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1 a way that is dependent on the AAV inverted terminal repeat sequence.
2 FTR gene was expressed from the AAV inverted terminal repeat sequence.
3 onsible for addition of the species-specific terminal repeat sequence.
4 by a kinetic PCR that quantitates HIV-1 long terminal repeat sequences.
5 consensus sequence in both protein and long terminal repeat sequences.
6 d IRs, Ad packaging signals, and Ad inverted terminal repeat sequences.
7 region flanked by multiple G + C-rich 801-bp terminal repeat sequences.
8 and extensive rearrangements in the inverted terminal repeat sequences.
9 nique coding region (LUR) flanked by GC-rich terminal repeat sequences.
10 s, as well as nearly identical flanking long terminal repeat sequences.
11 nversions, introduced into the HIV-1 U5 long terminal repeat sequence adjacent to and/or including th
12 primers generated by RNase H within the long terminal repeat sequence are found to have the capacity
15 , within viral constructs that lack inverted terminal repeat sequences, displays a high basal level o
17 duced splicing error that substitutes a long terminal repeat sequence for the carboxy-terminus of MIP
21 V-1 genomes (lacking less than 80 bp of long terminal repeat sequences) from primary isolates collect
22 d to wild-type virus, while exchange of long terminal repeat sequences had no significant effect.
24 tion is important for generation of the long terminal repeat sequences in the duplex DNA product of r
25 a reaction mediated by base-pairing between terminal repeat sequences in the RNA and their complemen
26 ing that recognition of the ends of the long terminal repeat sequence is required only in the early s
27 eneration Ad vectors containing AAV inverted terminal repeat sequences (ITRs) flanking a reporter gen
28 pression cassettes of interest with inverted terminal repeat sequences (ITRs) from adeno-associated v
29 e undertook a phylogenetic study of the long terminal repeat sequences of 15 HERV-K(HML-2) elements i
30 e YF vaccines showed no evidence of the long terminal repeat sequences of exogenous ALV subgroups A t
31 hat Himar1 transposase binds to the inverted terminal repeat sequences of its cognate transposon and
32 further increased by including the inverted terminal repeat sequences of the adeno-associated virus
33 ough Rep-mediated excision utilizing the AAV terminal repeat sequences present in the Ad/AAV hybrid v
34 immunoglobulin gene rearrangements and viral terminal repeat sequences revealed that the cell line an
36 -89.6P were limited to the env, tat, or long terminal repeat sequences, with most of the observed cha
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