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1 er sequences in the U3 region of the 3' long terminal repeat.
2 d eight, respectively, are duplicated in the terminal repeat.
3 ing of Tat at the TAR region of the HIV long terminal repeat.
4 differential DNA methylation at its 5'-long terminal repeat.
5 stranded RNA40 (ssRNA40) from the HIV-1 long terminal repeat.
6 obin locus within the self-inactivating long-terminal repeat.
7 F-kappaB and its recruitment to the HIV-long terminal repeat.
8 on through HIF-1 association with HIV-1 long terminal repeat.
9 lls using the mouse mammary tumor virus long terminal repeat.
10 IV Tat protein to transactivate the HIV long terminal repeat.
11 histone-modifying enzymes to the HIV-1 long terminal repeat.
12 form the capsid, and the right-hand inverted terminal repeat.
13 nt downstream transcription from the 3' long terminal repeat.
14 x that is associated with repressed HIV long terminal repeats.
15 ked by adeno-associated virus (AAV) inverted terminal repeats.
16 showed binding of RPA1 and RPA2 to the KSHV terminal repeats.
17 tures, followed by the nucleation of three N-terminal repeats.
18 gase, and this property is mediated by the C-terminal repeats.
19 aps that protect the hydrophobic core of the terminal repeats.
20 ipts is promoted by endogenous intronic long terminal repeats.
21 anded DNA (dsDNA) genome with large inverted terminal repeats.
22 y differ by the presence of 3(3R) or 4(4R) C-terminal repeats.
24 transcripts, a moderate reduction of 2-long terminal repeat (2-LTR) circles, and a relatively large
30 nal plasma CMV (P = 0.04), detectable 2-long terminal repeat (2-LTR), and lower nadir CD4(+) (P < 0.0
33 omes containing two copies of the viral long terminal repeat (2LTR circles) were analyzed in using dr
34 extension at the viral U5 end of the 3' long terminal repeat (3'-LTR), which is a poor substrate for
36 litates repression by LANA bound to the KSHV terminal repeats, a function that requires the MeCP2 C t
39 subunits (named CasC1-6), with its 5' and 3' terminal repeats anchored by CasD and CasE, respectively
42 d cryptic splice sites in the HIV 5iota long terminal repeat and gag gene as well as in the beta-glob
43 rictive chromatin structures at the HIV long terminal repeat and limiting P-TEFb levels contribute to
44 ons in the regulatory elements in their long terminal repeats and differed in a helical segment of en
45 eviously overlooked partly because they lack terminal repeats and do not create target site duplicati
46 The DNA of the element is marked by long terminal repeats and encodes a single large protein with
47 NMT3a/3b in suppressing retrotransposon long terminal repeats and long interspersed elements, respect
48 repetitive elements such as Alu, LINE, long-terminal repeats and simple tandem repeats are frequentl
49 ted replication fork protection at the viral terminal repeats and that Tim-dependent recombination-li
50 tational analysis of the HERV-K (HML-2) long terminal repeat, and treatments with agents that inhibit
51 ranulatus genome, especially Gypsy-like long terminal repeats, and there has also been an expansion i
53 c gene family expansions, the number of long-terminal-repeat-based retrotransposons, and mechanisms o
54 est of the genes, forming a part of inverted terminal repeat characteristic of linear mtDNA and linea
55 load was associated with undetectable 2-long terminal repeat circles (P < .001) and HIV-negative or i
56 a smaller increase in the number of two-long terminal repeat circles than for virus specifically bloc
57 duction (ie, HIV-specific antibodies, 2-long terminal repeat circles) and markers of immune activatio
58 ompetent reservoirs, proviral DNA, or 2-long-terminal repeat circles, although APOBEC3G, TRIM5alpha,
61 013), and frequency of detectable HIV 2-long terminal repeat circular DNA (P=.013) were significantly
62 normally in MX2-expressing cells, but 2-long terminal repeat circular forms of HIV-1 DNA are less abu
63 We demonstrate that the foamy virus long terminal repeats contain an insulator element that binds
64 )-RNA strand genome of retroviruses and long terminal repeat-containing retrotransposons reflects a m
65 un-described polarity effect, in which the N-terminal repeats contribute more to affinity than C-term
66 erally true that a T/SxxH motif close to the terminal repeats contributes little or even negatively t
68 motes Paf1C recruitment by phosphorylating C-terminal repeats (CTRs) in Spt5, and we show that Kin28
69 many 2C transcripts are initiated from long terminal repeats derived from endogenous retroviruses, s
70 nd recognize an unexpected abundance of long terminal repeat-derived and LINE1-mobilized transposed e
71 considerably reduced mLANA binding to viral terminal repeat DNA as assessed by electrophoretic mobil
72 ssay wherein both lytic (oriLyt) and latent (terminal repeat) DNA replication are evaluated simultane
73 ependent on the RNA polymerase II (RNAPII) C-terminal repeat domain (CTD) and the phosphorylation of
74 we found that Bur1 phosphorylates the Spt5 C-terminal repeat domain (CTD) both in vivo and in isolate
75 ytic subunit of the major elongation-phase C-terminal repeat domain (CTD) kinase in Saccharomyces cer
76 alter this Rtf1 domain or delete the Spt5 C-terminal repeat domain (CTR) disrupt the interaction bet
80 formins, Sca2 is monomeric, but has N- and C-terminal repeat domains (NRD and CRD) that interact with
83 demonstrate that DNA hypomethylation at long terminal repeat elements representing the most recent ge
84 lf-inactivating vectors devoid of viral long-terminal-repeat enhancers have proven safe; however, tra
85 HDAC3 contribute to repression of HIV-1 long terminal repeat expression in the HeLa P4/R5 cell line m
86 ion rate included the density of exons, long terminal repeats, GC content of the gene, and DNA methyl
87 ntiviral vectors with self-inactivating long terminal repeats, have been shown to have improved safet
94 c2(jeb)) due to a murine leukemia virus long terminal repeat insertion in Lamc2 (laminin gamma2 gene)
96 n immunodeficiency virus type 1 (HIV-1) long terminal repeat is present on both ends of the integrate
97 ow that this retrotransposon with LTRs (Long Terminal Repeats) is widely distributed among the Rubiac
98 e ubiquitous presence of a double-D inverted terminal repeat (ITR) structure, which implied a mechani
99 e D sequence, from both ends of the inverted terminal repeats (ITRs) in the adeno-associated virus se
100 cated that sequences at the envelope-3' long terminal repeat junction are required for proper export
101 476-bp fragment that spans the envelope-long terminal repeat junction had activity equivalent to the
103 milar to a repeat associated with a non-long terminal repeat-like element and is often found accompan
107 t, the two orthologs similarly restrict long terminal repeat (LTR) and non-LTR retrotransposons (MusD
108 NF-kappaB sites in the U3 region of the long terminal repeat (LTR) are critical for Cav-1-mediated in
109 /EBP) beta and C/EBP sites in the HIV-1 long terminal repeat (LTR) are crucial for HIV-1 replication
111 tone proteins at the HIV type 1 (HIV-1) long terminal repeat (LTR) by histone deactylases (HDACs) can
112 were assayed for total HIV-1 DNA and 2-long terminal repeat (LTR) circles by quantitative polymerase
113 found that repressive histone marks and long terminal repeat (LTR) DNA methylation could be detected
114 to the Repbase collection of known ERV/long terminal repeat (LTR) elements to annotate the retrovira
116 e transcriptional activity of the viral long terminal repeat (LTR) from Vpr-deficient proviruses was
118 we show that methylation of the HIV 5' long terminal repeat (LTR) in the latent viral reservoir of H
119 gs suggest that integration of the XMRV long terminal repeat (LTR) into host DNA could impart androge
120 , but rather transcription of the HIV-1 long terminal repeat (LTR) is increased in IL-4-producing CD4
121 entified a transcript that contains the long terminal repeat (LTR) of lambda-olt 2-1 and shows a simi
123 dition, we detected Tip110 at the HIV-1 long terminal repeat (LTR) promoter and found that Tip110 exp
124 t-mediated transactivation of the viral long terminal repeat (LTR) promoter is essential for HIV-1 tr
126 he G-quadruplex structures in the HIV-1 long terminal repeat (LTR) promoter suppresses viral transcri
127 by the mouse mammary tumor virus (MMTV) long terminal repeat (LTR) promoter were morphologically indi
128 this cell line was specific for the MLV long terminal repeat (LTR) promoter, as normal levels of repo
131 long interspersed elements (LINEs), and long terminal repeat (LTR) retroelements, which include endog
132 igated the phylogenetic distribution of long terminal repeat (LTR) retrotransposon, long interspersed
135 recombination with specific classes of long terminal repeat (LTR) retrotransposons and organize into
136 from these isoforms appeared to target long terminal repeat (LTR) retrotransposons and other unrelat
139 B homologues have been shown to silence long terminal repeat (LTR) retrotransposons by recruiting his
140 rly twofold increase in the deletion of long terminal repeat (LTR) retrotransposons via solo LTR form
142 genous retroviruses (ERVs), also called long terminal repeat (LTR) retrotransposons, begins with tran
148 tail here the contribution of different long terminal repeat (LTR) sequences for the establishment of
149 ter phenotypically resembles endogenous long terminal repeat (LTR) sequences, pointing to a select ro
150 1) integrase (IN) with DNA representing long terminal repeat (LTR) termini was previously assembled t
151 bp insulator located in the foamy virus long terminal repeat (LTR) that has high-affinity binding to
152 activates transcription from the viral long terminal repeat (LTR) through recruitment of cellular CR
153 ; (ii) the 5' end extending from the 5' long terminal repeat (LTR) to the beginning of the capsid (CA
154 ped to the NF-kappaB sites in the HIV-1 long terminal repeat (LTR) U3 and could be transferred to MLV
155 he transcriptional activity of the XMRV long terminal repeat (LTR) was found to be higher than the Mo
156 BRG1 associates with Tax at the HTLV-1 long terminal repeat (LTR), and coexpression of BRG1 and Tax
157 s of SIV polymerase (pol), STLV tax and long terminal repeat (LTR), and SFV pol and LTR sequences rev
158 owing the reduction in H3K27 at the HIV long terminal repeat (LTR), subsequent exposure to the HDACi
159 trinsic toggling of HIV's promoter, the long terminal repeat (LTR), to generate bimodal ON-OFF expres
160 lity group protein A1 (HMGA1) and viral long terminal repeat (LTR), which led to higher levels of HIV
161 a variety of retroelements, such as the long-terminal repeat (LTR)-containing MusD and Ty1 elements,
162 ntrinsic NF-kappaB activator, increased long terminal repeat (LTR)-dependent XMRV transcription.
165 ncreases HIV infection by enhancing HIV long terminal repeat (LTR)-driven transcription via the NF-ka
166 mas transiently transfected with an HIV long terminal repeat (LTR)-luciferase reporter that contained
167 elongation complex essential for HIV-1 long terminal repeat (LTR)-mediated and general cellular tran
183 We tag hPSCs by GFP, expressed by the long terminal repeat (LTR7) of HERVH endogenous retrovirus.
184 ammaRV/LV) with self-inactivating (SIN) long terminal repeats (LTRs) and internal moderate cellular p
185 ogenous retroviruses and their solitary long terminal repeats (LTRs) compose >40% of the human genome
186 contains identical 5' and 3' peripheral long terminal repeats (LTRs) containing bidirectional promote
190 insulators, and self-inactivating (SIN) long terminal repeats (LTRs) may have significantly reduced g
191 yza genomes, and based on the dating of long terminal repeats (LTRs) of FRetro3 it was amplified in t
192 of nucleotide substitution between two long terminal repeats (LTRs) of individual orthologous LTR-re
193 trong promoter/enhancer elements in the long terminal repeats (LTRs) of murine leukemia virus (MLV)-b
194 nabled the accurate prediction of 20.5% long terminal repeats (LTRs) that doubled the previous estima
195 ichment in transposable elements (TEs): long terminal repeats (LTRs) were randomly located across the
196 tering of telomeres and retrotransposon long terminal repeats (LTRs)) were observed throughout the ce
197 s retroviral elements (ERVs) containing long terminal repeats (LTRs), are silenced through trimethyla
200 transposable elements (TEs), including Long-Terminal-Repeats (LTRs) and SINE-VNTR-Alus (SVAs), that
201 ith transgenic NF-kappaB reporters (HIV-long terminal repeat/luciferase [HLL]), we found exaggerated
207 ave the hallmarks that characterize non-long terminal repeat (non-LTR) retrotransposons; they have al
208 trotransposition activity of the L1 non-long-terminal-repeat (non-LTR) retrotransposon in both HeLa a
209 promoter is not diminished, whereas the long terminal repeat of a retrovirus, like the ICP0 promoter,
213 found TINATs to be encoded in solitary long terminal repeats of the ERV9/LTR12 family, which are epi
214 owed that a minimal region in the ARC2 and N-terminal repeats of the LRR domain coordinate the activa
215 ontrol of the mouse mammary tumor virus long terminal repeat promoter, develop multifocal hyperplasia
217 ransferase, the cysteine protease, and the C-terminal repeat region can be aligned within three domai
219 a panel of ATIp mutants indicated that the C-terminal repeat region was required for inclusion format
223 P mice, which are transgenic for both a long terminal repeat-regulated full-length infectious HIV-1 p
224 lies of long interspersed element 1 and long terminal repeat retroelements, which are disparately met
227 -silencing pathways, particularly copia long terminal repeat retrotransposon in Drosophila melanogast
231 the GP(Y/F) domain in the IN of Tf1, a long terminal repeat retrotransposon of Schizosaccharomyces p
234 Like its retroviral relatives, the long terminal repeat retrotransposon Ty1 in the yeast Sacchar
237 he Saccharomyces cerevisiae genome, the long terminal repeat retrotransposon Ty3 is found at RNA poly
238 walnut short interspersed element (SINE) and terminal-repeat retrotransposon in miniature (TRIM) elem
239 Ms2 allele in the ms2 mutant has acquired a terminal-repeat retrotransposon in miniature (TRIM) elem
243 ansposable elements (TEs), particularly long terminal repeat retrotransposons (LTR-RTs), in recombina
245 ous end joining, mediates clustering of long terminal repeat retrotransposons at centromeres in fissi
248 ure (TRIMs) are a unique group of small long terminal repeat retrotransposons that are difficult to i
249 genome due to a rapid amplification of long terminal repeat retrotransposons that occurred 38 milli
250 T. halophila were found to contain five Long Terminal Repeat retrotransposons, MuDR DNA transposons a
251 repeat clusters are almost exclusively long terminal repeat retrotransposons, of which the paleontol
253 activity predominately driven by Gypsy long terminal repeat retrotransposons, which extended the low
258 genous retroviruses [ERVs] and 488 solo long terminal repeats [sLTRs]) within the C57BL/6J mouse geno
259 lication-competent avian leukosis virus long terminal repeat, splice acceptor (RCAS)/TVA system to tr
261 ee sites are located in a region of the KSHV terminal repeat subunit previously recognized as a minim
262 ial unfolding, when allowed to refold, the C-terminal repeats successively regain structures without
263 roviruses and proviruses devoid of a 5' long terminal repeat, suggesting that the expression of HERV-
265 st five nucleosomes, is found at the 5' long terminal repeat, the location and modification state of
266 the 3' end of the coding region and the long terminal repeat, this retrotransposon family contains a
267 was further located to a position within the terminal repeat (TR) and its deletion resulted in lower
270 1,162-amino-acid protein that acts on viral terminal repeat (TR) DNA to mediate KSHV episome persist
271 is an 1,162-amino-acid protein that tethers terminal repeat (TR) DNA to mitotic chromosomes to media
272 sociated herpesvirus (KSHV) initiates at the terminal repeat (TR) element and requires trans-acting e
279 he first of a highly variable reiteration of terminal repeat (TR) sequences that are joined by random
280 bound with high occupancy to the KSHV genome terminal repeats (TR) and to a few minor binding sites i
284 btelomere features such as the start of each terminal repeat tract, SRE identity and organization, an
285 and cohesin bind within 3 kb of the start of terminal repeat tracts at many, but not all, subtelomere
286 tallography have deciphered several inverted terminal repeat-transposase complexes that are intermedi
288 on by CHART revealed EBER2's presence at the terminal repeats (TRs) of the latent EBV genome, overlap
291 (nt) pri-miRNA, encoded within the BFV long terminal repeat U3 region, that is subsequently cleaved
293 anscription factors to the HIV provirus long terminal repeat using chromatin immunoprecipitation.
294 nctional annotations (e.g. centromeres, long terminal repeats) using 3D genome reconstructions from H
295 all eight open reading frames (ORFs) in the terminal repeat was investigated in IgM(+) WBC from koi
297 s of the viral envelope gene and the 3' long terminal repeat were tested in the presence and absence
298 ells and the replication-competent ASLV long terminal repeat with a splice acceptor/tv-a glioma mouse
299 d specific DNA sequences containing inverted terminal repeats with similarity to piggyBac transposons
300 iven the shift toward self-inactivating long terminal repeats with weaker promoters to control the ri
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