ライフサイエンスコーパス: telophase

1 d at the spindle midzone during anaphase and telophase.

2 ganization of SC35 is restored subsequent to telophase.

3 d in the zone of microtubule overlap late in telophase.

4 the proper spacing of daughter nuclei during telophase.

5 hase transition and closed 90 s later during telophase.

6 concentrates near the cleavage furrow during telophase.

7 ther, robust silencing is not observed until telophase.

8 coincident with nuclear envelope assembly in telophase.

9 osophila embryos, actin caps assemble during telophase.

10 d to kinetochores from late prophase to late telophase.

11 ntry of the proteins into the nucleus during telophase.

12 eentry into the newly formed nucleus in late telophase.

13 the cleavage furrow during prophase through telophase.

14 ase, and localizes to the spindle midbody at telophase.

15 ly the phragmoplast during late anaphase and telophase.

16 es with midzone microtubules in anaphase and telophase.

17 nding nucleoplasm before leaving in anaphase/telophase.

18 nd to the cleavage furrow and midbody during telophase.

19 t the onset of M-phase and reassemble during telophase.

20 ng prometaphase and back to the chromatin in telophase.

21 re most likely carried through anaphase into telophase.

22 with the cleavage furrow during anaphase and telophase.

23 hores in M phase cells from late prophase to telophase.

24 es gradually diminishes, and is gone by late telophase.

25 were significantly longer than wild type at telophase.

26 d foci (NDF) between early anaphase and late telophase.

27 midzone and the midbody during anaphase and telophase.

28 y region of the spindle in late anaphase and telophase.

29 as cells progress through anaphase and begin telophase.

30 nd remains centromere associated until after telophase.

31 usters in the period from metaphase to early telophase.

32 at the midzone of these same spindles during telophase.

33 itosis, is relocalized to the midbody during telophase.

34 and clustered near the poles in anaphase and telophase.

35 he volume of the equator during anaphase and telophase.

36 iled abscission of the cleavage furrow after telophase.

37 th by transiently elongating during anaphase/telophase.

38 rometaphase, but not metaphase, anaphase and telophase.

39 cortical dynein, followed by a reduction in telophase.

40 metaphase and translocated to the midbody at telophase.

41 r entry into prophase and that it resumes in telophase.

42 metaphase, and then quickly disappears after telophase.

43 ion zone that forms between sister-asters in telophase.

44 process of chromosome decondensation at late telophase.

45 re-associates with chromatin during anaphase/telophase.

46 se and remains excluded from DNA until early telophase.

47 res in early mitosis and near the spindle in telophase.

48 ) breaks down at prophase and reassembles at telophase.

49 e, peaks at metaphase, and decreases through telophase.

50 zone during anaphase and the mid-body during telophase.

51 fects at metaphase, but these are rescued by telophase.

52 aberrant microtubule bridges during anaphase/telophase.

53 s its kinase activity from metaphase through telophase.

54 with DNA replication, and dissociates by the telophase.

55 es in anaphase, and chromatin bridges during telophase.

56 ondensation was prematurely lost in anaphase/telophase.

57 c spot that persists until the completion of telophase.

58 d transiently released in early anaphase and telophase.

59 pindle during anaphase and to the midzone at telophase.

60 ed at the midline during late anaphase/early telophase.

61 ome earlier and accumulated gradually during telophase.

62 in the cancer cells during meta-, ana-, and telophases.

63 At the end of telophase, a portion of Cdc15p is located at the mother-

64 During telophase, a significant amount of Plk3 was also detecte

65 At telophase, active MAP kinase localized at the midbody.

66 rosomes and spindle poles from interphase to telophase and at the midbody during cytokinesis.

67 We find that CID assembly initiates at late telophase and continues during G1 phase in somatic tissu

68 toplasm and at the spindle poles, and during telophase and cytokinesis stimulated PSKs are present in

69 During telophase and cytokinesis, many Golgi stacks redistribut

70 bidopsis and rice cells undergoing anaphase, telophase and cytokinesis.

71 s detected weakly on midbody microtubules at telophase and cytokinesis.

72 at the contractile ring and mid-body during telophase and cytokinesis.

73 prophase, reassociating again at the end of telophase and cytokinesis.

74 ndle microtubules, where it remained through telophase and cytokinesis.

75 in cell cycle stages other than anaphase and telophase and Dbf2 kinase was prematurely active during

76 a lesser degree, Mcm2 onto chromatin during telophase and early G1 when Mcm2-7 are normally recruite

77 igh DII levels were observed in cells during telophase and early G1, suggesting that low auxin signal

78 xpansion during chromosome decondensation in telophase and early G1.

79 it fusome material to spindle midbodies near telophase and for normal fusome organization.

80 In telophase and G1 unbudded cells, no localization was obs

81 ar bodies (PNBs) appeared in nuclei in early telophase and gradually disappeared as nucleoli formed,

82 shed staining of anaphase and no staining of telophase and interphase centrosomes.

83 ained high near the equatorial plane through telophase and into cytokinesis, whereas the phosphorylat

84 re, we show that FIP3 binds to Cyk-4 at late telophase and that centralspindlin may be required for F

85 re both localized on midbody microtubules at telophase, and also interacted with each other during mi

86 cleavage furrow and midbody during anaphase, telophase, and cytokinesis, implicating a role in the co

87 ase-anaphase transition, impede anaphase and telophase, and impair a cell's ability to arrest in G1 o

88 tubules in interphase and the midbody during telophase, and its protein levels decrease as cells exit

89 s and spindle poles during metaphase through telophase, and partially co-localized with chromatin dur

90 ation of Runx foci is completely restored in telophase, and Runx proteins are equally partitioned int

91 ated Exocyst and ESCRT machinery during late telophase, and therefore that these two distinct facets

92 cm proteins with chromatin took place during telophase, approximately 30 min after the destruction of

93 is believed to be the result of a prolonged telophase arrest that has been recently identified in RN

94 llowing the change in centromere position in telophase-arrested cells upon depolymerization and subse

95 ndle during mitosis, in perichromatin during telophase, as well as in the midbody during cytokinesis.

96 cdc5-1 mutants arrest at telophase at the nonpermissive temperature due to the fa

97 ific displacement of H2A.Z from chromatin in telophase-blocked cells, regardless of the silencing sta

98 During telophase both isoforms colocalized to the contractile r

99 rest in interphase, prophase, metaphase, and telophase but not anaphase.

100 phase chromosomes and bind to chromosomes in telophase but not in metaphase.

101 Cells blocked in telophase (but not at metaphase) are also able to establ

102 ated suppression of LATS1 or MOB1A prolonged telophase, but had no effect on the length of the earlie

103 s and appeared in prenucleolar bodies during telophase, but it did not colocalize with p80-coilin in

104 e regulated, peaking in metaphase-anaphase B/telophase, but its function remains unknown.

105 Transcription is known to restart in bulk by telophase, but whether de novo transcription at the mito

106 interactions are not established until after telophase, by which time the nuclear envelope has reasse

107 ent in unbudded G1 cells, as well as in late telophase cells after spindle disassembly.

108 s of prolonged mitosis, we isolated anaphase-telophase cells that were just finishing a mitosis of no

109 centromere protein (INCENP) in anaphase and telophase cells.

110 explaining how cohesin can be reloaded onto telophase chromatin in the absence of securin and cyclin

111 gous chromosomes in paired late-anaphase and telophase chromosomal masses were highly correlated.

112 This telophase chromosome movement suggests that the surface

113 ked chromosome decondensation, and inhibited telophase chromosome movement.

114 t pBAF is associated with the core region of telophase chromosomes.

115 cytoplasm, partition stochastically, and in telophase coalesce to generate a functionally and struct

116 us and colocalize again with the DNA in late telophase, concomitantly with the appearance of the nucl

117 erine 19 phosphorylation during anaphase and telophase, consistent with an activating phosphorylation

118 nding protein 1 (53BP1) are absent until the telophase/cytokinesis stage.

119 suppressed in mitotic mammalian cells until telophase/cytokinesis.

120 ates the inclusion of lagging acentrics into telophase daughter nuclei.

121 e-segregating acentric chromosomes enter the telophase daughter nucleus.

122 Second, in telophase, decondensing chromosomes often moved rapidly

123 bility, but imp1delta mutant cells exhibit a telophase delay and mild temperature-sensitive lethality

124 tribution of midzone microtubule bundles and Telophase Disc 60 protein (TD60) rather than the positio

125 activation in vitro requires two cofactors, telophase disc-60kD (TD-60) and microtubules.

126 STB association lasts from G1/S through late telophase during the cell cycle.

127 aughter nuclei and the nucleolus during late telophase/early G1.

128 vels in cortices prepared from late anaphase/telophase embryos.

129 r RENT (regulator of nucleolar silencing and telophase exit) complex at the rDNA region.

130 e RENT (regulator of nucleolar silencing and telophase exit) silencing complex, and Fob1, which recru

131 d RENT (regulator of nucleolar silencing and telophase exit).

132 d RENT (regulator of nucleolar silencing and telophase exit).

133 d RENT (regulator of nucleolar silencing and telophase exit).

134 kinase (DDK) accumulates at kinetochores in telophase, facilitated by the Ctf19 kinetochore complex.

135 of microtubule dynamics, and no anaphase or telophase figures were observed.

136 at successfully proceed through anaphase and telophase, forming two daughter nuclei separated by a mi

137 ansition from early to late anaphase, and by telophase FP-PP1gamma also accumulates at the cleavage f

138 in "anaphase," and bundling into arrays in "telophase." Furrow induction usually occurs at multisite

139 n kinase that plays an important role in the telophase/G1 transition.

140 the role of DBF2 and MOB1 in controlling the telophase/G1 transition.

141 to abnormally high AIR-2 levels at the late telophase/G1 transition.

142 These factors localize to centromeres in telophase/G1, when new CENP-A chromatin is assembled.

143 In telophase, GM130 is dephosphorylated as the Golgi fragme

144 During telophase, Golgi cisternae are regenerated and stacked f

145 Cells in metaphase and telophase have no detectable focus.

146 Around telophase, Hof1p is phosphorylated and the double rings

147 In telophase, hRif1 localized to chromosomes, and in interp

148 servable defect occurs in microsporocytes at telophase I, where some chromosomes are scattered throug

149 he formation of radial microtubule arrays at telophase II and consequently leads to defects in postme

150 vated in vivo oocytes were enucleated at the telophase II stage, electrofused with donor somatic cell

151 disrupts the radial microtubule system after telophase II, and affects the proper establishment of nu

152 le as it transits into anaphase II and later telophase II, becoming associated with the midzone micro

153 was undetectable from late prophase I until telophase II.

154 progressed to pronuclear, MIII, and anaphase/telophase III stages.

155 se, whereas nucleation remained high through telophase, implying the presence of additional regulator

156 slow cleavage-furrow ingression during early telophase in dividing spermatocytes.

157 while it was concentrated at the midbody in telophase in meiotic oocytes.

158 the beginning of mitosis and reforms at late telophase in the daughter nuclei.

159 ng early mitosis and defective reassembly at telophase, increased formation of multiple spindle poles

160 specifically in the cleavage furrow late in telophase independent of contractile ring constriction.

161 rapidly and irreversibly disassemble during telophase is less clear.

162 ules show strong KRIT1 staining and, in late telophase, KRIT1 stains the midbody remnant most strongl

163 se stable binding of Mis18 to centromeres in telophase licenses them for CENP-A deposition.

164 y, An-Mad1 and An-Mlp1 redistribute from the telophase matrix and associate with segregated kinetocho

165 ssociation of ECT2 from the mid-body at late telophase may be required for the recruitment of FIP3 an

166 We show that, in telophase, MCM2 and MCM4 maintain transient interactions

167 mammalian pre-RC assembly takes place during telophase, mediated by post-translational modifications

168 dles and form distinct bands associated with telophase midbodies.

169 , cytokinesis was oriented transverse to the telophase mitotic array and was less well aligned with t

170 At telophase, MPP10 was found in cellular structures that r

171 At anaphase and telophase, myosin II moves to the cleavage furrow and ap

172 n regulating anaphase spindle elongation and telophase nuclear positioning via inhibition of Klp2, a

173 Telophase nuclei also played a predominant role in posit

174 Microtubules from the telophase nuclei interdigitated throughout the plane of

175 The reentry of processing complexes into telophase nuclei is suggested by the presence of pre-rRN

176 Telophase nuclei were competent for transcription and pr

177 In zygotes developing normally, telophase nuclei were positioned parallel to the polar g

178 patial correlations between the growth axis, telophase nuclei, and the division plane were analyzed i

179 mbiguously showing that from prometaphase to telophase of mammalian cells, most of the ER is organize

180 kinase signaling in one daughter cell during telophase of mitosis.

181 rrested eggs and because CENP-E reappears in telophase of mouse oocytes activated in the absence of p

182 r invagination channels at late prophase and telophase, potentially suggesting roles for such channel

183 olgin-positive acceptor compartment in early telophase preceded the accumulation of a Golgi glycosylt

184 During telophase, pY ERK associates with newly formed Golgi ves

185 e and continued to rise through anaphase and telophase, reaching a maximum of 7 times interphase rate

186 Loss of Clp1 from the cytoplasm in telophase renders cells sensitive to perturbation of the

187 embly and Cse4 exit during anaphase and late telophase, respectively.

188 its the degradation of mitotic cyclins after telophase, resulting eventually in cell death.

189 aphase bridges were observed to persist into telophase, resulting in chromosomal exclusion from the r

190 normal anaphase but then develop an abnormal telophase spindle and fail to undertake cytokinesis.

191 ced--relies on asymmetric positioning of the telophase spindle midzone, which specifies the cleavage

192 uired both to establish the structure of the telophase spindle to provide a framework for the assembl

193 addition, loss of ASE1 function destabilized telophase spindles, and expression of a nondegradable As

194 nvelope (e.g. metaphase, anaphase, and early telophase stages), these ARPs were excluded from the con

195 At anaphase/telophase, staining shifted to the midbody and the inter

196 associates with decondensing chromosomes in telophase, suggesting a role for YY1 in early marking of

197 levels that are elevated during anaphase and telophase, temporally correlating with H3-K9 acetylation

198 61F gains fusome-dependent interactions near telophase that mediate its incorporation into these stru

199 NT (for regulator of nucleolar silencing and telophase), that also contains Cdc14 and the silencing r

200 During telophase the NDF disappeared with a concomitant appeara

201 Brd4 binding to M/G1 genes increased at telophase, the end phase of mitosis, coinciding with inc

202 In late telophase, the Golgi ribbon began to be reformed by a dy

203 During telophase, the nuclear envelope (NE) reforms around daug

204 In telophase, the staining shifted from the centrosomes to

205 the division plane were not observed before telophase; the earliest division marker detected was a p

206 ly- and late-replicating chromatin from late telophase throughout G1-phase.

207 increased rapidly during the transition from telophase to cytokinesis, whereas cell volume increased

208 released from the nucleolus at late anaphase/telophase to dephosphorylate important regulators of Cdc

209 s required for cell cycle progression at the telophase to G1 cell cycle transition.

210 uired to maintain nuclear spacing during the telophase to interphase transition.

211 neighboring nonsister centrosomes during the telophase-to-interphase transition of the cortical divis

212 ciated from chromatin during the anaphase-to-telophase transition, coincident with the dissociation o

213 During the anaphase-telophase transition, lamin B1 begins to become concentr

214 from poles to midzone during the anaphase-to-telophase transition.

215 y the formation of four colinear clusters at telophase, two per daughter cell.

216 During metaphase and telophase, we suggest that its activity is downregulated

217 ic Golgi fragments, seen in prometaphase and telophase, were found to localize adjacent to endoplasmi

218 ession until some point during late anaphase/telophase when it is rapidly dephosphorylated.

219 , a putative methyl transferase, only during telophase when rDNA gene transcription and pre-rRNA meth

220 similarly on, and around, kinetochores until telophase when they transiently localize near the spindl

221 ome, are recruited to the cell plate at late telophase, when primary PD are formed, and remain associ

222 nexin 11 is recruited to the midbody in late telophase, where it forms part of the detergent-resistan

223 alizes on chromosomes from metaphase through telophase, whereas Ser-988-phosphorylated BRCA1 resides

224 OGT colocalized to the midbody during telophase with Aurora B.

225 ccumulated in nuclei in late anaphase and in telophase, with the exception of a pool of cIAP1 that as