ライフサイエンスコーパス: synaptic plasticity

1 the strength by which they connect neurons (synaptic plasticity).

2 lent' neural states (for example, short-term synaptic plasticity).

3 k parameters arising from learning-dependent synaptic plasticity.

4 ) due to its impact on synapse formation and synaptic plasticity.

5 um (Ca(2+)) signals to initiate long-lasting synaptic plasticity.

6 al stimulation highlight their importance in synaptic plasticity.

7 neuronal- and astrocytic metabolism, and (c) synaptic plasticity.

8 cortical development or experience-dependent synaptic plasticity.

9 tentiation (STP), a major form of short-term synaptic plasticity.

10 nsmission in humans that may favor increased synaptic plasticity.

11 release probability and abnormal short-term synaptic plasticity.

12 n extracellular matrix (ECM) remodelling and synaptic plasticity.

13 ical pathways, including those that regulate synaptic plasticity.

14 cal regions, a function that requires marked synaptic plasticity.

15 eton dynamics, injured neurons regeneration, synaptic plasticity.

16 ndent switch to a closed conformation during synaptic plasticity.

17 further source of Ca(2+) that contributes to synaptic plasticity.

18 nt and selective means to enhance memory and synaptic plasticity.

19 angement as a key determinant for inhibitory synaptic plasticity.

20 l neurons, which affects 'Hebbian' long-term synaptic plasticity.

21 d be an important dynamic process underlying synaptic plasticity.

22 hippocampal neurons, which affects 'Hebbian' synaptic plasticity.

23 in the cortex and hippocampus and to augment synaptic plasticity.

24 stimuli - is thought to be a consequence of synaptic plasticity.

25 trophysiological changes taking place during synaptic plasticity.

26 edure enhances fear learning by engaging VTA synaptic plasticity.

27 ecognition molecules have been implicated in synaptic plasticity.

28 n dendritic spine morphology associated with synaptic plasticity.

29 X), a common chemotherapeutic agent, impairs synaptic plasticity.

30 naling controls both synapse development and synaptic plasticity.

31 rged as an important regulator of excitatory synaptic plasticity.

32 into the role of the TA input in hippocampal synaptic plasticity.

33 ce lacking EAAC1 restores D1R expression and synaptic plasticity.

34 f the stimuli evoking the different forms of synaptic plasticity.

35 rm potentiation (LTP), a form of hippocampal synaptic plasticity.

36 ogy by contributing to neurotransmission and synaptic plasticity.

37 ular determinant for SC-driven neuromuscular synaptic plasticity.

38 h as vasodilation, platelet aggregation, and synaptic plasticity.

39 tal processes, including differentiation and synaptic plasticity.

40 reorganization of the actin cytoskeleton in synaptic plasticity.

41 NMDA receptors and is involved in mediating synaptic plasticity.

42 through many processes known collectively as synaptic plasticity.

43 and synaptic NMDAR and EphB2, and long-term synaptic plasticity.

44 ay thereby influence AMPAR clustering during synaptic plasticity.

45 specificity and the precision in structural synaptic plasticity.

46 HFD-induced metabolic changes and preserved synaptic plasticity.

47 ptor (D1R) expression, and ensures long-term synaptic plasticity.

48 ctions in NMDA receptor-mediated hippocampal synaptic plasticity.

49 release neuroactive molecules and influence synaptic plasticity.

50 thalamocortical system, similar to chemical synaptic plasticity.

51 ed for global cell-wide and pathway-specific synaptic plasticity.

52 and invasiveness, and define its new role in synaptic plasticity.

53 pine development and its modification during synaptic plasticity.

54 of its critical role in neurodevelopment and synaptic plasticity.

55 determinant for the expression of inhibitory synaptic plasticity.

56 ity ( 10(4)) and the peculiar time-dependent synaptic plasticity.

57 proteins in the prefrontal cortex related to synaptic plasticity.

58 n the induction of NMDA receptor independent synaptic plasticity.

59 d SR supporting neurotransmitter release and synaptic plasticity.

60 olishes post-tetanic potentiation, a form of synaptic plasticity.

61 ing, decreased synaptic activity and reduced synaptic plasticity.

62 inks between changes in binding kinetics and synaptic plasticity.

63 known to be critical for activity-dependent synaptic plasticity.

64 dom connectivity [7], sparse coding [8], and synaptic plasticity [9, 10]).

65 e kinase activations contribute to long-term synaptic plasticity, a cellular mechanism mediating long

66 This rule, named behavioral time scale synaptic plasticity, abruptly modifies inputs that were

67 We also discovered penetrant enhanced synaptic plasticity across mouse models that may be link

68 difying rules that govern activity-dependent synaptic plasticity, addictive drugs can derail the expe

69 ct physiological evidence linking obesity to synaptic plasticity akin to that occurring in addiction

70 ive morphological, electrophysiological, and synaptic plasticity alterations.

71 We postulate that electrical synaptic plasticity among inhibitory neurons directly in

72 lly modelling octopaminergic modification of synaptic plasticity among local neurons in the antennal

73 aling cascade modulates experience-dependent synaptic plasticity, among other processes.

74 ic structure and function are referred to as synaptic plasticity and are thought to be the basis of l

75 misfolding, and fibrillization of tau impair synaptic plasticity and cause degeneration.

76 Rho GTPases and other Rho-linked proteins in synaptic plasticity and cognitive function and dysfuncti

77 hibit betaAR-mediated effects on hippocampal synaptic plasticity and cognitive function.

78 ulate betaAR-mediated effects on hippocampal synaptic plasticity and cognitive function.

79 he MF projections plays an essential role in synaptic plasticity and contextual memory formation.

80 lator of striatal activity via modulation of synaptic plasticity and gene transcription.

81 after systemic administration and increases synaptic plasticity and hippocampal-dependent cognition

82 Synaptic plasticity and homeostatic regulation of synaps

83 improved cognitive flexibility, strengthened synaptic plasticity and induced stress resilience.

84 ating that NPY release modulates hippocampal synaptic plasticity and is impaired by predator scent st

85 Protein Kinase A (PKA) mediates synaptic plasticity and is widely implicated in learning

86 We review inhibitory synaptic plasticity and its roles in shaping both feedfo

87 n mGluR-LTD and is involved in many forms of synaptic plasticity and learning and memory.

88 een changes of brain state and modulation of synaptic plasticity and learning remains elusive.

89 he brain state into a shift of the regime of synaptic plasticity and learning.

90 state changes into a shift of the regime of synaptic plasticity and learning.

91 ral window of CaMKII activation required for synaptic plasticity and learning.

92 argement in AD and the earliest initiator of synaptic plasticity and long-term memory impairment.

93 The role of Arc in synaptic plasticity and memory consolidation has been in

94 ogical stimulation of BDNF signaling rescued synaptic plasticity and memory deficits in Ts65Dn mice.

95 Long-lasting forms of synaptic plasticity and memory require de novo protein s

96 in the healthy brain which, in turn, boosts synaptic plasticity and memory.SIGNIFICANCE STATEMENT Am

97 he AMPA receptor subunit GluA3 in cerebellar synaptic plasticity and motor learning in mice.

98 cidergic systems as well as abnormalities in synaptic plasticity and neural connectivity, are current

99 esults showed that antidepressants protected synaptic plasticity and neuronal circuitry from the effe

100 -1 and calpain-2, play opposite functions in synaptic plasticity and neuronal survival/death, which m

101 chidonylglycerol degradation, fully restored synaptic plasticity and normalized emotional and cogniti

102 to disruption of dendritic spine morphology, synaptic plasticity and plasticity-related gene expressi

103 aroused brain state to promote induction of synaptic plasticity and potentially to enhance spike tim

104 e inputs, allowing for selective reversal of synaptic plasticity and providing a cellular basis for d

105 We discuss the functional changes in synaptic plasticity and regenerative potential of the ag

106 etamine may be due to its ability to restore synaptic plasticity and related effects on sleep-wake an

107 covery of hippocampal adult neurogenesis and synaptic plasticity and restored cognitive performance i

108 sion, dopamine dynamics, and cocaine-induced synaptic plasticity and rewarding effects.

109 glutamate receptor expression, and impaired synaptic plasticity and spatial learning and memory in 3

110 day 21 (P21) and measured synaptic function, synaptic plasticity and spine numbers in acute hippocamp

111 ponse associated with changes in hippocampal synaptic plasticity and transient cognitive decline, how

112 st biochemical signal cascades important for synaptic plasticity and, ultimately, learning and memory

113 ablishment of the body axis, cell migration, synaptic plasticity, and a vast range of other biologica

114 vated stereotypy, decreased neurogenesis and synaptic plasticity, and abnormally reduced local excita

115 d by postnatal deficits in neuronal numbers, synaptic plasticity, and cognitive and motor function.

116 itory synaptic transmission, cocaine-induced synaptic plasticity, and drug-cue associative learning.

117 are key regulators of neuronal excitability, synaptic plasticity, and excitation-transcription coupli

118 1 showed abnormal excitatory synapse number, synaptic plasticity, and hippocampal-dependent learning

119 2+) channels regulate neuronal excitability, synaptic plasticity, and learning and memory.

120 pression completely restores synapse number, synaptic plasticity, and long-term memory.

121 odulation of neuritogenesis, synaptogenesis, synaptic plasticity, and memory consolidation during dev

122 in increased transmitter release, failure of synaptic plasticity, and memory loss.

123 luding alterations in synaptic transmission, synaptic plasticity, and spine density.

124 hes in cellular repair, neuronal networks in synaptic plasticity, and the distinction of molecular sy

125 CANCE STATEMENT The mechanisms of inhibitory synaptic plasticity are poorly understood, mainly becaus

126 stigate adaptations in synaptic function and synaptic plasticity arising from aSI.

127 ardation protein (FMRP) pathway may underlie synaptic plasticity associated with aggression escalatio

128 y identified fast-expressing form of Hebbian synaptic plasticity (associative short-term potentiation

129 pairments in learning and memory, as well as synaptic plasticity at an earlier age than mAPP mice.

130 During synaptic plasticity at excitatory synapses, numerous str

131 ute to the maintenance of different forms of synaptic plasticity at separate inputs, allowing for sel

132 siological changes during activity-dependent synaptic plasticity at the Drosophila neuromuscular junc

133 Here, we analyzed events of structural synaptic plasticity at the single-synapse level after di

134 synthesis in mature axons may play a role in synaptic plasticity, axonal arborization, or functional

135 the differences in dendritic integration and synaptic plasticity between dorsal and ventral CA1 pyram

136 me serine racemase after CCI injury improved synaptic plasticity, brain oscillations, and learning be

137 rtate receptors (NMDARs) plays a key role in synaptic plasticity, but excessive tonic NMDAR activatio

138 AKT is indirectly implicated in synaptic plasticity, but its direct role has not been st

139 n Drosophila, this mechanism is required for synaptic plasticity, but its role may extend beyond the

140 cation is crucial for neural development and synaptic plasticity, but the molecular players involved

141 AD brains, promotes memory loss and disrupts synaptic plasticity by reducing postsynaptic KIdney/BRAi

142 We examined modulation of synaptic plasticity by selective serotonin reuptake inhi

143 erexcitability and alterations in short-term synaptic plasticity consistent with enhanced neurotransm

144 nt history of activity and the threshold for synaptic plasticity conspire to effect divergent learnin

145 plasticity.SIGNIFICANCE STATEMENT Persistent synaptic plasticity contributes to the maintenance of lo

146 ce of amyloid plaques) to impair hippocampal synaptic plasticity, decrease synapses, induce tau hyper

147 erine reversed both the memory formation and synaptic plasticity deficits.

148 sting beneficial effects upon axonal health, synaptic plasticity, dementia-related amyloid-beta (Abet

149 We propose that synaptic plasticity determines synaptic connectivity map

150 rtex specific to dendrites, the main site of synaptic plasticity.Different stages of sleep, marked by

151 Synaptic plasticity (e.g., long-term potentiation [LTP])

152 We explore mechanisms through which synaptic plasticity, engrams, and long-range synchrony m

153 Here, we review data on synaptic plasticity, engrams, and network oscillations i

154 Across many studies, animals with enhanced synaptic plasticity exhibit either enhanced or impaired

155 ent plasticity (STDP) is a form of long-term synaptic plasticity exploiting the time relationship bet

156 The unusual form of synaptic plasticity expressed by the LPP (lppLTP) was pr

157 lutamate receptors is involved in regulating synaptic plasticity following aggressive experience.

158 fy distinct expression patterns and roles in synaptic plasticity for AKT isoforms in the hippocampus.

159 To separate the effects of APP-ICD on synaptic plasticity from Abeta-dependent effects, we cre

160 r level, immediate early genes are among the synaptic plasticity genes that are both induced by long-

161 central nervous system (e.g., neurogenesis, synaptic plasticity, glial cell development) and immune

162 roles of D-serine in NMDA receptor-mediated synaptic plasticity have been reported.

163 Although LTM is sustained by structural synaptic plasticity, how synapses integrate spaced stimu

164 LTM is supported by structural synaptic plasticity; however, how synapses integrate spa

165 we examined the role of MeCP2 in homeostatic synaptic plasticity (HSP) at excitatory synapses.

166 previously implicated in brain development, synaptic plasticity, immune function and/or schizophreni

167 NMDA receptor-dependent memory formation and synaptic plasticity impairments only in male progeny, wh

168 y for improving manual skills is mediated by synaptic plasticity in a region of motor cortex that, be

169 for AKT in multiple paradigms of hippocampal synaptic plasticity in area CA1.

170 ecific changes in intrinsic excitability and synaptic plasticity in basolateral amygdala neurons that

171 Synaptic plasticity in BLA neurons is essential for asso

172 essants have been shown to restore disrupted synaptic plasticity in both animal models and humans; ho

173 ngless/Wnt molecule is a potent regulator of synaptic plasticity in both vertebrates and invertebrate

174 lcohol craving that involves MMP-9-dependent synaptic plasticity in CeA.

175 Reflecting recent findings of fast Hebbian synaptic plasticity in cortex, we test whether a cortica

176 been demonstrated to play important roles in synaptic plasticity in cultured neurons.

177 d optogenetics to examine whether changes in synaptic plasticity in D1- versus D2-MSN GABAergic synap

178 ate the effect of chronic nicotine (cNIC) on synaptic plasticity in dopamine D2 receptor-expressing m

179 transmission and cocaine-induced inhibitory synaptic plasticity in dopamine neurons of the ventral t

180 al models but the functional implication for synaptic plasticity in HD remains unclear.

181 DAR) dysfunction, we analyzed NMDA-dependent synaptic plasticity in hippocampal slices from Tg(CJD) m

182 Expression of EEA1 restored homeostatic synaptic plasticity in Mecp2-deficient neurons, providin

183 repeated drinking bouts modulate differently synaptic plasticity in medium spiny neurons of the accum

184 hown to be essential for normal learning and synaptic plasticity in mice, but its requirement for hum

185 ntate gyrus (DG) granule cell reactivity and synaptic plasticity in naive and in MDA-kindled anaesthe

186 ether, these data reveal that cocaine-evoked synaptic plasticity in PL-mPFC is reversible in vivo, an

187 tle evidence exists to support the idea that synaptic plasticity in place cells is involved in formin

188 ta indicate that neuron-derived E2 modulates synaptic plasticity in rodent BLA sex-dependently.

189 Zuo, Panda et al., demonstrate adaptive synaptic plasticity in SmNiO3 perovskites to address cat

190 exposure to cocaine can trigger long-lasting synaptic plasticity in SPNs leading to behavioral altera

191 h reveals a sexually dimorphic regulation of synaptic plasticity in the BLA involving neuronal aromat

192 ecognition memory and attention, and reduced synaptic plasticity in the hippocampal-medial prefrontal

193 ctor-tropomyosin receptor kinase B-dependent synaptic plasticity in the hippocampus, which could acco

194 xtends our understanding of the mechanism of synaptic plasticity in the hypothalamus and suggests new

195 dent facilitation of extinction learning and synaptic plasticity in the IL-PFC involves functional in

196 Experience-driven synaptic plasticity in the lateral amygdala is thought t

197 earning rule that governs activity-dependent synaptic plasticity in the mouse prefrontal cortex, char

198 studies, we developed the working model that synaptic plasticity in the nucleus accumbens is central

199 her, our data indicate that nicotine-induced synaptic plasticity in the OFC and cognitive changes dep

200 We report that nicotine controls synaptic plasticity in the OFC through NRG3/ErbB4-depend

201 ABSTRACT: Synaptic plasticity in the striatum adjusts behaviour ad

202 cue associative learning and cocaine-induced synaptic plasticity in VTA dopamine neurons.

203 lescent aged mice identifies a novel form of synaptic plasticity in VTA GABA cells, and the synaptic

204 udy identifies a novel form of glutamatergic synaptic plasticity in VTA GABA neurons, a currently und

205 ion (PTP) is a widespread form of short-term synaptic plasticity in which a period of elevated presyn

206 ress via a modulation of Ca(2+) channels and synaptic plasticity independent of SERT.

207 ong-term depression (mGluR-LTD) is a form of synaptic plasticity induced at excitatory synapses by me

208 rise as a consequence of neuroadaptations in synaptic plasticity induced by chronic nicotine.

209 Long-term synaptic plasticity is a basic ability of the brain to d

210 ABSTRACT: Short-term synaptic plasticity is a critical regulator of neural ci

211 ll activity.SIGNIFICANCE STATEMENT Long-term synaptic plasticity is a fundamental property of the bra

212 Finally, synaptic plasticity is a key regulator of the rate and p

213 PP and that the Abeta-mediated impairment of synaptic plasticity is accompanied by presynaptic effect

214 Experience-driven synaptic plasticity is believed to underlie adaptive beh

215 We found that PN-KC synaptic plasticity is crucial in controlling the genera

216 sult shows that the regulation of excitatory synaptic plasticity is fundamentally altered in PE anima

217 The key trigger for Hebbian synaptic plasticity is influx of Ca2+ into postsynaptic

218 face diffusion as a trafficking mechanism in synaptic plasticity is still lacking.

219 A pivotal feature of long-lasting synaptic plasticity is the localization of RNAs and the

220 n networks of neurons by including dendrites.Synaptic plasticity is the neuronal mechanism underlying

221 One mechanism for synaptic plasticity is the regulated addition and remova

222 Synaptic plasticity is thought to be the principal neuro

223 Although the alteration of synaptic plasticity is thought to contribute to this beh

224 Synaptic plasticity is typically induced by Ca(2+) eleva

225 modify their synaptic connections, known as synaptic plasticity, is accepted as the cellular basis f

226 t homeostatic scaling-down, a global form of synaptic plasticity, is active during sleep to remodel s

227 Although RIbeta contributes to synaptic plasticity, it is the least studied isoform.

228 rget of rapamycin (mTOR) regulates long-term synaptic plasticity, learning, and memory by controlling

229 ecp2-deficient neurons also lack homeostatic synaptic plasticity, likely due to reduced levels of EEA

230 sm in neurons and astrocytes, and ultimately synaptic plasticity loss evident by a decreased long-ter

231 r proteins is required for normal short-term synaptic plasticity, LTP, and spatial learning and memor

232 (GSK3beta) activity, and increased levels of synaptic plasticity markers including brain derived neur

233 l-specific alterations in CREB signaling and synaptic plasticity may underlie certain nicotine withdr

234 opments of our understanding of circuits and synaptic plasticity mechanisms of drug relapse from stud

235 oscience (SFN) session entitled "Circuit and Synaptic Plasticity Mechanisms of Drug Relapse," we will

236 dels, we study how dendrites enable multiple synaptic plasticity mechanisms to coexist in a single ce

237 Sepers et al. show a selective deficit in synaptic plasticity mediated by the endocannabinoid anan

238 influence of menin on synapse formation and synaptic plasticity occur via modulation of nAChR channe

239 Synaptic plasticity occurs in response to intrinsic and

240 Experience-dependent synaptic plasticity occurs on DA neurons while an organi

241 Here, we tested synaptic plasticity of cortical excitatory synapses onto

242 ing the effects of binge alcohol drinking on synaptic plasticity of NAc MSNs differs markedly whether

243 Our results indicate that excitability and synaptic plasticity of subicular neurons relies heavily

244 rder to selectively manipulate glutamatergic synaptic plasticity on DA neurons.

245 dendritic pathology and improved axonal and synaptic plasticity on ventral horn motor neurons.

246 ur data provide a novel, causal link between synaptic plasticity onto DA neurons and fear learning.

247 However, whether synaptic plasticity onto DA neurons is causally involved

248 ecent studies implicate that Notch regulates synaptic plasticity or cognitive performance, the molecu

249 described, but the effect of these drugs on synaptic plasticity or memory has not been investigated.

250 in both neuronal excitability and short-term synaptic plasticity-parameters that critically govern ne

251 Activity-dependent synaptic plasticity plays a critical role in the refinem

252 orders including cognitive decline, impaired synaptic plasticity, reduced sociability, hyperactivity

253 opriate spike patterns could drive long-term synaptic plasticity remained unknown.

254 ds is activity dependent, this modulation of synaptic plasticity represents a negative feedback mecha

255 Many forms of synaptic plasticity require the local production of vola

256 A plausible synaptic plasticity rule for learning that balances weig

257 ritic trees, with important implications for synaptic plasticity rules and spine function.

258 w negative images are formed at the level of synaptic plasticity rules, cells, and circuits.

259 pose a model of spacing-dependent structural synaptic plasticity.SIGNIFICANCE STATEMENT Long-term mem

260 se constitutive activities sustain long-term synaptic plasticity.SIGNIFICANCE STATEMENT Persistent sy

261 neuronal excitability and altered short-term synaptic plasticity.SIGNIFICANCE STATEMENT Schizophrenia

262 r translation of mRNAs that are critical for synaptic plasticity.SIGNIFICANCE STATEMENT The elongatio

263 acking densin (densin KO) exhibit defects in synaptic plasticity, spatial memory, and increased anxie

264 Spike timing-dependent synaptic plasticity (STDP) serves as a key cellular corr

265 ay a critical role in spike-timing dependent synaptic plasticity (STDP).

266 Short-term synaptic plasticity (STP) critically affects the process

267 lternative models of WM maintenance based on synaptic plasticity, such as short-term nonassociative (

268 e models of 22q11DS have abnormal short-term synaptic plasticity that contributes to working-memory d

269 endowed with several forms of intrinsic and synaptic plasticity that could contribute to adaptive ch

270 is a key regulator of glucose metabolism and synaptic plasticity that is canonically regulated throug

271 rtant control points in mGluR-LTD, a form of synaptic plasticity that is compromised in a severe mono

272 stem (CNS), cholinergic transmission induces synaptic plasticity that is required for learning and me

273 ugs of abuse cause persistent alterations in synaptic plasticity that may underlie addiction behavior

274 Long-lasting forms of synaptic plasticity that underlie learning and memory re

275 LTD at the excitatory synapses, an important synaptic plasticity that weakens synaptic strength.

276 contribution of calpain-1 in another type of synaptic plasticity, the long-term depression (LTD) elic

277 e of stimulation (massed) induced structural synaptic plasticity, the same amount of stimulation divi

278 by insulin resistance and compromised brain synaptic plasticity through the impairment of insulin-se

279 Structural changes span from synaptic plasticity to adult neurogenesis, the latter be

280 hese results suggest that activity-dependent synaptic plasticity underlies the formation of many CA1

281 ntral amygdala neurons are essential for the synaptic plasticity underlying learning in the lateral a

282 to enable regulatory processes required for synaptic plasticity underlying learning, memory and cogn

283 Just as dopamine plays a critical role in synaptic plasticity underlying normal skill learning and

284 show that achieving cocaine use reversed the synaptic plasticity underpinning the motivation to seek

285 on of NF-kappaB signaling decreased cortical synaptic plasticity via HDAC2.

286 Nicotine-induced synaptic plasticity was also associated with accumulatio

287 After 2 weeks of daily alcohol binges, synaptic plasticity was profoundly altered.

288 thermore, after cocaine self-administration, synaptic plasticity was selectively lost in D2, but not

289 ty of glutamate release, although short-term synaptic plasticity was similar between the groups.

290 To relate these results to synaptic plasticity, we simulated a network of oscillato

291 ronal phosphoprotein of 32 kDa feedback, and synaptic plasticity were greater in 8- vs. 14-week-old r

292 smission, and both long-term and homeostatic synaptic plasticity were unchanged, suggesting that loss

293 may result from the effect of the spacing in synaptic plasticity, which appears to be a property not

294 ed cleavage maintain two forms of persistent synaptic plasticity, which are the cellular analogs of t

295 otein that plays a key role in bidirectional synaptic plasticity, which is a process important for le

296 phenotypes and corresponding impairments of synaptic plasticity, while the consolidation of new memo

297 bly influence mechanisms underlying forms of synaptic plasticity with a presynaptic locus.

298 Episodic memories initially require rapid synaptic plasticity within the hippocampus for their for

299 gests that AMPA glutamate-receptor-dependent synaptic plasticity within the NAc underlies aspects of

300 These mice display profound deficits in synaptic plasticity without any effects on basal synapti