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1 nsive (systolic blood pressure=81 mm Hg) and tachycardic (117/min), with an average shock time (systo
2 volved in the centrally mediated pressor and tachycardic actions of gamma-MSH, which, likely, are med
3 tor antagonist, AP-5, inhibited the pressor, tachycardic and irritation responses elicited by intrath
4 bined, their ability to suppress BMI-induced tachycardic and pressor responses was additive.
5 ect the pressor (DeltaMAP = 12 +/- 2 mm Hg), tachycardic (DeltaHR = 48 +/- 7 bpm) or venoconstrictor
6 t dose-dependent (0.1-12.5 nmol) pressor and tachycardic effects, which are much more pronounced afte
7 both for symptom reduction and prevention of tachycardic-induced cardiomyopathy; restoration and main
8 e the utility of the optimized platform in a tachycardic model of arrhythmogenesis, an aspect of card
9 dmyocardial myocytes isolated from normal or tachycardic pacing-induced failing canine hearts.
10 2+ handling in myocytes isolated from canine tachycardic pacing-induced failing hearts.
11                                              Tachycardic pigs given furosemide had significant accele
12 ntagonist (CP-99,994) in vivo attenuated the tachycardic response to hindlimb pinch.
13  into the AP elicited an initial pressor and tachycardic response which was followed by a depressor a
14 ial and subsequently a sustained pressor and tachycardic response, also exhibited a transient interve
15 icroM each) into the AP elicited pressor and tachycardic responses in unanesthetized decerebrate as w
16 S AAV2nNOSshRNA significantly reduced reflex tachycardic responses to acute hypotension while not aff
17                              The pressor and tachycardic responses to ARCN stimulation at below norma
18 vel about 1 mm rostral to it and pressor and tachycardic responses were elicited from the chemorecept
19 o the RVLM elicited increases in MAP and HR; tachycardic responses were relatively inconsistent.
20 cts on RVLM neurons resulting in pressor and tachycardic responses, (2) these responses were mediated
21  i.e., the cold stimulus induced pressor and tachycardic responses, augmented glutamate, and attenuat
22 tion and confrontation evoke bradycardic and tachycardic responses, respectively.
23 uced an immediate hypotensive, hyperdynamic, tachycardic state with progressive lactic acidosis.

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