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1 ide consisting of the first 25 residues from surfactant protein B.
2 ng at cellular sites consistent with that of surfactant protein B.
3 solated preparations or to altered levels of surfactant protein B.
4 ct with TTF-1 and regulate the expression of surfactant protein B, a protein required for lung functi
5 21-residue functional peptide mimic of lung surfactant protein B, an essential protein for lowering
7 nduced transcriptional activity of the mouse surfactant protein B and A (Sftpb and Sftpa) promoters i
8 nment of this fragment with type II-specific surfactant protein B and C promoters shows similar conse
9 ary acute lung injury, CPAP-30 yielded lower surfactant protein-B and higher type III procollagen exp
10 hown promise as functional analogues of lung surfactant proteins B and C (SP-B and SP-C), two helical
11 nts were made for PL and DAPL alone; with 3% surfactant proteins B and C (SP-B/C); with SP-B/C and 5%
13 factant (BC mixture = PL mixture + synthetic surfactant proteins B and C) at a dose of 100 mg alpha1-
14 ara cells, Clara cell secretory protein, and surfactant proteins B and C, without affecting airway ci
16 rfactant protein C, 95% for mucin-1, 93% for surfactant protein B, and 89% for the epithelial marker
18 Genetic variations in the gene coding for surfactant protein B are associated with more severe lun
21 atory adaptation at birth include hereditary surfactant protein B deficiency, mutations in surfactant
23 andem repeat polymorphism in intron 4 of the surfactant protein B gene and the surfactant protein B g
25 is study, the variable nuclear tandem repeat surfactant protein B gene polymorphism in intron 4 is as
26 e presence of variable nuclear tandem repeat surfactant protein B gene polymorphism was associated wi
34 s completely abolish expression of the human surfactant protein B (hSP-B) 1.5 kb lacZ reporter gene i
37 h surfactant protein A, B, and C content and surfactant protein-B mRNA expression in Stat3(DeltaDelta
38 mice was not associated with accumulation of surfactant proteins B or C, or their mRNAs, distinguishi
42 e aliquot was used to quantify levels of pro-surfactant protein B (pro-SFTPB), a previously establish
43 thyroid transcription factor (TTF)-1 and pro-surfactant protein-B (pro-SP-B), and mesenchymal (alpha-
44 3 is necessary for lamellar body biogenesis, surfactant protein-B processing, and lung development la
46 rowth factor-beta binding protein-4 (LTBP4), surfactant protein B (SFTPB), and transforming growth fa
47 P-2 enhanced the activity of the promoter of surfactant protein-B (Sftpb gene) but not other surfacta
48 and depth of each residue of lung pulmonary surfactant protein B (SP-B(1-25)) in a phospholipid bila
50 ification of structural changes of pulmonary surfactant protein B (SP-B) due to the heterogeneous rea
53 ted the -500 to +41 promoter activity of the surfactant protein B (SP-B) gene in respiratory epitheli
54 ming growth factor-beta (TGF-beta) represses surfactant protein B (Sp-B) gene transcription through a
71 the 102-amino acid C-terminal propeptide of surfactant protein B (SP-B) was analyzed by characterizi
73 from patients with ESRD (ESRD-HDL) included surfactant protein B (SP-B), apolipoprotein C-II, serum
74 tively charged to hydrophobic amino acids in surfactant protein B (SP-B), on the structure and collap
76 imulations of the N-terminal region of human surfactant protein-B (SP-B(1-25)) in dipalmitoylphosphat
82 mal patterns and numbers of cells expressing surfactant protein-B suggest that differentiation of typ
83 ation, as indicated by lack of expression of surfactant protein B, surfactant protein C, the Clara ce
84 eceptor for advanced glycation end products, surfactant protein-B, type III procollagen, and pro-casp
85 II alveolar epithelial cells were capable of surfactant protein-B uptake and stimulated surfactant re
87 ptide employed as a functional mimic of lung surfactant protein B, which successfully lowers surface
89 nts revealed a specific staining pattern for surfactant protein-B, which was the same pattern observe
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