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1 eliorated by treatment with the ER chaperone tauroursodeoxycholic acid.
2 oxybutyrate, or the anti-apoptotic bile acid tauroursodeoxycholic acid.
4 ion with increasing amounts of the bile salt tauroursodeoxycholic acid, Abcg5 became fully rate-limit
6 ER) stress was inhibited by pharmacological (tauroursodeoxycholic acid) and genetic (overexpression o
7 ical chaperones, sodium 4-phenylbutyrate and tauroursodeoxycholic acid, both with chaperone propertie
9 n contrast to taurochenodeoxycholic acid and tauroursodeoxycholic acid, GCDCA induced apoptosis in a
11 ugh induction of microRNA (miR)-34a, whereas tauroursodeoxycholic acid induced SIRT1 expression witho
12 nt of PCOS mice with an ER stress inhibitor, tauroursodeoxycholic acid or BGP-15, decreased interstit
13 posed to glycochenodeoxycholic acid (GCDCA), tauroursodeoxycholic acid, taurochenodeoxycholic acid, a
14 n vivo is converted to its taurine conjugate tauroursodeoxycholic acid (TUDC), is a mainstay for the
15 olic acid (TCA), glycocholic acid (GCA), and tauroursodeoxycholic acid (TUDCA) all activated ERK1/2 i
19 ed by co-infusion of the ER stress inhibitor tauroursodeoxycholic acid (TUDCA) into the lateral cereb
20 UDCA is conjugated to taurine in vivo, and tauroursodeoxycholic acid (TUDCA) is a potent hepatocell
21 hat acute inhibition of brain ER stress with tauroursodeoxycholic acid (TUDCA) partially reversed obe
22 virus, we found that the molecular chaperone tauroursodeoxycholic acid (TUDCA) significantly inhibits
24 es to insulin resistance, and treatment with tauroursodeoxycholic acid (TUDCA), a bile acid derivativ
25 is, we investigated the protective effect of tauroursodeoxycholic acid (TUDCA), a bile acid, on ER st
27 have previously reported that treatment with tauroursodeoxycholic acid (TUDCA), a hydrophilic bile ac
28 odel of progressive DN and treated mice with tauroursodeoxycholic acid (TUDCA), a specific inhibitor
31 ne the efficacy of an ER chemical chaperone, tauroursodeoxycholic acid (TUDCA), in preserving cones i
33 mical chaperones sodium 4-phenylbutyrate and tauroursodeoxycholic acid were found to reduce tunicamyc
34 ss signaling, like 4-phenylbutyrate (PBA) or tauroursodeoxycholic acid, will inhibit the disruption o
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