ライフサイエンスコーパス: susceptible cell

1 rus to receptors present on the surface of a susceptible cell.

2 cysteines are necessary for McpM to inhibit susceptible cells.

3 ubsequently confer cancer stemness traits to susceptible cells.

4 TLV-1) on both highly susceptible and poorly susceptible cells.

5 s that were infectious when transfected into susceptible cells.

6 ng viral destabilization after attachment to susceptible cells.

7 onstrate that the drug inhibits infection of susceptible cells.

8 e required for facilitating virus entry into susceptible cells.

9 the ligands TNF-alpha and TRAIL to kill the susceptible cells.

10 tic cytopathic effect by forming syncytia in susceptible cells.

11 minate the inserted sequences upon spread in susceptible cells.

12 n the formation of cation-selective pores in susceptible cells.

13 coreceptors for entry of HIV-1 strains into susceptible cells.

14 ae that inhibits protein synthesis and kills susceptible cells.

15 through stimulation of apoptotic programs in susceptible cells.

16 le Tva proteins to inhibit ALV(A) entry into susceptible cells.

17 te for P. falciparum) within a population of susceptible cells.

18 inding to RI and RII, and of PKA activity in susceptible cells.

19 gion of p6(Gag), Y36S-L41P, could not infect susceptible cells.

20 -Fas antibodies rapidly induces apoptosis in susceptible cells.

21 ral genome from the endosomal compartment of susceptible cells.

22 te degradation in the malignant hyperthermia-susceptible cells.

23 ation may be restricted by the quiescence of susceptible cells.

24 s essential for EV-D68 to bind to and infect susceptible cells.

25 ultimately leads to premature senescence of susceptible cells.

26 rus type 1 (HTLV-1) and can spread HTLV-1 to susceptible cells.

27 us was recovered after RNA transfection into susceptible cells.

28 virus particles which can be transmitted to susceptible cells.

29 tibodies that neutralize viral attachment to susceptible cells.

30 d viral integration and replication in HIV-1-susceptible cells.

31 ch are expressed to various degrees in HIV-1 susceptible cells.

32 ing transfection and subsequent infection of susceptible cells.

33 infect intraepithelial and subepithelial HIV-susceptible cells.

34 establishes sustained latent persistence in susceptible cells.

35 subunits that bind globotriaosylceramide on susceptible cells.

36 p53] host mRNA export following infection of susceptible cells.

37 icrodomains for entry into and egress out of susceptible cells.

38 xhibited similar one-step growth kinetics in susceptible cells.

39 tors and induces primarily necrotic death in susceptible cells.

40 s B virus (HBV) in attachment and entry into susceptible cells.

41 not needed for HBV attachment and entry into susceptible cells.

42 hrough binding the proteoglycan receptors of susceptible cells.

43 had infected the same small subpopulation of susceptible cells.

44 mplex intracellular pathway in order to kill susceptible cells.

45 verting enzyme 2, the functional receptor on susceptible cells.

46 receptors is essential for HSV infection of susceptible cells.

47 seminate viral particles to target organs or susceptible cells.

48 rains of primate immunodeficiency viruses to susceptible cells.

49 ate RNA degradation in neurons than in other susceptible cells.

50 anisms associated with the entry of EAV into susceptible cells.

51 ogenic potential of the Env protein in other susceptible cells.

52 between highly susceptible cells and poorly susceptible cells.

53 terminal repeat of p65 and p50 proteins from susceptible cells after exposure to HRF, and the binding

54 ansfer human immunodeficiency virus (HIV) to susceptible cells, although the underlying mechanism is

55 n E9 protein toxin to enter the cytoplasm of susceptible cells and kill them by hydrolysing their DNA

56 titers of HTLV-1 pseudotypes between highly susceptible cells and poorly susceptible cells.

57 viruses as they trigger cytotoxicity against susceptible cells and secrete proinflammatory cytokines

58 s-susceptible cells, binds preferentially to susceptible cells, and competes with mature virus for bi

59 sing, differences between O2-tolerant and O2-susceptible cells, and differences between acute and chr

60 for attachment and binding to receptor(s) on susceptible cells, and GP2, which mediates viral and cel

61 the leptin receptor conferred resistance in susceptible cells, and leptin stimulation enhanced prote

62 ereby decreasing the viral burden, access to susceptible cells, and the chronic activation of the imm

63 ciencies of infection of resistant cells and susceptible cells are similar, but resistant cells (C3H/

64 in AcH in aqueous solution and vesicles from susceptible cells, as well as the ability of trifluoroet

65 particle) upon interaction with receptors on susceptible cells at 37 degrees C.

66 , as well as the state of differentiation of susceptible cells at the time of infection, affects the

67 nt variable in this context is the number of susceptible cells available for virus replication.

68 binds with saturable character to poliovirus-susceptible cells, binds preferentially to susceptible c

69 of the Fas/FasL system induces apoptosis of susceptible cells, but may also lead to nuclear factor k

70 ptosis-inducing ligand) induces apoptosis in susceptible cells by binding to death receptors 4 (DR4)

71 Infection of susceptible cells by herpes simplex virus (HSV) can lead

72 Upon infection of susceptible cells by HIV-1, the conical capsid formed by

73 Hepatitis C virus (HCV) gains entry into susceptible cells by interacting with cell surface recep

74 HIV-1 can disseminate between susceptible cells by two mechanisms: cell-free infection

75 /RV) produce less genomic RNA than congenic, susceptible cells (C3H/He).

76 Expressing Abs on the surface of HIV-1-susceptible cells can be a new way to fight HIV-1.

77 The block to expression in less susceptible cells does not appear to result from the abi

78 dic platform was capable of delineating drug susceptible cells, drug tolerant, and drug resistant cel

79 Upon coculture with CD4(+)-susceptible cells, epithelial cells can effectively tran

80 qCXCL16 acts as an EAV entry receptor in EAV-susceptible cells, equine monocytes.

81 eumococci in mouse lungs were outcompeted by susceptible cells even during Cm treatment.

82 iate the lipid-mediated apoptotic cascade in susceptible cells, failed to either induce PT or release

83 In less susceptible cells, Fas transduces apoptosis-independent

84 to ensure the maintenance of a local pool of susceptible cells for additional rounds of virus replica

85 in the infant mucosa provide a large pool of susceptible cells for ingested HIV-1 at birth and during

86 WNV antibodies do not always protect susceptible cells from WNV infection in vitro.

87 The sequence data revealed that the EHV-1-susceptible cells had acquired an E. caballus MHC-I cDNA

88 s in many transformed cells due, in part, to susceptible cells harboring defects in the IFN system.

89 st in vitro, to promote virus replication in susceptible cells.IMPORTANCE AKAV and SBV are the etiolo

90 Understanding why and how susceptible cells in motor and nonmotor regions of the b

91 The mechanisms by which viruses kill susceptible cells in target organs and ultimately produc

92 of the dose required for 50% infectivity of susceptible cells in tissue culture (TCID(50)).

93 HIVs and simian immunodeficiency viruses to susceptible cells in trans.

94 ssays in measuring recent HIV-1 infection of susceptible cells in vivo needs to be reevaluated.

95 Skp2 becomes an essential survival gene when susceptible cells incur Rb1 deficiency.

96 How does the number of susceptible cells influence the growth potential of the

97 r understanding of how HIV is transmitted to susceptible cells is critical to devise effective strate

98 d by bacteria binds to membrane receptors of susceptible cells, is cleaved proteolytically, attaches

99 brane protein (p38.5) from a prototypical NK-susceptible cell line (K562) that preferentially bound t

100 Equivalent fractions from a second scrapie-susceptible cell line also stimulate conversion.

101 ells from infected tissues were mixed with a susceptible cell line, the subtype C isolates were outco

102 ed from gradient centrifugation of a scrapie-susceptible cell line.

103 xpressed on the surface of MARC-145, a PRRSV-susceptible cell line.

104 erlotinib decreased c-fos mRNA levels in the susceptible cell lines A431, CAL27, and HN11; however, b

105 ed 28S rRNA cleavage was detected in all MHV-susceptible cell lines and all MHV strains tested.

106 structure and assembly of PaV, we identified susceptible cell lines and developed a reverse genetic s

107 We also used susceptible cell lines and our modified cell blot proced

108 tudied in an attempt to identify potentially susceptible cell lines for virus propagation in vitro an

109 g that use of a combination of different HRV-susceptible cell lines is the best approach for the reco

110 Using genomic approaches, we show that the susceptible cell lines overexpress cytidine deaminase (C

111 In vitro, even in the most susceptible cell lines, more than 50% of cells latently

112 ar stomatitis virus, and influenza virus, in susceptible cell lines.

113 -FADD association and caspase-8 cleavage) in susceptible cell lines.

114 esistant mitochondria via elimination of Fas-susceptible cells may suggest the existence of a shared

115 infectivity, or it could block receptors on susceptible cells near the sites of transmission.

116 are small polypeptides (7-14 kDa) which kill susceptible cells of closely related fungal species.

117 In susceptible cells only, sigma antagonists evoke a rapid

118 ent modalities such as gene therapy of HIV-1-susceptible cell populations, must be capable of engende

119 ny antineoplastic agents induce apoptosis in susceptible cells raises the possibility that factors af

120 Transfection of these cosmid clones into MDV-susceptible cells resulted in the generation of a recomb

121 n-resistant revertants, isolated from highly susceptible cells, revealed a gene expression signature

122 To further understand how rotaviruses enter susceptible cells, six different polarized epithelial ce

123 R) as the receptor through which they infect susceptible cells, some CVB strains are known for their

124 y intermediate because it accumulates inside susceptible cells soon after infection and drugs which i

125 the activation or differentiation state of a susceptible cell such as a macrophage.

126 ation and secretion of H1B from target HIV-1 susceptible cells, suggesting that ubiquitinated H1B is

127 s with an EGFR complementary DNA renders non-susceptible cells susceptible to HCMV.

128 its increased binding to and infectivity for susceptible cell targets.

129 ant cancers contained a higher percentage of susceptible cells than cancers from noncastrated mice.

130 to elute unproductively from the surface of susceptible cells, their precise role remains unclear.

131 ute induction of EphA2 may purge genetically susceptible cells, thereby uncovering a more aggressive

132 Syringacin M kills susceptible cells through a highly specific phosphatase

133 Viruses are thought to spread across susceptible cells through an iterative process of infect

134 1b were serially diluted and cocultured with susceptible cells to amplify virus.

135 We determined whether exposure of susceptible cells to Tat protein of HIV could result in

136 he minimum inhibitory concentration (MIC) of susceptible cells to the MIC of the least susceptible, s

137 ld promote cell proliferation and predispose susceptible cells to tumorigenesis.

138 cell activation can be abrogated by exposing susceptible cells (tumor and mycobacteria-infected cells

139 ers in fetal brain development, are the most susceptible cell type for HCMV infection in the fetal br

140 cDNAs from this EHV-1-susceptible cell type were inserted into EHV-1-resistant

141 of regional and developmental variations in susceptible cell types and supportive microenvironments

142 Abnormal growth and maturation of susceptible cell types, particularly neurons and oligode

143 NFalpha family that can trigger apoptosis in susceptible cells via respective death receptors (DRs).

144 BDV G-mediated attachment to BDV-susceptible cells was cholesterol independent, but G loc

145 L activity levels in congenic resistant and susceptible cells were compared.

146 At the initiation of treatment, susceptible cells were either small and at early stages

147 heptapeptide facilitates McC transport into susceptible cells, where it is processed releasing a non

148 can be transmitted in cocultures to adherent susceptible cells, which become infected, express viral

149 MDV concurrently loses its ability to infect susceptible cells while gaining the capacity to infect i

150 Furthermore, pretreatment of susceptible cells with anti-beta(3) antibodies such as c

151 studies of IFN production after infection of susceptible cells with measles virus (MeV) have often re