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1 marily components of the metabolic syndrome (syndrome X).
2 lectively known as the metabolic syndrome or syndrome X.
3 w diet produce many of the features of human syndrome X.
4 tion is scant on the childhood predictors of syndrome X.
5 scular disease associated with the metabolic syndrome X.
6 ce fat distribution and variables related to syndrome X.
7 ssure were used as measures of components of Syndrome X.
8 ripheral resistance vessels of patients with syndrome X.
9 pulation reduces the public health impact of syndrome X.
10 full cluster of abnormalities that comprise syndrome X.
11 the metabolic abnormalities that constitute syndrome X.
12 diets should be avoided in the treatment of syndrome X.
13 pertension both independently and as part of syndrome X.
15 re not significantly higher in patients with syndrome X (4.8 vs. 4.0 pg/ml, p = 0.17), the vasoconstr
19 ty is a powerful predictor of development of syndrome X and underscore the importance of weight contr
21 lood flow of only 20 +/- 2% in patients with syndrome X compared with 35 +/- 3% in matched control su
22 tly increased in women (but not in men) with syndrome X, compared with women with no metabolic abnorm
23 ustering of cardiovascular risk variables of Syndrome X from childhood to adulthood were examined in
26 ent features of insulin resistance syndrome (Syndrome X) have been identified by factor analysis in m
27 luster of metabolic abnormalities defined as syndrome X (high blood glucose, high blood pressure, low
30 sults in X-linked hyper-immunoglobulin (Ig)M syndrome (X-HIGM), characterized by recurrent infections
31 diabetes and the often-associated metabolic syndrome X (hypertriglyceridemia, low serum high density
32 insulin to the adulthood risk of developing syndrome X in a biracial (black-white) community-based l
33 applied to the clustering characteristics of Syndrome X in a biracial (Black-White) community-based p
34 of the defining features of human metabolic Syndrome X, in which hypertension associates with insuli
35 full cluster of metabolic abnormalities from syndrome X is an important risk factor for cardiovascula
38 rotein, alpha thalassemia/mental retardation syndrome X linked, switch/sucrose nonfermentable pathway
39 aining alpha-thalassaemia/mental retardation syndrome X-linked (ATRX) and death-domain-associated pro
40 is the alpha-thalassaemia mental retardation syndrome X-linked (ATRX) homolog X-linked nuclear protei
42 roteins alpha thalassemia/mental retardation syndrome X-linked (ATRX) or death-domain associated prot
43 ponent, alpha-thalassemia/mental retardation syndrome X-linked (ATRX) protein, is predicted to be a t
45 les in the pathogenesis of Smith-Lemli-Opitz syndrome, X-linked dominant chondrodysplasia punctata, a
47 cardi-Goutieres syndrome, Hallervorden-Spatz syndrome, X-linked dystonia parkinsonism, deafness-dysto
49 ed that mutations in ARX cause X-linked West syndrome, X-linked myoclonic epilepsy with spasticity an
50 istinct clinical phenotypes (Wiskott-Aldrich syndrome/X-linked thrombocytopenia; intermittent thrombo
57 n of the cluster of metabolic abnormalities (syndrome X) should not distract our attention from estab
58 with CMR could clarify whether patients with syndrome X show evidence of myocardial ischemia (reduced
61 he degree of clustering of risk variables of Syndrome X varies with age from childhood to adulthood a
62 the full cluster of metabolic abnormalities (syndrome X) was low in the population as a whole, with o
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