ライフサイエンスコーパス: tarda

1 a, glomerulonephritis, and porphyria cutanea tarda.

2 commoner sporadic form of porphyria cutanea tarda.

3 sis, we isolated a noncytotoxic strain of E. tarda.

4 bility factor for sporadic porphyria cutanea tarda.

5 gh the skin lesions due to porphyria cutanea tarda.

6 ronic viral hepatitis, and porphyria cutanea tarda.

7 and a putative TonB homolog in Edwardsiella tarda.

8 cohol abuse, hepatitis, or porphyria cutanea tarda.

9 of reactive oxygen species than wild-type E. tarda.

10 (19 patients) and sporadic porphyria cutanea tarda (65 patients).

11 eria historically classified as Edwardsiella tarda actually represent three genetically distinct yet

12 ics of inherited disorders porphyria cutanea tarda and hereditary coproporphyria, respectively.

13 he dermatologic lesions in porphyria cutanea tarda, and diagnose and treat the erythropoietic porphyr

14 defined as typical motile fish-pathogenic E. tarda are synonymous with Edwardsiella piscicida, while

15 nic E. tarda isolates are consistent with E. tarda as it is currently defined.

16 pping to this region, including OFD1 and SED tarda, as well as Aicardi syndrome and Goltz syndrome.

17 ant to acquired (sporadic) porphyria cutanea tarda, because humans show greater than 60-fold genetic

18 een the HLA-A3 antigen and porphyria cutanea tarda but not by others.

19 ring and photosensitivity: porphyria cutanea tarda; congenital erythropoietic porphyria; hepatoerythr

20 A cosmid bearing 40 kb of E. tarda DNA, including wild-type copies of the E. tarda ho

21 E. tarda escapes from the endocytic vacuole within minutes

22 Familial porphyria cutanea tarda (f-PCT) results from the half-normal activity of u

23 ah pedigrees with familial porphyria cutanea tarda (F-PCT).

24 In this study, we report that the T3SS of E. tarda facilitates its survival and replication in murine

25 lement, suggesting that they arose in the E. tarda genome by horizontal transfer.

26 Porphyria cutanea tarda has been described in adult hemodialysis patients.

27 The type III secretion system (T3SS) of E. tarda has been identified as a key virulence factor that

28 ty is due to the previously characterized E. tarda hemolysin activity.

29 da DNA, including wild-type copies of the E. tarda homologs of the transporter-activator protein and

30 A case of porphyria cutanea tarda in an adult ESRD patient treated with continuous a

31 A proper diagnosis of porphyria cutanea tarda in hemodialysis patients requires fractionation of

32 The pathogenesis of porphyria cutanea tarda in this population is thought to be related to the

33 41 patients with sporadic porphyria cutanea tarda, in whom the frequency of microsatellite alleles t

34 genotypes in each type of porphyria cutanea tarda indicated that C282Y homozygosity is an important

35 present late in life with porphyria cutanea tarda, indicating that not all homozygotes present clini

36 We propose that the host controls E. tarda infection partially by detecting FliC translocated

37 rine bone marrow-derived macrophages, and E. tarda infection triggers pyroptosis of infected macropha

38 Porphyria cutanea tarda is a disorder of heme biosynthesis resulting from

39 Edwardsiella tarda is a Gram-negative enteric pathogen that causes he

40 Edwardsiella tarda is a Gram-negative enteric pathogen that causes he

41 Porphyria cutanea tarda is a liver disease characterized by elevated hepat

42 Sporadic porphyria cutanea tarda is a skin disease associated with hepatic siderosi

43 Porphyria cutanea tarda is a skin disease caused by photosensitization by

44 Edwardsiella tarda is an enteric pathogen that causes diarrhea, wound

45 wardsiella anguillarum Fish-nonpathogenic E. tarda isolates are consistent with E. tarda as it is cur

46 while atypical nonmotile fish-pathogenic E. tarda isolates are equivalent to Edwardsiella anguillaru

47 the frequency in sporadic porphyria cutanea tarda of mutations (Cys282Tyr, His63Asp) in a novel MHC

48 mbers of 530 patients with porphyria cutanea tarda (PCT) and 296 with acute intermittent porphyria (A

49 n is known to enhance both porphyria cutanea tarda (PCT) and experimental uroporphyria.

50 Two major risk factors for porphyria cutanea tarda (PCT) are alcohol consumption and homozygosity for

51 ors for the development of porphyria cutanea tarda (PCT) are alcohol consumption and homozygosity for

52 me, but not all countries, porphyria cutanea tarda (PCT) has been associated with chronic infection w

53 overload that is common in porphyria cutanea tarda (PCT) is uncertain.

54 ted in the pathogenesis of porphyria cutanea tarda (PCT), a disorder characterized by a photosensitiv

55 Porphyria cutanea tarda (PCT), a liver disease with skin lesions caused by

56 Porphyria cutanea tarda (PCT), the most common form of porphyria in humans

57 one-third of patients with porphyria cutanea tarda (PCT), the most common porphyria in humans, inheri

58 is common in patients with porphyria cutanea tarda (PCT).

59 hepatic manifestations of porphyria cutanea tarda (PCT).

60 rm of porphyria in humans, porphyria cutanea tarda (PCT).

61 ittent porphyria (AIP) and porphyria cutanea tarda (PCT).

62 orders involving the skin (porphyria cutanea tarda [PCT], vitiligo, and lichen planus); renal (membra

63 Deletion of the flagellin gene fliC of E. tarda results in decreased cytotoxicity for infected mac

64 lanus, Sjogren's syndrome, porphyria cutanea tarda, rheumatoid-like arthritis, or depression.

65 Spondyloepiphyseal dysplasia tarda (SEDL) is a genetically heterogeneous disorder cha

66 dlin gene cause spondyloepiphyseal dysplasia tarda (SEDT), a rare X-linked chondrodysplasia.

67 high sequence similarity with Orf29/30 of E. tarda strain EIB202, and is renamed EseJ.

68 c bacterial pathogens including Edwardsiella tarda, Streptococcus iniae, Yersinia ruckeri, Flavobacte

69 both familial and sporadic porphyria cutanea tarda, the effect being more marked in familial porphyri

70 the most common porphyria, porphyria cutanea tarda, the identification of an X-linked form of erythro

71 s leukemia and concomitant porphyria cutanea tarda was admitted to the hospital for consolidation che

72 Familial porphyria cutanea tarda was identified by mutational analysis of the uropo

73 ng more marked in familial porphyria cutanea tarda where anticipation was demonstrated in family stud

74 Efforts to study the interactions of E. tarda with tissue culture cells are complicated by the p

75 le to follow the entry and replication of E. tarda within tissue culture host cells.