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1 s the acquisition of antigens by B cells via surface immunoglobulin.
2 A and protein increase with cross-linking of surface immunoglobulin.
3 ess the B-lineage commitment factor Pax5 and surface immunoglobulins.
4 ines of GC origin identified 6 lines lacking surface immunoglobulin, a phenotype never seen among lin
5 e phosphorylation and became associated with surface immunoglobulin and CD79b, arguing for the involv
6 ative signaling in which co-cross-linking of surface immunoglobulin and Fc gammaRIIB1 results in sequ
7 in B cells and rendered them susceptible to surface immunoglobulin- and chemotherapeutic agent-induc
8 ymphoma cells with an antibody against their surface immunoglobulin (anti-Ig) induces apoptosis and h
11 induced in some cell lines upon ligation of surface immunoglobulin by mechanisms that include the ac
13 fection by tetradecanoyl phorbol acetate and surface immunoglobulin cross-linking and is activated by
15 utant virus was able to establish latency in surface immunoglobulin D-negative (sIgD(-)) B cells; by
17 signaling competence, BTK independence, and surface immunoglobulin dependence of the PLCG2 mutation
18 lerant B cells in the diminished capacity of surface immunoglobulin engagement to produce Fas resista
20 r (BCR)-type tumors that exhibited selective surface immunoglobulin expression and sensitivity to ent
21 with reduced percentages of both CD4(+) and surface immunoglobulin G-positive lymphocytes in the spl
23 e that expressed a mutant transgene encoding surface immunoglobulin (Ig), but which did not permit th
24 late times postinfection with cross-linking surface immunoglobulin (Ig), in conjunction with anti-CD
25 ach patient's tumor expressing a unique cell surface immunoglobulin (Ig), or B-cell receptor (BCR), t
27 rved that crosslinking, by antibody, of cell surface immunoglobulin induced G1 growth-arrest followed
28 Crosslinking of the B cell antigen receptor surface immunoglobulin induces tyrosine phosphorylation
30 Barr virus (EBV) lytic cycle by crosslinking surface immunoglobulin is inhibited by the immunosuppres
33 ymphoma cells with an antibody against their surface immunoglobulin M (anti-IgM) induces apoptosis an
34 GrB by IL-21 integrated signals mediated by surface immunoglobulin M (B-cell receptor) and Toll-like
35 racterized by a selective pressure to retain surface immunoglobulin M (IgM) BCR despite an active cla
36 s rapidly induced in peripheral B cells upon surface immunoglobulin M (IgM) cross-linking, CD40 signa
39 lignant B cells from apoptosis and increases surface immunoglobulin M (sIgM) expression on murine spl
40 erent states of anergy, indicated by reduced surface immunoglobulin M (sIgM) levels and signaling, co
41 UPR activation also correlated closely with surface immunoglobulin M (sIgM) signaling capacity in vi
42 when cells were stimulated with antibody to surface immunoglobulin M (sIgM), while in B cells from u
43 nd that it is characterized by low levels of surface immunoglobulin M and impairment of calcium mobil
44 ted VH genes (>5% deviation from germ line), surface immunoglobulin M ligation failed to induce recep
46 plasma cells (CD38hi, CD10-, CD19+, CD20lo, surface immunoglobulin negative, and cytoplasmic immunog
48 n gene rearrangements and express monoclonal surface immunoglobulin of either IgG or multiple heavy-c
51 ible patients were enrolled: 74 with B-cell (surface immunoglobulin-positive [Slg+] acute lymphoblast
53 ental alteration characterized by a block in surface immunoglobulin rearrangement resulting in BCR-ne
55 recognize foreign antigens by virtue of cell surface immunoglobulin receptors and are most effectivel
56 le and mice, that express rotavirus-specific surface immunoglobulin (RV-sIg) by flow cytometry with r
58 were simultaneously characterized for their surface immunoglobulin (sIg) isotypes and EBV genome cop
59 al demonstrate that sugar moieties linked to surface immunoglobulin (sIg) of follicular lymphoma (FL)
60 Cross-linking of the Fc receptor (FcR) to surface immunoglobulin (sIg) on B cells inhibits the inf
61 leukemia (CLL) cells express poor levels of surface immunoglobulin (sIg), and many are minimally act
62 (DC-SIGN) and mannose receptor, bound to FL surface immunoglobulin (sIg), generating an intracellula
63 ll receptor (BCR) for antigen is composed of surface immunoglobulin (sIg), which provides antigen spe
64 , we have conducted experiments in which the surface immunoglobulin (sIg)-mediated early cell signali
66 loss of LMP2A's dominant-negative effect on surface immunoglobulin signal transduction by monitoring
68 Btk may link engagement of receptors such as surface immunoglobulin to modulation of gene expression.
69 eins osteopontin and tenacsin C and the cell surface immunoglobulin vascular cell adhesion molecule-1
70 mphocytes, co-clustering of antigen receptor surface immunoglobulin with FcgammaRIIb promotes the neg
71 terparts, B-1 cells, CLL cells often express surface immunoglobulin with the capacity to bind autolog
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