ライフサイエンスコーパス: surfactant protein

1 e by proteolysis and through interactions of surfactant proteins.

2 cells correlated with the expression of the surfactant proteins.

3 skeleton, and as receptors for immunological surfactant proteins.

4 In the lungs, surfactant protein A (SP-A) and SP-D contribute to immun

5 Mice lacking surfactant protein A (SP-A) are susceptible to bacterial

6 The identification of surfactant protein A (SP-A) as an important innate immun

7 The host antimicrobial agent surfactant protein A (SP-A) effectively reduced ciliary

8 In the lung, surfactant protein A (SP-A) enhanced interleukin-4 (IL-4

9 Surfactant protein A (SP-A) enhances phagocytosis of Pse

10 We previously reported that mice lacking surfactant protein A (SP-A) have increased airway hyperr

11 The current study investigated the role of surfactant protein A (SP-A) in opsonization and clearanc

12 hypothesize a central role for the collectin surfactant protein A (SP-A) in regulating the developmen

13 onization of F. novicida with lung collectin surfactant protein A (SP-A) increased bacterial associat

14 Previous studies reported that surfactant protein A (SP-A) inhibits lymphocyte prolifer

15 Surfactant protein A (SP-A) is a hydrophilic glycoprotei

16 Surfactant protein A (SP-A) is an immune modulator that

17 Surfactant protein A (SP-A) is important for immune defe

18 The human surfactant protein A (SP-A) locus consists of two functi

19 Surfactant protein A (SP-A) modulates host responses to

20 Surfactant protein A (SP-A) plays a critical role in the

21 Pulmonary surfactant protein A (SP-A) plays a key role in innate l

22 Surfactant protein A (SP-A) plays a role in lung innate

23 Lung surfactant protein A (SP-A) plays an important function

24 Surfactant protein A (SP-A) plays an important role in t

25 Mass spectrometric characterization of the surfactant protein A (SP-A) receptor 210 (SP-R210) led t

26 st to immobilized human fibronectin (FN) and surfactant protein A (SP-A) under this condition.

27 Surfactant protein A (SP-A), a C-type lectin, plays an i

28 Surfactant protein A (SP-A), a major component of lung s

29 Pulmonary surfactant protein A (SP-A), a member of the collectin f

30 The lung collectin, surfactant protein A (SP-A), has emerged as an important

31 We observed that surfactant protein A (SP-A)-deficient mice have impaired

32 The PLA(2) activity is inhibited by surfactant protein A (SP-A).

33 stance to antibacterial effects of pulmonary surfactant protein A (SP-A).

34 al cells (AEC) in the presence or absence of surfactant protein A (SP-A).

35 Surfactant protein A (SPA), a lung-specific collectin, s

36 CARDS TX binds to human surfactant protein A and annexin A2 on airway epithelial

37 es even though several rare proteinopathies, surfactant protein A and C deficiencies, cause severe pu

38 Subsequently, surfactant lipids and/or surfactant protein A enhance CD36 transcript and protein

39 We have reported that surfactant protein A kills some Gram-negative organisms

40 Although surfactant protein A, B, and C content and surfactant pr

41 B. dermatitidis without or with normal BALF, surfactant protein A-deficient (SP-A-/-) or surfactant p

42 following exposure to surfactant lipids and surfactant protein A.

43 Surfactant proteins A (SP-A) and D (SP-D) play an import

44 The pulmonary collectins--surfactant proteins A (SP-A) and D (SP-D)--play importan

45 crobial actions of the pulmonary collectins, surfactant proteins A (SP-A) and D (SP-D).

46 ein (endothelial permeability) and preserved surfactant proteins A and B concentrations as compared w

47 Surfactant proteins A and D (SP-A and -D) play a role in

48 Surfactant proteins A and D (SP-A and SP-D) are collecti

49 vels of expression of mRNA for antimicrobial surfactant proteins A and D and sheep beta-defensin 1 we

50 uding lamellar body formation, expression of surfactant proteins A, B, and C, alpha-1-antitrypsin, an

51 interactions between M. pneumoniae and human surfactant protein-A (hSP-A).

52 ctivators 1 and 2 (SRC-1 and SRC-2) regulate surfactant protein-A (SP-A) and platelet-activating fact

53 Previous reports demonstrate that surfactant protein-A (SP-A) binds live M. pneumoniae and

54 Surfactant protein-A (SP-A) is an important antimicrobia

55 One component of this defense mechanism, surfactant protein-A (SP-A), exerts multifunctional role

56 eptor (VDR); soluble C-type lectins, such as surfactant protein-A (SP-A), SP-D, and mannose-binding l

57 Surfactant protein-A (SP-A), which is secreted by fetal

58 mniotic fluid (AF) macrophages, activated by surfactant protein-A (SP-A).

59 Enrichment of surfactant protein-A in the lung and surfactant of the m

60 , plasminogen activator inhibitor-1 (PAI-1), surfactant protein-A, surfactant protein-D, and intracel

61 e collagen-like domain distinguish the human surfactant protein A1 (SP-A1) variants from the SP-A2 va

62 ase reverse transcriptase promoter and human surfactant protein A1 promoter).

63 e gene, SFTPA2, within the interval encoding surfactant protein A2 (SP-A2).

64 heterozygous mutations in the gene encoding surfactant protein A2 (SP-A2, SFTPA2) are associated wit

65 analyze all common and rare variants of the surfactant protein A2, SP-A2, in both A549 cells and in

66 gs exhibited induction of genes that express surfactant proteins, ABCA3, GM-CSF, podoplanin, and cave

67 Our findings support a model in which the surfactant proteins accelerate adsorption by producing a

68 To determine if hydrophobic surfactant proteins affect the stability of pulmonary su

69 vided insight into the mechanisms underlying surfactant protein and alveolar homeostasis.

70 Pulmonary surfactant proteins and lipids are required for lung fun

71 Surfactant proteins and lipids were decreased or absent

72 miR-200 antagonists inhibited TTF-1 and surfactant proteins and up-regulated TGF-beta2 and ZEB1

73 The formation of surfactant-protein and surfactant-protein-organic anion

74 Gas microbubbles stabilized with lipids, surfactants, proteins and/or polymers are widely used cl

75 ndogenous TTF-1, blocked cAMP stimulation of surfactant proteins, and inhibited miR-200 expression, w

76 Mutations in the genes encoding the lung surfactant proteins are found in patients with interstit

77 Missense mutations of surfactant proteins are recognized as important causes o

78 Preliminary studies have identified pro-surfactant protein B (pro-SFTPB) to be a promising blood

79 e aliquot was used to quantify levels of pro-surfactant protein B (pro-SFTPB), a previously establish

80 Surfactant protein B (Sftpb) is a developmentally regula

81 rowth factor-beta binding protein-4 (LTBP4), surfactant protein B (SFTPB), and transforming growth fa

82 ification of structural changes of pulmonary surfactant protein B (SP-B) due to the heterogeneous rea

83 Surfactant protein B (SP-B) is a hydrophobic, 79 amino a

84 Pulmonary surfactant protein B (SP-B) is an essential protein for

85 Surfactant protein B (SP-B) is essential to the function

86 Surfactant protein B (SP-B) is one of two helical, amphi

87 Surfactant protein B (SP-B) is secreted into the airspac

88 Surfactant protein B (SP-B) proprotein contains three sa

89 from patients with ESRD (ESRD-HDL) included surfactant protein B (SP-B), apolipoprotein C-II, serum

90 tively charged to hydrophobic amino acids in surfactant protein B (SP-B), on the structure and collap

91 nduced transcriptional activity of the mouse surfactant protein B and A (Sftpb and Sftpa) promoters i

92 Genetic variations in the gene coding for surfactant protein B are associated with more severe lun

93 n 4 of the surfactant protein B gene and the surfactant protein B gene +1580 polymorphism.

94 andem repeat polymorphism in intron 4 of the surfactant protein B gene and the surfactant protein B g

95 is study, the variable nuclear tandem repeat surfactant protein B gene polymorphism in intron 4 is as

96 e presence of variable nuclear tandem repeat surfactant protein B gene polymorphism was associated wi

97 For the variable nuclear tandem repeat surfactant protein B gene polymorphism, patients were fo

98 m-tag single nucleotide polymorphisms in the surfactant protein B gene.

99 and nanoparticle, chain tilt angle, and the surfactant protein B peptide helix tilt angle.

100 ct with TTF-1 and regulate the expression of surfactant protein B, a protein required for lung functi

101 21-residue functional peptide mimic of lung surfactant protein B, an essential protein for lowering

102 SP-B1-25 is a shorter version of lung-surfactant protein B, an important component of lung sur

103 rfactant protein C, 95% for mucin-1, 93% for surfactant protein B, and 89% for the epithelial marker

104 recursor, pigment epithelium-derived factor, surfactant protein B, and serum amyloid A.

105 ation, as indicated by lack of expression of surfactant protein B, surfactant protein C, the Clara ce

106 ptide employed as a functional mimic of lung surfactant protein B, which successfully lowers surface

107 were reduced, with the exception of that for surfactant protein B, which was elevated.

108 ide consisting of the first 25 residues from surfactant protein B.

109 ng at cellular sites consistent with that of surfactant protein B.

110 type II cell differentiation, as measured by surfactant protein B/C mRNA and protein levels.

111 hown promise as functional analogues of lung surfactant proteins B and C (SP-B and SP-C), two helical

112 Surfactant proteins B and C and saturated phosphatidylch

113 ara cells, Clara cell secretory protein, and surfactant proteins B and C, without affecting airway ci

114 thyroid transcription factor (TTF)-1 and pro-surfactant protein-B (pro-SP-B), and mesenchymal (alpha-

115 P-2 enhanced the activity of the promoter of surfactant protein-B (Sftpb gene) but not other surfacta

116 nd use the method to measure the turnover of surfactant protein-B (SP-B).

117 ary acute lung injury, CPAP-30 yielded lower surfactant protein-B and higher type III procollagen exp

118 GF-beta represses transcription of pulmonary surfactant protein-B gene in lung epithelial cells.

119 h surfactant protein A, B, and C content and surfactant protein-B mRNA expression in Stat3(DeltaDelta

120 3 is necessary for lamellar body biogenesis, surfactant protein-B processing, and lung development la

121 II alveolar epithelial cells were capable of surfactant protein-B uptake and stimulated surfactant re

122 eceptor for advanced glycation end products, surfactant protein-B, type III procollagen, and pro-casp

123 nts revealed a specific staining pattern for surfactant protein-B, which was the same pattern observe

124 ) conditional expression of a mutant form of surfactant protein C (L188Q SFTPC) found in familial int

125 We used the mouse surfactant protein C (Sftpc) promoter to over-express th

126 cEGFP in the epithelium under the control of surfactant protein C (Sftpc) regulatory elements expands

127 Expression of mutant surfactant protein C (SFTPC) results in endoplasmic reti

128 wild-type (SFTPC+/+) controls, mice lacking surfactant protein C (SFTPC-/-) had greater lung neutrop

129 Biosynthesis of surfactant protein C (SP-C) by alveolar type 2 cells req

130 Surfactant protein C (SP-C) has been suggested to be an

131 To determine the role of surfactant protein C (SP-C) in host defense, SP-C-defici

132 d protein overlapped with those of TTF-1 and surfactant protein C (SP-C) in the respiratory epithelia

133 Surfactant protein C (SP-C) is a hydrophobic 35-amino ac

134 Surfactant protein C (SP-C) is a hydrophobic lipopeptide

135 Surfactant protein C (SP-C) is a novel amyloid protein f

136 ithelial type II cell-specific promoter--the surfactant protein C (SP-C) promoter.

137 R) retention, and degradation of the encoded surfactant protein C (SP-C) proprotein.

138 t cause misfolding of the encoded proprotein surfactant protein C (SP-C) trigger endoplasmic reticulu

139 Mice lacking the lung-specific protein surfactant protein C (Sp-C) were lethally irradiated, tr

140 Expression of surfactant protein C (SP-C), which is restricted to alve

141 conditionally target K-RasG12D expression in Surfactant Protein C (SPC)(+) alveolar type 2 cells and

142 , NKX2.1 protein, and its downstream target, surfactant protein C (SpC).

143 1 and downstream lung target genes including surfactant protein C and Clara cell secretory protein.

144 Inhibition of SCD reduced surfactant protein C expression and suppressed rhinoviru

145 Recent reports have linked mutations in the surfactant protein C gene (SFTPC) to familial forms of p

146 h a peptide model for collagen and pulmonary surfactant protein C have been simulated very closely by

147 liferative disease, including lung fibrosis (surfactant protein C precursor; pro-SP-C) and familial d

148 that express a lung-specific TSLP transgene (surfactant protein C promoter (SPC)-TSLP) develop a spon

149 xpressing PPARgamma under the control of the surfactant protein C promoter had reduced expression of

150 human DPP4 (hDPP4) under the control of the surfactant protein C promoter or cytokeratin 18 promoter

151 Transgenic mice using the human Surfactant Protein C promoter to drive the expression of

152 hrough use of the Cre/loxP system, the human surfactant protein C promoter, and the reverse tetracycl

153 leting the integrin from E10.5 onwards using surfactant protein C promoter-driven Cre.

154 ssed as a transgene under the control of the surfactant protein C promoter.

155 The BRICHOS domain from lung surfactant protein C proprotein (proSP-C) is required fo

156 nd that up to 97% of cells were positive for surfactant protein C, 95% for mucin-1, 93% for surfactan

157 helial markers (Clara cell-specific protein, surfactant protein C, and aquaporin-5), and lack of surf

158 dicated by an increase in the AEC II marker, surfactant protein C, and decreases in the AEC I markers

159 Mutations in the genes encoding surfactant protein C, SFTPC, and a member of the adenosi

160 lack of expression of surfactant protein B, surfactant protein C, the Clara cell secretory protein,

161 to four intratracheal doses of a recombinant surfactant protein C-based surfactant given within a per

162 We then used a Surfactant protein C-CreER(T2) knock-in allele to follow

163 Lungs of both naive surfactant protein C-MCP mice and influenza-infected WT

164 etic lineage-tracing experiments showed that surfactant protein C-positive (SFTPC-positive) AEC2s sel

165 Mutations in the gene encoding SP-C (surfactant protein C; SFTPC) have been linked to interst

166 ected against the ATII cell-specific antigen surfactant protein-C (SP-C) then administered to C57BL/6

167 Either deletion of TLR4 or HA synthase 2 in surfactant-protein-C-positive AEC2s leads to impaired re

168 rovide the most direct evidence yet that the surfactant proteins can induce negative curvature in lip

169 Our findings show that surfactant proteins can promote negative curvature, and

170 transcription of target genes, including the surfactant proteins critical for lung function.

171 tion analysis showed that baseline values of surfactant protein D (46.6 ng/mL vs 34.6 ng/mL, p=0.0018

172 + carbohydrate recognition domains of human surfactant protein D (NCRD) with CL-43 (RCL-43-NCRD) and

173 RATIONALE: Recombinant fragment of human surfactant protein D (rfhSP-D) has been shown to suppres

174 Mice deficient in surfactant protein D (Sftpd) develop progressive age-rel

175 Surfactant protein D (SP-D) and CD14 are important innat

176 Circulating levels of the biomarkers surfactant protein D (SP-D) and soluble receptor for adv

177 Lung surfactant protein D (SP-D) binds to Mycobacterium tuber

178 The innate host defense component surfactant protein D (SP-D) interacts with glycans on th

179 Surfactant protein D (SP-D) is a collectin family member

180 Surfactant protein D (SP-D) is a member of the collectin

181 Surfactant protein D (SP-D) is an important effector of

182 Surfactant protein D (SP-D) is an important regulator of

183 Surfactant protein D (SP-D) is an innate immune effector

184 Surfactant Protein D (SP-D) is an oligomerized C-type le

185 Surfactant protein D (SP-D) is critical for maintenance

186 he recognition of influenza A virus (IAV) by surfactant protein D (SP-D) is mediated by interactions

187 Our previous studies showed that surfactant protein D (SP-D) is present in human tear flu

188 Innate immune molecule surfactant protein D (SP-D) plays a critical role in hos

189 Surfactant protein D (SP-D) plays diverse and important

190 Surfactant protein D (SP-D) plays important roles in ant

191 Surfactant protein D (SP-D) plays important roles in inn

192 Surfactant protein D (SP-D) plays important roles in inn

193 Surfactant protein D (SP-D) plays important roles in lun

194 Surfactant protein D (SP-D) plays important roles in the

195 previous studies documenting that pulmonary surfactant protein D (SP-D) protects C. neoformans cells

196 recognition domains (CRDs) of lung collectin surfactant protein D (SP-D) recognize sugar patterns on

197 Surfactant protein D (SP-D) shows specific interactions

198 Surfactant protein D (SP-D), a component of innate immun

199 Pulmonary surfactant protein D (SP-D), a member of the collectin f

200 Polymorphisms of surfactant protein D (SP-D), an important molecule withi

201 ins, Clara cell secretory protein (CC16) and surfactant protein D (SP-D), and five systemic inflammat

202 The roles of NK cells, surfactant protein D (SP-D), and IFN-gamma, as well as t

203 emonstrated direct interactions of HNPs with surfactant protein D (SP-D), another important effector

204 Surfactant protein D (SP-D), shown to play a role in hos

205 The role of surfactant protein D (SP-D), which inhibits P. aeruginos

206 Surfactant protein D (SP-D)-deficient (SP-D-/-) mice exh

207 t on virulence in naive BALB/c, C57BL/6, and surfactant protein D (SP-D)-deficient mice.

208 a) production and the enhanced expression of surfactant protein D (SP-D).

209 Surfactant protein D (SPD) plays an important role in su

210 d, a tripartite fusion protein was made from surfactant protein D (SPD), HIV-1 Gag as a test antigen,

211 olar lavage and various serum markers (e.g., surfactant protein D and KL-6) each may provide useful i

212 soluble multitrimers of BAFF and APRIL using surfactant protein D as a scaffold, and we vaccinated mi

213 in, which evolved through duplication of the surfactant protein D gene in ruminants, prefers mannose

214 nt of peritoneal Mos with the lung collectin surfactant protein D inhibited AC uptake, and fluticason

215 Surfactant protein D is a pattern recognition molecule t

216 Surfactant protein D is characterized by two relatively

217 re generally not inhibited by the collectins surfactant protein D or mannose binding lectin because o

218 Surfactant protein D preferentially binds to glucose and

219 nary and activation-regulated chemokine, and surfactant protein D significantly increased the areas u

220 nary and activation-regulated chemokine, and surfactant protein D significantly strengthens this asso

221 e 2-trimer form, and fusion with the body of surfactant protein D was used to produce the 4-trimer fo

222 matrix metalloproteinase (MMP)-7, MMP-1, and surfactant protein D were assessed by ELISA.

223 ombinant homotrimeric fragment of human lung surfactant protein D with a series of bound ligands have

224 ptor for advanced glycation end products and surfactant protein D) and endothelial injury (angiopoiet

225 multitrimerized using the lung protein SP-D (surfactant protein D), enhancing immune responses.

226 KGF treatment increases BAL surfactant protein D, a marker of type II alveolar epith

227 eceptor for advanced glycation end products, surfactant protein D, angiopoietin-2, interleukin-6, int

228 a levels of fibrinogen, chemokine ligand 18, surfactant protein D, C-reactive protein, Clara cell sec

229 ding of three innate immune lectins, namely, surfactant protein D, human galectin-8, and Siglec-14, t

230 KGF increased BAL concentrations of surfactant protein D, matrix metalloproteinase (MMP)-9,

231 alysis, we identified four serum biomarkers (surfactant protein D, matrix metalloproteinase 7, CA19-9

232 es, including von Willebrand factor antigen, surfactant protein D, protein C, plasminogen activator i

233 infection, and, when combined with pulmonary surfactant protein D, their antiviral effects were addit

234 surfactant protein A-deficient (SP-A-/-) or surfactant protein D-deficient (SP-D-/-) BALF, or a mixt

235 on molecules in the collectin family, namely surfactant proteins D and A (Sp-D and Sp-A, respectively

236 Surfactant protein-D (SP-D) participates in several of t

237 The innate immune molecule surfactant protein-D (SP-D) plays an important regulator

238 Surfactant protein-D (SP-D), a member of the "collectin"

239 After challenge with high-dose surfactant protein-D (SP-D)-sensitive influenza A/Philad

240 production, and proteolysis of the collectin surfactant protein-D (SP-D).

241 In a subset, we compared plasma levels of surfactant protein-D and von Willebrand factor antigen b

242 ct of leukoreduced blood on plasma levels of surfactant protein-D or von Willebrand factor antigen wa

243 oluble tumor necrosis factor receptor-1, and surfactant protein-D) had nearly equivalent prognostic v

244 r inhibitor-1 (PAI-1), surfactant protein-A, surfactant protein-D, and intracellular adhesion molecul

245 n molecule-1, von Willebrand factor antigen, surfactant protein-D, and soluble tumor necrosis factor

246 indicate that at 47-53 mN/m, the hydrophobic surfactant proteins destabilize the compressed monolayer

247 Immunohistochemical analysis of surfactant protein expression in three patients revealed

248 fferentiation and an almost complete loss of surfactant protein expression.

249 h extracellular matrix components, including surfactant proteins, fibronectin, and mucin, which provi

250 Ranaspumin-2 (Rsn-2) is a surfactant protein found in the foam nests of the tungar

251 tspots near VMP1/MIR21 and indel hotspots in surfactant protein genes (SFTPA1, SFTPB, and SFTPC).

252 the proper formation of lamellar bodies and surfactant protein homeostasis.

253 the in vitro surface-active behavior of lung surfactant proteins in a mixed lipid film.

254 Examples include surfactant proteins in lung alveoli, albumin in liver pa

255 viral lesions and impaired the production of surfactant proteins in the alveolar epithelium.

256 also induced the sustained expression of the surfactant proteins in the lungs.

257 al foam formation is facilitated by specific surfactant proteins in the mixture, further stabilized b

258 factant protein-B (Sftpb gene) but not other surfactant proteins in vitro.

259 is a complex mixture of lipids and specific surfactant proteins, including the hydrophobic proteins

260 Other than constitutively expressed surfactant proteins, it is unknown whether alveolar epit

261 Mutations in surfactant proteins lead to pulmonary fibrosis and are a

262 However, surfactant protein levels were reduced, with the excepti

263 Identification of the surfactant proteins, lipid transporters, and transcripti

264 ions between highly glycosylated viruses and surfactant proteins may lead to an improved understandin

265 studies reported here, we tested whether the surfactant proteins might promote adsorption by inducing

266 philic proteins, but neither the hydrophobic surfactant proteins nor the phospholipids, induced agglo

267 Rarely, genetic defects in surfactant proteins or the common beta chain for the GM-

268 The formation of surfactant-protein and surfactant-protein-organic anion deposits is proposed on

269 Numerical density of surfactant protein positive cells was determined by immu

270 tion, decreased angiogenesis, and diminished surfactant protein production and may increase the risk

271 iate, supports the proposed model of how the surfactant proteins promote adsorption.

272 To determine how the hydrophobic surfactant proteins promote insertion of the surfactant

273 luid (LLF), which contains phospholipids and surfactant proteins, represents a first contact site wit

274 ession of lung-specific genes, including the surfactant proteins required for pulmonary function afte

275 s become available for probe partitioning in surfactant-protein solutions under visible light, while

276 is a truncated version of the full pulmonary surfactant protein SP-B, with dipalmitoylphosphatidylcho

277 tributed to SP-C or to the other hydrophobic surfactant protein SP-B.

278 The hydrophobic surfactant proteins SP-B and SP-C greatly accelerate the

279 rtitioning assay to assess the effect of the surfactant proteins SP-B and SP-C on cholesterol distrib

280 The hydrophobic surfactant proteins SP-B and SP-C promote rapid adsorpti

281 EKO lungs showed increased expression of the surfactant proteins Sp-B, Sp-C, and Sp-D, and displayed

282 Surfactant protein (SP) D is a member of the collectin f

283 hough many studies have shown that pulmonary surfactant protein (SP)-A functions in innate immunity,

284 Pulmonary surfactant protein (SP)-A is an endogenously produced li

285 Here, we study rare and common surfactant protein (SP)-A1 and SP-C variants, either dis

286 for a missense mutation in the gene encoding surfactant protein (SP)-A2 (SFTPA2) contains more TGF-be

287 Pulmonary surfactant protein (SP)-B plays a vital role in the form

288 om the N-terminal saposin-like domain of the surfactant protein (SP)-B proprotein, and SP-A are lung

289 eased amounts of surfactant phospholipid and surfactant protein (SP)-B.

290 ar epithelial cells under the control of the surfactant protein (SP)-C promoter develop pulmonary inf

291 Plasma surfactant protein (SP)-D > 31 ng/ml, matrix metalloprot

292 e alpha-defensins were also shown to bind to surfactant protein (SP)-D and reduce its antiviral activ

293 lso engineered MASP binding into a pulmonary surfactant protein (SP-A), which has the same domain str

294 Pulmonary surfactant proteins (SP-) A and D are innate immune patt

295 differentiation and expression of the major surfactant protein, SP-A, in mid-gestation human fetal l

296 ssion of the gene (SFTPA) encoding the major surfactant protein, SP-A, in midgestation human fetal lu

297 The hydrophobic surfactant proteins, SP-B and SP-C, greatly accelerate t

298 The hydrophobic surfactant proteins, SP-B and SP-C, have important roles

299 systemic hypercoagulant state, reduction of surfactant proteins SpC, SpB, and SpA, decline of lung c

300 othesized that collectins, such as pulmonary surfactant proteins (SPs) SP-A and SP-D and serum protei