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1 and was 30-fold above control levels at the terminal stage.
2 p53 or INK4a mutations upon progression to a terminal stage.
3 r myelin mutants, were not obvious until the terminal stage.
4 wn about SMG cell differentiation during the terminal stages.
5 ive, involving the entire cerebral mantle in terminal stages.
7 stic technology, roughly 80% of cases are in terminal stages at the time of diagnosis, and a large pr
9 oplasts contain both OspA and OspB and are a terminal stage in the bactericidal process induced by Fa
11 ur studies suggest that TIG3 facilitates the terminal stages in keratinocyte differentiation via acti
12 10,000-fold difference between SY and FU at terminal stages, indicating that PrP-res content does no
13 of congenic BALB/c.lambdaLIZ N5 mice in the terminal stage of a plasmacytoma induction experiment, 2
17 also facilitates the membrane fusion at the terminal stage of cytokinesis by controlling the localiz
20 entrosome-related abscission site during the terminal stage of cytokinesis, implicating RalA as a cri
22 t direct activation of genes involved in the terminal stage of differentiation may cause neuronal dif
25 ns in approximately 130 days and reached the terminal stage of disease in approximately 150 days.
32 nge alcohol accentuates loss of body mass at terminal stage of simian immunodeficiency virus (SIV) in
33 icles (PSVs) with the plasma membrane at the terminal stage of spore differentiation in Dictyostelium
34 mation of the membrane attack complex at the terminal stage of the complement activation cascades via
35 levels of human CD59, a CRP that acts at the terminal stage of the complement cascade, we present evi
37 a transcriptional repressor that acts at the terminal stage of the neuronal differentiation pathway a
39 eed conditions that mark the initial and the terminal stages of a given differentiation pathway and c
45 are a set of enzymes that participate in the terminal stages of bacterial peptidoglycan assembly.
46 were used to characterize progression of the terminal stages of Caulobacter crescentus cell division.
48 (CTBs) revealed that HCMV infection impedes terminal stages of differentiation and invasion by vario
49 trations decreased to undetectable levels at terminal stages of disease in animals that succumbed to
51 rain tissues, edema was detected only at the terminal stages of disease, likely caused by the leakage
55 rized and is believed to be critical for the terminal stages of erythroid differentiation, but the ad
56 proliferation to differentiation during the terminal stages of erythropoiesis is a tightly controlle
58 hosphatase inhibitor okadaic acid during the terminal stages of expression to create a hyperphosphory
60 orter to hydrolyze Glc-6-P to glucose in the terminal stages of glycogenolysis and gluconeogenesis.
61 bits a selective role being required for the terminal stages of granulocyte development but is dispen
64 uent reason for sedation was delirium in the terminal stages of illness (median, 57.1%; range, 13.8%
65 ted rhesus macaques, at the asymptomatic and terminal stages of infection, were cultured in ethanol 2
66 nce of spontaneous diabetes, 5) arrested the terminal stages of islet cell destruction, and 6) disrup
69 provide an understanding of tumor growth at terminal stages of malignancy, they do not address tumor
70 ptors may have a specific role in regulating terminal stages of myelination, such as myelin membrane
71 iferation leading to astrogliosis during the terminal stages of neurodegeneration in Sandhoff disease
73 hat receptor expression is restricted to the terminal stages of olfactory neuron differentiation, and
77 e megakaryocytes resulted in a defect in the terminal stages of platelet production and had a mild ef
85 expression patterns between the initial and terminal stages of the differentiation pathway, coupled
86 The paradox is that with the exception of terminal stages of the disease or chemotherapy treatment
88 ed metal enrichment events coincide with the terminal stages of Tiwanaku culture (1000 to 1200 A.D.)
90 ficult to identify by analyzing cells within terminal stage tumors, whose identity could be concealed
91 nd retinal pigment epithelium in all but the terminal stages, when subtle changes were noted within t
92 of non-GABAergic to GABAergic neurons in the terminal stages, which implies that non-GABAergic neuron
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