ライフサイエンスコーパス: surgical stress

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1 le to the negative sequela of anesthetic and surgical stress.

2 tors within the cecum of mice in response to surgical stress.

3 ogens in the intestinal tract in response to surgical stress.

4 N, SON and MnPO of rats with HF during acute surgical stress.

5 sis due to intestinal P. aeruginosa in which surgical stress (30% hepatectomy) was combined with dire

6 ated behavioural analysis could be detecting surgical stress and/or post-surgical pain.

7 ivated during HF and are sensitive to 'acute surgical stress' and may contribute to the elevated leve

8 documented in Crohn disease, celiac disease, surgical stress, and intestinal obstruction and are asso

9 of the Estimation of Physiologic Ability and Surgical Stress (E-PASS) risk model in a Western PHC coh

10 se data provide direct in vivo evidence that surgical stress impairs global NK cell function.

11 eurons in the PVN, SON and MnPO due to acute surgical stress in rats with HF, and (2) vagal afferents

12 Furthermore, surgical stress in the form of hepatectomy significantly

13 uced withdrawal reflex) and antinociception (surgical stress index, skin conductance algesimeter) wer

14 Furthermore, we demonstrated that surgical stress or cecal ligation and puncture caused a

15 rioperative pulmonary function, blunting the surgical stress response and improved analgesia.

16 Protein can modulate the surgical stress response and postoperative catabolism.

17 r improve outcome presumably by blocking the surgical stress response.

18 Multisystem injury and major surgical stress result in a hypermetabolic state with ac

19 Clinical markers of surgical stress that were collected included duration of

20 ely attenuated the catecholamine response to surgical stress throughout the postoperative study perio

21 gy of the anomaly and the general effects of surgical stress, tocolytic agents, and anesthesia.

22 Using a mouse model of surgical stress, we transferred surgically stressed NK c

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