1 may be enhanced in response to local lactate
surfeit,
alone or relative to glucose deficiency, and th
2 Prospective evidence of both reward
surfeit and reward deficit pathways to increased body we
3 ng, suggesting qualitatively distinct reward
surfeit and reward deficit pathways to obesity.
4 habit formation, reward deficits and stress
surfeits,
and compromised executive function in three st
5 The Fugu
Surfeit gene homologs appear to be associated with CpG-r
6 Here, Fugu gene homologs of all six
Surfeit genes (Surf-1 to Surf-6) have been cloned and se
7 The organisation and juxtaposition of the
Surfeit genes are conserved between human and mouse.
8 has been shown previously that, whereas the
Surfeit genes are tightly clustered at a single locus in
9 e associated with CpG-rich islands, like the
Surfeit genes in higher vertebrates, but these Fugu CpG
10 seven Fugu genes which are closely linked to
Surfeit genes in two regions of the Fugu genome and have
11 , approximately 2-4 Mb proximal to the human
Surfeit genes.
12 The human
Surfeit locus contains at least six tightly clustered ge
13 d determined its genomic organisation in the
Surfeit locus.
14 Counter to the energy
surfeit model of obesity, results suggest that increasin
15 been no such prospective tests of the reward
surfeit model of these motivated behaviors.
16 nset, providing novel support for the reward
surfeit model.
17 We find that a
surfeit of autosomal retroposed genes originated from pa
18 ized about a diet that allows ingestion of a
surfeit of calories that are burned off effortlessly by
19 However, such a
surfeit of control raises an additional choice problem:
20 Thus, like its loss, a
surfeit of endogenous p220 function represents a threat
21 serve than their wild-type sisters, and this
surfeit of follicles is maintained in advanced chronolog
22 The brief
surfeit of MYC activity was accompanied by evidence of g
23 The
surfeit of protein structural data enables development o
24 f RepA excess are prevented by introducing a
surfeit of RepA binding sites.
25 y activated were characterized by a relative
surfeit of stalled polymerases at the TSS, which resolve
26 Low energy flux, but not energy
surfeit,
predicted future increases in body fat in both
27 es that have prospectively tested the energy
surfeit theory of obesity with the use of objectively es
28 opamine signaling, consonant with the reward-
surfeit theory of obesity.