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   1 ease 11 L/min and 8 L/min) than those taking terbutaline.                                            
     2 e randomly assigned formoterol 4.5 microg or terbutaline 0.5 mg as needed by Turbuhaler in daily dose
     3 5 microg provided better asthma control than terbutaline 0.5 mg in patients requiring moderate doses 
     4 horbol 12-myristate 13-acetate (PMA, 10 nM), terbutaline (0.1 mM), or ATP (1 mM), the binding of SP-A
     5 voked NO release (isoproterenol; dobutamine; terbutaline; 10(-9) to 10(-5) m) was blocked by the NOS 
  
  
     8 R responses in newborn rats, we administered terbutaline, a beta2AR agonist, on postnatal day 2 and e
  
    10 ) M), the putative beta 2-adrenergic agonist terbutaline also caused preconstricted arterioles and ve
  
  
    13 h2 cells were exposed to the beta2AR agonist terbutaline before activation by Ag-presenting B cells. 
  
  
  
    17 re isometric tension after administration of terbutaline (concentration range, 10(-8) to 10(-4) M), d
  
    19  to determine whether other bronchodilators (terbutaline, diltiazem, and aminophylline) relax bronchi
  
  
    22 acting beta-agonist formoterol compared with terbutaline, each taken as needed, in patients with mode
    23 rnatants, the lower level of IL-2 present in terbutaline-exposed culture supernatants supported the p
  
  
    26 pre-exposed to Ag and/or the beta 2AR ligand terbutaline for 24 h before being activated by either a 
    27     The antagonist propranolol competed with terbutaline for beta2AR binding sites and expectedly rig
  
    29 acellular cAMP were similar in both subsets, terbutaline induced an increase in cAMP levels in Th1 ce
    30 ntagonist atenolol (10(-6) M) did not affect terbutaline-induced dilation in preconstricted arteriole
  
  
    33 ist, butoxamine, suggests that the effect of terbutaline is mediated by activation of beta2-adrenergi
    34 hese structurally related bronchodilators is terbutaline; it is administered as a prodrug, bambuterol
    35 > .05) was the most efficacious, followed by terbutaline (maximum relaxation, 72%+/-13% [proximal], 5
    36 2)-adrenergic receptor ligands (epinephrine, terbutaline, metaproterenol, salmeterol, propranolol, al
  
  
  
    40 ells to either the beta 2AR-selective ligand terbutaline or the sympathetic neurotransmitter norepine
  
  
    43 ing beta2-agonists albuterol, fenoterol, and terbutaline provide rapid as-needed symptom relief and s
  
  
    46  of dobutamine (selective beta1-agonist) and terbutaline (selective beta2-agonist) on glycerol releas
  
  
  
    50 vivo tests using zebrafish models found that terbutaline sulfate prevents defects in axons and neurom
  
    52  Interestingly, the prediction suggests that terbutaline sulfate, which is widely used for asthma, is
  
  
    55 ) responses to administration of intravenous terbutaline (TRB) before and after 5 days of low dietary
    56  and combined effects of maternal stress and terbutaline (used to arrest preterm labor), autism risk 
    57 chiral separation of milligram quantities of terbutaline using sulfated cyclodextrin as a chiral addi
  
  
    60 aline and adrenaline than when salbutamol or terbutaline were present (e.g., log KD propranolol -8.65
    61 d of clenbuterol, cimaterol, procaterol, and terbutaline which acted as full agonists for cAMP produc
    62 atients may be treated with theophylline and terbutaline, which clinical experience suggests may redu
  
    64  epinephrine > or = formoterol = fenoterol > terbutaline = zinterol = albuterol > salmeterol > dobuta
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