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1 efined as enlargement of airspaces distal to terminal bronchioles accompanied by destruction of alveo
3 otein (MIP)-2 in epithelial cells lining the terminal bronchioles and alveolar ducts as well as macro
4 During infection, P. aeruginosa enters the terminal bronchioles and alveoli and comes into contact
5 m tuberculosis is initially deposited in the terminal bronchioles and alveoli, as well as following r
7 ent, CCSP-expressing stem cell population in terminal bronchioles and support the notion that regiosp
9 t in Clara cells of wild-type and COX-1(-/-) terminal bronchioles and was strongly induced 24 hours a
11 identifies type II cells, Clara cells in the terminal bronchioles, and putative bronchoalveolar stem
12 icroCT has made it possible to show that the terminal bronchioles are narrowed and destroyed before t
13 ilagenous rings, neuroepithelial bodies, and terminal bronchioles/bronchoalveolar duct junctions.
14 emphysematous tissue destruction, number of terminal bronchioles, infiltrating inflammatory cells, a
15 aged cells) was significantly greater in the terminal bronchioles of CYP2A13/2F1-humanized mice than
17 total number and cross-sectional area of the terminal bronchioles or in alveolar dimensions (mean lin
21 Micro-CT showed that the total number of terminal bronchioles was decreased (2.9/ml [2.6-4.4] vs.
22 ed, and the cross-sectional area of the open terminal bronchioles was reduced (0.093 mm(2) [0.084-0.1
23 t alveolar duct bifurcations and in adjacent terminal bronchioles was significantly reduced in the 12
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