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1 , and an accuracy of 94% in the diagnosis of testicular tumors.
2 pelvis, paratesticular rhabdomyosarcoma, and testicular tumors.
3 phasis on Wilms tumor, rhabdomyosarcoma, and testicular tumors.
4 ainst a protein found in normal brain and in testicular tumors.
5 cisplatin is particularly effective against testicular tumors.
6 lar tumor cell lines and in 1 of 11 sporadic testicular tumors.
7 There is a significant association of TM and testicular tumors.
8 orders-and in rare cases to the formation of testicular tumors.
10 Thirty-four of the 50 lesions (68%) were testicular tumors and 16 (32%) were of nontumorous origi
11 the presence of hard lesions in all cases of testicular tumors and three cases of nontumorous lesions
13 nstrated PODXL is overexpressed in malignant testicular tumor, and cellular depletion of PODXL result
14 tion, presence and degree of TM, presence of testicular tumor, and patient age was prospectively reco
18 lation of the zinc finger protein 93 and the testicular tumor-associated paraneoplastic Ma antigen (P
19 ata bases were searched for pathology-proven testicular tumors (benign or malignant germ cell or stro
20 that has enjoyed remarkable success against testicular tumors, but dose limiting side-effects have l
21 e expanded (CAG)n tracts in DNA from 5 of 11 testicular tumor cell lines and in 1 of 11 sporadic test
23 ors, only one colorectal carcinoma and three testicular tumors displayed LKB1 promoter hypermethylati
24 6 cases and 913 controls in the Servicemen's Testicular Tumor Environmental and Endocrine Determinant
25 es and 790 controls from the US Servicemen's Testicular Tumor Environmental and Endocrine Determinant
26 among participants in the U.S. Servicemen's Testicular Tumor Environmental and Endocrine Determinant
27 nd 928 controls enrolled in the Servicemen's Testicular Tumor Environmental and Endocrine Determinant
28 ions or microlithiasis and all patients with testicular tumors from pathology database were identifie
31 emonstrate a higher incidence of spontaneous testicular tumors in XPC-/- TrpS3-/- double mutant mice
36 verse transcription-PCR, is very low in most testicular tumors, particularly those of germ cell origi
38 agments identified in human breast tumor and testicular tumor RLGS profiles and in the cloning of an
39 nce of disease outside the boundaries of the Testicular Tumor Study Group template, the incidence of
41 was detected in four spermatocytic seminomas-testicular tumors that most likely originate from germ c
42 d expression was detected in 10 nonseminomas-testicular tumors that originate from the same precursor
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