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7 mptoms are associated with the occurrence of temporomandibular disorder (TMD), using the OPPERA prosp
10 n elderly general populations, prevalence of temporomandibular disorder was 7% (1-31) and abdominal p
11 uring evaluations of 610 new patients with a temporomandibular disorder who also reported a history o
12 clusal abnormalities have been implicated in temporomandibular disorder, it is not known if these ris
13 wel syndrome, multiple chemical sensitivity, temporomandibular disorder, tension headache, interstiti
15 examined using Diagnostic Criteria (DC) for Temporomandibular Disorders (TMD) and magnetic resonance
23 to the symptomatology of female-predominant temporomandibular disorders (TMDs) inflammatory pain.
24 This article uses the example of a study of temporomandibular disorders (TMDs), investigating causal
30 igraine such as medication-overuse headache, temporomandibular disorders, obstructive sleep apnea and
33 tes has roles in degenerative remodelling of temporomandibular joint (TMJ) and to determine associate
34 cells (BMSCs) on osteoarthritis (OA) of the temporomandibular joint (TMJ) and to explore the role of
41 , we determined whether the mutation affects temporomandibular joint (TMJ) development and growth.
42 n and hedgehog signaling, but their roles in temporomandibular joint (TMJ) development are unknown.
45 een used for the management of patients with temporomandibular joint (TMJ) disc displacement without
48 chanical fatigue-related degeneration of the temporomandibular joint (TMJ) disc may be promoted by tr
51 nical loading on solute transport in porcine temporomandibular joint (TMJ) discs using the electrical
55 previous data suggested the hypothesis that temporomandibular joint (TMJ) eminence shapes develop id
56 onatal mice and assessed the consequences on temporomandibular joint (TMJ) growth and organization ov
58 association between more advanced stages of temporomandibular joint (TMJ) intra-articular disorders
62 artilage stem cells (FCSCs) derived from the temporomandibular joint (TMJ) mandibular condyle that ge
64 (CFA) was injected unilaterally into the rat temporomandibular joint (TMJ) or perioral (PO) skin to p
67 of the US population will seek treatment for temporomandibular joint (TMJ) symptoms, typically occurr
68 es have indicated a positive response of the temporomandibular joint (TMJ) to mandibular advancement,
69 ditions, including those affecting the human temporomandibular joint (TMJ), but the underlying molecu
76 artilage, mandible, the articulating disc of temporomandibular joint and branchial arch nerve ganglia
78 ardized videotapes showing palpations of the temporomandibular joint and muscles of mastication and r
80 cterized by pain and reduced function in the temporomandibular joint and/or associated masticatory mu
81 rome characterized by variable micrognathia, temporomandibular joint ankylosis, cleft palate, and a c
82 lyarticular disease are all risk factors for temporomandibular joint arthritis but may underpredict t
83 cal and radiographic signs, and treatment of temporomandibular joint arthritis in children with juven
85 currently the gold standard in detection of temporomandibular joint arthritis, and treatment with in
86 Given the paucity of clinical symptoms in temporomandibular joint arthritis, detection of temporom
89 lysis of these data demonstrated significant temporomandibular joint disc-engineering potential for P
90 e arthritis in the previous 6 months (mCSA), temporomandibular joint disease (mCSA and section modulu
91 2.3 times, the risk of developing myogenous temporomandibular joint disorder (TMD), a common musculo
94 corticosteroids holds promise for control of temporomandibular joint inflammation and prevention of a
95 poromandibular joint arthritis, detection of temporomandibular joint inflammation using contrast-enha
96 dibular joint arthritis but may underpredict temporomandibular joint involvement in juvenile idiopath
98 tstanding review of the anatomy of the human temporomandibular joint is presented by Piette [5.].
101 -/-) MCCs, we discovered the early basis for temporomandibular joint osteoarthritis arises from abnor
103 d function, we used a genetic mouse model of temporomandibular joint osteoarthritis that is deficient
105 mpirical evidence suggests that the "normal" temporomandibular joint produces noise during function.
107 the filling of the cavity (posterior to the temporomandibular joint) coincides with the moment of ma
108 lutionary innovations in the ear region, the temporomandibular joint, and the brain vault evolved inc
109 uffered physiological saline solution, TMJ = temporomandibular joint, mu(T) =tractional coefficient,
113 plants and chondrocytes, derived from bovine temporomandibular joints (TMJ), were examined for matrix
115 ites (UACs) elicited OA-like lesions in mice temporomandibular joints (TMJs), displaying as subchondr
116 rus (FIV) (Cre) vector in the right and left temporomandibular joints (TMJs), or in the cisterna magn
118 imaging and by using the maxillary teeth and temporomandibular joints as a guide to finish the recons
119 A single injection of FIV(HuMOR) into the temporomandibular joints of Col1-IL-1beta(XAT)-transgeni
120 incisors, alveolar bone loss and compressed temporomandibular joints, in addition to abnormal skull
121 fficult joints to examine, such as the hips, temporomandibular joints, small joints of the feet, and
126 of masticatory muscle activation on pain in temporomandibular muscle and joint disorders (TMJD) is c
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