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1 a suggest similarity to rheumatoid arthritis synovia.
2 tis are being applied to psoriatic arthritis synovia.
3 by histomorphometry in healthy and arthritic synovia.
4 al cells extracted from RA and nonrheumatoid synovia.
5 Both GNS and FLNA were highly expressed in synovia.
6 l cells was detected in RA and nonrheumatoid synovia.
7 ced this binding in RA but not nonrheumatoid synovia.
8 lls was demonstrated in RA and nonrheumatoid synovia.
9 ation and matrix degradation in the inflamed synovia.
10 was performed to identify RAG+ cells within synovia.
11 a, and it was absent from all of the healthy synovia.
12 actant; a rat homolog for human IL-8) in the synovia.
13 ed and the PMN activation state in the joint synovia.
14 hain rearrangements in about one-third of RA synovia.
15 gests the presence of immature B cells in RA synovia.
18 D200R was found to be expressed in arthritic synovia and in lymph nodes, yet no changes in T cell cyt
21 arkedly lower expression of HIF-1alpha in OA synovia, and it was absent from all of the healthy synov
23 ver, the overexpression of cathepsin K in RA synovia due to an increase in the number of cathepsin K-
27 sectional evaluations of psoriatic arthritis synovia in the context of other arthritides have been pe
28 r, in human macrophages and fibroblasts from synovia of individuals with rheumatoid arthritis, tenasc
30 eted chemoattractant that is abundant in the synovia of patients with juvenile idiopathic arthritis (
32 with acute graft-versus-host disease and the synovia of patients with rheumatoid arthritis during act
36 omics to detect HLA-DR-presented peptides in synovia or peripheral blood mononuclear cells and identi
37 nerated in other tissues and then migrate to synovia, perhaps due to cross-reactive joint-specific Ag
38 positive for IL-8 mRNA in multiple tissues (synovia, pericardium, and peritoneum), and 10 dogs expre
39 abundantly by macrophages in most rheumatoid synovia, predominantly close to the intimal layer but al
41 sublining and vascularized areas of inflamed synovia showed a highly significant negative correlation
43 macrophage-like cells in normal, OA, and RA synovia suggests a constitutive expression of this prote
45 cept for lesser expression of VCAM-1 in Lyme synovia, the levels of expression of these adhesion mole
46 to endothelial HSPGs in RA and nonrheumatoid synovia was determined by heparinase treatment followed
48 ascular growth is enhanced in osteoarthritic synovia when infiltrating macrophages generate angiogeni
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