ライフサイエンスコーパス: temporal artery

1 pathologic evidence of AL-amyloidosis of the temporal arteries.

2 ctive inhibitor of cytokine transcription in temporal arteries.

3 ith cytokine mRNA expression in the affected temporal arteries.

4 The absence of any temporal artery abnormality was the only clinical factor

5 In temporal arteries affected by giant cell arteritis, DCs

6 The remaining 26 histologically positive temporal arteries and all 29 histologically negative art

7 in the head and neck involve the superficial temporal artery and its branches, and they typically occ

8 the left ophthalmic artery and anterior deep temporal artery as a potential route for microspheres mi

9 Predictive physical findings included temporal artery beading (positive LR, 4.6; 95% CI, 1.1-1

10 ella-zoster virus antigen) was detectable in temporal artery biopsies taken from individuals with gia

11 ted GCA was examined in peripheral blood and temporal artery biopsies with protein quantification ass

12 iopsy-positive GCA underwent two consecutive temporal artery biopsies, one prior to therapy and one w

13 erpes zoster antigen was detected on several temporal artery biopsies.

14 In 53.0% of patients (98 of 185), temporal artery biopsy ( TAB temporal artery biopsy ) wa

15 comparison with the diagnostic standard TAB temporal artery biopsy ( TAB temporal artery biopsy subc

16 rd to age, frequency of positive findings on temporal artery biopsy (placebo 87%, MTX 79%), or comorb

17 The temporal artery biopsy (TAB) has long been the standard

18 Temporal artery biopsy (TAB) remains the gold standard f

19 We prospectively examined temporal artery biopsy (TAB) tissue from 50 consecutive

20 Temporal artery biopsy (TAB), performed for the diagnosi

21 ts (98 of 185), temporal artery biopsy ( TAB temporal artery biopsy ) was performed (diagnostic stand

22 In 56.5% of patients with TAB temporal artery biopsy -positive results (35 of 62), MR

23 y ) was performed (diagnostic standard [ TAB temporal artery biopsy ]).

24 l in predicting the likelihood of a positive temporal artery biopsy among patients with a clinical su

25 genesis of the disease but have not replaced temporal artery biopsy as the gold standard for securing

26 However, a temporal artery biopsy excluded GCA, showing segmental s

27 Temporal artery biopsy findings were negative in 42% of

28 ), and 28 patients had negative results of a temporal artery biopsy for GCA (group 2).

29 1 core studies, 39% of patients referred for temporal artery biopsy had positive results.

30 In GCA, temporal artery biopsy may not be required in patients w

31 cent of the control samples were obtained by temporal artery biopsy performed within 1 year of the bi

32 Temporal artery biopsy practices vary greatly among trea

33 Temporal artery biopsy remains the diagnostic procedure

34 In GCA, temporal artery biopsy remains the standard for definiti

35 owed a significant association of VZV DNA to temporal artery biopsy samples positive for GCA compared

36 Controls had negative temporal artery biopsy specimens during the same 32-year

37 Temporal artery biopsy specimens from patients with GCA

38 The inflammatory response in temporal artery biopsy specimens was characterized by se

39 In a randomized masked study, 64 temporal artery biopsy specimens were analyzed by PCR fo

40 stochemical, and ultrastructural analyses of temporal artery biopsy specimens.

41 88.7% and specificity was 75.0% for the TAB temporal artery biopsy subcohort (first observer).

42 giant cell arteritis -negative results ( TAB temporal artery biopsy subcohort and total study cohort,

43 ic standard TAB temporal artery biopsy ( TAB temporal artery biopsy subcohort).

44 ers, with good interobserver agreement ( TAB temporal artery biopsy subcohort, kappa = 0.718; total s

45 by angiography and 74 control patients with temporal artery biopsy-proven GCA without large vessel i

46 nts suspected of having GCA should undergo a temporal artery biopsy.

47 Vascular tissue was obtained at the time of temporal artery biopsy.

48 Anastomosis of superficial temporal artery branch to a middle cerebral artery corti

49 Normal temporal arteries contain immature DCs that are located

50 Intimal hyperplasia of the temporal artery correlated with ischemic complications o

51 GCA is self-sustained in temporal arteries engrafted into SCID mice, providing a

52 erpes zoster antigen was detected in 3 of 25 temporal arteries from patients with biopsy-proven GCA.

53 Sixty arteries (the superior and inferior temporal arteries) from 30 eyes of 30 patients (17 femal

54 cells cause inflammation of engrafted human temporal arteries, glucocorticoids were similarly select

55 Temporal artery grafts were harvested and cytokine messa

56 the cellular functions in the infiltrates of temporal arteries impart a basis for rational therapy.

57 chimeras were created by engrafting inflamed temporal arteries into SCID mice.

58 oidosis may present with AION, high ESR, and temporal artery involvement, mimicking GCA.

59 chain (AL) amyloidosis may rarely affect the temporal arteries, mimicking giant cell arteritis, while

60 s observed at the media-adventitia border in temporal arteries of CeAD patients suggest a predisposin

61 mparison of tissue cytokine transcription in temporal arteries of giant cell arteritis patients with

62 ith productive VZV infection in cerebral and temporal arteries, respectively, we evaluated human aort

63 on analyses of giant cell arteritis-affected temporal arteries revealed abundant expression of the NO

64 ocytes/macrophages in the circulation and in temporal arteries revealed glucocorticoid-mediated suppr

65 Transcriptome analysis of GCA-affected temporal arteries revealed low expression of the coinhib

66 Administration of dexamethasone to temporal artery-SCID chimeras for 1 wk induced a partial

67 In human temporal artery-SCID chimeras, lipopolysaccharides stimu

68 Adoptive T cell transfer into temporal artery-SCID mouse chimeras demonstrated that DC

69 of tissue-infiltrating macrophages in human temporal artery-SCID mouse chimeras disrupted nitrotyros

70 Temporal artery-severe combined immunodeficiency (SCID)

71 R and HNE was explored by treating human GCA temporal artery-severe combined immunodeficiency (SCID)

72 Temporal artery specimens from patients with giant cell

73 , gene expression in inflamed and unaffected temporal artery specimens was compared by differential d

74 Diagnosis is confirmed by temporal artery (TA) biopsy, although biopsy results are

75 The temporal artery tap maneuver was performed on 324 caroti

76 ) antigen was found in all of 4 GCA-positive temporal arteries (TAs) but was not present in any of 13

77 PCR was positive for VZV DNA in 9 (26%) temporal arteries tested that showed histologic evidence

78 Expression of cytokine messenger RNA in temporal artery tissue from patients with large-vessel a

79 Inflammatory cytokines were expressed in all temporal artery tissues.

80 nent carotid occlusion after the superficial temporal artery to middle cerebral artery bypass graft o

81 subclavian, carotid, mesenteric, iliac, and temporal arteries) to initiate innate and adaptive immun

82 Sections of formalin-fixed paraffin-embedded temporal arteries were examined first by hematoxylin-eos

83 of neoangiogenesis in giant cell arteritis, temporal arteries were examined for the extent and local

84 mptoms (headache, scalp tenderness, abnormal temporal arteries) were negatively associated with large

85 sence of parvovirus B19 and herpesviruses in temporal arteries with giant cell arteritis have yielded