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1 l cells which is characteristic for virulent swine influenza viruses.
2 modon hispidus) are susceptible to avian and swine influenza viruses.
3 nd might reassort with currently circulating swine influenza viruses.
4 es and antigenically distinct from reference swine influenza viruses.
6 lb09]), with that of the 1918-like classical swine influenza virus (A/swine/Iowa/1930 [IA30]) in the
7 y shown to enhance the replication of a 1976 swine influenza virus also significantly improved the re
8 th the reconstructed 1918 virus, a 1976 H1N1 swine influenza virus, and a highly pathogenic H5N1 viru
9 uman H1N1 influenza A virus strains, several swine influenza viruses, and influenza B viruses but wer
10 uenza viruses, unlike other human, avian and swine influenza viruses, are resistant to the antiviral
11 logic factors that limit the transmission of swine influenza viruses between humans are unresolved.
12 large-scale genomic characterization of 290 swine influenza viruses collected from 14 European count
13 y (typically found in avian and classic H1N1 swine influenza viruses), conferring binding to human- a
14 CD8(+) T-cell epitopes in NP of human versus swine influenza virus, consistent with the idea that the
16 ses--containing genes from avian, human, and swine influenza viruses--emerged and became enzootic amo
17 ttle known about the host barriers that keep swine influenza viruses from entering the human populati
18 se data highlight the increased diversity of swine influenza viruses in the United States and would i
19 n that infection in humans with the pandemic swine influenza virus induces antibodies with specificit
22 e its absence from some animal (particularly swine) influenza virus isolates, variable expression in
23 nes can be recombined from human, avian, and swine influenza viruses, leading to triple reassortants.
25 recursor strains from the triple-reassortant swine influenza virus lineage, which cause only sporadic
27 However, when transferred into avian and swine influenza viruses, only partial ts and attenuation
28 , and nonstructural genes being of classical swine influenza virus origin, and the PA and PB2 polymer
30 The recent flu epidemic caused by an H1N1 swine influenza virus presents an opportunity to examine
31 Biosystems 7500 Fast platform, using the CDC swine influenza virus real-time RT-PCR detection panel (
34 raction between Mycoplasma hyopneumoniae and swine influenza virus (SIV) in the induction of pneumoni
35 dentifying a student with triple-reassortant swine influenza virus (SIV) infection and pig exposure a
36 ole of the NS1 protein in the virulence of a swine influenza virus (SIV) isolate in pigs by using rev
43 rotected completely against lethal avian and swine influenza virus strains in mice, and induced robus
47 to a representative human triple-reassortant swine influenza virus that has circulated in pigs in the
48 e epitopes in parallel lineages of human and swine influenza viruses that have been diverging since r
52 ing a panel of 28 distinct human, avian, and swine influenza viruses, we found that only a small subs
53 ls which recovered from exposure to virulent swine influenza virus were completely resistant to infec
54 onstrate here that an engineered reassortant swine influenza virus, with the same gene constellation
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