ライフサイエンスコーパス: t

1 SETBP1 in aCML, and SF3B1-JAK2 in MDS/MPN-RS-T).

2 ucture of PDE6 complexed to GTP-bound Galpha(T).

3 ilencing mechanism of transposable elements (TEs).

4 3 providing electrostatic stabilization of G-T*.

5 es were nearly 40% at 1.5 T and < 12% at 3.0 T.

6 I(2) was 98% at 1.5 T and 3.0 T.

7 quid with a magnetic field B(S) = 10(12+/-1) T.

8 re found in the promoters of TBT, DBTNBT and TS.

9 tial benefits to the host, the virus, or the TEs.

10 on the advantages and limitations of current T(1) contrast agents and the potential of IONPs to serve

11 xes cannot compete with Gd(III) complexes as T(1) MRI contrast agents.

12 xBox(4+) for the population of the low-lying T(1) state.

13 We first analyze the E(T(1)), E(S(1)), and E(T(2)) of benzene and cyclobutadien

14 ly, we discovered that G. stearothermophilus T-1 can also utilize lactose and galactosyl-glycerol via

15 For these organs, the mean pancreatic T(2) values were nearly 40% at 1.5 T and < 12% at 3.0 T.

16 We first analyze the E(T(1)), E(S(1)), and E(T(2)) of benzene and cyclobutadiene (CBD) as excited-sta

17 ude that both atypical 3q26/MECOM and inv(3)/t(3;3) can be classified as a single entity of 3q26-rear

18 Phylogenetic analysis showed Asp t 36 to be highly conserved with close similarity to the

19 The resultant radiance bound by the T(4) law limits the ability to regulate radiative heat.

20 nd ecosystem respiration (RE) in response to T(a) and EF anomalies were compared for different forest

21 actor RASGRP1 is frequently overexpressed in T-ALL patients.

22 ing in T-cell acute lymphoblastic leukaemia (T-ALL), and the involvement of BCL6 in other types of le

23 s study is important to our understanding of T-ALL.

24 signaling, could have a suppressive role in T-ALL.

25 ancreatic T(2) values were nearly 40% at 1.5 T and < 12% at 3.0 T.

26 tion therapy or craniotomy who underwent 1.5-T and 3-T same-plane T1-weighted MRI (in any order).

27 I(2) was 98% at 1.5 T and 3.0 T.

28 CR5(-) memory Th cells as well as regulatory T and B cells were increased.

29 nctional pathway alpha-diversity between CTX-T and CTX-N infants.

30 model was trained to distinguish between non-TEs and TEs in plants.

31 anding of neurodevelopmental trajectories in TS and carry implications for future applications aimed

32 Cytotoxic T lymphocytes (T) and natural killer cells are the main cytotoxic kille

33 B, T, and myeloid cells were analyzed before anti-CD20 admi

34 A simple descriptor, mu/t, where mu and t are the octahedral and tolerance factors, respectively

35 omparison to prior work indicates that these TS are similar in nature to those for the alkaline hydro

36 lts, FPR, FNR, and FL); (2) total deviation (TD) at each location; and (3) computationally derived ar

37 ), CD45RO-/lo, CD62L(-), CD27lo) with higher T-bet expression.

38 data suggest that mTOR activity is linked to T-bet in Ag-expCD4(+) T cells but that reduction in mTOR

39 s show no large rate enhancements due to the t-butyl group in 7.

40 air of recurrent cortico-thalamo-cortical (C-T-C) loops.

41 G), grape seed and olive (O) or grape total (T), called ESG, ESO and EST, respectively.

42 findings define AP-1 as the key link between T cell activation and chromatin remodeling.

43 pinocytosis that increases in magnitude upon T cell activation to support T cell growth even under am

44 I and III responses, early CD4(+) and CD8(+) T cell activation, and counterregulation by the co-recep

45 ld and a conceptually simple model of CD8(+) T cell Ag recognition, in which Ag dose and affinity do

46 When a T cell and an antigen-presenting cell form an immunologi

47 rly clonal dynamics imprint the hierarchy of T cell clone sizes with implications for pathogen defens

48 nd that the immunodominance of high-affinity T cell clones declined during the chronic infection phas

49 in Tph1 deficient ILC2s including inducible T cell co-stimulator (Icos).

50 rforin contributed to both CD8+ and CD4+ CAR T cell cytotoxicity but was not required for in vitro or

51 ions harboring genes with prominent roles in T cell development in both strains.

52 te Notch, a critical regulator of B cell and T cell development.

53 role for LDH in modulating cytokine-mediated T cell differentiation and underscore the therapeutic po

54 a, we develop a quantitative theory of human T cell dynamics compatible with the statistical laws of

55 R signal strength is able to regulate CD8(+) T cell effector cytokine R production independent of TCR

56 T cell expansion and differentiation are critically depe

57 proach is often limited by the extent of CAR-T cell expansion in vivo.

58 g of the Th1/Th2 paradigm ignited the CD4(+) T cell field.

59 To understand how these compounds alter T cell function, we assessed their therapeutic activity

60 magnitude upon T cell activation to support T cell growth even under amino acid (AA) replete conditi

61 regnancy is associated with recovery of CD4+ T cell immunity.

62 tages of tumor inception to subvert adaptive T cell immunity.

63 on in complex biological settings, including T cell immunology.

64 ition to suppressing viremia, bNAbs may have T cell immunomodulatory effects as seen for other forms

65 nd neoantigen load) and the degree of CD8(+) T cell infiltration were not associated with clinical re

66 ith lower levels of Th1 cytokines, decreased T cell infiltration, increased B cell numbers, and decre

67 in ligase Peli1 as an important regulator of T cell metabolism and antitumor immunity.

68 th enhanced cytolytic potential and requires T cell migration from lymph nodes for therapeutic effica

69 Although T cell migration is most frequently defined in the conte

70 ntiation programs in the human CD8(+) memory T cell pool, with potentially broad implications for the

71 enance of extraordinarily large CMV-specific T cell populations.

72 ts an important role for B cells in indirect T cell priming and further emphasizes the advantage of c

73 discuss the innovative designs of novel CAR T cell products that are being developed to increase and

74 Cytokines that stimulate T cell proliferation, such as interleukin (IL)-15, have

75 T cell reactivity against SARS-CoV-2 was observed in une

76 Activation of the T cell receptor (TCR) results in binding of the adapter

77 matical modeling and statistical analyses of T cell receptor sequencing data, we develop a quantitati

78 entially enhance our understanding of CD4(+) T cell recognition.

79 xhausted CD8(+) T cells, it fails to restore T cell repertoire diversity.IMPORTANCE Checkpoint inhibi

80 onclusion, we show that MEKi leads to CD8(+) T cell reprogramming into T(SCM) that acts as a reservoi

81 iming and effector phases, provokes systemic T cell responses against dominant and subdominant neoant

82 Total and spike-specific T cell responses correlated with spike-specific antibody

83 late and coordinate alphabeta and gammadelta T cell responses remains unknown.

84 pic model of TCR signaling in which multiple T cell responses share a common rate-limiting threshold

85 SARS-CoV-2-specific T cell responses were driven by TCR clusters shared betw

86 ive range, multifunctional CD8(+) and CD4(+) T cell responses with S protein-specific killing activit

87 nate immune responses and adaptive cytotoxic T cell responses.

88 icrobiome shifts and enhanced intestinal CD8 T cell responses.

89 elicit broadly protective CD4(+) and CD8(+) T cell responses.

90 cells and the induction of protective CD8(+) T cell responses.

91 A distinct TEMRA CD8(+) T cell subpopulation was identified that was characteriz

92 The discovery of CD4(+) T cell subset-defining master transcription factors and

93 The application of adoptive T cell therapies, including those using chimeric antigen

94 munotherapy and has general implications for T cell-based immunotherapies.

95 f transiently inhibiting LDH during adoptive T cell-based immunotherapy, with an unanticipated cooper

96 This can complement existing T cell-directed immunotherapy, providing a promising app

97 eir therapeutic activity in a mouse model of T cell-mediated autoimmunity that mimics multiple sclero

98 alization are important determinants of CD8+ T cell-mediated efficacy against SIV.

99 sing macrophage-specific (CD14) but not CD4+ T cell-specific (CD3) antibodies, suggesting that M-trop

100 Is) after chimeric antigen receptor-modified T-cell (CAR-T-cell) therapy are limited.

101 Noninvasive strategies detecting T-cell activation would allow for early diagnosis and po

102 Loss of Yap in T cells results in enhanced T-cell activation, differentiation, and function, which

103 rtance of inappropriate NOTCH1 signalling in T-cell acute lymphoblastic leukaemia (T-ALL), and the in

104 rate that alphaCD3 alone induced substantial T-cell depletion, impacting both conventional T cells (T

105 r virus (EBV) is associated with a number of T-cell diseases, including some peripheral T-cell lympho

106 c predisposition, epidermal dysfunction, and T-cell driven inflammation.

107 e reasons, a cataloging and appraisal of the T-cell epitopes targeted in type 1 diabetes was complete

108 ssue and is related to the B7/CD28 family of T-cell immune checkpoint markers.

109 eat me" signal for macrophages) and PD-L1 (a T-cell inactivator) on their surface.

110 f T-cell diseases, including some peripheral T-cell lymphomas, hemophagocytic lymphohistiocytosis, an

111 not promote resurrection of exhausted CD4(+) T-cell memory in chronic infection.

112 with a substantial increase in the number of T-cell receptor (TCR) sequences and their cognate antige

113 ng through multiple receptors, including the T-cell receptor (TCR), co-receptors, and cytokine recept

114 sed to measure the frequency of EBV-specific T-cell responses between groups following stimulation wi

115 B*57:01-restricted, HIV epitope-specific CD8 T-cell responses showed beneficial functional patterns a

116 The Matrix-M1 adjuvant induced CD4+ T-cell responses that were biased toward a Th1 phenotype

117 Total memory T-cell responses were measured after anti-CD3 or vaccini

118 Ankara vector to induce HBV-specific B- and T-cell responses.

119 imeric antigen receptor-modified T-cell (CAR-T-cell) therapy are limited.

120 gamma-delta T cells (CD3(+)TCRgd(+)), CD8(+) T cells (CD3(+)CD8(+)CD161(+)PD1(+)), and memory B cells

121 Subsets of gamma-delta T cells (CD3(+)TCRgd(+)), CD8(+) T cells (CD3(+)CD8(+)CD

122 ed the phenotype characteristics of effector T cells (CD45RA(+), CD45RO-/lo, CD62L(-), CD27lo) with h

123 -cell depletion, impacting both conventional T cells (T(conv)) and T(regs), subsequently followed by

124 Nuclear Factor of Activated T cells 5 (NFAT5) is a transcription factor (TF) that me

125 ssing and priming for both CD4(+) and CD8(+) T cells and of the direct orchestration of their cross-t

126 Inhibition of NOX4 increased CD8(+) T cells and restored responsiveness to immune therapy, s

127 In conclusion, orchestrated T cells are able to regulate osteoclasts at the early st

128 mary, the current study suggests that CD4(+) T cells are critical for controlling acute-stage poliomy

129 Although CD4+ T cells are implicated in MS pathogenesis and have been

130 n contrast, increased frequency of EM CD8(+) T cells associated with reduced risk of graft failure.

131 developed in the absence of NOD-PerIg CD8(+) T cells but required CD4(+) T cells.

132 activity is linked to T-bet in Ag-expCD4(+) T cells but that reduction in mTOR activity may not dire

133 Activation of cytotoxic CD8(+) T cells by cross-priming DC contributes to exacerbation

134 Activation of gammadelta T cells can be elicited by butyrophilin and butyrophilin

135 T cells can sometimes acquire properties of a memory cel

136 comorbidity had larger numbers of activated T cells compared with patients who had fewer risk factor

137 ngs demonstrate that STAT1 signaling and CD8 T cells concomitantly act to mitigate MuPyV-encephalopat

138 cific cytokines produced by autoreactive CD4 T cells contribute to the pathogenesis of MS.

139 CD4+ T cells derived from individuals with latent Mtb infecti

140 transfer studies indicate that these memory T cells develop in a cell-intrinsic manner following thy

141 cells for metabolic resources often renders T cells dysfunctional.

142 r vaccinia virus (VV) stimulation to measure T cells elicited after childhood smallpox vaccination.

143 Here we show that primary mouse and human T cells engage in macropinocytosis that increases in mag

144 Gal-9(+) T cells exhibited the phenotype characteristics of effec

145 T cells from infant mice were predominantly immature, in

146 rectly or indirectly excluding effector CD8+ T cells from the tumor microenvironment.

147 The vaccine-induced T cells had a cytotoxic phenotype and were capable of tr

148 In those whose T cells had the capacity to respond, older patients with

149 CAR-T cells have shown encouraging activity against recurren

150 We were able to detect SARS-CoV-2-specific T cells in 10 of 10 COVID-19 patients with mild symptoms

151 m patients with MS points to a role for CD8+ T cells in disease pathogenesis.

152 ich are then eradicated by CD19-specific CAR-T cells in immunodeficient and immunocompetent mouse mod

153 ppress the activity of pancreas autoreactive T cells in newly hyperglycemic non-obese diabetic (NOD)

154 pands the proportion of proliferating CD8(+) T cells in the tumor with enhanced cytolytic potential a

155 We have examined the priming of naive CD4 T cells in vitro at fever temperatures, and we report no

156 However, whether T cells induced by one viral species cross-react with ot

157 ctional consequence of LEC priming of CD8(+) T cells is unknown.

158 can be found in the peripheral blood CD4(+) T cells of patients at all stages of HIV-1 infection.

159 s could benefit from such products, since no T cells recognizing any EBV-derived peptide in this comm

160 cDC1s in expansion of tumor-specific CD8(+) T cells remains unclear.

161 NINJA will enable studies of how T cells respond to defined neoantigens in the context of

162 Finally, deletion of Dot1L in T cells resulted in an impaired immune response.

163 Loss of Yap in T cells results in enhanced T-cell activation, different

164 iated with potent antiviral function: memory T cells secreted cytokines and expanded upon antigen re-

165 Using G9Calpha(-/-)CD8(+) T cells specific for proinsulin, we studied the mechanis

166 These drawbacks can be circumvented by CAR-T cells targeting tumour-specific driver gene mutations,

167 Human skin contains a population of memory T cells that supports tissue homeostasis and provides pr

168 In support, adoptive transfer of old CD4(+) T cells that were transfected with a lentiviral vector i

169 us and influences the susceptibility of CD4+ T cells to HIV-1 replication.

170 ranslates in vivo to an improved ability for T cells to infiltrate and repress tumors.

171 ved drug FTY720 increased the sensitivity of T cells to the cytokine interleukin-2 (IL-2) through a p

172 en-specific progeny of individual naive CD8+ T cells to the T effector (TEFF), T circulating memory (

173 In tracking the journey of T cells traversing from the thymus to the periphery and

174 However, Batf3 (-/-) T cells underwent increased apoptosis during contraction

175 The number of intravascular CD3+CD8+ T cells was influenced by CM status (CM+ > CM-, P = 0.00

176 T cells were sampled for up to 11 weeks to capture stead

177 Resting CMV-specific CD8 T cells were terminally differentiated and expressed hig

178 CD29 also marked T cells with cytotoxic gene expression from different ti

179 T(SCM) that acts as a reservoir for effector T cells with potent therapeutic characteristics.

180 ssible to defeat this mechanism and activate T cells with solution ligands by cross-linking pMHC or u

181 ffinity analyses of cytomegalovirus-specific T cells, and through the generation and in vivo monitori

182 th factor-beta receptor 2 (TGFBR2) in CD4(+) T cells, but not CD8(+) T cells, halts cancer progressio

183 e cancer- and virus-induced exhausted CD8(+) T cells, by enhancing the quality and survival of immune

184 2 (TGFBR2) in CD4(+) T cells, but not CD8(+) T cells, halts cancer progression as a result of tissue

185 )CD8(-)TCRalphabeta(+), double-negative (DN) T cells, in mouse secondary lymphoid organs.

186 PD-L1 pathway reinvigorates exhausted CD8(+) T cells, it fails to restore T cell repertoire diversity

187 response eQTLs (reQTLs) in myeloid cells and T cells, respectively.

188 ing chimeric antigen receptor (CAR)-modified T cells, to solid tumors requires combinatorial strategi

189 pitopes targeted by clusters of Mtb-specific T cells, we carried out a screen of 3,724 distinct prote

190 ell killing by freshly isolated human CD8(+) T cells, which represent a challenging but valuable mode

191 activity of chimeric antigen receptor (CAR) T cells.

192 rain has the unique ability to infect mature T cells.

193 rticular, ET were enriched in polyfunctional T cells.

194 tion of CD68/CD206 on MNPs and CD69/CD103 on T cells.

195 ursors of M2 macrophages, DCs and regulatory T cells.

196 ntigen-presenting cells (aAPCs) and reporter T cells.

197 gen-mediated signals to human Vgamma9Vdelta2 T cells.

198 plication-competent virus cultured from CD4+ T cells.

199 is in Fcgr2b(+), but not Fcgr2b(-/-), CD8(+) T cells.

200 ements drive gene expression in primary CD4+ T cells.

201 epertoire diversity of virus-specific CD8(+) T cells.

202 atory objectives included tracking of edited T cells.

203 NOD-PerIg CD8(+) T cells but required CD4(+) T cells.

204 opic cytokine produced predominantly by CD4+ T cells.

205 ons/glia for the brain data and granulocytes/T cells/B cells/monocytes for the blood data.

206 IV DNA isolated from peripheral blood CD4(+) T-cells at weeks 16 and 18 after randomisation.

207 with this hypothesis, mice transplanted with T-cells co-expressing NOTCH1 and NRARP develop leukemia

208 role of the different subsets of gammadelta T-cells detected in the skin in steady-state, psoriasis,

209 ing the risk of progression using naive CD4+ T-cells was predictive of progression along the whole IA

210 a look back, to see how science has and hasn't changed.

211 naive CD8+ T cells to the T effector (TEFF), T circulating memory (TCIRCM), and TRM pools by lineage-

212 , indicated CO(2) sequestration rates of 2-4 t CO(2) /ha, 1-5 years after a single application of bas

213 of T(regs) Despite robust depletion of host T(conv) and host T(regs), donor T(regs) failed to engraf

214 letion, impacting both conventional T cells (T(conv)) and T(regs), subsequently followed by more rapi

215 while conjugation of long-chain Vi generates T-dependent antigens, the conjugates also retain T-indep

216 analysis, non-negative matrix factorization, t-distributed stochastic neighbor embedding, and uniform

217 s in the field of mass spectrometry imaging: t-distributed stochastic neighborhood embedding (t-SNE)

218 esidual errors with a heavy-tailed Student's t-distribution to estimate a manifold that is robust to

219 Transposable elements (TEs) drive genome evolution and are a notable source of

220 geny of individual naive CD8+ T cells to the T effector (TEFF), T circulating memory (TCIRCM), and TR

221 l persistence is particularly significant in T(EM) cells.

222 tion of T(h)1 gene expression profiles in GC T(fh) cells.

223 Thioflavin T fluorimetry estimates rapid and near-stoichiometric co

224 Bcl6 is required for the development of T follicular helper cells and T follicular regulatory (T

225 development of T follicular helper cells and T follicular regulatory (Tfr) cells that regulate germin

226 rnatives on how to establish tau positivity (T+) for multiple tau-imaging tracers in order to reach a

227 ering-related genes, such as FLOWERING LOCUS T (FT), FLOWERING LOCUS C (FLC), AGAMOUS (AG) and APETAL

228 thymidine analogs along with the natural A, T, G and C bases during DNA synthesis, which allows for

229 apicoplast has a key role in heme biology in T. gondii and is important for both mitochondrial and ge

230 We have recently reported that the putative T. gondii CGL gene encodes a functional enzyme.

231 ns of GRA12 to the molecular pathogenesis of T. gondii infection were examined in vitro and in vivo.

232 vident in immunodeficient mice infected with T. gondii, as associated with high expression level (P <

233 The wG-T->G-T* tautomerization is predicted to be endoergic in

234 y epithelial activation, ILC2 expansion, and T(H) 2 differentiation.

235 Anti-T(H) 2 therapies have the potential to effectively reduc

236 ted GC B cell responses and the promotion of T(h)1 gene expression profiles in GC T(fh) cells.

237 T(H)17 cells are believed to orchestrate MS pathology, i

238 s mTOR activation in IL-17(+) gammadelta and T(H)17 cells.

239 ignificantly increased GM-CSF production and T(H)2 cell differentiation.

240 Follicular T helper (TFH) cells provide B-cell help and are crucial

241 ation resolution, including dampening of the T helper 1 response, alternative activation of macrophag

242 T-IFTA was similarly associated with decreased DC-GS.

243 s trained to distinguish between non-TEs and TEs in plants.

244 s aimed at predicting the clinical course of TS in individuals over development.

245 pendent antigens, the conjugates also retain T-independent properties, leading to detrimental effects

246 is shows that the thermophoretic mobility (D(T)) is thermophobic in sign and increases linearly with

247 ght, while the least (1.69%) was observed in T. kotschyanus grown under red-blue light.

248 Analysis of 13 T-lineage acute lymphoblastic leukemias identified a rec

249 use line is an ideal tool to study cytotoxic T lymphocyte biology and to optimize personalized immuno

250 e achieved with immunotherapy that relies on T lymphocyte-mediated recognition of tumor antigens.

251 Cytotoxic T lymphocytes (T) and natural killer cells are the main

252 s had significantly increased percentages of T lymphocytes and higher levels of a wide array of infla

253 indicated that CD20(+) B lymphocytes, CD8(+) T lymphocytes, and CD11c(+) cells are susceptible to IAV

254 activation thresholds (CCL1/3/4/5/XCL1); and T(M) chemokine profiles modulated by persisting viral Ag

255 8(+) and CD4(+)T(M) subsets; long-term CD8(+)T(M) maintenance is associated with a pronounced increas

256 es dominate the earliest stages of the CD8(+)T(M) recall response because of expeditious synthesis/se

257 tably, constitutive CCL5 expression by CD8(+)T(M) serves as a unique functional imprint of prior anti

258 ifies highly polyfunctional CD8(+) and CD4(+)T(M) subsets; long-term CD8(+)T(M) maintenance is associ

259 ng an anomalous peak in specific heat at low T, magnetic phase transitions, and no mixed valency), Yb

260 Mucosal-associated invariant T (MAIT) cells are important for immune responses agains

261 g memory, all animals performed a reinforced T-maze alternation task, then a more challenging version

262 The highest thymol (66%) was found in T. migricus exposed to blue light, while the least (1.69

263 nging efficacy studies were performed on a 1 T MRI scanner using a transgenic APP/PSEN1 mouse model o

264 three-dimensionally printed molds by using 3-T MRI with DR-CSI and were then sliced to create coregis

265 p leukemia later than mice transplanted with T-NOTCH1 cells.

266 Here, we report that the immobilization of TD-NTs in size-exclusive hydrogel resins simultaneously

267 ositive viral test with a cycle threshold (C(T) ) of <35 or seroconverted during the follow-up period

268 t up to two-thirds at risk to inherit LS don't participate.

269 of Tilia cordata Mill, T.x europaea L., and T. platyphyllos Scop.

270 whilst much is known about the existence of T-R conflicts, our understanding of the genetic and temp

271 ll, we find that sex-specific differences in T(reg) cells from VAT are determined by the tissue niche

272 bsequently followed by more rapid rebound of T(regs) Despite robust depletion of host T(conv) and hos

273 tion of host T(conv) and host T(regs), donor T(regs) failed to engraft even with interleukin-2 (IL-2)

274 te robust depletion of host T(conv) and host T(regs), donor T(regs) failed to engraft even with inter

275 ting both conventional T cells (T(conv)) and T(regs), subsequently followed by more rapid rebound of

276 We have previously shown that the ts residue signature of the Russian A/Leningrad/17/57 H2

277 Ex-T(RM) cells, former intestinal T(RM) cells that rejoined the circulating pool, heritabl

278 Ex-T(RM) cells, former intestinal T(RM) cells that rejoined

279 heightened capacity to redifferentiate into T(RM) cells.

280 rapy or craniotomy who underwent 1.5-T and 3-T same-plane T1-weighted MRI (in any order).

281 Ki leads to CD8(+) T cell reprogramming into T(SCM) that acts as a reservoir for effector T cells wit

282 we report a chromosome-scale assembly of the T. sinense genome.

283 stributed stochastic neighborhood embedding (t-SNE) and uniform manifold approximation and projection

284 ctor b (P-TEFb), composed of CDK9 and cyclin T, stimulates transcriptional elongation by RNA polymera

285 V using the MOLLI T1 mapping sequence at 1.5 T.Supplemental material is available for this article.(C

286 the time-specific associations were notable: t-tau 8 to 16 years, and Nf-L and GFAP 4 to 8 years prio

287 The wG-T->G-T* tautomerization is predicted to be endoergic in aqueo

288 Using high-resolution 7 T time-of-flight angiography we manually classified hipp

289 PI3Kdelta is a key regulator of regulatory T (Treg) cell function.

290 or the function of thymus-derived regulatory T (Treg) cells (ie, FOXP3), resulting in impaired Treg f

291 a(v)3.1 and Ca(v)3.2 and produced functional T-type VGCC currents when patch clamped.

292 regulation of 12-OPDA and JA in response to T. virens colonization results in ISR induction.

293 [(11)C]UCB-J V(T) was significantly lower in the frontal and anterior c

294 Using a temperature-sensitive allele (Sac1(ts)), we show that Sac1 is required for structural integ

295 A simple descriptor, mu/t, where mu and t are the octahedral and tolerance facto

296 erwent transpedal MR lymphangiography at 1.5 T with T1-weighted imaging after interstitial pedal of g

297 pared to previously reported pure Ti(3) C(2) T(x) films.

298 Here, it is reported on Ti(3) C(2) T(x) MXene microstrip transmission lines with low-energy

299 hould include flowers of Tilia cordata Mill, T.x europaea L., and T. platyphyllos Scop.

300 gas SCVF is estimated to reach about 75,000 t/y based on the existing inventory calculation; however