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1 ance, leading to a striking reshaping of the synaptic potential.
2 threshold for action potential generation by synaptic potential.
3 ol also displayed large-amplitude inhibitory synaptic potentials.
4 ynaptic currents to generate rapid and small synaptic potentials.
5 5-HT inhibited spontaneous synaptic potentials.
6 es may contribute to repolarization of large synaptic potentials.
7 over which they effectively summate incoming synaptic potentials.
8 cellular excitability and the integration of synaptic potentials.
9 onsiderable complexity to the integration of synaptic potentials.
10 tional barrages of excitatory and inhibitory synaptic potentials.
11 ptic inputs from other neurons by generating synaptic potentials.
12 t barrages of both excitatory and inhibitory synaptic potentials.
13 nction is to generate outputs in response to synaptic potentials.
14 bitory barrages and narrower visually evoked synaptic potentials.
15 ne potential and the temporal integration of synaptic potentials.
16 ficant increase in the field excitatory post-synaptic potentials.
17 ritic space constant for the auditory-evoked synaptic potentials.
18 membrane potential, evoked quantal release, synaptic potentials, acetylcholine receptor channel kine
19 -B synaptic potentials and suppressed single synaptic potentials after the cessation of the artificia
20 followed by persistent synaptic depression (synaptic potential amplitude reduced by 22% at 30 min).
21 ficant decrease of the field excitatory post-synaptic potentials amplitude in 60-day animals, but not
22 riable depolarization that summates with the synaptic potential and can act as a trigger for action p
23 roach capable of resolving sequentially both synaptic potentials and action potential firing in large
24 ording with the ability to detect both small synaptic potentials and action potentials, we developed
26 late currents) and the kinetic properties of synaptic potentials and currents were similar to control
27 at spines have an electrical role, filtering synaptic potentials and electrically isolating inputs fr
30 pond to stimuli by integrating generator and synaptic potentials and generating action potentials.
31 in synaptic potency reduced the duration of synaptic potentials and limited temporal summation of af
32 ndrites modulate the summation of excitatory synaptic potentials and prevent the generation of dendri
33 intrinsic jitter in spike triggering, evoked synaptic potentials and spikes had surprisingly similar
34 synaptic transmission, properties of evoked synaptic potentials and spontaneous synaptic currents we
35 attern: DSI strongly enhanced summated VSI-B synaptic potentials and suppressed single synaptic poten
36 ce of sodium action potential (AP)-dependent synaptic potentials and synaptic currents in >50% of the
37 bserved a monotonic decline in extracellular synaptic potentials and their initial slope over the tem
38 eurons minimizes spike-induced distortion of synaptic potentials and thus preserves the accuracy of b
40 lutamate receptors (mGluRs) can modify field-synaptic-potentials and protein kinase activity in rat a
41 d excitatory components of the bidirectional synaptic potentials are dependent upon AMPA (GluA) recep
43 he depolarizing drive, and the amplitudes of synaptic potentials are generally thought to reflect the
46 . find that spontaneously released miniature synaptic potentials arise from a pool of vesicles distin
49 ctive desynchronization reduces summation of synaptic potentials at target AOS cells and thus provide
52 the presence of 1% halothane, the excitatory synaptic potential between VD4 and the PeA(E) cells was
53 completely abolished, whereas the inhibitory synaptic potential between VD4 and the PeA(I) cells was
54 he first characterization of visually evoked synaptic potentials between the visual thalamus and visu
57 om CA3 neurons revealed bursts of excitatory synaptic potentials coincident with the network bursts r
58 larly with higher frequency signals (such as synaptic potentials compared with steady-state signals),
59 ynaptic transmission such as multi-component synaptic potentials, cotransmission, and neuromodulation
60 s in the auditory cortex of mice and measure synaptic potentials/currents, membrane potentials, and s
62 dulla that receive increased excitatory post-synaptic potentials during inspiration, but also have sp
63 were recorded as field potentials and focal synaptic potentials during systematic exploration of the
64 ntrast, mono- and disynaptic excitatory post-synaptic potentials elicited from the medial longitudina
66 c but not dendritic post-synaptic excitatory synaptic potential (EPSP) integration in CA1 pyramidal c
67 sible reduction of the field excitatory post-synaptic potential (EPSP) slope in response to low-frequ
69 and increased amplitudes of excitatory post-synaptic potentials (EPSPs) in adult rat hippocampal sli
71 d at layer 6 (L6), whereas evoked excitatory synaptic potentials (EPSPs) were recorded from L5 neuron
72 in long-term depression (LTD) of excitatory synaptic potentials (EPSPs), long-term decrease of popul
73 tic response, slope of field excitatory post-synaptic potentials (EPSPs), or the input/output functio
76 (15 microM) facilitated the excitatory field synaptic potential evoked in the BLA by stimulation of t
77 (10 microM) facilitated the excitatory field synaptic potential evoked in the BLA by stimulation of t
78 DSI spike trains heterosynaptically enhanced synaptic potentials evoked by another CPG neuron, ventra
79 about half the cells demonstrated excitatory synaptic potentials evoked by extracellular stimulation.
80 heart muscle contractility, or amplitudes of synaptic potentials evoked by stimulating postganglionic
81 PSPs occurred coincident with fast nicotinic synaptic potentials evoked by the myenteric projections
83 sensory maps to synaptogenesis, we recorded synaptic potentials evoked by whisker deflection in laye
84 axonal pattern of arborization, spontaneous synaptic potentials, evoked postsynaptic currents, patte
85 We tested this by recording CST-evoked focal synaptic potentials, extracellularly, in the cervical en
86 a lasting increase in field excitatory post-synaptic potential (fEPSP) slope in area CA1 following t
87 only small effects on field excitatory post-synaptic potentials (fEPSP) in slices from 60-day animal
88 , by monitoring evoked field excitatory post-synaptic potentials (fEPSP), and slice nicotinamide aden
89 Recordings of hippocampal field excitatory synaptic potentials (fEPSPs) showed that prenatal exposu
92 viding 122 regions of maximum negative focal synaptic potentials (FSPs) of mean amplitude 7.51 microV
93 ement of terminal potentials (TPs) and focal synaptic potentials (FSPs), recorded with spike-triggere
95 action potential duration, dampen excitatory synaptic potentials, impede temporal summation, and rais
96 indicating that they represent synchronized synaptic potentials impinging on layer II-III neurons.
99 activated during subthreshold glutamatergic synaptic potentials in a principal cell generate an exci
100 eir activity reduces the amplitude of evoked synaptic potentials in an NMDA receptor (NMDAR)-dependen
101 sed Ih in corticocollicular neurons, whereas synaptic potentials in L2/3-->corticocallosal connection
103 re the predominant entry site for excitatory synaptic potentials in most types of central neurons.
104 ns in the sensory layers revealed excitatory synaptic potentials in response to glutamate application
105 hese models we have recorded visually evoked synaptic potentials in simple cells while cooling the co
107 ewed as occurring through the interaction of synaptic potentials in the dendrite and soma, followed b
108 The recordings of field excitatory post-synaptic potentials in the hippocampal CA1 subfield demo
110 agonists depressed the field excitatory post-synaptic potentials in the SC-CA1 pathway, but had no si
111 rrents in dorsal horn neurons and population synaptic potentials in ventral roots provided evidence t
113 the facilitating effects of field excitatory synaptic potential induced by the agonist were reduced t
114 the facilitating effects of field excitatory synaptic potential induced by the agonist were reduced t
116 pyramidal neurons modulate spike firing and synaptic potential integration by influencing the membra
117 ) can be mediated by GABA(B) inhibitory post-synaptic potentials (IPSPs) or slow after-hyperpolarizat
123 that intrinsic properties can override fast synaptic potentials, mediate circuit reconfiguration, an
126 supraoptic nucleus (SON) were used to study synaptic potentials mediated by nicotinic acetylcholine
129 to the presynaptically mediated decreases in synaptic potentials previously demonstrated in vitro wit
130 with a foot-shock resulted in enhanced post-synaptic potential (PSP) responses to the odour and incr
133 induced a transient depression of the evoked synaptic potential recorded in the rat hippocampal CA1 r
135 ron somata, the amplitude of afferent-evoked synaptic potentials recorded in motor neurons was reduce
136 ent 1.7-fold enhancement in the amplitude of synaptic potentials recorded intracellularly in basolate
137 urons well beyond the duration of excitatory synaptic potentials resulting from activation of the tri
142 t, the effect of somatic K(V)7/M-channels on synaptic potential summation was dependent upon the neur
143 rd high-frequency bursting activity and slow synaptic potentials that are hard to resolve by multicel
144 These oscillations reflect synchronized synaptic potentials that entrain the discharge of neuron
145 ansmit visual information by means of graded synaptic potentials that spread to the synaptic terminal
146 terconnections can generate giant excitatory synaptic potentials that support the bursting behaviour
147 als generated, but also the amplitude of the synaptic potentials that these action potentials initiat
148 developing and sustained depression of field synaptic potentials that was quantitatively similar in b
149 lso drastically decreases the isolated NMDAR synaptic potentials, these responses are still enhanced
153 es the resistance of Schaffer collateral-CA1 synaptic potentials to further, longer periods of hypoxi
154 1-43 imaging and intracellular recordings of synaptic potentials to measure the time course of endocy
155 ocytosis), intracellular recording of evoked synaptic potentials (to measure acetylcholine release),
156 the detection of either action potentials or synaptic potentials, to follow high versus low rates of
157 ious components of excitatory and inhibitory synaptic potentials, using intracellular current- and vo
158 of EtOH, the amplitude of dorsal root-evoked synaptic potentials was reduced, the frequency of sponta
159 ed, temporal summation at the soma of distal synaptic potentials was similar to that seen with curren
161 depolarization persisted in TTX or when fast synaptic potentials were blocked, indicating that it rep
162 sociated with spatially uniform subthreshold synaptic potentials were comparable but not equal in amp
164 an early (2 min) consequence of OGD, evoked synaptic potentials were lost, and K, Cl, P, and Mg conc
167 he lateral geniculate nucleus (LGN) generate synaptic potentials, which sometimes produce spikes sent
168 with tetrodotoxin and by blocking inhibitory synaptic potentials with bicuculline and strychnine.
169 scillations consisted of compound inhibitory synaptic potentials with initial IPSPs with slow kinetic
171 to the spinal cord elicited Ca(2+)-dependent synaptic potentials with short latencies in reticulospin
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