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1 nsive (systolic blood pressure=81 mm Hg) and tachycardic (117/min), with an average shock time (systo
2 volved in the centrally mediated pressor and tachycardic actions of gamma-MSH, which, likely, are med
3 tor antagonist, AP-5, inhibited the pressor, tachycardic and irritation responses elicited by intrath
5 ect the pressor (DeltaMAP = 12 +/- 2 mm Hg), tachycardic (DeltaHR = 48 +/- 7 bpm) or venoconstrictor
6 t dose-dependent (0.1-12.5 nmol) pressor and tachycardic effects, which are much more pronounced afte
7 both for symptom reduction and prevention of tachycardic-induced cardiomyopathy; restoration and main
8 e the utility of the optimized platform in a tachycardic model of arrhythmogenesis, an aspect of card
13 into the AP elicited an initial pressor and tachycardic response which was followed by a depressor a
14 ial and subsequently a sustained pressor and tachycardic response, also exhibited a transient interve
15 icroM each) into the AP elicited pressor and tachycardic responses in unanesthetized decerebrate as w
16 S AAV2nNOSshRNA significantly reduced reflex tachycardic responses to acute hypotension while not aff
18 vel about 1 mm rostral to it and pressor and tachycardic responses were elicited from the chemorecept
20 cts on RVLM neurons resulting in pressor and tachycardic responses, (2) these responses were mediated
21 i.e., the cold stimulus induced pressor and tachycardic responses, augmented glutamate, and attenuat
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