ライフサイエンスコーパス: ternary complex factor

1 k-1 transcription factor, a component of the ternary complex factor.

2 including the serum response factor and the ternary complex factor.

3 Elk-1, a principal ternary complex factor and Ets domain-containing protein

4 transcription factor Elk-1 is a component of ternary complex factor and regulates gene expression in

5 ubgroup of ETS proteins that can function as ternary complex factors and further implicate a novel fu

6 cluding the serum response factor (SRF), the ternary complex factor, and the CCAAT/enhancer-binding p

7 SRE, including serum response factor (SRF), ternary complex factors, and CCAAT/enhancer-binding prot

8 or (SRF), which functions with an ETS domain ternary complex factor as a growth-factor-activated tran

9 Therefore, like the ternary complex factor, C/EBPbeta may be recruited to th

10 -activated protein (MAP) kinase activity and ternary complex factor-dependent transcription.

11 tion of ribosomal S6 kinase-1 (Rsk-1) or the ternary complex factor Elk-1 (TCF/Elk-1), both of which

12 factor (EGF)-induced transactivation of the ternary complex factor Elk-1 using a GAL4-Elk-1 construc

13 sident nuclear proteins that bind them (e.g. ternary complex factor/ELK1; serum response factor, cAMP

14 that create binding motifs for E-twenty six/ternary complex factors (Ets/TCF), affected 65.4% of the

15 C/EBPbeta can interact with both SRF and the ternary complex factor family member Elk-1, but only in

16 ctor activity, focusing on the ERK-regulated ternary complex factor family of SRF partner proteins.

17 ia JNK/SAPK-mediated phosphorylation of TCF (ternary complex factor), for c-fos induction [6-8], and

18 An activated ternary complex factor has a similar effect.

19 sphorylation, substrate phosphorylation, and ternary complex factor-independent gene expression.

20 ignal transduction pathway that leads to the ternary complex factor-independent transcriptional activ

21 of a subfamily of ETS domain proteins called ternary complex factors, it is only Elk-1 but not SAP-1

22 e and a non-mitogen-activated protein kinase/ternary complex factor pathway that targets the serum re

23 ogen-activated protein kinase activation and ternary complex factor phosphorylation.

24 together, this suggests that MIP1 acts as a ternary complex factor specifically with C-function and

25 e present study demonstrates that of several ternary complex factor subfamily members, only Elk-3 rep

26 mediators of inflammatory responses, and the ternary complex factor subfamily of Ets proteins has bot

27 to ELK1, an ETS transcription factor of the ternary complex factor subfamily.

28 nown to bind Serum Response Factor (SRF) and ternary complex factors such as Elk-1.

29 es the serum response factor (SRF), a 62 kDa ternary complex factor (TCF) and a serum response elemen

30 It forms a complex with the ternary complex factor (TCF) and with a dimer of the ser

31 tor (SRF) binding element (CArG box) and the ternary complex factor (TCF) binding element (Ets box).

32 fos transcription, and in HeLa TK- cells the Ternary Complex Factor (TCF) binding site contributes su

33 -activated protein kinase phosphorylation of ternary complex factor (TCF) but is also regulated by a

34 show that a dominant-negative mutant of the ternary complex factor (TCF) Elk-1 attenuated the upregu

35 c-fos promoter, SRF recruits members of the ternary complex factor (TCF) family of Ets domain protei

36 Both MAL and the ternary complex factor (TCF) family of SRF cofactors int

37 y contain a binding site for a member of the ternary complex factor (TCF) family.

38 /EBPbeta is dependent on SRF binding but not ternary complex factor (TCF) formation.

39 DNA binding by the ternary complex factor (TCF) subfamily of ETS-domain tra

40 The ternary complex factor (TCF) subfamily of ETS-domain tra

41 The ternary complex factor (TCF) subfamily of ETS-transcript

42 against Elk-1, suggesting that it contains a ternary complex factor (TCF) such as Elk-1 in addition t

43 x comprising the serum response factor and a ternary complex factor (TCF).

44 as-induced phosphorylation and activation of ternary complex factors (TCF), physiological substrates

45 The ERK-regulated ternary complex factors (TCFs) act with the transcriptio

46 ular signal-regulated kinase (ERK)-regulated ternary complex factors (TCFs) and the actin-regulated m

47 Ternary complex factors (TCFs) bind to the serum respons

48 e transcriptional coactivator myocardin, and ternary complex factors (TCFs) of the ETS-domain family,

49 s downstream of TAOs, the effects of TAO2 on ternary complex factors (TCFs) were investigated.

50 e also been implicated in IEG induction, the ternary complex factors (TCFs), ELK1, Sap1, and Net, and

51 such as members of the myocardin family and ternary complex factors (TCFs), respectively.

52 required for the binding of the Ets-related ternary complex factors (TCFs), such as Elk-1, indicatin

53 This causes activation of ternary complex factors (TCFs), which regulate c-fos gen

54 lated by serum responsive factors (SRFs) and ternary complex factors (TCFs).

55 The ternary complex factors (TCFs; Elk1, Net, and Sap-1) are

56 The ternary complex factors (TCFs; SAP-1, Elk-1, and Net) ar