ライフサイエンスコーパス: synovial membrane

1 pha, interleukin (IL)-1beta, and IL-6 in the synovial membrane.

2 ) from measures of pathologic changes in the synovial membrane.

3 ve osteoclast precursor cells emerged in the synovial membrane.

4 pression in, and PGE(2) production from, the synovial membrane.

5 is a critical factor for GC formation in the synovial membrane.

6 ssa articular cartilage, articular disc, and synovial membrane.

7 y disease with primary manifestations in the synovial membrane.

8 ion of synovial cells and hyperplasia of the synovial membrane.

9 the expression of the immune response in the synovial membrane.

10 hocytes, macrophages and synoviocytes in the synovial membrane.

11 fects of sCT in joint tissues, including the synovial membrane and cartilage.

12 were localized at the periosteum next to the synovial membrane and in subchondral bone channels.

13 g heterotypic signaling between cells of the synovial membrane and infiltrating lymphocytes in regula

14 lammatory phenotype in fibroblasts from both synovial membrane and infra-patellar fat pad and therefo

15 Strong immunohistochemical staining of the synovial membrane and subsynovial tissue was apparent in

16 rminal center response within the rheumatoid synovial membrane and the existence of similar structure

17 from the sprouted sympathetic fibers in the synovial membrane and upper dermis contribute to the pai

18 arge bones close to the joint spaces, and in synovial membranes and carpal tunnel tissue.

19 Fibroblasts isolated from synovial membranes and infra-patellar fat pad of patient

20 e IL-1alpha transcripts was also detected in synovial membranes and was coordinately up-regulated in

21 pression of COX-2 and PGE(2) in OA meniscus, synovial membrane, and osteophytic fibrocartilage explan

22 s of human OA articular cartilage, meniscus, synovial membrane, and osteophytic fibrocartilage were o

23 -inflammation in target organs such as skin, synovial membrane, and the central nervous system.

24 in the superficial and deeper layers of the synovial membrane as well as a proliferation of synovial

25 One possibility is that the synovial membrane becomes gradually repopulated with imm

26 degradation of cartilage proteoglycans in OA synovial membrane-cartilage cocultures was blocked by th

27 ced cartilage proteoglycan degradation in OA synovial membrane-cartilage cocultures.

28 Conditioned media from synovial membrane cell cultures stimulated human macroph

29 nzymes MMP-1, MMP-2, MMP-3, and MMP-13 in RA synovial membrane cell cultures.

30 flammatory cytokines, VEGF, and MMPs from RA synovial membrane cells was examined.

31 In addition, release of VEGF from RA synovial membrane cells was selectively up-regulated by

32 Dissociated RA synovial membrane cells were cultured in the presence of

33 Synovial membrane cells were dissociated from connective

34 onfirmed that PBEF immunolocalized in apical synovial membrane cells, endothelial cells, adipocytes,

35 In RA synovial membrane cells, IKK-2 inhibition had no effect

36 IkappaBalpha were used to infect ex vivo RA synovial membrane cultures and synovial fibroblasts obta

37 am signaling adaptors MyD88 and Mal/TIRAP in synovial membrane cultures from RA tissue.

38 n of TNFalpha and interleukin-1 (IL-1) in RA synovial membrane cultures reduced VEGF release by 45% (

39 primary human monocytes, macrophages, and RA synovial membrane cultures with p38 MAPK inhibitor compo

40 an peripheral blood monocytes and rheumatoid synovial membrane cultures.

41 feron-gamma-inducible protein 10 in human RA synovial membrane cultures.

42 r the first time in a prospective study that synovial membrane cytokine mRNA expression is predictive

43 In all RA synovial membranes examined (n = 9), BMPRI- and BMPRII-e

44 Canine synovial membrane explants were exposed to high- or low-

45 Synovial membranes from patients with SeA (9 specimens;

46 ow been demonstrated in rheumatoid arthritis synovial membrane, including members of the IL-1 superfa

47 and have demonstrated its expression in the synovial membrane lining layer by immunohistochemistry.

48 Eighteen RA synovial membrane MNC suspensions were cultured for 2 da

49 In ex vivo-cultured RA synovial membrane MNCs, although a limited role of TNFRI

50 In ex vivo-cultured RA synovial membrane MNCs, selective blockade of TNFRI inhi

51 erent methods consistently demonstrated that synovial membrane mRNA levels of IL-1beta, TNFalpha, IL-

52 n (V(H)) genes of B cells recovered from the synovial membrane of five OA patients with marked B cell

53 mutations in p53 have been described in the synovial membrane of rheumatoid arthritis patients, the

54 rget genes in a dose-dependent manner in the synovial membrane of TMJ.

55 Dendritic cell networks in the inflamed synovial membrane optimize antigen collection, storage,

56 s observed between fibroblasts isolated from synovial membrane or infra-patellar fat pad.

57 Sequential sections of wax-embedded synovial membrane samples were obtained from 10 patients

58 as undertaken to analyze their expression in synovial membrane (SM) of patients with psoriatic arthri

59 fic for ADAMTS-4_v1 was found to bind to the synovial membrane surface on cryosections, and the prote

60 and concentration polarization of HA at the synovial membrane surface.

61 IL-11 was found in RA synovial membranes, synovial fluids, and blood sera.

62 lts from a T cell-driven inflammation in the synovial membrane that is frequently associated with the

63 an recruit and activate T lymphocytes in the synovial membrane, thereby contributing to RA pathogenes

64 ohistochemistry in rheumatoid arthritis (RA) synovial membrane tissue and in paw tissue from arthriti

65 e element that defines susceptibility of the synovial membrane to lymphoid organogenesis.

66 ew precursors are continuously formed in the synovial membrane to replenish the osteoclast pool.

67 after gadolinium administration, to quantify synovial membrane volume (SV) as a measure of synovial p

68 Synovial membrane was obtained by knee biopsy and assess

69 l salivary gland, kidney cortex, dermis, and synovial membrane were completely restored, whereas only

70 Cultures of human RA synovial membranes were treated with SSRIs, and cytokine