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1 -box riboswitch in complex with an uncharged tRNAGly.
2 cognate amino acids; GARS ligates glycine to tRNA(Gly).
3 ' region of the mRNA by binding of uncharged tRNA(Gly).
4 tion from thrombin digestion was afforded by tRNA(gly).
5 me and to be amplified by mutant variants of tRNA(Gly)(2) known to diminish bypassing efficiency.
6                           Mutant variants of tRNA(Gly)(2) that impair bypassing mediate stop-hopping
7 ubstantially impairing the ability of mutant tRNA(Gly)(2) variants to re-pair with the mRNA by sub-op
8  Mutations affecting ribosomal protein L9 or tRNA(Gly)(2), the tRNA that decodes GGA, alter the effic
9 onal slowdown is not attributable to altered tRNA(Gly) aminoacylation, and cannot be rescued by Droso
10 d from specific tRNA loci (e.g., the nuclear tRNA(Gly) and tRNA(Leu), the mitochondrial tRNA(Val) and
11 s of tRFs derived from tRNA(Glu), tRNA(Asp), tRNA(Gly), and tRNA(Tyr) that, upon induction, suppress
12 ecifier Sequence of the glyQS leader RNA and tRNA(Gly) anticodon to test the effect of all possible p
13 ressed by the insertion of a structured RNA (tRNA(Gly)) between fimE and the fim switch, indicating t
14                    We found that addition of tRNA(Gly) can promote antitermination as long as the tRN
15 tion by uncharged tRNA unless the leader RNA-tRNA(Gly) complexes contained the complete antiterminato
16                                              tRNA(Gly)-dependent antitermination of the Bacillus subt
17                                              tRNA(Gly)-dependent antitermination of the Bacillus subt
18 turase, and addition of Gly by DhpK in a Gly-tRNA(Gly)-dependent manner completes the in vitro biosyn
19  lies within the 5' region of the M. xanthus tRNA(Gly) gene.
20  strand exchange occurs and which overlaps a tRNAGly gene at attB.
21 r to one-half as many were found upstream of tRNA(Gly) genes in a hos2delta or set3delta mutant than
22 us Ty1 elements into the upstream regions of tRNA(Gly) genes was substantially decreased.
23 ation of the six mutant mispair sites across tRNA(Gly) in many organisms points to a fundamental stru
24          Structural mapping studies revealed tRNA(Gly)-induced protection in the glyQS leader RNA at
25                           A mimic of charged tRNA(Gly) inhibited antitermination by uncharged tRNA un
26 ic counterpart in charging the mitochondrial tRNA(Gly) isoacceptor, which carries a defective TpsiC h
27                           The consequent Gly-tRNA(Gly) 'misediting paradox' is resolved by EF-Tu in t
28 ealed that MuLV prefers to select tRNA(Pro), tRNA(Gly), or tRNA(Arg).
29 tion of a feature of the Mypoplasma mycoides tRNAGly responsible for non-discriminate decoding, a C a
30  identity elements into the Escherichia coli tRNA(Gly) scaffold endowed facile phosphoserylation acti
31 tablished conditions for specific binding of tRNA(Gly) to glyQS leader RNA generated by phage T7 RNA
32 expression of SR1 glycyl-tRNA synthetase and tRNA(Gly)UCA in Escherichia coli yields significant beta
33                      SR1 encodes a divergent tRNA(Gly)UCA with an opal-decoding anticodon.
34          SR1 glycyl-tRNA synthetase acylates tRNA(Gly)UCA with glycine in vitro with similar activity
35 o with similar activity compared with normal tRNA(Gly)UCC.
36                                   Changes in tRNA(Gly) upon binding to glyQS leader RNA were detected
37 ant G-U and A-C mispairs in Escherichia coli tRNA(Gly) using genetic and bioinformatic tools and show
38 ated glyQS T-box in complex with its cognate tRNAGly was derived based on the molecular envelope.
39 ia coli EF-Tu, whereas Ala-tRNA(Ala) and Gly-tRNA(Gly) were unaffected.
40 ochemical evidence for direct interaction of tRNA(Gly) with full-length in vitro transcribed glyQS le
41 damental structure-function signature within tRNA(Gly) with possible analogous missions in other RNAs

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