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1 er sequences in the U3 region of the 3' long terminal repeat.
2 d eight, respectively, are duplicated in the terminal repeat.
3 ing of Tat at the TAR region of the HIV long terminal repeat.
4  differential DNA methylation at its 5'-long terminal repeat.
5 stranded RNA40 (ssRNA40) from the HIV-1 long terminal repeat.
6 obin locus within the self-inactivating long-terminal repeat.
7 F-kappaB and its recruitment to the HIV-long terminal repeat.
8 on through HIF-1 association with HIV-1 long terminal repeat.
9 lls using the mouse mammary tumor virus long terminal repeat.
10 IV Tat protein to transactivate the HIV long terminal repeat.
11  histone-modifying enzymes to the HIV-1 long terminal repeat.
12 form the capsid, and the right-hand inverted terminal repeat.
13 nt downstream transcription from the 3' long terminal repeat.
14 x that is associated with repressed HIV long terminal repeats.
15 ked by adeno-associated virus (AAV) inverted terminal repeats.
16  showed binding of RPA1 and RPA2 to the KSHV terminal repeats.
17 tures, followed by the nucleation of three N-terminal repeats.
18 gase, and this property is mediated by the C-terminal repeats.
19 aps that protect the hydrophobic core of the terminal repeats.
20 ipts is promoted by endogenous intronic long terminal repeats.
21 anded DNA (dsDNA) genome with large inverted terminal repeats.
22 y differ by the presence of 3(3R) or 4(4R) C-terminal repeats.
23 tor genomes that consisted of linear, 1-long-terminal-repeat (1-LTR), and 2-LTR circular DNAs.
24  transcripts, a moderate reduction of 2-long terminal repeat (2-LTR) circles, and a relatively large
25                  HIV-1 DNA, including 2-long terminal repeat (2-LTR) circles, and multiply spliced (m
26                                     Two-long terminal repeat (2-LTR) circles, which are formed in the
27 efined by an increase in the level of 2-long terminal repeat (2-LTR) circles.
28 DNA to circularization in the form of 2-long terminal repeat (2-LTR) circles.
29 al HIV DNA, integrated HIV DNA, and two long terminal repeat (2-LTR) circles.
30 nal plasma CMV (P = 0.04), detectable 2-long terminal repeat (2-LTR), and lower nadir CD4(+) (P < 0.0
31                        An increase in 2-long-terminal-repeat (2-LTR) circles in the depleted FACT com
32 egrated viral DNA, particularly the two-long-terminal-repeat (2-LTR) circles.
33 omes containing two copies of the viral long terminal repeat (2LTR circles) were analyzed in using dr
34 extension at the viral U5 end of the 3' long terminal repeat (3'-LTR), which is a poor substrate for
35  and eIF3f specifically targeted the 3' long terminal repeat (3'LTR) region in the viral mRNA.
36 litates repression by LANA bound to the KSHV terminal repeats, a function that requires the MeCP2 C t
37 ugs and tested these compounds on HIV-1 long terminal repeat-activated transcription.
38                         Two degenerate amino-terminal repeats also interact with the DNA.
39 subunits (named CasC1-6), with its 5' and 3' terminal repeats anchored by CasD and CasE, respectively
40 semble that is highly polarized toward the C-terminal repeat and cap.
41      Both P-TEFb recruitment to the HIV long terminal repeat and enhanced HIV processivity were block
42 d cryptic splice sites in the HIV 5iota long terminal repeat and gag gene as well as in the beta-glob
43 rictive chromatin structures at the HIV long terminal repeat and limiting P-TEFb levels contribute to
44 ons in the regulatory elements in their long terminal repeats and differed in a helical segment of en
45 eviously overlooked partly because they lack terminal repeats and do not create target site duplicati
46     The DNA of the element is marked by long terminal repeats and encodes a single large protein with
47 NMT3a/3b in suppressing retrotransposon long terminal repeats and long interspersed elements, respect
48  repetitive elements such as Alu, LINE, long-terminal repeats and simple tandem repeats are frequentl
49 ted replication fork protection at the viral terminal repeats and that Tim-dependent recombination-li
50 tational analysis of the HERV-K (HML-2) long terminal repeat, and treatments with agents that inhibit
51 ranulatus genome, especially Gypsy-like long terminal repeats, and there has also been an expansion i
52 ut any nucleation step by using the intact N-terminal repeats as a template.
53 c gene family expansions, the number of long-terminal-repeat-based retrotransposons, and mechanisms o
54 est of the genes, forming a part of inverted terminal repeat characteristic of linear mtDNA and linea
55 load was associated with undetectable 2-long terminal repeat circles (P < .001) and HIV-negative or i
56 a smaller increase in the number of two-long terminal repeat circles than for virus specifically bloc
57 duction (ie, HIV-specific antibodies, 2-long terminal repeat circles) and markers of immune activatio
58 ompetent reservoirs, proviral DNA, or 2-long-terminal repeat circles, although APOBEC3G, TRIM5alpha,
59  RNA transcription or more detectable 2-long terminal repeat circles.
60 y processed viral ends and abortive two-long-terminal-repeat circles.
61 013), and frequency of detectable HIV 2-long terminal repeat circular DNA (P=.013) were significantly
62 normally in MX2-expressing cells, but 2-long terminal repeat circular forms of HIV-1 DNA are less abu
63     We demonstrate that the foamy virus long terminal repeats contain an insulator element that binds
64 )-RNA strand genome of retroviruses and long terminal repeat-containing retrotransposons reflects a m
65 un-described polarity effect, in which the N-terminal repeats contribute more to affinity than C-term
66 erally true that a T/SxxH motif close to the terminal repeats contributes little or even negatively t
67                  We further found that the C-terminal repeat (CTR) domain of Spt5, which is dispensab
68 motes Paf1C recruitment by phosphorylating C-terminal repeats (CTRs) in Spt5, and we show that Kin28
69  many 2C transcripts are initiated from long terminal repeats derived from endogenous retroviruses, s
70 nd recognize an unexpected abundance of long terminal repeat-derived and LINE1-mobilized transposed e
71  considerably reduced mLANA binding to viral terminal repeat DNA as assessed by electrophoretic mobil
72 ssay wherein both lytic (oriLyt) and latent (terminal repeat) DNA replication are evaluated simultane
73 ependent on the RNA polymerase II (RNAPII) C-terminal repeat domain (CTD) and the phosphorylation of
74 we found that Bur1 phosphorylates the Spt5 C-terminal repeat domain (CTD) both in vivo and in isolate
75 ytic subunit of the major elongation-phase C-terminal repeat domain (CTD) kinase in Saccharomyces cer
76  alter this Rtf1 domain or delete the Spt5 C-terminal repeat domain (CTR) disrupt the interaction bet
77 n with an N-terminal WD domain (WDD) and a C-terminal repeat domain (RPD).
78          The NMR solution structure of the N-terminal repeat domain of ClpC (N-ClpCR) comprises two s
79 dicated that Egf1.0 binds PAPs through its C-terminal repeat domain.
80 formins, Sca2 is monomeric, but has N- and C-terminal repeat domains (NRD and CRD) that interact with
81                          We show that a long terminal repeat element inserted into intron 35 exposes
82 e as well as the transposon cargo flanked by terminal repeat element sequences.
83 demonstrate that DNA hypomethylation at long terminal repeat elements representing the most recent ge
84 lf-inactivating vectors devoid of viral long-terminal-repeat enhancers have proven safe; however, tra
85 HDAC3 contribute to repression of HIV-1 long terminal repeat expression in the HeLa P4/R5 cell line m
86 ion rate included the density of exons, long terminal repeats, GC content of the gene, and DNA methyl
87 ntiviral vectors with self-inactivating long terminal repeats, have been shown to have improved safet
88                   (iii) However, the carboxy-terminal repeat in the VEEV HVD is indispensable for VEE
89 Tat was unable to transactivate the HIV long terminal repeat in U937 human macrophages.
90  shock factor binding HREs within their long terminal repeats in seven Brassicaceae species.
91                           Insertions of long terminal repeats in the past 5 million years are respons
92 BRCT repeats (breast cancer gene 1 [BRCA1] C terminal repeats) in the Dpb11 protein [5, 6].
93  of unique antibodies identified bound the C-terminal repeat-in-toxin (RTX) domain.
94 c2(jeb)) due to a murine leukemia virus long terminal repeat insertion in Lamc2 (laminin gamma2 gene)
95 ep 78/68 polypeptide in conjunction with AAV terminal repeat integrating elements.
96 n immunodeficiency virus type 1 (HIV-1) long terminal repeat is present on both ends of the integrate
97 ow that this retrotransposon with LTRs (Long Terminal Repeats) is widely distributed among the Rubiac
98 e ubiquitous presence of a double-D inverted terminal repeat (ITR) structure, which implied a mechani
99 e D sequence, from both ends of the inverted terminal repeats (ITRs) in the adeno-associated virus se
100 cated that sequences at the envelope-3' long terminal repeat junction are required for proper export
101 476-bp fragment that spans the envelope-long terminal repeat junction had activity equivalent to the
102               Using the analysis of two long terminal repeat junctions in HIV-infected cells, we show
103 milar to a repeat associated with a non-long terminal repeat-like element and is often found accompan
104 tant inhibition of NF-kappaB and HTLV-1 long terminal repeat (LTR) activation.
105 s have been reported to stimulate HIV-1 long terminal repeat (LTR) activity.
106                               : MGEScan-long terminal repeat (LTR) and MGEScan-non-LTR are successful
107 t, the two orthologs similarly restrict long terminal repeat (LTR) and non-LTR retrotransposons (MusD
108 NF-kappaB sites in the U3 region of the long terminal repeat (LTR) are critical for Cav-1-mediated in
109 /EBP) beta and C/EBP sites in the HIV-1 long terminal repeat (LTR) are crucial for HIV-1 replication
110 e RNA polymerase II (RNAP II) on the 5' long terminal repeat (LTR) but not on the 3' LTR.
111 tone proteins at the HIV type 1 (HIV-1) long terminal repeat (LTR) by histone deactylases (HDACs) can
112  were assayed for total HIV-1 DNA and 2-long terminal repeat (LTR) circles by quantitative polymerase
113 found that repressive histone marks and long terminal repeat (LTR) DNA methylation could be detected
114  to the Repbase collection of known ERV/long terminal repeat (LTR) elements to annotate the retrovira
115  or octamer complex with the viral cDNA long terminal repeat (LTR) ends termed an intasome.
116 e transcriptional activity of the viral long terminal repeat (LTR) from Vpr-deficient proviruses was
117 ut 11.7% with intact genomes and normal long terminal repeat (LTR) function.
118  we show that methylation of the HIV 5' long terminal repeat (LTR) in the latent viral reservoir of H
119 gs suggest that integration of the XMRV long terminal repeat (LTR) into host DNA could impart androge
120 , but rather transcription of the HIV-1 long terminal repeat (LTR) is increased in IL-4-producing CD4
121 entified a transcript that contains the long terminal repeat (LTR) of lambda-olt 2-1 and shows a simi
122  demonstrate that HspBP1 inhibits HIV-1 long terminal repeat (LTR) promoter activity.
123 dition, we detected Tip110 at the HIV-1 long terminal repeat (LTR) promoter and found that Tip110 exp
124 t-mediated transactivation of the viral long terminal repeat (LTR) promoter is essential for HIV-1 tr
125 ng of viral transcription driven by the long terminal repeat (LTR) promoter of HIV-1.
126 he G-quadruplex structures in the HIV-1 long terminal repeat (LTR) promoter suppresses viral transcri
127 by the mouse mammary tumor virus (MMTV) long terminal repeat (LTR) promoter were morphologically indi
128 this cell line was specific for the MLV long terminal repeat (LTR) promoter, as normal levels of repo
129  by the transcriptional activity of its long terminal repeat (LTR) region.
130 coding the viral proteins is flanked by long terminal repeat (LTR) regions from the retrovirus.
131 long interspersed elements (LINEs), and long terminal repeat (LTR) retroelements, which include endog
132 igated the phylogenetic distribution of long terminal repeat (LTR) retrotransposon, long interspersed
133 cing of all 13 intact copies of the Tf2 long terminal repeat (LTR) retrotransposon.
134 s known repeats including a majority of long terminal repeat (LTR) retrotransposons (13.67%).
135  recombination with specific classes of long terminal repeat (LTR) retrotransposons and organize into
136  from these isoforms appeared to target long terminal repeat (LTR) retrotransposons and other unrelat
137                                         Long terminal repeat (LTR) retrotransposons are an abundant c
138                                         Long terminal repeat (LTR) retrotransposons are closely relat
139 B homologues have been shown to silence long terminal repeat (LTR) retrotransposons by recruiting his
140 rly twofold increase in the deletion of long terminal repeat (LTR) retrotransposons via solo LTR form
141                            BEL/Pao-like long-terminal repeat (LTR) retrotransposons were annotated fr
142 genous retroviruses (ERVs), also called long terminal repeat (LTR) retrotransposons, begins with tran
143                                         Long terminal repeat (LTR) retrotransposons, the most abundan
144 me surveillance role by controlling Tf2 long terminal repeat (LTR) retrotransposons.
145 transposable elements, 77% of which are long terminal repeat (LTR) retrotransposons.
146 dant elements in eukaryotes are the non long terminal repeat (LTR) retrotransposons.
147 ithin the Ty3/gypsy-like superfamily of long terminal repeat (LTR) retrotransposons.
148 tail here the contribution of different long terminal repeat (LTR) sequences for the establishment of
149 ter phenotypically resembles endogenous long terminal repeat (LTR) sequences, pointing to a select ro
150 1) integrase (IN) with DNA representing long terminal repeat (LTR) termini was previously assembled t
151 bp insulator located in the foamy virus long terminal repeat (LTR) that has high-affinity binding to
152  activates transcription from the viral long terminal repeat (LTR) through recruitment of cellular CR
153 ; (ii) the 5' end extending from the 5' long terminal repeat (LTR) to the beginning of the capsid (CA
154 ped to the NF-kappaB sites in the HIV-1 long terminal repeat (LTR) U3 and could be transferred to MLV
155 he transcriptional activity of the XMRV long terminal repeat (LTR) was found to be higher than the Mo
156  BRG1 associates with Tax at the HTLV-1 long terminal repeat (LTR), and coexpression of BRG1 and Tax
157 s of SIV polymerase (pol), STLV tax and long terminal repeat (LTR), and SFV pol and LTR sequences rev
158 owing the reduction in H3K27 at the HIV long terminal repeat (LTR), subsequent exposure to the HDACi
159 trinsic toggling of HIV's promoter, the long terminal repeat (LTR), to generate bimodal ON-OFF expres
160 lity group protein A1 (HMGA1) and viral long terminal repeat (LTR), which led to higher levels of HIV
161 a variety of retroelements, such as the long-terminal repeat (LTR)-containing MusD and Ty1 elements,
162 ntrinsic NF-kappaB activator, increased long terminal repeat (LTR)-dependent XMRV transcription.
163                            We show that long terminal repeat (LTR)-derived transcripts contribute ext
164               We found two instances of long terminal repeat (LTR)-driven provirus transcription but
165 ncreases HIV infection by enhancing HIV long terminal repeat (LTR)-driven transcription via the NF-ka
166 mas transiently transfected with an HIV long terminal repeat (LTR)-luciferase reporter that contained
167  elongation complex essential for HIV-1 long terminal repeat (LTR)-mediated and general cellular tran
168                           In the mouse, long terminal repeat (LTR)-retrotransposons, or endogenous re
169 ntromeric sequence, uncovered 45 intact long terminal repeat (LTR)-retrotransposons.
170 E, in the surface glycoprotein (SU) and long terminal repeat (LTR).
171 ranscription initiation site on the HIV long terminal repeat (LTR).
172 lation of histones located at the viral long terminal repeat (LTR).
173 me2) that colocalizes with a retroviral long terminal repeat (LTR).
174 ment (HRE) localized in the proviral 5' long terminal repeat (LTR).
175 s well as the coactivator CBP, with the long terminal repeat (LTR).
176 IM1 complex for activation of the viral long terminal repeat (LTR).
177 he CD28-responsive element of the HIV-1 long terminal repeat (LTR).
178 tivating the HIV-1 promoter, termed the long terminal repeat (LTR).
179 sed nuclear elements (LINE), but not in long terminal repeats (LTR).
180 ects on nuclear histone acetylation and long-terminal-repeat (LTR) transcription.
181 rom their well-characterized effects of long-terminal-repeat (LTR)-driven gene expression.
182 tion of, the integrated viral promoter (long terminal repeat [LTR]).
183   We tag hPSCs by GFP, expressed by the long terminal repeat (LTR7) of HERVH endogenous retrovirus.
184 ammaRV/LV) with self-inactivating (SIN) long terminal repeats (LTRs) and internal moderate cellular p
185 ogenous retroviruses and their solitary long terminal repeats (LTRs) compose >40% of the human genome
186 contains identical 5' and 3' peripheral long terminal repeats (LTRs) containing bidirectional promote
187  intrachromosomal recombination between long terminal repeats (LTRs) flanking GAP1.
188            Retrotransposons often carry long terminal repeats (LTRs) for retrovirus-like reverse tran
189             Retrotransposons containing long terminal repeats (LTRs) form a substantial fraction of e
190 insulators, and self-inactivating (SIN) long terminal repeats (LTRs) may have significantly reduced g
191 yza genomes, and based on the dating of long terminal repeats (LTRs) of FRetro3 it was amplified in t
192  of nucleotide substitution between two long terminal repeats (LTRs) of individual orthologous LTR-re
193 trong promoter/enhancer elements in the long terminal repeats (LTRs) of murine leukemia virus (MLV)-b
194 nabled the accurate prediction of 20.5% long terminal repeats (LTRs) that doubled the previous estima
195 ichment in transposable elements (TEs): long terminal repeats (LTRs) were randomly located across the
196 tering of telomeres and retrotransposon long terminal repeats (LTRs)) were observed throughout the ce
197 s retroviral elements (ERVs) containing long terminal repeats (LTRs), are silenced through trimethyla
198 tains identical DNA sequences, known as long terminal repeats (LTRs), at its 5' and 3' ends.
199 his is flanked by two loxP sites in its long terminal repeats (LTRs).
200  transposable elements (TEs), including Long-Terminal-Repeats (LTRs) and SINE-VNTR-Alus (SVAs), that
201 ith transgenic NF-kappaB reporters (HIV-long terminal repeat/luciferase [HLL]), we found exaggerated
202 of the mouse mammary tumor virus (MMTV) long-terminal repeat (MMT mice).
203                                 The non-long terminal repeat (non-LTR) retrotransposon R2 is inserted
204                                     Non-long terminal repeat (non-LTR) retrotransposons are a class o
205                                     Non-long terminal repeat (non-LTR) retrotransposons are highly ab
206                                Many non-long terminal repeat (non-LTR) retrotransposons lack internal
207 ave the hallmarks that characterize non-long terminal repeat (non-LTR) retrotransposons; they have al
208 trotransposition activity of the L1 non-long-terminal-repeat (non-LTR) retrotransposon in both HeLa a
209 promoter is not diminished, whereas the long terminal repeat of a retrovirus, like the ICP0 promoter,
210       Additionally, mutation of the inverted terminal repeat of the rAAV genome, which has been propo
211  breakpoint precisely located at an inverted terminal repeat of the retained starting element.
212                                     The long terminal repeats of lymphomagenic P-MLVs are differentia
213  found TINATs to be encoded in solitary long terminal repeats of the ERV9/LTR12 family, which are epi
214 owed that a minimal region in the ARC2 and N-terminal repeats of the LRR domain coordinate the activa
215 ontrol of the mouse mammary tumor virus long terminal repeat promoter, develop multifocal hyperplasia
216 rotein enhances Tat-mediated HIV-1 LTR (long terminal repeat) promoter activity.
217 ransferase, the cysteine protease, and the C-terminal repeat region can be aligned within three domai
218                                        The C-terminal repeat region of Spt5, which has been implicate
219 a panel of ATIp mutants indicated that the C-terminal repeat region was required for inclusion format
220  coverage was obtained in the highly GC-rich terminal repeat regions.
221  the resulting BAC had only one of the 18-kb terminal repeated regions.
222 , generating DNA products with heterogeneous terminal repeat registers.
223 P mice, which are transgenic for both a long terminal repeat-regulated full-length infectious HIV-1 p
224 lies of long interspersed element 1 and long terminal repeat retroelements, which are disparately met
225                                   Large long terminal repeat retrotransposon clusters occupy signific
226 ecules, including coverage of the major Long Terminal Repeat retrotransposon families.
227 -silencing pathways, particularly copia long terminal repeat retrotransposon in Drosophila melanogast
228                                  Tf1, a long-terminal repeat retrotransposon in Schizosaccharomyces p
229              Previous studies of Tf1, a long terminal repeat retrotransposon in Schizosaccharomyces p
230                                  Ty1, a long terminal repeat retrotransposon of Saccharomyces, is str
231  the GP(Y/F) domain in the IN of Tf1, a long terminal repeat retrotransposon of Schizosaccharomyces p
232  gene duplication event mediated by the long terminal repeat retrotransposon Rider.
233               This reduced capacity for long terminal repeat retrotransposon silencing and removal in
234      Like its retroviral relatives, the long terminal repeat retrotransposon Ty1 in the yeast Sacchar
235            The Saccharomyces cerevisiae long terminal repeat retrotransposon Ty3 assembles its Gag3 a
236            The Saccharomyces cerevisiae long terminal repeat retrotransposon Ty3 integrates within on
237 he Saccharomyces cerevisiae genome, the long terminal repeat retrotransposon Ty3 is found at RNA poly
238 walnut short interspersed element (SINE) and terminal-repeat retrotransposon in miniature (TRIM) elem
239  Ms2 allele in the ms2 mutant has acquired a terminal-repeat retrotransposon in miniature (TRIM) elem
240           We initially investigated 510 long terminal repeat-retrotransposon (LTR-RT) families compri
241                           Analyzing the long terminal repeat-retrotransposon (LTR-RT) type of TE, we
242                                         Long terminal repeat retrotransposons (LTR-RTs) are prevalent
243 ansposable elements (TEs), particularly long terminal repeat retrotransposons (LTR-RTs), in recombina
244 marily by the periodic amplification of long terminal repeat retrotransposons (LTR-RTs).
245 ous end joining, mediates clustering of long terminal repeat retrotransposons at centromeres in fissi
246                                              Terminal repeat retrotransposons in miniature (TRIMs) ar
247 and evolutionary and genomic studies of long terminal repeat retrotransposons in other genomes.
248 ure (TRIMs) are a unique group of small long terminal repeat retrotransposons that are difficult to i
249  genome due to a rapid amplification of long terminal repeat retrotransposons that occurred 38 milli
250 T. halophila were found to contain five Long Terminal Repeat retrotransposons, MuDR DNA transposons a
251  repeat clusters are almost exclusively long terminal repeat retrotransposons, of which the paleontol
252                            Unlike other long terminal repeat retrotransposons, TRIMs are enriched in
253  activity predominately driven by Gypsy long terminal repeat retrotransposons, which extended the low
254 y of the replication strategies used by long terminal repeat retrotransposons.
255 erived from internal deletions of large long terminal repeat retrotransposons.
256                   Structural analysis of EBV terminal repeats revealed a banding pattern consistent w
257               Finally, analysis of KERV long terminal repeat sequences using massively parallel seque
258 genous retroviruses [ERVs] and 488 solo long terminal repeats [sLTRs]) within the C57BL/6J mouse geno
259 lication-competent avian leukosis virus long terminal repeat, splice acceptor (RCAS)/TVA system to tr
260                                        Short terminal repeats (STRs) were performed simultaneously on
261 ee sites are located in a region of the KSHV terminal repeat subunit previously recognized as a minim
262 ial unfolding, when allowed to refold, the C-terminal repeats successively regain structures without
263 roviruses and proviruses devoid of a 5' long terminal repeat, suggesting that the expression of HERV-
264 all single-stranded DNA genome with inverted terminal repeats that form hairpin structures.
265 st five nucleosomes, is found at the 5' long terminal repeat, the location and modification state of
266 the 3' end of the coding region and the long terminal repeat, this retrotransposon family contains a
267 was further located to a position within the terminal repeat (TR) and its deletion resulted in lower
268                           LANA binds to KSHV terminal repeat (TR) DNA and simultaneously associates w
269                           LANA binds to KSHV terminal repeat (TR) DNA and tethers the viral episomes
270  1,162-amino-acid protein that acts on viral terminal repeat (TR) DNA to mediate KSHV episome persist
271  is an 1,162-amino-acid protein that tethers terminal repeat (TR) DNA to mitotic chromosomes to media
272 sociated herpesvirus (KSHV) initiates at the terminal repeat (TR) element and requires trans-acting e
273          The prevalence of piggyBac(PB)-like terminal repeat (TR) elements in the human genome raises
274                        LANA acts on the KSHV terminal repeat (TR) elements to mediate KSHV episome ma
275              LANA binds cooperatively to the terminal repeat (TR) region of the viral episome via adj
276 d LANA-mediated recruitment of NAP1L1 at the terminal repeat (TR) region of the viral genome.
277 d nuclear antigen (LANA), which binds in the terminal repeat (TR) region of the viral genome.
278 with the BAC vector cassette inserted in the terminal repeat (TR) region.
279 he first of a highly variable reiteration of terminal repeat (TR) sequences that are joined by random
280 bound with high occupancy to the KSHV genome terminal repeats (TR) and to a few minor binding sites i
281 s (KSHV) genomes is mediated in cis by their terminal repeats (TR).
282 f the KSHV-encoded LANA protein to the viral terminal repeats (TR).
283            LANA self-associates to bind KSHV terminal-repeat (TR) DNA and to mediate its replication.
284 btelomere features such as the start of each terminal repeat tract, SRE identity and organization, an
285 and cohesin bind within 3 kb of the start of terminal repeat tracts at many, but not all, subtelomere
286 tallography have deciphered several inverted terminal repeat-transposase complexes that are intermedi
287 y vectors package a transgene flanked by the terminal repeats (TRs) of AAV type 2 (AAV2).
288 on by CHART revealed EBER2's presence at the terminal repeats (TRs) of the latent EBV genome, overlap
289  the latent origin, which resides within the terminal repeats (TRs) of the viral genome.
290 esulted in reduced binding of LANA with KSHV terminal repeats (TRs).
291  (nt) pri-miRNA, encoded within the BFV long terminal repeat U3 region, that is subsequently cleaved
292 red to the cell surface is mediated by the C-terminal repeating units.
293 anscription factors to the HIV provirus long terminal repeat using chromatin immunoprecipitation.
294 nctional annotations (e.g. centromeres, long terminal repeats) using 3D genome reconstructions from H
295  all eight open reading frames (ORFs) in the terminal repeat was investigated in IgM(+) WBC from koi
296                           A pair of inverted terminal repeats was detected in YSLPV1, suggesting that
297 s of the viral envelope gene and the 3' long terminal repeat were tested in the presence and absence
298 ells and the replication-competent ASLV long terminal repeat with a splice acceptor/tv-a glioma mouse
299 d specific DNA sequences containing inverted terminal repeats with similarity to piggyBac transposons
300 iven the shift toward self-inactivating long terminal repeats with weaker promoters to control the ri

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