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1 cts associated with the lack of a functional tat gene.
2 sity for the sicker child, especially in the tat gene.
3 he wild-type Tat gene or the attenuated H13L Tat gene.
4 lin M (IgM) and human immunodeficiency virus tat genes.
5 l clones transfected with either full-length tat gene (101 amino acids), or in control cells containi
6 human immunodeficiency virus type 1 (HIV-1) Tat gene, a potent transactivator of viral and cellular
7 tion of a T cell line, H9 cells with the HIV-tat gene also resulted in an activation of JNK that was
8 licing of a substrate derived from the HIV-1 tat gene and have shown that hnRNP H family members are
11 now demonstrate that the requirement for the tat gene can be offset by placing constitutive promoters
12 se transcription seen with HIV-1 lacking the tat gene could not be complemented when the target cells
18 determined that three of the four identified tat genes in Haloferax volcanii are essential for viabil
19 ing transfection of wild-type but not mutant tat genes into cell lines producing HIV-1 lacking tat, t
21 taken to investigate the effect of the HIV-1 tat gene on the expression of inducible nitric-oxide syn
23 uman immunodeficiency virus, type 1 (HIV-1), tat gene stimulates transcriptional activity of the HRES
24 were obtained so that the abilities of these tat genes to stimulate HIV-1 gene expression and reverse
25 ation-defective herpes simplex virus (HSV)-1 tat gene transfer vector, dSTat, was used to transiently
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