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1 .e., anxiety, tachycardia, hypertension, and tachypnea).
2 ted to an outside institution with fever and tachypnea.
3 8) were associated with faster resolution of tachypnea.
4 ed the firing of pre-I neurons, resulting in tachypnea.
5 g for ruling out pneumonia is the absence of tachypnea.
6 /= 1.1 joule/L, extubation proceeded despite tachypnea.
7 onspecific symptoms of cough, coryza, and/or tachypnea.
8                    At the time of admission, tachypnea (27% vs 60%; OR, 0.24; P = .031) and respirato
9 poxemia (45% vs 26%), crackles (69% vs 62%), tachypnea (85% vs 80%), or fever (20% vs 16%) and less l
10     Patients with more severe HF had greater tachypnea and a smaller tidal volume (VT) at a given exe
11 ed work of breathing are more important than tachypnea and auscultatory findings.
12 on were concerns over patient discomfort and tachypnea and concerns over hypercapnia, acidosis, and h
13                                              Tachypnea and hypoxemia resolved faster in older childre
14       We report an infant who presented with tachypnea and interstitial infiltrates on chest radiogra
15 a SUNDS victim who suffered sudden nocturnal tachypnea and lacked pathogenic variants in known arrhyt
16 ansgenic recipients of CD8(+) CTLs exhibited tachypnea and progressive weight loss, becoming moribund
17 r, increased locomotion, grooming, diarrhea, tachypnea and ptosis.
18 o had sepsis, defined as fever, tachycardia, tachypnea, and acute failure of at least one organ syste
19 is, respiratory distress syndrome, transient tachypnea, and apnea.
20 rogression, particularly the onset of fever, tachypnea, and bacteremia, should be useful for evaluati
21 ereafter, the infected rats exhibited fever, tachypnea, and hypertension that persisted for 24 to 36
22 zed by increased frequency due to periods of tachypnea, and increased apneas, as in RTT patients.
23 iary atresia developed fevers, hematochezia, tachypnea, and laboratory evidence of hepatitis and panc
24       Signs of illness included tachycardia, tachypnea, and leukopenia.
25 ed seconds, and were accompanied by moaning, tachypnea, and oxygen desaturation.
26                 Oxygen saturation of </=90%, tachypnea, and tachycardia were each associated with an
27                                              Tachypnea as a marker of respiratory distress is sensiti
28                                  Reliance on tachypnea as a preextubation trial failure criterion is
29           This study evaluates the impact of tachypnea as an indicator of ventilatory failure during
30 suggesting respiratory distress or transient tachypnea at lower catecholamine levels.
31 ection (pneumonia and cellulitis), response (tachypnea, bandemia, and tachycardia), and organ dysfunc
32  the ventral part of the lateral PAG induced tachypnea but inhibited pre-I cell firing, whereas stimu
33                                       Fever, tachypnea, cough, rhinorrhea, retractions of the chest w
34 G was normal and identical on each side, but tachypnea could not be elicited in the pre-BotC of SSP-S
35 developed an identical illness consisting of tachypnea, decreased activity, and hunched posture.
36 eexposure baseline (SAW(grp)) or exaggerated tachypnea during exercise.
37 onger duration of fever, higher morbidity of tachypnea/dyspnea, pleural effusion, diarrhea, hepatospl
38                           Cough, rhinorrhea, tachypnea, fever, abnormal breath sounds, and hypoxia we
39 The parents reported a history of persistent tachypnea, grunting, and episodic nonproductive cough wi
40 ach for systolic hypotension [</=100 mm Hg], tachypnea [&gt;/=22/min], or altered mentation).
41 anic-like responses, defined as tachycardia, tachypnea, hypertension, and increased anxiety as measur
42  dose-dependent blockade of the tachycardia, tachypnea, hypertension, and SI responses after lactate
43 ed acute lung injury had fever, tachycardia, tachypnea, hypotension, and prolonged hypoxemia compared
44  the restrictive changes leading to exercise tachypnea in HF patients.
45 tibiotics may be unnecessary for physiologic tachypnea in otherwise well newborns.
46 ory response syndrome criteria (tachycardia, tachypnea, leukocytosis, and fever) in surgical ICU pati
47                                   Persistent tachypnea of infancy (PTI) is a specific clinical entity
48 me (OR = 1.09; 95% CI, 1.01-1.19), transient tachypnea of the newborn (OR = 1.10; 95% CI, 1.02-1.19),
49 s syndrome, apnea, sepsis, anemia, transient tachypnea of the newborn, infective pneumonia, asphyxia,
50  Severe respiratory complications, transient tachypnea of the newborn, surfactant use, and bronchopul
51                 In intact rats, DLH produced tachypnea only when injected into the pre-Botzinger comp
52  recording the presence of terminal illness, tachypnea or hypoxemia, septic shock, platelet count <15
53 io, 6.1; 95% confidence interval, 3.6-10.2), tachypnea or hypoxia (2.7, 1.6-4.3), septic shock (2.7,
54 , absence of a febrile response (P = 0.003), tachypnea (OR 1.9, P = 0.001), leukopenia or leukocytosi
55  hypotension (OR = 4.8; 95% CI, 2.8 to 8.3), tachypnea (OR = 2.9; 95% CI, 1.7 to 4.9), diabetes melli
56 ein was associated with a longer duration of tachypnea (P = 0.044).
57 rvable differences in the physical response (tachypnea, piloerection, lethargy, etc), or intra-abdomi
58 entilator inflation time (TI,vent) can cause tachypnea, probably as a response to lung inflation.
59 hypothesis that a decrease in TI,vent causes tachypnea, prolongation of exhalation, and a decrease in
60 hese, 97 were successfully extubated despite tachypnea ranging from 32 to 56 breaths/min, when combin
61 sitivity, 80%-92%; specificity, 47%-54%) and tachypnea (respiratory rate >40 breaths/min; LR, 1.5 [95
62                                              Tachypnea, respiratory distress, deep breathing, shock,
63 e blood culture or the combination of fever, tachypnea, tachycardia, clinically suspected infection,
64                    Common physical signs are tachypnea, tachycardia, rales, and cyanosis.
65         The dolphin showed clinical signs of tachypnea, transient dyspnea, and mild tachycardia and d
66                 Some of the stimulus for the tachypnea was possibly due to increased imposed work of
67                                           If tachypnea was the reason for failure, work of breathing
68  reduce TI,vent in patients with COPD caused tachypnea, yet prolonged the time for exhalation with co

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