressing
44                          RESEARCH DESIGN AND Sural nerve myelinated fiber density (MFD), nerve conduc
45                         Neither the proximal sural nerve nor the motor tibial nerve exhibit axon loss
46 M phosphorylation by 2.5-fold (P < 0.001) in sural nerve of BB rats.
47  increases in total levels of p38 and JNK in sural nerve of type I and II diabetic patients.
48                                          The sural nerve of untreated diabetic rats showed a 50% decr
49                   The clinical phenotype and sural nerve pathology in these two families differs in s
50                                     Detailed sural nerve pathology is presented in both cases.
51   Stimulation of the gastrocnemius nerve and sural nerve revealed significant convergence of muscle a
52                               In this study, sural nerve segments from individuals with PMP22 duplica
53 n delivered to tibialis anterior (TA) MNs by sural nerve stimulation.
54 ts were seen with field potentials evoked by sural nerve stimulation.
55 p) elicited by electrical stimulation of the sural nerve was measured in six normal adults.
56 were exposed and in situ recordings from the sural nerve were performed to determine compound C-fiber
57                                  Sections of sural nerve were removed from amputated legs of patients
58                                       In the sural nerve, 20% of the unmyelinated axon profiles immun
59 lation of a 54-kDa isoform of JNK in DRG and sural nerve, and this correlated with elevated c-Jun and
60 ipsilateral foot, and also the contralateral sural nerve, decreased presynaptic inhibition.
61                                       In the sural nerve, where the large majority of myelinated fibe
62 egment of the medial cutaneous branch of the sural nerve.
63 0.01) and mean axonal size (P < 0.05) in the sural nerve.
64 ers and profuse regenerative activity in the sural nerve.
65 he peroneal nerve, the tibial nerve, and the sural nerve.
66 on was also increased in the DRG, but not in sural nerve.
67 10 sensory potentials were newly observed in sural nerves after treatment.
68 pathies and compared these with normal human sural nerves and those from patients with Guillain-Barre
69 iments were performed on 50 samples of human sural nerves collected during a 52-week clinical trial.
70 tation in mtDNA was more prevalent in distal sural nerves compared to dorsal root ganglia.
71 urofilament distances (NNND) in the axons of sural nerves from patients with anti-MAG paraproteinaemi
72 e sensory latency of both the left and right sural nerves improved on the basis of faster median cond
73 f HIV-SN, freshly isolated mitochondria from sural nerves of macaques infected with a neurovirulent s
74 mtDNA common deletion mutation in postmortem sural nerves of patients with HIV-SN as compared to unin
75 s from four cryopreserved normal adult human sural nerves referenced to the Genome Reference Consorti
76 icantly lower C-fiber conduction velocity in sural nerves than uninfected animals and the magnitude o
77            JAM-C was also expressed in human sural nerves with an expression profile similar to that
78                              In normal human sural nerves, axonal NNND was correlated with axonal dia
79       In the largely cutaneous saphenous and sural nerves, the naked mole-rat had the lowest C:A-fibe
80 Unit isolation was by partial section of the sural or lateral plantar nerves.
81  large myelinated nerve fibers, specifically sural or sciatic nerve conduction velocities, but signif
82 hy, metabolic syndrome associated with lower sural sensory nerve action potential amplitudes.
83 oral dispersion and conduction block, (5) no sural sparing, (6) greater number of fibrillation potent
84  dispersion and conduction block, and absent sural sparing.

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