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1 e molecular pathway of retinoic acid-induced teratogenesis.
2 ility of the embryo and developing organs to teratogenesis.
3 B1 appears to play a role in dioxin-mediated teratogenesis.
4 erapeutic strategies to prevent Zika-induced teratogenesis.
5 bit increased incidence of radiation-induced teratogenesis.
6 hanisms underlying copper deficiency-induced teratogenesis.
7 malidomide and Lenalidomide on angiogenesis, teratogenesis, and neurite outgrowth, known detrimental
8 are more sensitive to retinoic acid-induced teratogenesis, and retinoid target genes are expressed a
11 resistant to the acute toxicity and cardiac teratogenesis caused by high levels of polycyclic aromat
13 is; some subpopulations resisted the cardiac teratogenesis caused by the challenges at doses that sti
18 se findings suggest that cyclopamine-induced teratogenesis is due to a more direct antagonism of Shh
22 developing embryo; therefore, mechanisms of teratogenesis may be approached within the context of th
24 ice that differ in susceptibility to ethanol teratogenesis showed corresponding differences in MAPK a
25 always correlate strongly with resistance to teratogenesis; some subpopulations resisted the cardiac
26 onstrating the importance of maternal AHR in teratogenesis, these data may have implications that rea
28 To elucidate their roles in dioxin-induced teratogenesis, we compared Cyp1a1(-/-), Cyp1a2(-/-), and
29 ression may cause some aspects of RA-induced teratogenesis, we studied the mechanism underlying the i
30 tionship between p53-dependent apoptosis and teratogenesis, we subjected day 8 mouse embryos with dif
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