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1 d by initially unorganized embryos lacking a suspensor.
2  expressed either in the proembryo or in the suspensor.
3 oper establishment in the absence of a basal suspensor.
4 cell and a basal cell that gives rise to the suspensor.
5 aughter cell forms the mostly extraembryonic suspensor.
6 ent developmental domains, the proembryo and suspensor.
7 basal daughter cell into the extra-embryonic suspensor.
8  pole from its normal position on top of the suspensor.
9 roper and abnormal cell divisions within the suspensor.
10 stead of differentiating the extra-embryonic suspensor.
11 al mass towards the distal end of the embryo suspensor.
12 tion suggests preferential expression in the suspensor.
13 s of the whole embryo, the proembryo and the suspensor.
14 pmental delay in another mutant with shorter suspensors.
15                               Defects in the suspensor, a normally transient structure derived from t
16 tion alleles cause exaggerated growth of the suspensor and can suppress embryonic development to a de
17  Cell division was also aberrant both in the suspensor and embryo proper.
18  These results confirm the importance of the suspensor and suspensor-driven auxin transport in patter
19           The defective embryos have shorter suspensors and reduced growth along the longitudinal axi
20 proper formation from the distal cell of the suspensor, and a pathway characterized by initially unor
21  from double fertilization (i.e. the embryo, suspensor, and endosperm) and in apomictic, somatic, and
22 zation in flowering plants (i.e. the embryo, suspensor, and endosperm).
23  sufficient to activate transcription in the suspensor, and that a sequence (TTGGT) between the 10-bp
24 ize a root independent of MONOPTEROS and the suspensor around a new boundary marked by the auxin maxi
25        This creates a useful system to study suspensor biology.
26 protein, was shifted in the basal embryo and suspensor but does not support a strong direct link to a
27 aspase mcII-Pa, a key protease essential for suspensor cell death.
28  embryonal mass cells to thick cables in the suspensor cells in which the microtubule network is comp
29 n one pole and the terminally differentiated suspensor cells on the other, separated by the embryonal
30 g embryonal mass, while it was absent in the suspensor cells.
31  cells and the microtubules disrupted in the suspensor cells.
32 ike" cellular protuberances with an enlarged suspensor characteristic of other raspberry embryo mutan
33 r to necrotic death, resulting in failure of suspensor differentiation and embryonic arrest.
34  confirm the importance of the suspensor and suspensor-driven auxin transport in patterning, but also
35  how genes are activated specifically in the suspensor during early embryo development.
36  genes are activated specifically within the suspensor during early embryo development.
37 eptor-like kinase SHORT SUSPENSOR to promote suspensor elongation through the YODA mitogen-activated
38 us during early stages of development in the suspensor, embryo, and endosperms.
39      Cells of the embryo proper, but not the suspensor, exhibit abnormalities in size and shape.
40 gotic-like pathway, characterized by initial suspensor formation followed by embryo proper formation
41 er simple Arabidopsis (Arabidopsis thaliana) suspensor has on embryogenesis.
42  nutrient transport has been ascribed to the suspensor in species with prominent suspensor structures
43 es and other gymnosperms form well-developed suspensors in somatic embryogenic cultures.
44 es required to activate transcription in the suspensor, including the 10-bp motif (GAAAAGCGAA) and a
45           We conclude that the length of the suspensor is crucial for fast developmental progression
46 ell divisions in the basal embryo domain and suspensor led to diverse defects during embryogenesis in
47 cts in the early embryo and markedly reduced suspensor length.
48      LEC2 is required for the maintenance of suspensor morphology, specification of cotyledon identit
49                                          The suspensor needs the lowest levels, followed by the root
50 re required to activate transcription in the suspensor of a plant embryo after fertilization.
51 541, that accumulate specifically within the suspensor of globular-stage embryos.
52 r, we show that vacuolar death in the embryo suspensor of Norway spruce requires autophagy.
53 ar mechanisms by which the embryo proper and suspensor of plant embryos activate specific gene sets s
54 ar mechanisms by which the embryo proper and suspensor of plant embryos activate specific gene sets s
55 ted specifically within the basal region and suspensor of preglobular tobacco embryos.
56 e basal cells that normally give rise to the suspensor proliferate abnormally, giving rise to multipl
57 sion, leading to defects in the cells of the suspensor, root and hypocotyl precursors, and provascula
58 of the 5 tandem repeats is required to drive suspensor-specific transcription.
59                                Subsequently, suspensor-specific WOX8 expression disappears while proe
60 d kinase (IRAK)/Pelle-like kinase gene SHORT SUSPENSOR (SSP) regulates this pathway through a previou
61  pattern formation and associated PCD in the suspensor, strongly suggesting that the actin network is
62 d to the suspensor in species with prominent suspensor structures.
63 er level within the large basal cells of the suspensor that anchor the embryo to the surrounding seed
64 domain required for transcription within the suspensor that contains several copies of a conserved mo
65 g plant embryogenesis is the extra-embryonic suspensor that positions the embryo in the lumen of the
66  of PtNIP1;1 transcript in embryo proper and suspensor tissues by reverse transcription-polymerase ch
67 originating from the proembryo instructs the suspensor to generate the root stem cell niche.
68 stically with the receptor-like kinase SHORT SUSPENSOR to promote suspensor elongation through the YO
69 f the G564 upstream region is sufficient for suspensor transcription and contains at least three requ
70 Region 2 and Fifth motifs) is sufficient for suspensor transcription, and represents a cis-regulatory
71  comparing motif sequences shown to activate suspensor transcription.
72 letion of these repeats results in a loss of suspensor transcription.
73  the 10-bp-like motifs is also necessary for suspensor transcription.
74 regulatory module that are required for G564 suspensor transcription: the Fifth motif (5'-GAGTTA-3')
75 64 mRNA accumulates uniformly throughout the suspensor, whereas C541 mRNA accumulates to a higher lev
76 ort for embryo proper specification from the suspensor, while the suspensorless pathway is polar auxi

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