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1 ly: either accelerating it or initiating net sutural bone resorption for various therapeutic objectiv
2 th autosomal dominant congenital nuclear and sutural cataracts was identified and characterized.
3 ome characterized by late-closing fontanels, sutural cataracts, facial dysmorphisms and skeletal defe
4 2.3, in a family with congenital nuclear and sutural cataracts.
5 1 also results in changes in the identity of sutural cells prior to overt osteogenic differentiation,
6  is expressed in a layer of mesoderm-derived sutural cells that lie along the osteogenic-non-osteogen
7  the mutant neurocranium, we found a midline sutural defect and craniosynostosis with abnormal osteob
8 pe, impaired secondary fiber cell migration, sutural defects and thinning of the posterior capsule wh
9 sults show that lens opacities and posterior sutural defects occur when mutant R116C alphaA-crystalli
10  are sufficient to induce lens opacities and sutural defects.
11                             Cranio-lenticulo-sutural dysplasia (CLSD) is an autosomal recessive syndr
12                             Cranio-lenticulo-sutural dysplasia (CLSD) was recently shown to be caused
13 phenotype of individuals with cranio-lentico-sutural dysplasia (CLSD).
14  skeletal disorders such as cranio-lenticulo-sutural dysplasia and osteogenesis imperfect, caused by
15 As SEC23A lesions cause the cranio-lenticulo-sutural dysplasia syndrome, crusher provides the first v
16 382L in human Sec23A causes cranio-lenticulo-sutural dysplasia, and we suggest that this mutation dis
17 ollagen deposition defects, cranio-lenticulo-sutural dysplasia, or chylomicron retention disease, but
18 entified from patients with cranio-lenticulo-sutural dysplasia, we discovered that the SEC23-SEC31 in
19 nical stimuli have the potential to modulate sutural growth effectively: either accelerating it or in
20                                              Sutural growth is accelerated upon small doses of oscill
21                                        Thus, sutural growth is determined by hereditary and mechanica
22  mechanical stimuli can effectively modulate sutural growth.
23 rstanding of how mechanical stimuli modulate sutural growth.
24 ne hypoplasia and persistent widening of the sutural joints.
25 nx2-II, and pSmad1/5/8 expression as well as sutural mesenchymal cell proliferation.
26 ones and conversely, what is the fate of the sutural mesenchymal cells when calvarial bones approxima
27 tical event of CS is the early loss of basal sutural mesenchyme as the osteogenic fronts, expressing
28 p-regulation of osteopontin and Fgfr1 in the sutural mesenchyme, down-regulation of Fgfr2, and inhibi
29 uding premature osteogenic conversion of the sutural mesenchyme.
30  Notch2 and the Notch effector, Hes1, in the sutural mesenchyme.
31                The average elastic moduli of sutural mineralization fronts 30 micro m away were signi
32        The elastic properties of sutures and sutural mineralization fronts are potentially useful for
33 acial sutures and their immediately adjacent sutural mineralization fronts have different capacities
34 maticotemporal (ZTS)-and their corresponding sutural mineralization fronts in 8 young New Zealand Whi
35          Our approach consists of defining a sutural morphospace, assigning functional fields to that
36  by nearly threefold through enhancements in sutural remodeling compared with unmodified E2 administr
37 sile and compressive strains induce anabolic sutural responses beyond natural growth.
38  waveforms induce corresponding waveforms of sutural strain.

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