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1 d to reduce hospitalization and mortality in systolic heart failure.
2 nd is at least as common in the community as systolic heart failure.
3 that is in clinical trials for treatment of systolic heart failure.
4 ailability and muscle power in patients with systolic heart failure.
5 iorespiratory fitness (CRF) in patients with systolic heart failure.
6 ure in patients with decompensated end-stage systolic heart failure.
7 ure in patients with decompensated end-stage systolic heart failure.
8 reasingly prevalent in patients with chronic systolic heart failure.
9 vabradine reduces afterload of patients with systolic heart failure.
10 ct of isolated HR reduction in patients with systolic heart failure.
11 ardiac contractility is a central feature of systolic heart failure.
12 ivator, omecamtiv mecarbil, in patients with systolic heart failure.
13 n may provide a new therapeutic approach for systolic heart failure.
14 rral cohort representing a broad spectrum of systolic heart failure.
15 stic and prognostic utility in patients with systolic heart failure.
16 ing is the principal cause of progression of systolic heart failure.
17 in children and adolescents with symptomatic systolic heart failure.
18 having symptoms that could be attributed to systolic heart failure.
19 emerged as an important therapy for advanced systolic heart failure.
20 art failure hospitalization in patients with systolic heart failure.
21 e, and reduced cost of care in patients with systolic heart failure.
22 trial was conducted of 6800 outpatients with systolic heart failure.
23 prognostic implications among patients with systolic heart failure.
24 rom 39 patients with decompensated end-stage systolic heart failure (92% male), aged 56 +/- 13 years.
26 daily with placebo in patients with ischemic systolic heart failure according to baseline high sensit
31 valuated and is used in select patients with systolic heart failure and chronic stable angina without
34 of ventricular systole are characteristic of systolic heart failure and might be improved by a new th
36 -blind trial, we assigned 2278 patients with systolic heart failure and mild-to-moderate anemia (hemo
39 iews data from randomized clinical trials in systolic heart failure and the interactions between base
40 eatment for patients with moderate to severe systolic heart failure and ventricular dyssynchrony.
41 ents that clinically have both diastolic and systolic heart failure and will soon become the most com
42 ined ventricular tachycardia with syncope or systolic heart failure as a result of ischemic or nonisc
43 ntagonists improve outcomes in patients with systolic heart failure but may induce worsening of renal
46 an exercise training trial for patients with systolic heart failure, cardiopulmonary exercise tests w
47 brillator (ICD) in patients with symptomatic systolic heart failure caused by coronary artery disease
49 m coenzyme Q in 1,191 patients with ischemic systolic heart failure enrolled in CORONA (Controlled Ro
50 y profile) in patients with mild symptoms of systolic heart failure, even in those already receiving
51 s with New York Heart Association class I/II systolic heart failure exhibit left ventricular (LV) rev
53 h greater proportional representation in the systolic heart failure group; all 12 SNPs were confirmed
55 ished adjunctive treatment for patients with systolic heart failure (HF) and ventricular dyssynchrony
56 ng at night ("sleep apnea") in patients with systolic heart failure (HF) have failed to improve progn
59 ommend digoxin for patients with symptomatic systolic heart failure (HF) receiving optimal medical th
60 remodeling associated with the transition to systolic heart failure (HF) were examined in the spontan
67 tractile failure during cardiac ischemia and systolic heart failure, in part due to decreased excitat
72 trolled trial, 556 patients with symptomatic systolic heart failure (left ventricular ejection fracti
73 CD implantation in patients with symptomatic systolic heart failure not caused by coronary artery dis
76 echocardiographic analysis in patients with systolic heart failure obtained from a variety of invest
77 efibrillators] in Patients With Non-Ischemic Systolic Heart Failure on Mortality) did not demonstrate
79 ducted a randomized, controlled trial of 223 systolic heart failure patients and compared the effects
82 ment of patients with moderately symptomatic systolic heart failure, patients experienced thromboembo
84 ansplantation in ESRD patients with advanced systolic heart failure results in an increase in LVEF, i
85 ontrols and 1,117 Caucasian individuals with systolic heart failure revealed 12 SNPs in the cardiovas
86 gnificantly lower BNP levels than those with systolic heart failure (S-CHF) (413 pg/ml vs. 821 pg/ml,
87 e retrospective cohort had 664 patients with systolic heart failure (SHF) and 399 patients with diast
88 ol group of 35 subjects and 60 patients with systolic heart failure (SHF) were included for compariso
92 enefit of prophylactic ICDs in patients with systolic heart failure that is not due to coronary arter
95 o either placebo or ivabradine in the SHIFT (Systolic Heart Failure Treatment With the If Inhibitor I
96 vabradine and outcomes in chronic HF (SHIFT [Systolic Heart Failure Treatment With the IF Inhibitor I
98 prevention ICD in patients with nonischemic systolic heart failure warrants further investigation.
99 ure in patients with decompensated end-stage systolic heart failure was recently challenged, but the
102 au patterns may be observed in patients with systolic heart failure who cannot further increase .Q at
103 tors (PROSe-ICD) enrolled 1189 patients with systolic heart failure who underwent ICD implantation fo
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