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1 s the acquisition of antigens by B cells via surface immunoglobulin.
2 A and protein increase with cross-linking of surface immunoglobulin.
3 ess the B-lineage commitment factor Pax5 and surface immunoglobulins.
4 ines of GC origin identified 6 lines lacking surface immunoglobulin, a phenotype never seen among lin
5 e phosphorylation and became associated with surface immunoglobulin and CD79b, arguing for the involv
6 ative signaling in which co-cross-linking of surface immunoglobulin and Fc gammaRIIB1 results in sequ
7  in B cells and rendered them susceptible to surface immunoglobulin- and chemotherapeutic agent-induc
8 ymphoma cells with an antibody against their surface immunoglobulin (anti-Ig) induces apoptosis and h
9     Binding of the tetramers occurred by the surface immunoglobulin antigen receptor with little or n
10 f relatively mature lymphoid phenotype, with surface immunoglobulins being abundantly expressed.
11  induced in some cell lines upon ligation of surface immunoglobulin by mechanisms that include the ac
12                                Engagement of surface immunoglobulin by specific (HEL) antigen failed
13 fection by tetradecanoyl phorbol acetate and surface immunoglobulin cross-linking and is activated by
14 nd activation of lytic replication following surface immunoglobulin cross-linking.
15 utant virus was able to establish latency in surface immunoglobulin D-negative (sIgD(-)) B cells; by
16  latent gammaHV68 is largely confined to the surface immunoglobulin D-negative subset of B cells.
17  signaling competence, BTK independence, and surface immunoglobulin dependence of the PLCG2 mutation
18 lerant B cells in the diminished capacity of surface immunoglobulin engagement to produce Fas resista
19  were monoclonal, morphologically mature and surface immunoglobulin expressing B cells.
20 r (BCR)-type tumors that exhibited selective surface immunoglobulin expression and sensitivity to ent
21  with reduced percentages of both CD4(+) and surface immunoglobulin G-positive lymphocytes in the spl
22 dition of calcium ionophore, and ligation of surface immunoglobulin (Ig) by antibody.
23 e that expressed a mutant transgene encoding surface immunoglobulin (Ig), but which did not permit th
24  late times postinfection with cross-linking surface immunoglobulin (Ig), in conjunction with anti-CD
25 ach patient's tumor expressing a unique cell surface immunoglobulin (Ig), or B-cell receptor (BCR), t
26                                Cross-linking surface immunoglobulin (Ig)M on the WEHI-231 B-cell lymp
27 rved that crosslinking, by antibody, of cell surface immunoglobulin induced G1 growth-arrest followed
28  Crosslinking of the B cell antigen receptor surface immunoglobulin induces tyrosine phosphorylation
29         In developing B cells, expression of surface immunoglobulin is an important signal to termina
30 Barr virus (EBV) lytic cycle by crosslinking surface immunoglobulin is inhibited by the immunosuppres
31 ases of typical HCL, all expressing multiple surface immunoglobulin isotypes, were analyzed.
32         In addition, coligation of CD19 with surface immunoglobulin leads to augmented Akt activity i
33 ymphoma cells with an antibody against their surface immunoglobulin M (anti-IgM) induces apoptosis an
34  GrB by IL-21 integrated signals mediated by surface immunoglobulin M (B-cell receptor) and Toll-like
35 racterized by a selective pressure to retain surface immunoglobulin M (IgM) BCR despite an active cla
36 s rapidly induced in peripheral B cells upon surface immunoglobulin M (IgM) cross-linking, CD40 signa
37 genic signals including the cross-linking of surface immunoglobulin M (IgM).
38 f magnitude as they acquire higher levels of surface immunoglobulin M (IgM).
39 lignant B cells from apoptosis and increases surface immunoglobulin M (sIgM) expression on murine spl
40 erent states of anergy, indicated by reduced surface immunoglobulin M (sIgM) levels and signaling, co
41  UPR activation also correlated closely with surface immunoglobulin M (sIgM) signaling capacity in vi
42  when cells were stimulated with antibody to surface immunoglobulin M (sIgM), while in B cells from u
43 nd that it is characterized by low levels of surface immunoglobulin M and impairment of calcium mobil
44 ted VH genes (>5% deviation from germ line), surface immunoglobulin M ligation failed to induce recep
45 by induction of NF-kappa B and expression of surface immunoglobulin M).
46  plasma cells (CD38hi, CD10-, CD19+, CD20lo, surface immunoglobulin negative, and cytoplasmic immunog
47                      Furthermore, 3 of the 6 surface immunoglobulin-negative GC lines carried inactiv
48 n gene rearrangements and express monoclonal surface immunoglobulin of either IgG or multiple heavy-c
49                                Engagement of surface immunoglobulin on mature B cells leads to rescue
50 activation of Akt following cross-linking of surface immunoglobulin or Igbeta.
51 ible patients were enrolled: 74 with B-cell (surface immunoglobulin-positive [Slg+] acute lymphoblast
52                                           In surface immunoglobulin-positive B cells (Ramos), ligatio
53 ental alteration characterized by a block in surface immunoglobulin rearrangement resulting in BCR-ne
54 cell help, along with ligation of the B cell surface immunoglobulin receptor by antigen.
55 recognize foreign antigens by virtue of cell surface immunoglobulin receptors and are most effectivel
56 le and mice, that express rotavirus-specific surface immunoglobulin (RV-sIg) by flow cytometry with r
57                                EWI-2, a cell surface immunoglobulin SF protein of unknown function, a
58  were simultaneously characterized for their surface immunoglobulin (sIg) isotypes and EBV genome cop
59 al demonstrate that sugar moieties linked to surface immunoglobulin (sIg) of follicular lymphoma (FL)
60    Cross-linking of the Fc receptor (FcR) to surface immunoglobulin (sIg) on B cells inhibits the inf
61  leukemia (CLL) cells express poor levels of surface immunoglobulin (sIg), and many are minimally act
62  (DC-SIGN) and mannose receptor, bound to FL surface immunoglobulin (sIg), generating an intracellula
63 ll receptor (BCR) for antigen is composed of surface immunoglobulin (sIg), which provides antigen spe
64 , we have conducted experiments in which the surface immunoglobulin (sIg)-mediated early cell signali
65 er upon stimulation of the antigen receptor, surface immunoglobulin (sIg).
66  loss of LMP2A's dominant-negative effect on surface immunoglobulin signal transduction by monitoring
67                                CDO is a cell surface immunoglobulin superfamily member that positivel
68 Btk may link engagement of receptors such as surface immunoglobulin to modulation of gene expression.
69 eins osteopontin and tenacsin C and the cell surface immunoglobulin vascular cell adhesion molecule-1
70 mphocytes, co-clustering of antigen receptor surface immunoglobulin with FcgammaRIIb promotes the neg
71 terparts, B-1 cells, CLL cells often express surface immunoglobulin with the capacity to bind autolog
72      This selection favors B cell expressing surface immunoglobulins with VHa2 structures in the firs

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