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1 f IgG1-, IgG2a-, and IgG2b-dependent passive systemic anaphylaxis.
2 are less sensitive to IgE-mediated, passive, systemic anaphylaxis.
3 ivo, and ROCK inhibitors protect from lethal systemic anaphylaxis.
4  that PAG is a positive regulator of passive systemic anaphylaxis.
5 asses have been shown to enable induction of systemic anaphylaxis.
6 tion and to ovalbumin (OVA) allergen induced systemic anaphylaxis.
7  airway inflammation, and active and passive systemic anaphylaxis.
8 says ex vivo and in a mouse model of passive systemic anaphylaxis.
9 esponsible for FcgammaRIIA-dependent passive systemic anaphylaxis.
10 ct OX40L contribution in an in vivo model of systemic anaphylaxis.
11 on, and do not develop IgE-dependent passive systemic anaphylaxis.
12 axis in the ear and joint as well as passive systemic anaphylaxis.
13 osal tissues, thereby minimizing the risk of systemic anaphylaxis.
14 a leakage syndromes, including angioedema or systemic anaphylaxis.
15 ntly greater and faster death rate in active systemic anaphylaxis.
16 gy, food allergy, conjunctivitis, and severe systemic anaphylaxis.
17  mice were resistant to IgE-mediated passive systemic anaphylaxis.
18 ce in both active and IgG1-dependent passive systemic anaphylaxis.
19 ining their fates in mice undergoing passive systemic anaphylaxis.
20  human intravenous immunoglobulin and active systemic anaphylaxis after immunization and challenge.
21 he jejunum, increased serum IL-4 levels, and systemic anaphylaxis after oral challenge, as evidenced
22 ary for the development of passive local and systemic anaphylaxis and airway inflammation.
23 and S1P(2) on mast cells are determinants of systemic anaphylaxis and associated pulmonary edema and
24 ine the capabilities of hFcgammaRs to induce systemic anaphylaxis and identify the cell types and med
25 on FcepsilonRI-MC-mediated passive local and systemic anaphylaxis and on airway inflammation.
26 n shown to mediate an alternative pathway of systemic anaphylaxis and to participate in allergic skin
27 ergies, urticaria, nonhereditary angioedema, systemic anaphylaxis, and allergic conjunctivitis are as
28 sposes individuals to develop asthma, severe systemic anaphylaxis, and atopic dermatitis, is usually
29 cytopenia, neutrophils to be responsible for systemic anaphylaxis, and both cell types to be responsi
30 d IgE-mediated histamine release and passive systemic anaphylaxis, and Drebrin(-/-) mast cells also e
31 re known for their roles in allergy, asthma, systemic anaphylaxis, and inflammatory disease.
32  Furthermore, in a low-dose model of passive systemic anaphylaxis, antigen-dependent decrease in body
33 rgic reactions such as passive cutaneous and systemic anaphylaxis are markedly impaired in Gab2-/- mi
34 d with PN, they exhibited mast-cell-mediated systemic anaphylaxis, as indicated by hypothermia and in
35  than their normal counterparts to a passive systemic anaphylaxis challenge.
36        We recently found in mice that active systemic anaphylaxis depends on IgG and IgG receptors (F
37  surface IgE and triggered degranulation and systemic anaphylaxis in allergen-naive WT but not in IL-
38 aphylaxis; and attenuate dansyl IgE-mediated systemic anaphylaxis in human FcepsilonRIalpha transgeni
39                           Studies of passive systemic anaphylaxis in IL-10-transgenic mice showed tha
40                              We confirm that systemic anaphylaxis in mice can be mediated largely thr
41  of an IgG1-dependent alternative pathway of systemic anaphylaxis in mice.
42  sensitized to exhibit IgE-dependent passive systemic anaphylaxis in vivo and to investigate the anti
43  cell degranulation and efficiently inhibits systemic anaphylaxis in vivo.
44 vascular hyperpermeability (AVH) and passive systemic anaphylaxis-induced hypothermia and edema.
45        Also, in 2 different models of active systemic anaphylaxis, inhibition of NOS, PI3K, or Akt or
46                                              Systemic anaphylaxis is generally recognized as a severe
47 g an AD-HIES mutation were hyporesponsive to systemic anaphylaxis models.
48 in passive cutaneous anaphylaxis and passive systemic anaphylaxis mouse models, and in ovalbumin-indu
49                                              Systemic anaphylaxis occurred in Was(-/-) mice (95%) wit
50 nt specific IgE-mediated responses and drive systemic anaphylaxis on ingestion challenge.
51 ral antigen dose determine whether diarrhea, systemic anaphylaxis, or both are induced, and ingested
52                                      Passive systemic anaphylaxis (PSA) induced HDAC3 expression and
53         Furthermore, antigen-induced passive systemic anaphylaxis (PSA) was analysed in PEP(+/+) and
54 aling in the context of IgG-mediated passive systemic anaphylaxis (PSA).
55  and profound depletion of plasma S1P during systemic anaphylaxis rendered both platelet- and erythro
56  a human mast cell antigen-dependent passive systemic anaphylaxis response in primed mice.
57 d mast cell granularity and impaired passive systemic anaphylaxis responses.
58 tations of food allergy include diarrhea and systemic anaphylaxis (shock), which can occur together o
59 of passive cutaneous anaphylaxis and passive systemic anaphylaxis that can be used to investigate the
60 allergen-specific IgG confers sensitivity to systemic anaphylaxis that relies on IgG Fc receptors (Fc
61 h trinitrophenyl-BSA intravenously to induce systemic anaphylaxis that was monitored by using rectal
62 n smooth muscle cells, also leads to reduced systemic anaphylaxis, the most severe form of allergic r
63                  In response to IgE-mediated systemic anaphylaxis, the symptoms and decreases in core
64                               Intestinal and systemic anaphylaxis was assessed, and the role of the h
65                                      Passive systemic anaphylaxis was induced by injection of heat-ag
66                                      Passive systemic anaphylaxis was induced in female and male mice
67                                  Severity of systemic anaphylaxis was measured by changes in body tem
68                                   A model of systemic anaphylaxis was studied in transgenic mice carr
69                                In a model of systemic anaphylaxis, we found no difference between WT
70 a of V3 mastocytosis mice undergoing passive systemic anaphylaxis, we used this in vivo model system
71 th ovalbumin; models of allergic diarrhea or systemic anaphylaxis were studied.

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