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1 take, induce pica, and produce a conditioned taste aversion.
2 d feeding and the formation of a conditioned taste aversion.
3 he development of a LiCl-induced conditioned taste aversion.
4 erone secretion, body weight, or conditioned taste aversion.
5 "good" to "bad" as occurs during conditioned taste aversion.
6 ince they successfully expressed conditioned taste aversion.
7 ns had no impact on LiCl-induced conditioned taste aversion.
8 d intake in rats without causing conditioned taste aversion.
9 oiding taste stimuli following a conditioned taste aversion.
10 nt used in laboratory studies of conditioned taste aversion.
11 pect to the neural substrates of conditioned taste aversion.
12 n of latent inhibition (LI) of a conditioned taste aversion.
13 kness as icv TTR did not cause a conditioned taste aversion.
14 choice behavior when it was guided by innate taste aversion.
15 sponses including development of conditioned taste aversion.
16 ) aversive effects as indexed by conditioned taste aversion.
17 nduces pica, and produces robust conditioned taste aversions.
18 dence that drugs of abuse induce conditioned taste aversions.
21 , nociception, and extinction of conditioned taste aversion and an appetitive instrumental response.
23 t genetic correlations were observed between taste aversion and ethanol-related behaviors measured in
27 ocked behavioral expression of a conditioned taste aversion and this was evident not only when lesion
28 conditioned fear responses and a conditioned taste aversion as well as enhanced performance in an att
31 atability, the current results indicate that taste aversions based on either lithium or activity redu
33 f paraquat at a dose sufficient to condition taste aversion, but produce no other signs of overt toxi
35 d that amygdala was absolutely necessary for taste aversions conditioned with the intraoral method bu
39 was then accomplished in 1 group by inducing taste aversion; controls received either saline or unpai
43 field is sufficient to induce a conditioned taste aversion (CTA) and induce brainstem expression of
44 g sodium depletion, and a normal conditioned taste aversion (CTA) for alanine when paired with lithiu
49 ucleus (PBN) failed to acquire a conditioned taste aversion (CTA) induced by lithium chloride (LiCl)
58 s widely regarded as integral to conditioned taste aversion (CTA) retention, a link that has been pri
65 nist, was infused into IC before conditioned taste aversion (CTA) training with a familiar taste.
66 correlate of the expression of a conditioned taste aversion (CTA) when conditioning occurs using tast
67 a herbicide capable of eliciting conditioned taste aversion (CTA), a behavioral response characterist
68 readily acquired a LiCl-induced conditioned taste aversion (CTA), the suppressive effects of sucrose
85 thium chloride (referred to as a conditioned taste aversion, CTA); (2) access to a very palatable con
86 f permanent forebrain lesions on conditioned taste aversions (CTAs) and conditioned odor aversions (C
87 ) lesions attenuate LiCl-induced conditioned taste aversions (CTAs) by disruption of information abou
88 the acquisition and retention of conditioned taste aversions (CTAs) in rodents, but large lesions in
90 da tympani nerves (CTX) received conditioned taste aversions (CTAs) to the free fatty acids (FFAs), l
92 havioral experiments testing for conditioned taste aversion did not confirm that SEB challenge promot
93 e acquisition of ethanol-induced conditioned taste aversion, ethanol-induced conditioned place prefer
94 ncluding active place avoidance, conditioned taste aversion, fear conditioning and spatial learning.
97 rin taste developed a persistent conditioned taste aversion in both preference and taste reactivity t
100 parabrachial nucleus (PBN) failed to learn a taste aversion induced by lithium chloride (LiCl) toxico
102 support the hypothesis that ethanol-induced taste aversion is mediated by the drug's rewarding prope
103 e GI tract suggests a new account of delayed taste aversion learning as well as learning about the po
105 the light CS in the first training phase or taste aversion learning in the second training phase.
108 H3, c-Fos induction, and amygdalar-dependent taste aversion learning is constrained by endogenous his
111 rained first to reject it (i.e., conditioned taste aversion learning) and then to enjoy it again (i.e
112 itioning represents a molecular correlate of taste aversion learning, i.e. the formerly neutral CS no
113 details of the neural circuitry involved in taste aversion learning, including its anatomical distri
118 thdrawal with low drinking, high conditioned taste aversion, low tolerance to EtOH-induced hypothermi
120 egulates ingestive behavior without inducing taste aversion may open the possibility of a therapeutic
122 lso attenuated the extinction of established taste aversion memory without altering the initial assoc
126 l training procedures and either conditioned taste aversion or satiation devaluation procedures.
127 ubstrates of LI as assessed in a conditioned taste aversion paradigm by comparing regional c-Fos acti
132 elay in extinguishing an ethanol-conditioned taste aversion, suggesting that they drink less ethanol
134 xperiment 4) or consumption on a conditioned taste aversion test that does not elicit antipredator re
137 ransection (CTX) acquired a LiCl-conditioned taste aversion to 0.1 M NaCl at the same rate as control
139 lued the food unconditioned stimulus (US) by taste aversion to differentiate stimulus-stimulus(CS-US)
140 tration of GLP-1 did not cause a conditioned taste aversion to saccharin, suggesting that the anorexi
141 ate was confirmed in rats with a conditioned taste aversion to sucrose: after paired exposure to sucr
144 acquisition and expression of a conditioned taste aversion was assessed using two different conditio
146 l can induce memory given that a conditioned taste aversion was obtained for a novel taste, presented
149 y pairing it with lithium chloride (acquired taste aversion), while the other distinctive flavor was
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