ライフサイエンスコーパス: temporal lobe

1 ht be associated with WML, especially in the temporal lobe.

2 pplied a virtual lesion to the left anterior temporal lobe.

3 ccipitotemporal face areas and left anterior temporal lobe.

4 ed with increased (18)F-AV1451 in the medial temporal lobe.

5 vealed a cluster spanning the right anterior temporal lobe.

6 al mantle, particularly the inferior-lateral temporal lobe.

7 rvention targeting seizure foci, such as the temporal lobe.

8 musician with a low-grade tumor in the right temporal lobe.

9 and less sparse than elsewhere in the medial temporal lobe.

10 -fornix in regions located within the medial temporal lobe.

11 by progressive atrophy of the frontal and/or temporal lobes.

12 and different locations within the bilateral temporal lobes.

13 ex, angular gyrus, posterior MTG, and medial temporal lobes.

14 s in gene expression between the frontal and temporal lobes.

15 clei, cerebellum, and frontal, parietal, and temporal lobes.

16 aled that damage predominated in frontal and temporal lobes.

17 it activity in distinct regions of the human temporal lobes.

18 rder predominantly affecting the frontal and temporal lobes.

19 nnected with memory structures in the medial temporal lobe [4].

20 ons involved were the frontal lobes (30.4%), temporal lobes (8.69%), basal ganglia (22%), cerebellum(

21 xamining identity representation in anterior temporal lobe, a likely region for the computing of iden

22 nforce the conclusion that the left anterior temporal lobe, a region ignored by classic aphasiology,

23 were disclosed in the right ventral anterior temporal lobe, a region that is undersampled in neuroima

24 ly less pronounced involvement of the medial temporal lobe, abnormal cerebrospinal fluid amyloid-beta

25 s of structural abnormalities in frontal and temporal lobes, amygdala, and insula are less consistent

26 with notably focal involvement of the medial temporal lobe and a slow steady progression, likely repr

27 ng that was confined primarily to the medial temporal lobe and adjacent neocortical regions.

28 relationships (i.e. episodic memory: medial temporal lobe and angular gyrus; semantic memory: left a

29 etween rather than within the visual, medial temporal lobe and default mode networks, whereas during

30 Moreover, tau deposits in the temporal lobe and distributed heteromodal areas were tig

31 emonstrated that cortical dysfunction in the temporal lobe and fusiform gyrus may be related to epile

32 us with higher left-right stretch around the temporal lobe and interhemispheric fissure, anterior-pos

33 efined seven regions of interest in anterior temporal lobe and occipital lobe sections corresponding

34 isal network (SAN), anchored in the anterior temporal lobe and subgenual cingulate, were estimated.

35 l regions; language: left posterior superior temporal lobe and supramarginal gyrus; executive functio

36 ocalised volume loss in the thalamus, medial temporal lobe and temporal neocortex.

37 This elevation was most prominent in the temporal lobe and temporoparietal junction, but extended

38 s (the 'peak clusters') were in the anterior temporal lobe and the precentral gyrus.

39 ease, there is focal tauopathy in the medial temporal lobes and adjacent cortices.

40 on smaller WMVs were present in frontal and temporal lobes and corpus callosum (all p values <0.01).

41 us, posterior cingulate gyrus, cortex of the temporal lobes and corpus callosum, and fractional aniso

42 Based on evidence that the medial temporal lobes and prefrontal cortex represent contextua

43 d through increased activation in the medial temporal lobes and prefrontal cortex, and more consisten

44 e of involvement of the frontal and anterior temporal lobes and the presence of asymmetry could be us

45 e of involvement of the frontal and anterior temporal lobes and the presence of asymmetry could be us

46 cipital lobe, orbitofrontal cortex, anterior temporal lobe, and caudate nucleus than PCA, and PCA sho

47 rain, basal ganglia, basal forebrain, medial temporal lobe, and discrete cortical regions.

48 [0.11]), less severe atrophy of the lateral temporal lobe, and lower mean (SD) cerebrospinal fluid l

49 osterior cingulate gyrus, lateral and medial temporal lobe, and occipital lobe as well as caudate and

50 etween functional areas in the occipital and temporal lobe, and the fusiform face area (FFA) and ante

51 inputs from extrastriate cortex, the medial temporal lobe, and three subcortical structures (the pul

52 nd ND, tau levels in the medial and inferior temporal lobes, and longitudinal cognition were examined

53 PTSD exhibit hypersynchrony in a circuit of temporal lobe areas associated with learning and memory

54 Thus, two temporal lobe areas extend the core face-processing netw

55 and clinical studies that the human anterior temporal lobe (ATL) is a semantic representational hub.

56 The anterior temporal lobe (ATL) makes a critical contribution to sem

57 Here, we asked whether the anterior temporal lobe (ATL) serves as a hub for a distributed ne

58 egion for object perception, or the anterior temporal lobe (ATL), a "hub" of supramodal conceptual pr

59 nition depends on subregions of the anterior temporal lobe (ATL).

60 Associations between cognition, medial temporal lobe atrophy (MTA), lesion volumes, and global

61 35) by using the Scheltens' scale for medial temporal lobe atrophy (MTA), the Koedam's scale for post

62 rophy (RR=2.59, 95% CI=1.95 to 3.44), medial temporal lobe atrophy (RR=2.11, 95% CI=1.70 to 2.63) and

63 date index, global cortical (GCA) and medial temporal lobe atrophy scores and single voxel (basal gan

64 In the MRI data, mesial temporal lobe atrophy was determined by the Scheltens me

65 normal CSF tau level, hippocampal and medial temporal lobe atrophy, entorhinal atrophy, depression, d

66 INTERPRETATION: Medial temporal lobe AV-1451 uptake distinguishes AD dementia f

67 Medial temporal lobe AV-1451 uptake distinguishes AD dementia f

68 lp-negative seizures arising from the mesial temporal lobe based on scalp electroencephalogram networ

69 rus, beta = -0.001 [SE, 0.027]; and inferior temporal lobe, beta = -0.004 [SE, 0.027]; P >/= .88) and

70 rus, beta = -0.074 [SE, 0.030]; and inferior temporal lobe, beta = -0.083 [SE, 0.031]; P </= .02).

71 y settings, the hippocampus and other medial temporal lobe brain structures have been shown to repres

72 hanges in the hippocampus and related medial temporal lobe circuitry-brain areas that are important f

73 riences, typical neurons in the human medial temporal lobe code for a considerable range of objects,

74 anterior and posterior cingulate, insula and temporal lobes (Cohen's d effect sizes: -0.10 to -0.14).

75 Stimulations in the medial temporal lobe components of the DN evoked relatively lat

76 ences in the salience network and the medial temporal lobe contribute to memory impairment in PD.

77 ieved with the volumetric measurement of the temporal lobe cortex - the values were 90% and 93%, resp

78 The volumetric measurement of the temporal lobe cortex had the highest correct classificat

79 to the hippocampus, and not found in medial temporal lobe cortex, category-selective areas of the vi

80 supported by similar interaction effects on temporal lobe cortical thickness (whole-brain voxelwise

81 Reduced total cortical volume and temporal lobe cortical thickness and greater lateral ven

82 slexia and semantic impairment from anterior temporal lobe degeneration, surface errors in our patien

83 y exhibits a preferred posterior to anterior temporal lobe direction, consistent across participants.

84 The tau deposits were grouped in the temporal lobe, distributed in heteromodal areas, medial

85 was predicted by a common profile of medial temporal lobe downregulation involving the anterior hipp

86 ices, temporoparietal junction, and anterior temporal lobes during social thought: rationality, socia

87 such changes are merely secondary to medial temporal lobe dysfunctions is challenged.

88 e epilepsy, the remaining 15 cases for extra-temporal lobe epilepsies.

89 e studied 36 patients with unilateral medial temporal lobe epilepsy (19 right) before and 3 and 12 mo

90 ed protein, cause autosomal dominant lateral temporal lobe epilepsy (ADLTE).

91 ls (DGCs) are altered in experimental mesial temporal lobe epilepsy (mTLE) and whether their integrat

92 chronically lost in a mouse model of mesial temporal lobe epilepsy (MTLE) as well as in hippocampal

93 As mesial temporal lobe epilepsy (mTLE) has been recognized as a n

94 The cause of mesial temporal lobe epilepsy (MTLE) is often unknown.

95 Mesial temporal lobe epilepsy (mTLE) is the most common focal e

96 Patients with mesial temporal lobe epilepsy (MTLE) without MRI abnormalities

97 tment of patients with drug-resistant mesial temporal lobe epilepsy (mTLE).

98 Seizures in temporal lobe epilepsy (TLE) disturb brain networks and

99 of TLE.SIGNIFICANCE STATEMENT Development of temporal lobe epilepsy (TLE) generally takes years after

100 Temporal lobe epilepsy (TLE) is a common epilepsy syndro

101 Mesial temporal lobe epilepsy (TLE) is characterized by stereot

102 Temporal lobe epilepsy (TLE) is one of the most common d

103 Temporal lobe epilepsy (TLE) is one of the most common f

104 Temporal lobe epilepsy (TLE) is the most frequent form o

105 Drug-resistant temporal lobe epilepsy (TLE) often requires thorough inv

106 ersely related in the DG of individuals with temporal lobe epilepsy (TLE) or AD and correlate with pe

107 Although most temporal lobe epilepsy (TLE) patients show marked hippoc

108 Medically refractory unilateral temporal lobe epilepsy (TLE) with hippocampal sclerosis

109 itivity to localize hippocampal pathology in temporal lobe epilepsy (TLE), but has rarely been evalua

110 Temporal lobe epilepsy (TLE), the most common form of ac

111 blish further parallels between human medial temporal lobe epilepsy and a naturally occurring conditi

112 logy is associated with cognitive decline in temporal lobe epilepsy and explored this through clinico

113 A levels in the hippocampus of patients with temporal lobe epilepsy and in neural tissues from animal

114 tify the transcriptomic signature of chornic temporal lobe epilepsy and the drugs that reverse it.

115 and UFRs were observed only in patients with temporal lobe epilepsy and were recorded exclusively fro

116 We studied 43 patients with mesial temporal lobe epilepsy associated with hippocampal scler

117 vide new insights for why some patients with temporal lobe epilepsy continue to experience postoperat

118 The studies assessing SPECT use in temporal lobe epilepsy did not reveal a correlation with

119 clinical trials enrolling 118 patients with temporal lobe epilepsy found greater freedom from seizur

120 proved verbal learning in patients with left temporal lobe epilepsy from preoperatively to 12 months

121 Human temporal lobe epilepsy has been associated with dysfunct

122 lation-sensitive microRNA were identified in temporal lobe epilepsy including MIR27A, miR-193a-5p (MI

123 Temporal lobe epilepsy is a common and challenging clini

124 Temporal lobe epilepsy is a common and frequently intrac

125 One in 26 people develops epilepsy, and temporal lobe epilepsy is a common form.

126 Temporal lobe epilepsy is common and can be difficult to

127 Temporal lobe epilepsy is the most common and drug-resis

128 s been suggested that the pathophysiology of temporal lobe epilepsy may relate to abnormalities in va

129 pocampi from mice with epilepsy (pilocarpine-temporal lobe epilepsy model) and 100 healthy control hi

130 more, despite induction of MF sprouting in a temporal lobe epilepsy model, KARs were not recruited to

131 Patients with temporal lobe epilepsy often display cognitive comorbidi

132 Temporal lobe epilepsy or limbic epilepsy lacks effectiv

133 mory-encoding network in both left and right temporal lobe epilepsy patients across both verbal and v

134 Ictal events occurring in temporal lobe epilepsy patients and in experimental mode

135 Left temporal lobe epilepsy patients showed increased right a

136 Humans with medial temporal lobe epilepsy present with white matter patholo

137 Patients with right temporal lobe epilepsy showed increased left anterior hi

138 We show in a rat model of temporal lobe epilepsy that spontaneous hippocampal IEDs

139 ppocampus and neocortex of rats with chronic temporal lobe epilepsy to demonstrate that subsets of ce

140 This study evaluated a mouse model of temporal lobe epilepsy to test which pathological change

141 coordinating gene networks in patients with temporal lobe epilepsy will help to identify novel thera

142 in every two patients with pharmacoresistant temporal lobe epilepsy will not be rendered completely s

143 compared miRNA expression patterns in mesial temporal lobe epilepsy with and without hippocampal scle

144 s) in a large consecutive series with mesial temporal lobe epilepsy with hippocampal sclerosis (MTLE-

145 e prolonged febrile seizures group developed temporal lobe epilepsy with mesial temporal sclerosis.

146 Unique methylation profiles were evident in temporal lobe epilepsy with or without hippocampal scler

147 that the hippocampus of patients with mesial temporal lobe epilepsy with sclerosis is completely devo

148 pping confirmed that in the course of mesial temporal lobe epilepsy with sclerosis, astrocytes acquir

149 erogeneous forms of epilepsy including human temporal lobe epilepsy, a mouse model of acquired tempor

150 ral lobe epilepsy, a mouse model of acquired temporal lobe epilepsy, and a mouse model of monogenic D

151 e basolateral amygdala kindling rat model of temporal lobe epilepsy, and it led to prolongation of th

152 has clear clinical utility in patients with temporal lobe epilepsy, and its potential easily transla

153 In a rat model of temporal lobe epilepsy, basket cell-to-granule cell (BC-

154 ibute to the pathogenesis and maintenance of temporal lobe epilepsy, but the underlying cell and mole

155 ory formation (e.g., pattern separation) and temporal lobe epilepsy, little is known about activity-d

156 These mice had mesial-temporal lobe epilepsy, microcephaly and corpus callosum

157 In a mouse model of temporal lobe epilepsy, multicellular calcium imaging re

158 nt in neuroinflammation, aggravating stroke, temporal lobe epilepsy, neuropathic pain, and various ne

159 During temporal lobe epilepsy, the homeostatic regulation of in

160 FOR A SCIENTIFIC COMMENTARY ON THIS ARTICLE: Temporal lobe epilepsy, the most prevalent form of chron

161 Fifty-one patients (77%) had surgery for temporal lobe epilepsy, the remaining 15 cases for extra

162 and propagation of temporal lobe seizures in temporal lobe epilepsy, using diffusion tensor imaging a

163 ngs suggest an epilepsy-related tauopathy in temporal lobe epilepsy, which contributes to accelerated

164 tent postoperative seizures in patients with temporal lobe epilepsy.

165 endent in vivo rat model systems relevant to temporal lobe epilepsy.

166 for further testing as a potential cause of temporal lobe epilepsy.

167 ucture of BC-->GC synapses in a rat model of temporal lobe epilepsy.

168 t seizures on the model of pharmacoresistant temporal lobe epilepsy.

169 ntrol seizures in up to 80% of patients with temporal lobe epilepsy.

170 ide (LPS) administration in mice with mesial temporal lobe epilepsy.

171 ges 50 and 65 years to treat drug-refractory temporal lobe epilepsy.

172 ocampus from patients with pharmacoresistant temporal lobe epilepsy.

173 ting of mossy fiber axons, both hallmarks of temporal lobe epilepsy.

174 ion with mossy fibre sprouting, a feature of temporal lobe epilepsy.

175 h seizure susceptibility in mice with mesial temporal lobe epilepsy.

176 l circuits in experimental models of chronic temporal lobe epilepsy.

177 about the recurrent excitation hypothesis of temporal lobe epilepsy.

178 thesized to contribute to the development of temporal lobe epilepsy.

179 roximately one-third of patients with mesial temporal lobe epilepsy.

180 rain region in the pathophysiology of mesial temporal lobe epilepsy.

181 Second, we tested 20 refractory temporal lobe epileptic patients (11 women) with unilate

182 nto the dentate gyrus, potentially mediating temporal lobe epileptogenesis.

183 tems in the parahippocampal gyrus and medial temporal lobe, especially involving the perirhinal corte

184 We found that the lateral anterior temporal lobe exhibited increased activation when partic

185 ral representation for imagined words in the temporal lobe, frontal lobe and sensorimotor cortex, con

186 system with a particular emphasis on medial temporal lobe function.

187 lia (g = 0.39; 95% CI, 0.09-0.70) and medial temporal lobe (g = 0.32; 95% CI, 0.12-0.52).

188 Though memory dependent on the medial temporal lobes has been shown to drive attention, how ot

189 rors that seen on the lateral surface of the temporal lobe in macaques, suggesting that the entire tr

190 ht hemisphere regions, as follows: bilateral temporal lobe, including temporal pole, fusiform gyrus,

191 The medial temporal lobe, including the hippocampus, has been impli

192 us, basal forebrain, occipital, parietal and temporal lobes, including the hippocampus, were associat

193 at incipient protein pathology in the medial temporal lobe instigates the loss of episodic memory in

194 is concordant with the seizure origin in the temporal lobe is a significant factor associated with a

195 suggest development of tau beyond the mesial temporal lobe is associated with, and may be dependent o

196 vement of the parieto-occipital, frontal and temporal lobes is common in PRES.

197 related to contextual learning; the anterior temporal lobe, known to be an amodal hub for semantic pr

198 PND (non-displaceable binding potential)) in temporal lobes, lateralising according to their clinical

199 tinct subregions of the left medial anterior temporal lobe (LATL).

200 selective subregions of medial left anterior temporal lobe (LATL); (2) tools, but not animals, were e

201 served in the corpus callosum, cingulum, and temporal lobe likely constitute the neural substrate for

202 m (P < 0.001) involving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontopar

203 m (P < 0.001) involving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontopar

204 the early-onset group, and more focal medial temporal lobe loss in the late-onset group.

205 ysical interaction preference, which, in the temporal lobe, mapped onto a fine-grain pattern of objec

206 in WS patients indicates a severe failure of temporal lobe maturation during infancy.

207 antly lower gray matter volume in the medial temporal lobe (maximum z score = 5.2 and cluster size of

208 for studying higher-level vision and medial temporal lobe memory.

209 Both in the occipital and temporal lobe, metabolites with higher physiological con

210 ntral medial prefrontal cortex, and superior temporal lobe, mostly on the left side.

211 ncert with other spatial cells in the medial temporal lobe (MTL) [3-6], they provide a representation

212 eriments, patients with damage to the medial temporal lobe (MTL) and healthy controls produced detail

213 esponses of individual neurons in the medial temporal lobe (MTL) are attenuated, delayed, and lengthe

214 The medial temporal lobe (MTL) contains "concept cells" that respon

215 Given the critical role of the medial temporal lobe (MTL) in episodic memory, age-related chan

216 The medial temporal lobe (MTL) is an early site of tau accumulation

217 Neural circuitry in the medial temporal lobe (MTL) is critically involved in mental tim

218 By contrast, one patient with large medial temporal lobe (MTL) lesions performed poorly overall in

219 beta) and tau proteins and atrophy of medial temporal lobe (MTL) structures crucial to memory formati

220 re has been interest in the idea that medial temporal lobe (MTL) structures might be especially impor

221 o delineate the alERC/pmERC and other medial temporal lobe (MTL) subregions.

222 data indicate that tau tangles in the medial temporal lobe (MTL) underlie episodic-memory impairments

223 Neurons in the medial temporal lobe (MTL), a critical area for declarative mem

224 vity of neuronal ensembles within the medial temporal lobe (MTL).

225 The temporal lobe network (TLN), and subcortical network (SC

226 We find that suppression in a medial temporal lobe network changes trial-by-trial in proporti

227 cuit dysfunction could erode the function of temporal lobe networks and ultimately contribute to cogn

228 examine depth electrode recordings from the temporal lobe of 13 presurgical epilepsy patients perfor

229 ith cell-type-specific imaging in the medial temporal lobe of cognitively assessed, aged rhesus macaq

230 characterized by atrophy in the frontal and temporal lobes of the brain.

231 haracterize the effects of DBS in the medial temporal lobe on human memory.

232 and ventral tegmental area (SN/VTA), medial temporal lobe, or subsequent memory performance.

233 of the lateral ventricle and in the inferior temporal lobe; or (2) the presence of a subcortical U-fi

234 ) than with SUVRCB (Pearson r: from 0.51 for temporal lobe [P = 0.002] to 0.82 for precuneus [P < 0.0

235 ork, such as the insula, cingulate cortices, temporal lobe, parietal lobe, and medial frontal lobe.

236 nd the fusiform face area (FFA) and anterior temporal lobe play key roles in the recognition of facia

237 The temporal lobes play a prominent role in declarative memo

238 otor neuron disease, eight type C with right temporal lobe presentations, and 11 unclassifiable inclu

239 in AD, which typically begins in the medial temporal lobe progressing along the cortical default mod

240 requency theta power are seen across lateral temporal lobe recording sites and persist throughout the

241 cipir standard uptake value ratios in mesial temporal lobe regions (amygdala, hippocampus/choroid ple

242 in other hippocampal (e.g., CA3) and medial temporal lobe regions (e.g., entorhinal cortex).

243 ng) but decreased in domain-specific ventral temporal lobe regions (temporal pole for word matching a

244 ia and highlight the importance of different temporal lobe regions for the recovery of amusia after s

245 icantly worse aphasia, particularly when key temporal lobe regions were isolated into segregated modu

246 l connectivity between these distinct medial temporal lobe regions with the ventral tegmental area (V

247 investigate how in vivo tau accumulation in temporal lobe regions, Abeta, and MTL atrophy contribute

248 report and reduce the BOLD signal in lateral temporal lobe regions.

249 f small/large objects), whereas the anterior temporal lobe represents semantically based aspects (obj

250 ing test scores (r = -0.50) over 1-year post-temporal lobe resection (P < 0.05).

251 on tissue from 33 patients who had undergone temporal lobe resection between ages 50 and 65 years to

252 Anterior temporal lobe resection can control seizures in up to 80

253 dementia-motor neuron disease who underwent temporal lobe resection for epilepsy 5 years prior to he

254 t) before and 3 and 12 months after anterior temporal lobe resection.

255 This model thus allows direct control of the temporal lobe seizure threshold via endogenous pathways,

256 Alzheimer's disease, in which occult mesial temporal lobe seizures are suspected to play a significa

257 etector specifically recognizes focal mesial temporal lobe seizures based on scalp electroencephalogr

258 s to the hippocampus, we control the risk of temporal lobe seizures during a specific time period.

259 We identified 25 scalp-negative mesial temporal lobe seizures in 10 patients and obtained contr

260 tis were observed in all patients, including temporal lobe seizures in 16 patients (88.9%) and memory

261 Focal temporal lobe seizures in humans and in several experime

262 portant in the generation and propagation of temporal lobe seizures in temporal lobe epilepsy, using

263 eizure alarms, 80% had scalp-negative mesial temporal lobe seizures.

264 Whereas the anterior temporal lobe served as the major network hub for face p

265 ccumulation of early tauopathy in the medial temporal lobe, specifically in the entorhinal cortex, an

266 y auditory areas and moving laterally on the temporal lobe: spectral features are found in the core o

267 The amygdala is a medial temporal lobe structure implicated in social and emotion

268 s there was activation in lateral and medial temporal lobe structures, posterior cingulate cortex, pr

269 rt, from declines in the integrity of medial temporal lobe structures, such as the hippocampus, but a

270 dine reliably induces seizure-like events in temporal lobe structures.

271 F 18 was found across neocortical and mesial temporal lobe structures.

272 Among temporal lobe subregions, episodic memory was most stron

273 and whether this occurs in structures in the temporal lobe, supplying cortical inputs to the hippocam

274 ot be rendered completely seizure-free after temporal lobe surgery.

275 c importance of SPECT in patients undergoing temporal lobe surgery.

276 of memory representations within the medial temporal lobe system.

277 The medial temporal-lobe system is essential for the formation and

278 ntral pathway, including the dorsal anterior temporal lobe (TEd), object-sensitive neurons are interm

279 60] years) adolescence, predominantly in the temporal lobes (temporal cortex: random field theory cor

280 ting, bilaterally, the lateral aspect of the temporal lobe, the temporo-parieto-occipital junction an

281 fected regions were the inferior and lateral temporal lobes, the same regions where the first signs o

282 ing presurgical evaluation and proceeding to temporal lobe (TL) resection were studied.

283 f music perception/reproduction and the left temporal lobe to language semantics; both these regions

284 TLE subjects had lateralized atrophy of most temporal lobe tracts, and hippocampal volumes in HS-TLE

285 in key regions implicated in memory (medial temporal lobes), visuospatial function (occipital, right

286 Temporal lobe volume had a negative correlation with ser

287 infection, magnetic resonance imaging-based temporal lobe volume.

288 Volumetric reduction in the medial temporal lobe was correlated with PS symptom severity.

289 The temporal lobe was involved in 71.9% of operations.

290 ophrenia, whereas elevated Glx in the medial temporal lobe was seen with chronic schizophrenia (g = 0

291 Temporal lobe was the most common NHS location (16 of 51

292 Tau levels in the medial and inferior temporal lobes were indistinguishable between the SNAP a

293 ed tracer retention in regions of the medial temporal lobe, which predicted worse episodic memory per

294 n the human medial frontal cortex and medial temporal lobe while subjects held up to three items in m

295 The responses were recorded over the right temporal lobe while the infants were presented with enga

296 ional tissue characteristics of preoperative temporal lobe white matter tracts known to be important

297 s showed elevated FTP uptake in the anterior temporal lobe with a leftward asymmetry that was not obs

298 ons distributed throughout the occipital and temporal lobes with a right hemisphere advantage.

299 ta (Abeta) levels that was restricted to the temporal lobe, with the greatest increase seen in the hi

300 in deep cortical regions such as the mesial temporal lobes without showing any obvious signs of seiz