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1 GVHD (n = 86) (the status was uncertain in 1 surviving patient).
2 t discharge (home or another hospital, among surviving patients).
3 e and 266 were reassessed at 4 years (94% of surviving patients).
4 provement of motor function over time in the surviving patient.
5 hs with a median follow-up of 115 months for surviving patients.
6 of clinical visit or telephone interview for surviving patients.
7 nths (range, 1-264 months) and 51 months for surviving patients.
8 criteria; median follow-up was 51 months for surviving patients.
9 linical status was ascertained in 167 of 171 surviving patients.
10 to evaluate the relative risk of relapse in surviving patients.
11 on nonfatal events was available for 95% of surviving patients.
12 lief and improved quality of life in all the surviving patients.
13 nformation on the current clinical status of surviving patients.
14 No relapse has been seen in surviving patients.
15 oid donor chimerism was documented in 80% of surviving patients.
16 and an extremely low percentage of long-term surviving patients.
17 ugs on the risk of clinical deterioration in surviving patients.
18 d donor chimerism was documented in 52 (93%) surviving patients.
19 of IgG responses was significantly higher in surviving patients.
20 was 3.3 years (range, 0.6 to 4.4 years) for surviving patients.
21 -up of 43 months (range, 34-54) for the four surviving patients.
22 tion of chronic graft-versus-host disease in surviving patients.
23 , and the median follow-up was 57 months for surviving patients.
24 ow-up of 59 months (range, 27-78 months) for surviving patients.
25 32 months for all patients and 42 months for surviving patients.
26 wever, 6 months after surgery, only 4 of 243 surviving patients (1.6%) had a persistent language defi
29 tic stress-related symptoms in relatives and surviving patients 12 months after intensive care unit d
30 died of hypertrophic cardiomyopathy, and 216 surviving patients (20 percent) had severe, disabling sy
31 ith a median follow-up time of 43 months for surviving patients, 20 patients have had disease progres
33 atients, and the 1-year cohort included 7014 surviving patients (3454 women [49.2%] and 3560 men [50.
38 .5% of patients surviving at 5 1/2 years; in surviving patients, 89% of homografts have continued to
39 ctional analysis was performed on 220 of 239 surviving patients (92%) to determine the late incidence
42 ort- and long-term health status outcomes of surviving patients after TAVR in the context of an unsel
47 y an extensive echocardiography protocol: in surviving patients and homografts, three valved conduits
48 aftment was achieved in nearly all evaluable surviving patients and was seen even after unconditioned
52 erval of 5.3 years (range 1 to 18.2), all 28 surviving patients are free of exercise limitation (func
58 functionally a crossover study in which only surviving patients can cross over in one direction (towa
64 ntation existed for 16 abnormalities (29% of surviving patients) during a mean follow-up of 3.2 years
65 e ability to KCH criteria but identified non-surviving patients earlier (4 [3--13] vs 10 [3.5--19.5]
70 subset of 31 patients with fatal HPS and 20 surviving patients for whom samples were available withi
71 bnormalities, but at the end of the trial no surviving patient had any detectable visual deficits rel
72 (1.7%) had major stroke, and 313 (92.6%) of surviving patients had good functional status (New York
77 after orthotopic liver transplantation, all surviving patients had relief of their pain, distention,
79 m the first week after the onset of HPS, all surviving patients had SNV-specific IgG responses, compa
88 netic abnormalities have been observed among surviving patients in both arms (2 of 14 ATG versus 1 of
89 year, 16 of 55 (29.1%) and 23 of 36 (63.9%) surviving patients in the early and delayed groups, resp
93 ive regimen was well tolerated, and in the 9 surviving patients it provided durable engraftment and w
94 c and clinical follow-up was obtained for 36 surviving patients (mean = 8 months; range: 1 week-5 yea
96 follow-up of 11.9 years (range 9.7-14.5 for surviving patients), men assigned immediate ADT had a si
99 7.9 years (range, 0.3 to 10.1 years) for 355 surviving patients, no differences were observed in OS (
102 ts were enrolled, with a median follow-up of surviving patients of 3.8 years (3-year EFS for all pati
106 iod of 5.6 years was assessed among all 1691 surviving patients (phase 1) and subsequently among 854
107 related symptom score after 12 months in the surviving patients (prediary 34.6 +/- 15.9, diary 21 +/-
108 a median follow-up duration of 24 months for surviving patients (range, 3 to 131 months), 112 of 289
117 sed between 1961 and March 1992, 77 (6.6% of surviving patients tested thus far) have evidence of HCV
118 dian lactate was significantly higher in non-surviving patients than in survivors both in the early s
119 With a median follow-up of 46 months for surviving patients, the 5-year probability of chronic GV
121 With a median follow-up of 26.3 months among surviving patients, the cumulative incidence of chronic
123 With a median follow-up of 15 months in surviving patients, the incidence of total grade 3+ toxi
126 median follow-up time of 79.5 months for all surviving patients, the median OS had not been reached f
127 With a minimum follow-up of 19 months in surviving patients, the median survival and 1-year survi
131 In addition, psychomotor development in surviving patients treated with betaine was normal in al
151 December 15, 2015, median follow-up for 263 surviving patients was 47.4 months (range, 0-110.7 month
163 aspartate aminotransferase levels in the 154 surviving patients were 0.5 mg/dL and 34 international u
164 8 year follow-up study was conducted wherein surviving patients were contacted by phone to evaluate s
168 fter treatment, and at last follow-up, all 6 surviving patients were free of blood product support an
169 es of death were "sudden" or "unknown." Most surviving patients were gainfully employed or attending
170 1.1% for transfemoral patients, and 73.5% of surviving patients were in New York Heart Association (N
179 years following the study year, only 52% of surviving patients who were initially prescribed lipid-l
180 r) have evidence of HCV infection, whereas 4 surviving patients who were transfused after March 1992
182 s, fungal or otherwise, were reported in the surviving patients, who ranged in age from 37 to 75 year
183 early-treated patients but in none of the 19 surviving patients with delayed treatment (P < .001).
185 ibition prevents the clinical progression of surviving patients with heart failure more effectively t
187 istribution of higher IgG antibody titers in surviving patients with HPS suggests that production of
189 pitalization for congestive heart failure in surviving patients with matched data decreased from 0.59
190 s of IFN- alpha were significantly higher in surviving patients with SEBOV infection, whereas the lev
193 4) and 83% of upper-arm AVFs (341 of 411) in surviving patients without thrombosis or AVF interventio
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