ライフサイエンスコーパス: synovial fluid

1 aracterization of exosomes miRNAs from human synovial fluid.

2 HMVEC migration and/or tube formation by RA synovial fluid.

3 ation of activated human T cells toward this synovial fluid.

4 e turnover were examined in serum, urine, or synovial fluid.

5 duces the induction of HMVEC migration by RA synovial fluid.

6 uid compared with RA serum or osteoarthritis synovial fluid.

7 8 was higher in RA synovial fluid than in OA synovial fluid.

8 tes at the concentrations detected in the RA synovial fluid.

9 ly reduced monocyte migration mediated by RA synovial fluid.

10 7 is expressed in the RA synovial tissue and synovial fluid.

11 gly correlated with the level of gp96 in the synovial fluid.

12 suggesting that cleaved OPN is released into synovial fluid.

13 at the alpha-enolase was citrullinated in RA synovial fluid.

14 are common components of osteoarthritis (OA) synovial fluid.

15 ative method for identifying BCP crystals in synovial fluid.

16 se-beta) are present in rheumatoid arthritis synovial fluid.

17 the quantities of synthetic BCP crystals in synovial fluid.

18 able methods for identifying BCP crystals in synovial fluid.

19 oncentrations of the amino acid glutamate in synovial fluid.

20 n unity (alpha approximately 0.6) for normal synovial fluid.

21 ular interactions between lubricin and HA in synovial fluid.

22 tion may require lubricin supplementation in synovial fluid.

23 found in biological fluids such as urine and synovial fluid.

24 tic tool had low sensitivity when applied to synovial fluid.

25 gradation and release of components into the synovial fluid.

26 imilar concentrations of IL-7 detected in RA synovial fluid.

27 tics and were pre-coated with human blood or synovial fluid.

28 sphatidylcholine type phospholipids in human synovial fluid.

29 lymph nodes, and later after infusion in the synovial fluid.

30 lated with the total leukocyte counts in the synovial fluids.

31 Samples of serum and knee joint synovial fluid (275 knees) were obtained from 159 patien

32 including the blood, peritoneal fluid, bone, synovial fluid, a perianal abscess, and an arm wound.

33 CXCL10/CXCR3 axis, with CXCL10 increasing in synovial fluids after injury and Cxcr3(-/-) mice being p

34 ound that HA contributes to the formation of synovial fluid aggregates, and HysA can disrupt aggregat

35 tractants CCL15 and CCL23 by incubation with synovial fluid, although the responsible proteases could

36 Current methods of diagnosing SA rely on synovial fluid analysis and culture which are known to b

37 endation: ACP recommends that clinicians use synovial fluid analysis when clinical judgment indicates

38 nd lubricin is a HA-binding protein found in synovial fluid and at cartilage surfaces, where it contr

39 ogy and polymerase chain reaction testing of synovial fluid and biopsy material.

40 vated levels of PGE(2) have been reported in synovial fluid and cartilage from patients with osteoart

41 tion of cleaved chemerins was much higher in synovial fluid and cerebrospinal fluid samples than in p

42 vides high signal intensity in cartilage and synovial fluid and is a promising technique for imaging

43 Quantification of calreticulin in plasma and synovial fluid and of calreticulin-FasL binding was perf

44 hic arthritis to both mononuclear cells from synovial fluid and PBMC fails to reduce the production o

45 ts receptor was significantly elevated in RA synovial fluid and peripheral blood macrophages as well

46 pression was studied in neutrophils from the synovial fluid and peripheral blood of patients with rhe

47 The correlation between synovial fluid and serum COMP levels was significant (r

48 Synovial fluid and serum samples were collected to measu

49 The presence of rVSV in synovial fluid and skin lesions confirmed causality.

50 Its performance is similar to that of synovial fluid and superior to those of saline and Synvi

51 splant 21 months prior to presentation whose synovial fluid and surgical specimens grew Phomopsis bou

52 is an important modulator of TNF-alpha in RA synovial fluid and that TLR5 expression on these cells s

53 odulating transcription of TNF-alpha from RA synovial fluid and the strong correlation of TLR5 and TN

54 Expression of visfatin/PBEF in synovial fluid and tissue of RA patients was detected by

55 ronic acid (HA) is abundant in cartilage and synovial fluid and widely thought to play a principal ro

56 tals are common components of osteoarthritic synovial fluids and define subsets of patients with infl

57 ugh proteomic and transcriptomic analyses of synovial fluids and membranes from individuals with oste

58 PCR and culture of periprosthetic tissue and synovial fluid (and serology).

59 ls of inflammatory biomarkers, both locally (synovial fluid) and systemically (serum and plasma).

60 cimens, including cerebrospinal fluid (CSF), synovial fluid, and abscess tissue, from 469 case patien

61 ritis was performed, examining ankle tissue, synovial fluid, and peripheral blood leukocytes.

62 man body fluids such as cerebrospinal fluid, synovial fluid, and peritoneal fluid.

63 ation products have been measured in plasma, synovial fluid, and synovial tissues of patients.

64 uingly, the repertoires of FOXP3(+) Tregs in synovial fluid are highly overlapping with CD25(+)FOXP3(

65 3 in chondrocytes and their release into the synovial fluid are significantly increased.

66 ity of proteins showed overexpression in JIA synovial fluid as compared with noninflammatory control

67 The virus was identified in one synovial-fluid aspirate and in skin vesicles of 2 other

68 ensated polarized light microscope (CPLM) in synovial fluid aspirated from the patient's joint.

69 eived antibiotics within the month preceding synovial fluid aspiration (48 of whom had PJI), PCR pane

70 hat the subdiffusive and elastic behavior of synovial fluid, at physiological shear rates, was absent

71 ein we show that CCL15 is processed in human synovial fluid by matrix metalloproteinases (MMPs) and s

72 d BMP-2 productions were measured in patient synovial fluids by enzyme-linked immunosorbent assay.

73 en the 2 patient groups were observed in the synovial fluid CD4+CD25(high) population, a cell subset

74 The cocultures of platelets with synovial fluid cells from rheumatoid arthritis patients

75 Synovial fluid cells were analyzed by polymerase chain r

76 Endogenous concentration of bFGF in synovial fluids collected from arthritis patients and as

77 Synovial fluid COMP levels correlated most strongly with

78 Synovial fluid COMP levels correlated strongly with 2 in

79 ion of sJAM-C is also elevated locally in RA synovial fluid compared with RA serum or osteoarthritis

80 The synovial fluid concentrations of FSTL-1 were 2-3-fold hi

81 Joint lubrication, synovial fluid conservation and many pathophysiological

82 The synovial fluid contains activated inflammatory macrophag

83 TLR-5 to endogenous ligands expressed in RA synovial fluid contributed to endothelial cell infiltrat

84 Calreticulin concentrations in the synovial fluid correlated with the tender and swollen jo

85 radiographic tests, such as serum urate and synovial fluid crystal analysis and radiographic or ultr

86 assay were 95% and 97%, respectively, versus synovial fluid culture results.

87 piration (48 of whom had PJI), PCR panel and synovial fluid culture sensitivities were 64.5% and 85.4

88 d tissue cultures and pre- and postoperative synovial fluid cultures were all negative.

89 Multicolor characterizations in synovial fluid demonstrated CXCL13 expression in antigen

90 Molecular profiling of synovial fluid derived exosomal miRNAs may increase our

91 of physiologic concentrations of plasma- or synovial fluid-derived antiproteinases.

92 an ultrafilter that reflects HA during trans-synovial fluid drainage.

93 of both pathways can more strongly impair RA synovial fluid-driven monocyte migration and osteoclast

94 may decrease transport between cartilage and synovial fluid during compression.

95 ed septic joints should be aspirated and the synovial fluid examined by microscopy for the presence o

96 In conclusion, synovial fluid exosomal miRNA content is altered in pati

97 The synovial fluid exosomes share similar characteristics (s

98 ling, and reports newly identified serum and synovial fluid FAs as predictive biomarkers of OA in obe

99 These results highlight the importance of synovial fluid for the expression of citrullinated autoa

100 s with or without IL-1beta or osteoarthritic synovial fluids for 48 h.

101 pneumoniae was associated with the increased synovial fluids found in arthritic flares but was not fo

102 scent sera from 91 EM patients, in serum and synovial fluid from 141 LA patients, and in serum from 5

103 describe the evaluation of culture-negative synovial fluid from a 3-year-old boy by PCR and electros

104 ues of unity for simple HA solutions and for synovial fluid from an individual who genetically lacked

105 IL-17 is involved in neutrophil recruitment, synovial fluid from arthritic joints showed a comparable

106 (HCs) with hyaluronan was first described in synovial fluid from arthritic patients and later describ

107 In addition, sera and synovial fluid from children with oligoarticular, polyar

108 nd this process was prevented by addition of synovial fluid from JIA patients, through an IL-6-indepe

109 ontaining proteins and hemolytic activity of synovial fluid from patients suffering from rheumatoid a

110 g revision surgery were higher than those in synovial fluid from patients undergoing primary surgery.

111 Synovial fluid from patients undergoing revision surgery

112 rmally produced by the chemokines present in synovial fluid from patients with active rheumatoid arth

113 ated T cells toward the chemokine mixture in synovial fluid from patients with active rheumatoid arth

114 rullinated polypeptides were detected in the synovial fluid from patients with RA and patients with S

115 ge were detected by Western blot analysis in synovial fluid from patients with rheumatoid arthritis a

116 RNase activity was increased in synovial fluid from RA and OA patients compared with pso

117 ated SOX5 levels were higher in synovium and synovial fluid from RA compared to osteoarthritis patien

118 crp30 and its globular fragment (gAcrp30) in synovial fluid from rheumatoid arthritis patients.

119 , complement activation was also detected in synovial fluid from RRV-infected patients.

120 gp96 was increased in synovial fluid from the joints of RA compared with disea

121 and naturally occurring chemoattractants in synovial fluid from the rheumatoid synovium, including C

122 DEK autoantibodies and protein are found in synovial fluids from JIA patients.

123 reas it was elevated approximately 2-fold in synovial fluids from patients with arthritis.

124 were isolated by affinity chromatography of synovial fluids from patients with rheumatoid arthritis,

125 L-34, TGF-beta1, and BMP-2 were expressed in synovial fluids from RA patients.

126 bowel disease (IBD), and reduced TNFalpha in synovial fluids from RA patients.

127 Succinate is abundant in synovial fluids from rheumatoid arthritis (RA) patients,

128 Moreover, calcium crystal positive synovial fluids from some OA patients exhibited inflamma

129 Lubricin is a mucinous, synovial fluid glycoprotein that enables near frictionle

130 After washing out the endogenous synovial fluid HA (miscibility coefficient 0.4), secreti

131 fluid uric acid was strongly correlated with synovial fluid IL-18 and IL-1beta.

132 Furthermore, synovial fluid IL-18 was associated with a 3-y change in

133 tivated inflammasomes and the association of synovial fluid IL-18 with OA progression, lend strong su

134 y both radiograph and bone scintigraphy, and synovial fluid IL-1beta was associated with OA severity

135 Finally, RA synovial fluid immunoneutralized with anti-IL-17 and ant

136 Interestingly, we show that RA synovial fluids immunoneutralized for IL-17 and CCL2/MCP

137 llular matrix of all body tissues, including synovial fluid in joints, in which it behaves as a filte

138 It is assumed that the interaction with synovial fluid in the biocompartment leads to significan

139 As we identified DEK in synovial fluids in JIA patients, we now investigate how

140 RA synovial fluid induced the activation of macrophages and

141 Neutralizing anti-JAM-C Abs inhibited RA synovial fluid-induced HMVEC chemotaxis and sJAM-C-induc

142 RA synovial fluid-induced macrophage and HEK-TLR-2 activati

143 Synovial fluid is a semidilute hyaluronate (HA) polymer

144 rical items, physical examination, serum, or synovial fluid laboratory data for diagnosing septic art

145 tion of peripheral blood neutrophils with RA synovial fluid led to TNFalpha-dependent shedding of BLy

146 tibiotic-refractory Lyme arthritis have high synovial fluid levels of proinflammatory chemokines and

147 Synovial fluid likely reflects the proteins present in t

148 o determine the associations among serum and synovial fluid lipid levels with OA, synovitis, adipokin

149 eins are a prerequisite for agglomeration in synovial fluid, low activity of the Agr regulatory syste

150 Neutralization of RA synovial fluid macrophage cell surface gp96 inhibited th

151 ermore, expression of TLR5 is elevated in RA synovial fluid macrophages and RA peripheral blood monoc

152 ealthy donors and treated with TNF and using synovial fluid macrophages derived from patients with RA

153 ecursors obtained from mouse bone marrow and synovial fluid macrophages derived from RA patients were

154 RA synovial tissue and synovial fluid macrophages expressed CCR7, which was inc

155 Synovial fluid macrophages from RA patients were refract

156 The expression of TLR2, but not TLR4, on synovial fluid macrophages strongly correlated with the

157 RA synovial fluid macrophages were isolated by CD14 negativ

158 l, interquartile range [IQR] 14.8 ng/ml) and synovial fluid (median 10.3 ng/ml, IQR 12.0 ng/ml) of RA

159 plasma (median 3.1 ng/ml, IQR 1.3 ng/ml) and synovial fluid (median 2.9 ng/ml, IQR 0.9 ng/ml) of pati

160 175)-specific T cells in samples of PBMC and synovial fluid mononuclear cells (SFMC) from 13 DRB1*040

161 oviocytes (FLSs) and TNF-alpha production in synovial fluid mononuclear cells (SFMCs) derived from hu

162 17-producing T cells in peripheral blood and synovial fluid mononuclear cells from 36 children with J

163 healthy controls, whereas BLyS expression on synovial fluid neutrophils was very low.

164 CCL4, CCL5, CXCL9, and CXCL10; all levels in synovial fluid obtained from patients undergoing revisio

165 Macrophages from the synovial fluid of 5 patients with RA and 3 with seronega

166 In synovial fluid of arthritic joints, MMR was expressed on

167 hai protein, and neutrophil migration toward synovial fluid of arthritis patients was inhibited by tr

168 of the medium such as that occurring in the synovial fluid of DRA patients.

169 lar traps (NETs) are found abundantly in the synovial fluid of gout patients.

170 process was also effective for HC-HA in the synovial fluid of human rheumatoid arthritis patients (i

171 essed on macrophages in vitro and in vivo in synovial fluid of inflamed paws, whereas expression is r

172 56(bright)CD16(-) NK cells isolated from the synovial fluid of juvenile idiopathic arthritis patients

173 Although leukotrienes are present in the synovial fluid of Lyme disease patients, their role in t

174 how that the HC-HA complex is present in the synovial fluid of mice subjected to systemic and monoart

175 SPADE was used to analyze EVs present in the synovial fluid of patients with inflammatory arthritis.

176 PBMC and mononuclear cells obtained from the synovial fluid of patients with juvenile idiopathic arth

177 nd which has been previously observed in the synovial fluid of patients with Lyme arthritis.

178 e-linked immunosorbent assays, we tested the synovial fluid of patients with RA, osteoarthritis (OA),

179 andins (PGs) are found in high levels in the synovial fluid of patients with rheumatoid arthritis, an

180 1alpha was found in peri-implant tissues and synovial fluid of people with failing Metal-on-Metal hip

181 d whether IgA immune complexes in plasma and synovial fluid of RA patients activate neutrophils.

182 Herein, we studied macrophages from the synovial fluid of RA patients and observed a significant

183 and citrullinated proteins are found in the synovial fluid of RA patients.

184 t at higher concentrations in the plasma and synovial fluid of RA patients.

185 The RANKL/OPG ratio was disrupted in the synovial fluid of RRV patients, and this was accompanied

186 Expression of cytokines was elevated in the synovial fluid of some patients.

187 We detected ITCs in the synovial fluid of the high glucosinolate group, but not

188 d the greater levels of IL-6 detected in the synovial fluid of the obese OA patients.

189 The concentration of adrenomedullin in the synovial fluid of untreated Lyme arthritis patients was

190 and synovial macrophages were purified from synovial fluids of JIA patients.

191 cells, leukemic cells, and granulocytes from synovial fluids of patients with rheumatoid arthritis.

192 in arthritic flares but was not found in the synovial fluids of trauma patients.

193 d the effects of IL-1beta and osteoarthritic synovial fluids on anabolic gene expression and increase

194 e and (iv) exposure to body fluids (blood or synovial fluid) on release kinetics and efficacy of anti

195 In human serum and synovial fluid, only those samples from children with th

196 Neutralization of either CCL21 in RA synovial fluid or CCR7 in HMVECs significantly reduced t

197 Finally, neutralization of IL-17 in RA synovial fluid or its receptors on monocytes significant

198 Neutralization of either IL-17 in RA synovial fluids or IL-17 receptor C on HMVECs significan

199 Pa was lower in RA synovial fluid than in OA synovial fluid (P < 0.05; n = 8), and free IL-18 was hig

200 gnificantly higher concentrations of ECGF in synovial fluid (P<0.0001) and more often had ECGF antibo

201 Periprosthetic tissue and/or synovial fluid PCR has been previously studied for prost

202 Sensitivities of the synovial fluid PCR panel and culture were 55.6% and 76.1

203 e purpose of this study was to delineate the synovial fluid proteome and determine whether protein ex

204 The synovial fluid proteome of the samples was delineated.

205 saminoglycan measurements from cartilage and synovial fluid regions.

206 duced suppression is due to resistance of RA synovial fluid responder T cells to Treg inhibition.

207 s from the responder T cell population in RA synovial fluid restored Treg-mediated suppression.

208 The role of lubricin in synovial fluid rheology is not known.

209 Proteins from an RA synovial fluid sample were separated by 2-dimensional el

210 On the same synovial fluid sample, a polymorphonuclear cell count of

211 detected Streptobacillus moniliformis in the synovial fluid sample.

212 ic PCR assay panel using 284 prosthetic knee synovial fluid samples collected from patients presentin

213 Matched peripheral blood and synovial fluid samples from 15 patients with antibiotic-

214 The method was applied to human synovial fluid samples from osteo- and rheumatoid arthri

215 One hundred twenty-one clinical synovial fluid samples from patients presenting with sus

216 Levels of IL-18 and IL-18BPa in synovial fluid samples from patients with osteoarthritis

217 ated levels of OPN-R and OPN-L were found in synovial fluid samples from RA patients, but not in samp

218 Synovial fluid samples obtained from children with oligo

219 um-specific PCR testing was performed on all synovial fluid samples to confirm the U. parvum detectio

220 Blood and/or waste synovial fluid samples were collected from children with

221 A small set of characterized synovial fluid samples were compared by staining with al

222 r) embedded in normal and lubricin-deficient synovial fluid samples were tracked separately by using

223 Vitamin A metabolite levels were elevated in synovial fluid, serum, and cartilage from patients with

224 tein levels of IL-1alpha and IL-1beta in the synovial fluid, serum, and joint tissues.

225 )) is an abundant mucin-like glycoprotein in synovial fluid (SF) and a major component responsible fo

226 The concentration of histamine in synovial fluid (SF) and sera in patients with RA was mea

227 sed by enzyme-linked immunosorbent assays in synovial fluid (SF) and serum samples obtained from pati

228 Since synovial fluid (SF) bathes joint cartilage and synovium,

229 To determine whether the synovial fluid (SF) constituents hyaluronan (HA), proteo

230 Arthritic synovial fluid (SF) contains mesenchymal stem cells (MSC

231 0 patients with erythema migrans (EM) and in synovial fluid (SF) from 63 patients with Lyme arthritis

232 lasma, serum, cerebrospinal fluid (CSF), and synovial fluid (SF) from both preclinical species and hu

233 lly bound to C4d were identified from pooled synovial fluid (SF) from four rheumatoid arthritis (RA)

234 and usually presents as multiple bands when synovial fluid (SF) from OA patients is analyzed by zymo

235 ther a TLR-2- or TLR-4-stimulating factor in synovial fluid (SF) from patients with early knee OA wit

236 CCL19 and CCL21 levels in synovial fluid (SF) from patients with osteoarthritis (O

237 It is noteworthy that synovial fluid (SF) from patients with rheumatoid arthri

238 The infrapatellar fat pad (FP) and synovial fluid (SF) in the knee serve as reservoirs of m

239 ), and sulfated glycosaminoglycans (sGAG) in synovial fluid (SF) lavage specimens and synovial tissue

240 Synovial fluid (SF) macrophages purified from 24 patient

241 from peripheral blood mononuclear cells and synovial fluid (SF) macrophages were determined by tartr

242 enumerated in the peripheral blood (PB) and synovial fluid (SF) of 12 patients with antibiotic-refra

243 Since DEK has been identified in the synovial fluid (SF) of patients with JIA, this study was

244 + monocytes in the peripheral blood (PB) and synovial fluid (SF) of patients with RA were investigate

245 Cells were isolated from the synovial fluid (SF) of RA patients or patients with othe

246 nd subjected to relative lateral motion with synovial fluid (SF) or phosphate buffered saline (PBS) a

247 ne and cytokine levels were determined in 65 synovial fluid (SF) samples and 7 synovial tissue (ST) s

248 The lubricating ability of synovial fluid (SF) samples from humans with genetic lub

249 CXCL16 was markedly elevated in RA synovial fluid (SF) samples, being as high as 145 ng/ml.

250 ine the functional roles of one of the major synovial fluid (SF) T cell subsets, Vgamma9(+)Vdelta2(+)

251 idence time of hyaluronan (HA) in knee joint synovial fluid (SF) was investigated using a rabbit ante

252 Both synthetic peptide and synovial fluid (SF) were used as sources of antigen.

253 CD4+ T cells from the peripheral blood (PB), synovial fluid (SF), and synovial tissue of RA patients.

254 Expression of NGF/NGFR was examined in synovial fluid (SF), FLS, peripheral blood (PB)-derived

255 tly demonstrated that mechanical shearing of synovial fluid (SF), induced during joint motion, rapidl

256 nally strong biofilmlike aggregates in human synovial fluid (SF), to an extent significantly exceedin

257 tically qualified for use in human urine and synovial fluid (SF).

258 ted intragraft with CXCL16-immunodepleted RA synovial fluid (SF).

259 OA synovial fluids (SF) stimulated TLR2 and TLR4 receptors

260 Proteomic analysis of synovial fluid showed significantly distinct profiles be

261 -time PCR, we studied 24 blood samples and 2 synovial fluid specimens from 20 patients with persisten

262 pray ionization mass spectrometry applied to synovial fluid specimens had an 81% sensitivity and a 95

263 of CD4 T cells in 2 samples of inflammatory synovial fluid, suggesting a potential role in the patho

264 We quantitated NET levels in gout synovial fluid supernatants and detected enzymatically a

265 Tregs from RA synovial fluid suppressed autologous responder T cells;

266 taneous CXCL13 secretion were detected in RA synovial fluid T cells but were not detected (or were de

267 IL-18BPa was lower in RA synovial fluid than in OA synovial fluid (P < 0.05; n =

268 .05; n = 8), and free IL-18 was higher in RA synovial fluid than in OA synovial fluid.

269 riguingly, the LMW form was more abundant in synovial fluid than in serum from both rheumatoid arthri

270 ated mediators were often >10-fold higher in synovial fluid than serum.

271 Synovial fluid, tissue, and sonicate fluid culture and s

272 e real-time PCR and sequencing to culture of synovial fluid, tissue, and sonicate fluid for the micro

273 Synovial fluid uric acid and IL-18 were strongly and pos

274 We conclude that synovial fluid uric acid is a marker of knee OA severity

275 t of subjects with knee osteoarthritis (OA), synovial fluid uric acid was strongly correlated with sy

276 The correlation of synovial fluid uric acid with the two cytokines (IL-18 a

277 early-phase bone scintigraphic findings and synovial fluid volume.

278 ase bone scan scores (P = 0.015), as well as synovial fluid volumes (P < 0.0001).

279 urement of nitrite/nitrate in the plasma and synovial fluid was carried out by chemiluminescence assa

280 flammatory cells involved in osteoarthritis, synovial fluid was collected early after disease inducti

281 Analysis of synovial fluid was consistent with infection, but cultur

282 itionally, expression of both CPB and C5a in synovial fluid was higher in patients with RA than in th

283 Synovial fluid was obtained from 20 patients with RA, 20

284 RA synovial fluid was used to activate macrophages and HEK-

285 mmary likelihood ratio (LR) increased as the synovial fluid WBC count increased (for counts <25,000/m

286 Of the galectin family members present in synovial fluid, we find that galectin-3 is a specific, h

287 Serum and synovial fluid were collected for lipidomic and adipokin

288 ions of SZP and proinflammatory cytokines in synovial fluid were determined by ELISA.

289 SZP expression and aspirate volume of synovial fluid were higher in OA patients than in normal

290 Samples of articular cartilage and synovial fluid were obtained from patients undergoing el

291 Synthetic BCP crystals in synovial fluid were quantified fluorimetrically using ox

292 heral blood neutrophils cultured with 50% RA synovial fluid were study for membrane expression of BLy

293 ules, including PRELP, are released into the synovial fluid where they may interact with components o

294 BCS serves as an in vitro substitute for the synovial fluid which forms a lubricant in the actual ort

295 the clinician, the 2 most powerful were the synovial fluid white blood cell (WBC) count and percenta

296 ng the diagnostic utility of quantifying the synovial fluid white cell count, with two recent systema

297 nts derived from cartilage and released into synovial fluid will allow discrimination between differe

298 We conclude that lubricin provides synovial fluid with an ability to dissipate strain energ

299 survival and suggest that supplementation of synovial fluid with lubricin may be an effective treatme

300 les, six clinical specimens (five blood, one synovial fluid) yielded an atypical oppA1 PCR product, b