ライフサイエンスコーパス: testicular germ cell tumor

1 emain for treatment of certain categories of testicular germ cell tumors.

2 emain for treatment of certain categories of testicular germ cell tumors.

3 emain for treatment of certain categories of testicular germ cell tumors.

4 delayed toxicities of initial treatments for testicular germ cell tumors.

5 c events that occur in vivo, particularly in testicular germ cell tumors.

6 m patients with pathological stage II or III testicular germ cell tumors.

7 models for seminomatous and nonseminomatous testicular germ cell tumors.

8 own that taller men are at increased risk of testicular germ-cell tumors.

9 be a major cause of cisplatin resistance in testicular germ cell tumors and may also be implicated i

10 The genetic nature of testicular germ cell tumors and the molecular mechanisms

11 FAM50B expression is deregulated in testicular germ cell tumors, and loss of imprinting occu

12 Attenuations in the rising incidence of testicular germ cell tumors are beginning to be observed

13 We analyzed three pairs of testicular germ cell tumor cell lines using Affymetrix e

14 Analysis of testicular germ cell tumor clinical samples by quantitat

15 on during human spermatogenesis and in human testicular germ cell tumors considered to be precursors

16 ow-up strategies for the different stages of testicular germ cell tumors continue to be defined and r

17 line chemotherapy for patients with relapsed testicular germ cell tumors (GCTs).

18 onducted a genome-wide association study for testicular germ cell tumor, genotyping 298,782 SNPs in 9

19 The medical treatment of advanced testicular germ cell tumors has changed over the past 30

20 Patients with clinical stage I testicular germ cell tumors have been managed with adjuv

21 discusses several important developments in testicular germ cell tumors in the last year.

22 verview on several important developments in testicular germ cell tumors in the past year is provided

23 discusses several important developments in testicular germ cell tumors in the past year.

24 To discuss several important developments in testicular germ cell tumors in the past year.

25 discusses several important developments in testicular germ cell tumors in the past year.

26 etected mutations in 1 of 14 nonseminomatous testicular germ-cell tumors, in 2 of 5 embryonal rhabdom

27 e associations between chlordane isomers and testicular germ cell tumors, it is reasonable to assume

28 Secondly, a subset of testicular germ cell tumors, known as non-seminomas, oft

29 nced stages, recent studies demonstrate that testicular germ cell tumor mortalities can vary signific

30 rognosticate survival outcomes in metastatic testicular germ cell tumor (MT-GCT), but how the initial

31 splays genetic linkage to the development of testicular germ cell tumors of adolescents and adults (T

32 t in vertebrates that has been implicated in testicular germ cell tumors of mouse and human.

33 t overlapping regions of imbalance (SORI) in testicular germ cell tumors other than the 12p region, w

34 IGF-1 or IGFBP-3 concentrations and risk of testicular germ-cell tumors (p > 0.05).

35 al morphology of nonseminomatous subtypes of testicular germ cell tumors remain to be established.

36 The authors examined associations between testicular germ-cell tumor risk and circulating concentr

37 bolic consequences of therapies in long-term testicular germ cell tumor survivors are being further c

38 Complex genetic factors underlie testicular germ cell tumor (TGCT) development.

39 Genome-wide association (GWA) studies of testicular germ cell tumor (TGCT) have identified 18 sus

40 Testicular germ cell tumor (TGCT) is the most common can

41 Testicular germ cell tumor (TGCT) is the most common can

42 Unconventional inheritance for testicular germ cell tumor (TGCT) risk both in humans an

43 ted Pde11a exhibit male infertility, a known testicular germ cell tumor (TGCT) risk factor.

44 (Dnd1) gene are the most potent modifiers of testicular germ cell tumor (TGCT) susceptibility in mice

45 the only genetic modifier known to suppress testicular germ cell tumor (TGCT) susceptibility in mice

46 Several genetic variants act as modifiers of testicular germ cell tumor (TGCT) susceptibility in the

47 onducted a genome-wide association study for testicular germ cell tumor (TGCT), genotyping 307,666 SN

48 ysis to identify new susceptibility loci for testicular germ cell tumor (TGCT).

49 reported 19 distinct susceptibility loci for testicular germ cell tumor (TGCT).

50 published genome-wide association studies of testicular germ cell tumor (TGCT; 3,558 cases and 13,970

51 as been marked disparity in the incidence of testicular germ cell tumors (TGCT) among white and black

52 Testicular germ cell tumors (TGCT) are considered a para

53 Testicular germ cell tumors (TGCT) are sex limited, occu

54 Testicular germ cell tumors (TGCT) are the most common s

55 Testicular germ cell tumors (TGCT) are the most frequent

56 Testicular germ cell tumors (TGCT) generally respond wel

57 Susceptibility to testicular germ cell tumors (TGCT) has a significant her

58 Testicular germ cell tumors (TGCT) have been expected to

59 out the genetic control of susceptibility to testicular germ cell tumors (TGCT) in humans or mice.

60 Testicular germ cell tumors (TGCT) originate from germ c

61 Testicular germ cell tumors (TGCT) represent the most co

62 lity is a risk factor for the development of testicular germ cell tumors (TGCT), but the initiating e

63 in pathway also influences susceptibility to testicular germ cell tumors (TGCT), the most common test

64 ormonal balance and thereby increase risk of testicular germ cell tumors (TGCT).

65 y model, we analyzed population-wide data on testicular germ-cell tumor (TGCT) status in 1,135,320 tw

66 Testicular germ cell tumors (TGCTs) are highly responsiv

67 Testicular germ cell tumors (TGCTs) are the most common

68 Testicular germ cell tumors (TGCTs) arise despite posses

69 The genetic basis for susceptibility to testicular germ cell tumors (TGCTs) has been remarkably

70 dend1 (Dnd1) gene enhances susceptibility to testicular germ cell tumors (TGCTs) in mice, in part by

71 osis, management, and risk stratification of testicular germ cell tumors (TGCTs) in the past year.

72 The etiology of testicular germ cell tumors (TGCTs) is poorly understood

73 f 12p material is invariably associated with testicular germ cell tumors (TGCTs) of adolescents and a

74 discusses several important developments in testicular germ cell tumors (TGCTs) over the past year.

75 Testicular germ cell tumors (TGCTs) share germline ances

76 Susceptibility to spontaneous testicular germ cell tumors (TGCTs), a common cancer aff

77 ansformed understanding of susceptibility to testicular germ cell tumors (TGCTs), but much of the her

78 mutant testes also are misregulated in human testicular germ cell tumors (TGCTs), suggesting similar

79 Testicular germ cell tumors (TGCTs), the most common neo

80 ly used anticancer drug for the treatment of testicular germ cell tumors (TGCTs).

81 ariants associated with an increased risk of testicular germ cell tumors (TGCTs).

82 3 is linked to the known hypersensitivity of testicular germ cell tumors to chemotherapy.

83 tential relevance to the hypersensitivity of testicular germ cell tumors to cisplatin, are discussed.

84 r controls but were indeed found in 7% of 43 testicular germ cell tumor trios; this percentage exceed

85 s, we studied its expression levels in human testicular germ cell tumors using patient tissues, model

86 The treatment of advanced testicular germ cell tumors with cisplatin combination c