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1 marily components of the metabolic syndrome (syndrome X).
2 lectively known as the metabolic syndrome or syndrome X.
3 w diet produce many of the features of human syndrome X.
4 tion is scant on the childhood predictors of syndrome X.
5 scular disease associated with the metabolic syndrome X.
6 ce fat distribution and variables related to syndrome X.
7 ssure were used as measures of components of Syndrome X.
8 ripheral resistance vessels of patients with syndrome X.
9 pulation reduces the public health impact of syndrome X.
10  full cluster of abnormalities that comprise syndrome X.
11  the metabolic abnormalities that constitute syndrome X.
12  diets should be avoided in the treatment of syndrome X.
13 pertension both independently and as part of syndrome X.
14                           More patients with syndrome X (17/18 [94%]) developed chest pain during ade
15 re not significantly higher in patients with syndrome X (4.8 vs. 4.0 pg/ml, p = 0.17), the vasoconstr
16 cclusion plethysmography in 10 patients with syndrome X and 10 matched control subjects.
17  (2.63 versus 2.53; P=0.60) in patients with syndrome X and controls, respectively.
18                                              Syndrome X and microvascular angina are a heterogenous g
19 ty is a powerful predictor of development of syndrome X and underscore the importance of weight contr
20                       Eighteen patients with syndrome X (chest pain, abnormal exercise treadmill test
21 lood flow of only 20 +/- 2% in patients with syndrome X compared with 35 +/- 3% in matched control su
22 tly increased in women (but not in men) with syndrome X, compared with women with no metabolic abnorm
23 ustering of cardiovascular risk variables of Syndrome X from childhood to adulthood were examined in
24                                Patients with syndrome X had normal basal and stimulated nitric oxide
25  of dyslipidemia and diabetes (components of syndrome X) have become global health epidemics.
26 ent features of insulin resistance syndrome (Syndrome X) have been identified by factor analysis in m
27 luster of metabolic abnormalities defined as syndrome X (high blood glucose, high blood pressure, low
28             Patients with X-linked hyper-IgM syndrome (X-HIGM) due to CD40 ligand (CD40L) mutations a
29             Subjects with X-linked hyper-IgM syndrome (X-HIgM) have a markedly reduced frequency of C
30 sults in X-linked hyper-immunoglobulin (Ig)M syndrome (X-HIGM), characterized by recurrent infections
31  diabetes and the often-associated metabolic syndrome X (hypertriglyceridemia, low serum high density
32  insulin to the adulthood risk of developing syndrome X in a biracial (black-white) community-based l
33 applied to the clustering characteristics of Syndrome X in a biracial (Black-White) community-based p
34  of the defining features of human metabolic Syndrome X, in which hypertension associates with insuli
35 full cluster of metabolic abnormalities from syndrome X is an important risk factor for cardiovascula
36                   These results suggest that Syndrome X is characterized by the linking of a metaboli
37 e occurrence of insulin resistance syndrome (syndrome X) is common in the general population.
38 rotein, alpha thalassemia/mental retardation syndrome X linked, switch/sucrose nonfermentable pathway
39 aining alpha-thalassaemia/mental retardation syndrome X-linked (ATRX) and death-domain-associated pro
40 is the alpha-thalassaemia mental retardation syndrome X-linked (ATRX) homolog X-linked nuclear protei
41         Alpha thalassemia/mental retardation syndrome X-linked (ATRX) is a member of the SWI/SNF prot
42 roteins alpha thalassemia/mental retardation syndrome X-linked (ATRX) or death-domain associated prot
43 ponent, alpha-thalassemia/mental retardation syndrome X-linked (ATRX) protein, is predicted to be a t
44 d ATRX (alpha thalassemia/mental retardation syndrome X-linked).
45 les in the pathogenesis of Smith-Lemli-Opitz syndrome, X-linked dominant chondrodysplasia punctata, a
46 increased cancer susceptibility in the human syndrome, X-linked dyskeratosis congenita (X-DC).
47 cardi-Goutieres syndrome, Hallervorden-Spatz syndrome, X-linked dystonia parkinsonism, deafness-dysto
48                         In craniofrontonasal syndrome, X-linked mosaicism for ephrin-B1 expression ha
49 ed that mutations in ARX cause X-linked West syndrome, X-linked myoclonic epilepsy with spasticity an
50 istinct clinical phenotypes (Wiskott-Aldrich syndrome/X-linked thrombocytopenia; intermittent thrombo
51                   These results suggest that syndrome X may be comprised of separable PPARgamma-depen
52                                Patients with syndrome X may have a generalized disorder of vascular a
53               In 1987 when Reaven introduced syndrome X (metabolic syndrome, or MS), we were studying
54 line in physical functioning than either the syndrome X or primary mediator components alone.
55                                              Syndrome X refers to a cluster of abnormalities, associa
56 ology of chest pain in patients with cardiac syndrome X remains controversial.
57 n of the cluster of metabolic abnormalities (syndrome X) should not distract our attention from estab
58 with CMR could clarify whether patients with syndrome X show evidence of myocardial ischemia (reduced
59                                Patients with syndrome X show greater sensitivity to chest pain compar
60                                              Syndrome X, typified by obesity, insulin resistance (IR)
61 he degree of clustering of risk variables of Syndrome X varies with age from childhood to adulthood a
62 the full cluster of metabolic abnormalities (syndrome X) was low in the population as a whole, with o

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