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1 arietal cells then predominantly to daughter tapetal cells.
2 ollen coat and premature degeneration of the tapetal cells.
3 lationship between the development of ml and tapetal cells.
4 densely stained cytoplasm typical of normal tapetal cells.
5 within the large cytoplasmic lipid bodies of tapetal cells.
6 ) are contributed by surrounding sporophytic tapetal cells.
7 localized to endoplasmic reticulum of anther tapetal cells.
8 me genes may be expressed in the sporophytic tapetal cells and in gametophytic tissues, they are regu
9 Transgenic plants exhibited GUS activity in tapetal cells and pollen of the developing anthers indic
10 that produces excess microsporocytes, lacks tapetal cells, and abnormally maintains middle layer cel
12 ly from the mitochondria into the cytosol of tapetal cells before the gross morphological changes ass
18 to suppress trans-differentiation of somatic tapetal cells into meiocytes, we find that mac1 anthers
22 ouble mutant anthers lack development of the tapetal cell layer, which accounts for the microspore ab
24 ese vacuolar inclusions were not observed in tapetal cells of double mutants of abcg26 and genes enco
27 e differentiation of the microsporocytes and tapetal cells, suggesting that EMS1 mediates signals tha
28 morphology beginning at anther stage 4, with tapetal cells that have excess and/or enlarged vacuoles
29 ail to elongate, and there are fewer, larger tapetal cells that retain, rather than secrete, their co
30 ere ABCG26-exported polyketides traffic from tapetal cells to form the sporopollenin backbone, in coo
31 nsport and assembly of exine components from tapetal cells to microspores in the intact anthers of Ar
32 asm in sunflower causes premature PCD of the tapetal cells, which then extends to other anther tissue
33 ned by biphasic protein expression in anther tapetal cells, with an initial peak around pollen meiosi
34 n, accumulated large fluorescent vacuoles in tapetal cells, with corresponding loss of fluorescence o
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