ライフサイエンスコーパス: syrian hamster

1 ecreted protein 2 was analyzed in the Golden Syrian hamster.

2 ons that mediate mating behavior in the male Syrian hamster.

3 the median and dorsal raphe nuclei), in the Syrian hamster.

4 pleting hepatotoxin, was administered to the Syrian hamster.

5 yloids from three species: human, mouse, and Syrian hamster.

6 arious behaviors and neural functions in the Syrian hamster.

7 ence stimulates offensive aggression in male Syrian hamsters.

8 lity of SARS CoV to infect 5-week-old Golden Syrian hamsters.

9 associated with functional specialization in Syrian hamsters.

10 n of a key hormone, prolactin, in refractory Syrian hamsters.

11 hepatic sterol 27-hydroxylase mRNA levels in Syrian hamsters.

12 ical component of conditioned defeat in male Syrian hamsters.

13 e fibers with biotinylated dextran in golden Syrian hamsters.

14 le of such a change is conditioned defeat in Syrian hamsters.

15 hway of bile acid synthesis, in the liver of Syrian hamsters.

16 thed flagella was isolated from the feces of Syrian hamsters.

17 ortens period length of circadian rhythms in Syrian hamsters.

18 flagella isolated from feces of asymptomatic Syrian hamsters.

19 from the inflamed stomachs and ceca of adult Syrian hamsters.

20 mide (GlcCer) in circulating lipoproteins in Syrian hamsters.

21 eases serotonin receptor populations in male Syrian hamsters.

22 s were identified in juvenile and adult male Syrian hamsters.

23 d no effect against other scrapie strains in Syrian hamsters.

24 the expression of conditioned defeat in male Syrian hamsters.

25 evaluated by a toxin neutralization assay in Syrian hamsters.

26 eight different prion strains propagated in Syrian hamsters.

27 g(MH2M) mice] and subsequent transmission to Syrian hamsters.

28 erolemia and early atherosclerosis in Golden Syrian hamsters.

29 uction of kidney tumors by estradiol in male Syrian hamsters.

30 Copenhageni strain Fiocruz L1-130 in Golden Syrian hamsters.

31 ffects of endotoxin and cytokines on CETP in Syrian hamsters.

32 antavirus is Andes virus (ANDV) infection of Syrian hamsters.

33 ns and removed periodically for bioassays in Syrian hamsters.

34 3-CTV exhibited no toxicity in the brains of Syrian hamsters.

35 lescence facilitates offensive aggression in Syrian hamsters.

36 typhlocolitis in aging (18- to 24-month-old) Syrian hamsters.

37 virus pulmonary syndrome (HPS) in humans and Syrian hamsters.

38 nation inhibition (HAI) titers than RD-Ad in Syrian hamsters.

39 as to replication and pathogenesis in golden Syrian hamsters.

40 e brains of young, middle-aged, and old male Syrian hamsters.

41 on reward processes and copulation in female Syrian hamsters.

42 dian pacemaker and peripheral oscillators in Syrian hamsters.

43 ed three related hypotheses; namely, that in Syrian hamsters: (1) PS potentiation can be elicited bel

44 Single episodes of arousal of Syrian hamsters 2 h before projected activity onset (i.e

45 is peptide has been investigated in the male Syrian hamster, a species in which brain nuclei controll

46 ets of retinal fibers in the early postnatal Syrian hamster, a well-characterized developmental model

47 , most studies have been conducted in golden Syrian hamsters, a species particularly sensitive to acu

48 asured in rapid and slow acetylator congenic Syrian hamsters administered 3,2' -dimethyl-4-aminobiphe

49 dministration of (L)- Met-methyl-d(3) to the Syrian hamster after GSH had been depleted by BB resulte

50 These findings indicate that human and Syrian hamster alpha(v)beta(3) integrins are key recepto

51 he treatment of SARS was evaluated in golden Syrian hamsters, an animal model that supports SARS-CoV

52 ithin the forebrain and midbrain of the male Syrian hamster and addresses the question of whether enk

53 hrough PSI domain residues conserved in both Syrian hamster and human beta(3) integrins.

54 SI domain revealed eight differences between Syrian hamster and human beta(3) integrins.

55 ulated expression of COX-2 in HIT-T15 cells, Syrian hamster and human islets, and other Syrian hamste

56 onditions on reconfiguration dynamics of the Syrian hamster and rabbit prion proteins.

57 protein (PrP) amyloids from human, mouse and Syrian hamster and show that their structural difference

58 e-resistant PrP was reported in the urine of Syrian hamsters and humans with prion disease.

59 uated in animal models of dyslipidemia using Syrian hamsters and male Beagle dogs, and all these comp

60 P were sparse within the SCN of Siberian and Syrian hamsters and mouse but were intense in the ventra

61 rticle sequences closest to those present in Syrian hamsters and not mice were also detected in the J

62 stent immune responses than RD-Ad vectors in Syrian hamsters and rhesus macaques.

63 Syrian hamsters are permissive for the replication of sp

64 Syrian hamsters are susceptible to infection with certai

65 In Syrian hamsters, arginine vasopressin (AVP) plays a crit

66 ed in the domestic cat as Orange, and in the Syrian hamster as Sex-linked yellow (Sly), but are curio

67 Twenty-four male Golden-Syrian hamsters at 3 months of age were randomly divided

68 atic nucleus (SCN) in urethane-anaesthetized Syrian hamsters at different times of the light-dark cyc

69 Syrian hamsters become anemic and exhibit delayed growth

70 Sequencing the Syrian hamster beta(3) integrin PSI domain revealed eigh

71 I domain polypeptides derived from human and Syrian hamster beta(3) subunits, but not murine or bovin

72 used for mucosal intranasal immunization of Syrian hamsters, both SC-Ad and RC-Ad expressed transgen

73 Exposure of Sc237 prions in Syrian hamster brain homogenates to 1% SDS and 0.5% AcOH

74 ously distributed across the neuraxis of the Syrian hamster brain.

75 he distribution of 5-HT(3A) receptors in the Syrian hamster brain.

76 c were obtained when CLDs were isolated from Syrian hamster brains.

77 strain could also be transmitted directly to Syrian hamsters, but when derived in this way, its prope

78 n of bile acid metabolism in the male Golden Syrian hamster by measuring the rate of fecal bile acid

79 kinetics of leptospiral infection in Golden Syrian hamsters by a sensitive quantitative real-time PC

80 ubjective day, circadian activity rhythms in Syrian hamsters can be phase advanced by a variety of st

81 DFTD, and as well as the soft-shell clam and Syrian hamster, can advance studies of tumor biology.

82 und that human adenovirus replicates well in Syrian hamster cell lines and confirmed replication in t

83 lved in structural alterations in neoplastic Syrian hamster cells transformed by chemical carcinogens

84 echanisms activated cph in other MCA-treated Syrian hamster cells.

85 respectively, and was 95% homologous to the Syrian hamster cells.

86 nt with the production of the JEV vaccine in Syrian hamster cells.

87 Complexes of the Syrian hamster cellular prion protein (PrPC) and synthet

88 This strain was also avirulent in the Syrian hamster challenge model.

89 slow (Bio.82.73/ H-Pat(s) acetylator female Syrian hamsters congenic at the NAT2 locus received a s.

90 PrP(Sc) from the brains of Syrian hamsters contains the same set of glycans as PrP(

91 To address this question, we gave male Syrian hamsters daily injections of the cell birthdate m

92 ANDV infection of human endothelial cells or Syrian hamster-derived BHK-21 cells was selectively inhi

93 Here, we report that Syrian hamster dermal fibroblasts possess substantial le

94 scribed lethal HPS animal model (i.e., adult Syrian hamsters develop HPS and die within 10 to 15 days

95 Depletion of T cells from Syrian hamsters did not significantly influence early ev

96 ring long "summer-like" photoperiods, female Syrian hamsters display a regular 4-day estrous cycle.

97 ficacious in the clinically validated golden Syrian hamster ear animal model, was designed to be deli

98 Male Syrian hamsters either did or did not experience endogen

99 transformation system of carcinogen-treated Syrian hamster embryo (SHE) cell cultures represents mul

100 In Syrian hamster embryo (SHE) fibroblasts, epidermal growt

101 In Syrian hamster embryo (SHE) fibroblasts, epidermal growt

102 le of c-Fos in apoptosis was examined in two Syrian hamster embryo cell lines (sup+I and sup-II) and

103 and comparisons were made between these two Syrian hamster embryo cell lines.

104 o(a)pyrene [B(a)P]-induced transformation of Syrian hamster embryo cells and benzo(a)pyrene diol-epox

105 anscripts expressed in normal and neoplastic Syrian hamster embryo cells in culture (5.0, 3.5 and 2.0

106 A preneoplastic variant of Syrian hamster embryo cells, sup(+), exhibits decreased

107 Cph was isolated from neoplastic Syrian hamster embryo fibroblasts initiated by 3-methylc

108 Treatment of Syrian hamster embryo fibroblasts with a single dose of

109 sforming mutants of avian pp60(c-src) from a Syrian hamster embryo-derived cell line, 10W, transfecte

110 LTP) was examined in hippocampal slices from Syrian hamsters entrained to a LD 14:10 cycle.

111 Adult male Syrian hamsters equipped with a surgically implanted gui

112 In Syrian hamsters, even a single social defeat results in

113 that the SCN, IGL, MRN, and DRN of the male Syrian hamster exhibited specific binding of [3H]8-OH-DP

114 ness to estradiol and progesterone in female Syrian hamsters exposed to a short photoperiod is associ

115 eks of the study (HFD-FBX4w group) to Golden Syrian hamsters fed a high-fat diet (HFD) for 8 weeks we

116 Chronic administration of estrogen to male Syrian hamsters for 7.0 to 9.0 months induces a high fre

117 -HT(3A) receptors across the neuraxis of the Syrian hamster forebrain using immunohistochemistry.

118 We have now isolated Syrian hamster full-length cDNAs for the cph oncogene an

119 Unlike rats, studies in Syrian hamsters have failed to detect any alterations in

120 Golden Syrian hamster hearts were heterotopically transplanted

121 lly transplanted with brown Norway or Golden Syrian hamster hearts were treated for 50 or 75 days wit

122 ependent phosphodiesterase (PDE) activity in Syrian hamster hearts with hypertrophic cardiomyopathy (

123 dues from the kinase recognition sequence of Syrian hamster HMG-CoA reductase (E.C. 1.1.1.34).

124 The activity of the class I Syrian hamster HMG-CoA reductase is regulated by phospho

125 The three conserved lysines of Syrian hamster HMG-CoA reductase were mutated to alanine

126 When injected unilaterally into the SCN of Syrian hamsters housed in constant darkness, OFQ/N (1-50

127 nditioned defeat is a social defeat model in Syrian hamsters in which individuals display increased s

128 tamate agonist NMDA directly into the SCN of Syrian hamsters induced significant phase delays at circ

129 Syrian hamsters infected with ANDV, but not SNV, develop

130 ids in purified fractions from the brains of Syrian hamsters infected with Sc237 prions.

131 about PrP 27-30 purified from the brains of Syrian hamsters infected with scrapie.

132 y, no signs of prion infection were found in Syrian hamsters inoculated with rPrP fibrils that resemb

133 ing and sequence analysis of portions of the Syrian hamster interleukin 2 (IL-2), IL-4, gamma interfe

134 Using a Syrian hamster intraperitoneal model of infection, we de

135 Thus, the immunosuppressed Syrian hamster is a powerful model to evaluate anti-Ad d

136 Our findings show that the Syrian hamster is a promising immunocompetent model that

137 p process for estrogen carcinogenesis in the Syrian hamster kidney is proposed.

138 ls: the db/db mouse type 2 diabetes model, a Syrian hamster lipid model, and a dog lipid model.

139 In the male Syrian hamster, mating is dependent on chemosensory and

140 For CRM data acquired from midsagittal Syrian hamster ( Mesocricetus auratus ) brain cryosectio

141 In the Syrian hamster (Mesocricetus auratus) glutamate activity

142 The Syrian hamster (Mesocricetus auratus) has been extensive

143 Remarkably, infection of the Syrian hamster (Mesocricetus auratus) with L. donovani r

144 tems modulating this behavior using pubertal Syrian hamsters (Mesocricetus auratus) as an adolescent-

145 al neurons from postnatal day (P)0-P2 golden Syrian hamsters (Mesocricetus auratus) of either sex to

146 Male Syrian hamsters (Mesocricetus auratus) treated with low-

147 Cocaine-treated Syrian hamsters (Mesocricetus auratus) were tested for a

148 In Syrian hamsters (Mesocricetus auratus), cells in a subre

149 In Syrian hamsters (Mesocricetus auratus), social defeat pr

150 ehavioral effects on intermale aggression in Syrian hamsters (Mesocricetus auratus).

151 of various neurotransmitter systems in male Syrian hamsters (Mesocricetus auratus).

152 or protein-1 (AP-1) activity in both primary Syrian hamster mesothelial cells (SHM) and primary human

153 Therefore, the Syrian hamster model may become a valuable tool for the

154 tant showed enhanced virulence in the Golden Syrian hamster model of acute C. difficile infection.

155 Here, we describe a lethal Syrian hamster model of EHF using mouse-adapted Ebola vi

156 tahcp6) and tested them for virulence in the Syrian hamster model of infection.

157 er membrane lipoprotein LipL41 in the Golden Syrian hamster model of leptospirosis.

158 associated pathological findings in a golden Syrian hamster model of SARS-CoV infection.

159 To this end, we developed a novel Syrian hamster model that is both immunocompetent and re

160 In the golden Syrian hamster model, p.o. sodium bicarbonate supplement

161 sease; however, by directly manipulating the Syrian hamster model, we continue to eliminate individua

162 -specific antibodies than RD in a permissive Syrian hamster model.

163 its protective efficacy in the common lethal Syrian hamster model.

164 integrins in hantavirus pathogenesis in the Syrian hamster model.

165 tissue spread of the vector, we employed the Syrian hamster model.

166 nicity of ATCC 23344 in the BALB/c mouse and Syrian hamster models of infection.

167 examine the therapeutic and toxic effects in Syrian hamster models of pancreatic cancer (PaCa).

168 e clock mutation (tau) was discovered in the Syrian hamster more than a decade ago and, using the pow

169 ssion to transgenic mice expressing chimeric Syrian hamster/mouse (MH2M) prion protein (PrP) genes [T

170 of residues within the PSI domains of human, Syrian hamster, murine, and bovine beta(3) integrins ide

171 As in the analogous Syrian hamster mutant enzyme S871D, P. mevalonii mutant

172 and MS-8209 prolonged the incubation time in Syrian hamsters of the 263K strain of scrapie, but AmB h

173 ve entraining stimulus in fetal and neonatal Syrian hamsters of the same developmental ages used to p

174 In a Syrian hamster pancreatic cancer model, tumors similar t

175 However, expression of the Syrian hamster PCPH and mt-PCPH proteins in haploid yeas

176 of peptides based on residues 109-122 of the Syrian hamster prion protein (H1) with a range of substi

177 -ray crystallographic structures of the anti-Syrian hamster prion protein (SHaPrP) monoclonal Fab 3F4

178 Using recombinant Syrian hamster prion protein [SHaPrP(29-231)], we invest

179 sion cross sections measured for recombinant Syrian hamster prion protein PrP(90-231).

180 The binding of the Syrian hamster prion protein, SHaPrP(90-231), to model l

181 lts of previous HDX studies on the human and Syrian hamster prion proteins at a higher pH, various se

182 (rPrP) of 142 residues corresponding to the Syrian hamster PrP 27-30 was expressed in Escherichia co

183 recombinant protein (rPrP) corresponding to Syrian hamster PrP 27-30 was expressed in Escherichia co

184 d transgenic mice expressing either mouse or Syrian hamster PrP exclusively in muscle.

185 amination of the dynamics of two recombinant Syrian hamster PrP fragments, PrP(29-231) and PrP(90-231

186 experiments using transgenic mice expressing Syrian hamster PrP in neurons only, MS-8209 extended the

187 this present work, recombinant, full-length Syrian hamster PrP is investigated using EPR methodologi

188 , the secondary structure of the recombinant Syrian hamster PrP of residues 29-231 [PrP(29-231)] is i

189 cellular prion protein (PrPC) and synthetic Syrian hamster PrP peptides were found to mimic many of

190 Studies of an analogous peptide from Syrian hamster PrP verify that sequence alterations in r

191 t similar to those found in the structure of Syrian hamster PrP(90-231).

192 We have investigated the conformation of Syrian hamster PrP(C) on the surface of transfected CHO

193 Syrian hamsters readily form dominant-subordinate relati

194 amyloid states were prepared in vitro using Syrian hamster recombinant PrP (rPrP) in the absence of

195 In Syrian hamsters, reproductive behaviors are initiated in

196 In vitro amplification of Syrian hamster Sc237 PrP(Sc) displays an optimum pH of a

197 ions of serotonergic and other agents to the Syrian hamster SCN with durations equivalent to the incr

198 l-length prion protein PrP (residues 23-231, Syrian hamster sequence).

199 Synthetic peptides included: (i) Syrian hamster (SHa) hydrophobic core, SHa106-122 (KTNMK

200 NMR has been used to refine the structure of Syrian hamster (SHa) prion protein rPrP(90-231), which i

201 s 120-144 of human (Hu), bank vole (BV), and Syrian hamster (SHa) prion protein, from disordered mono

202 olling susceptibility to foreign prions, two Syrian hamster (SHa) prion strains, Sc237 and DY, were t

203 Syrian hamster (SHa) PrP genes of varying length ranging

204 Selection against recombinant (rec) PrP of Syrian hamster (SHa) sequence 90-231 folded into an alph

205 ission of prions through feces, uninoculated Syrian hamsters (SHas) were cohabitated with or exposed

206 Dermal fibroblasts from the Syrian hamster (SHD cells) are exceptionally resistant t

207 nostaining generalized to the SCN and IGL of Syrian hamster, Siberian hamster, and mouse.

208 cating efficiently in normally nonpermissive Syrian hamster smooth muscle (DDT-1), Chinese hamster ov

209 In contrast, in the TG-resistant Syrian hamster smooth muscle cell line DDT/TG 4 microM,

210 ted a series of cell lines from the parental Syrian hamster smooth muscle cell line DDT1-MF2that are

211 t carcinoma cells MCF-7 and MDA-MB-468 and a Syrian hamster smooth muscle cell line DDT1MF2 and were

212 under moderate stringency conditions with a Syrian hamster-specific cph probe.

213 tinguishable from the previously established Syrian hamster strain Sc237, despite having been derived

214 ect virus replication or morbidity in golden Syrian hamsters, suggesting that the gene 7 products are

215 the Mitf gene of the anophthalmic white Wh) Syrian hamster that destabilizes its mRNA and prevents t

216 severe systemic disease in immunosuppressed Syrian hamsters that is similar to that seen in immunoco

217 Golden Syrian hamsters that were intranasally inoculated with S

218 After transmission to Syrian hamsters, the Me7 strain was indistinguishable fr

219 xpression of the cph proto-oncogene in adult Syrian hamster tissues by northern hybridization using c

220 , Syrian hamster and human islets, and other Syrian hamster tissues.

221 d the brain transcriptome of male and female Syrian hamsters to generate the necessary resources to c

222 se fluoxetine during adolescence predisposes Syrian hamsters to offensive aggression, with demonstrab

223 and decondensation induced by the binding of Syrian hamster transition proteins TP1 and TP2 and prota

224 In the present study, male Syrian hamsters treated with TRZ (5 mg/kg) at ZT6 signif

225 hK2 was expressed in the adenovirus-induced Syrian hamster tumor cell line AV12-664 (AV12-hK2).

226 lian cells, recombinant PSA was expressed in Syrian hamster tumor cell line AV12-664 (AV12-PSA).

227 Estrogen-induced Syrian hamster tumors in the kidney represent a useful m

228 n membrane currents in single cells from the Syrian hamster vas deferens cell line DDT1MF-2 were inve

229 In Syrian hamsters, vasopressin (AVP) controls a form of sc

230 In Syrian hamsters, vasopressin (AVP) in the medial preopti

231 teral inoculation into the sciatic nerves of Syrian hamsters was investigated.

232 Young male Golden Syrian hamsters, weighing 90 to 120 g.

233 Golden Syrian hamsters were immunized at 3 and 6 weeks of age w

234 Syrian hamsters were inoculated intraperitoneally with b

235 STAT1(-)(/-) mice, Hartley guinea pigs, and Syrian hamsters were inoculated intraperitoneally with R

236 Syrian hamsters were split under LDLD with dimly lit nig

237 nogenesis, freshly isolated islets from male Syrian hamsters were transplanted into the right submand

238 Male Syrian hamsters were treated with AAS throughout adolesc

239 Syrian hamsters were treated, intratracheally, with clod

240 Panama, cause a differential pathogenesis in Syrian hamsters, which could be a useful model for under

241 Intratracheal infection of Syrian hamsters with Ad14p1 caused a marked, patchy bron

242 Infection of Syrian hamsters with ANDV results in a disease that clos

243 inkage map for the euchromatic region of the Syrian hamster X chromosome that places Sly in a region