ライフサイエンスコーパス: tert-butylhydroquinone

1 igargin (Tg), cyclopiazonic acid, and 2,5-di-tert-butylhydroquinone.

2 lasmic reticulum Ca(2)(+) ATPase with 2,5-di-tert-butylhydroquinone.

3 hibited by prototypical inducers arsenic and tert-butylhydroquinone.

4 activation was induced with the electrophile tert-butylhydroquinone.

5 human UGT1 locus (Tg-UGT1) were treated with tert-butylhydroquinone.

6 and this activation was further increased by tert-butylhydroquinone.

7 but this activation was no longer induced by tert-butylhydroquinone.

8 ured cells treated with pro-oxidants such as tert-butylhydroquinone, 2,3-dimethoxy-1, 4-naphthoquinon

9 tert-Butylhydroquinone and a mixture containing ascorbic

10 epG2 cells were treated with the prooxidants tert-butylhydroquinone and beta-naphthoflavone, cellular

11 pump (thapsigargin, cyclopiazonic acid, and tert-butylhydroquinone), and prolonged incubation of cel

12 The reduced forms of quinone, tert-butylhydroquinone, and 5-imino-daunorubicin do not

13 variety of oxidants, including sulforaphane, tert-butylhydroquinone, and H2O2, could effectively indu

14 Known inducers of phase II enzymes, such as tert-butylhydroquinone, beta-naphthoflavone, and sulfora

15 protein kinase C in HepG2 cells treated with tert-butylhydroquinone, beta-naphthoflavone, or 12-O-tet

16 Thapsigargin and 2,5-di-tert-butylhydroquinone (BHQ) blocked insulin signaling,

17 ion of a model hydroquinone compound, 2,5-di-tert-butylhydroquinone, by LPO/H(2)O(2) is also dependen

18 f2-ARE pathway by the known chemical inducer tert-butylhydroquinone can protect against 6-OHDA in vit

19 demonstrate that a common food preservative, tert-butylhydroquinone, can activate Nrf2 in T cells, as

20 ased basal NQO1 activity and no induction by tert-butylhydroquinone compared with Nrf2(+/+) astrocyte

21 cleus, arsenic, but not phenolic antioxidant tert-butylhydroquinone, dissociated Nrf2 from Keap1 and

22 ere increased in hepatoma cells treated with tert-butylhydroquinone for 2 h as measured by flow cytom

23 SN50 also blocked tert-butylhydroquinone-induced nuclear fluorescence of G

24 knockdown of CHD6 reduced both the basal and tert-butylhydroquinone-inducible expression of NQO1, a p

25 hepatoblastoma (HepG2) cells to antioxidant tert-butylhydroquinone led to induction of Bcl-2.

26 of Nrf2 were increased in cells treated with tert-butylhydroquinone or beta-naphthoflavone by a post-

27 atment of Hepa cells with hydrogen peroxide, tert-butylhydroquinone, or zinc suggested a rapid increa

28 Oxidative stress (tert-butylhydroquinone) rapidly induced metallothionein-

29 ement (ARE) that was positively regulated by tert-butylhydroquinone, sulforaphane, and hemin with res

30 er, a reported potential antiviral compound, tert-butylhydroquinone (t-b-HQ), showed no significant e

31 Treatment with the antioxidant tert-Butylhydroquinone (t-BHQ) leads to induction of Cul

32 itin H transcription was robustly induced in tert-butylhydroquinone (t-BHQ)-treated Jurkat cells via

33 50-fold following treatment with 50 mumol/L tert-butylhydroquinone (t-BHQ).

34 ion and induction in response to antioxidant tert-butylhydroquinone (t-BHQ).

35 tert-Butylhydroquinone (TBH) exerted a dose- and time-de

36 tert-Butylhydroquinone (TBH) induces human GCLC via Nrf2

37 (DEM), D,L-buthionine sulfoximine (BSO), or tert-butylhydroquinone (TBH).

38 In this study, tert-butylhydroquinone (tBHQ) and sulforaphane were used

39 the actions of the prototypical Nrf2 inducer tert-butylhydroquinone (tBHQ) and two biotin-tagged, thi

40 e (BHA), butylated hydroxytoluene (BHT), and tert-butylhydroquinone (TBHQ) are synthetic antioxidants

41 activation of Nrf2 by sulforaphane (SF) and tert-butylhydroquinone (tBHQ) depends upon Keap1-C151 an

42 ivation of the human NQO1-ARE (hNQO1-ARE) by tert-butylhydroquinone (tBHQ) is mediated by phosphatidy

43 The enzyme-mediated grafting of tert-butylhydroquinone (TBHQ) onto chitosan and further

44 ci was enhanced by phenobarbital, but not by tert-butylhydroquinone (tBHQ) or paraquat.

45 ) and murine hepatoma (Hepa1c1c7) cells with tert-butylhydroquinone (tBHQ) or sulforaphane (SUL), two

46 resent study was designed to investigate how tert-butylhydroquinone (tBHQ) prevents hydrogen peroxide

47 pG2 and murine hepatoma Hepa1c1c7 cells with tert-butylhydroquinone (tBHQ) stimulated the activity of

48 Tert-butylhydroquinone (TBHQ) was tested for potential c

49 ted by antiestrogen to the same extent as by tert-butylhydroquinone (TBHQ), a known activator of EpRE

50 A major metabolite of BHA, tert-butylhydroquinone (tBHQ), also activated ERK2 but w

51 We examined the effect of tert-butylhydroquinone (tBHQ), an Nrf2-ARE signaling pat

52 llular fractionation revealed that PMA, like tert-butylhydroquinone (tBHQ), promoted the nuclear loca

53 how that phorbol myristate acetate (PMA) and tert-butylhydroquinone (tBHQ), which induce oxidative st

54 combinations (75:25, 50:50, and 25:75), and tert-butylhydroquinone (TBHQ).

55 globin mRNA production by the NRF2 activator Tert-butylhydroquinone (tBHQ).

56 er antioxidant activities than 0.02% (1.1mM) tert-butylhydroquinone (TBHQ).

57 perfused with vehicle or the AP-1 stimulator tert-butylhydroquinone (tBHQ).

58 tion of both the CYP2C9 and CYP2C19 genes by tert-butylhydroquinone (tBHQ).

59 In addition, antioxidant (tert-butylhydroquinone) treatment destabilized the Nrf2-

60 ated that H2O2 (hydrogen peroxide) or t-BHQ (tert-butylhydroquinone) treatment increased total protei

61 sponse to heme, cadmium, zinc, arsenite, and tert-butylhydroquinone was inhibited by 85-95%.

62 sensitive to tert-butylhydroperoxide but not tert-butylhydroquinone, whereas knockdown of Nrf2b did n

63 t from other electrophilic compounds such as tert-butylhydroquinone, which activates the antioxidant-

64 hat interaction of anthracyclines and 2,5-di-tert-butylhydroquinone with LPO/H(2)O(2)/NO(2)(-) genera

65 whereas Nrf2 activation by sulforaphane and tert-butylhydroquinone with subsequent HO-1 induction en