ライフサイエンスコーパス: tetani

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1 following prolonged tetanic stimulation (40 tetani).

2 d to 1.8 +/- 0.06 mm s(1) over the last five tetani.

3 on of either drug alone, induced LTD without tetani.

4 ency stimuli were preceded by high-frequency tetani.

5 es to intact frog muscle fibres during fused tetani.

6 the frog, was investigated during isometric tetani.

7 se caused by a toxin produced by Clostridium tetani-a Gram-positive bacillus found in high concentrat

8 due to Clostridium botulinum and Clostridium tetani among users of illegal injected drugs (IDUs) occu

9 artifacts, even while segments were in fused tetani and developing maximum tensions of more than 600

10 flashes/1000 mum(2).100 s before and after 5 tetani) and markedly decreased (to 7.7 +/- 1.6 flashes/1

11 ist blocked the LTD induction by serotonin + tetani, and co-application of a group I mGluR agonist an

12 ses, ColG from C. histolyticum, ColT from C. tetani, and ColQ1 from the Bacillus cereus strain Q1, wh

13 evelopment that exhibited summation, unfused tetani, and fused tetani in a frequency-dependent manner

14 An immunoassay for the determination of anti-tetani antibodies has been developed using a screen prin

15 ution factor should be small, while the anti-tetani antibody incubation time should be long.

16 ed during a series of thirty 0.2 s isometric tetani at L(o), the length optimal for force.

17 every 100 ms during electrically stimulated tetani at muscle lengths that varied 1.5-fold and at the

18 During tetani, birefringence increased with approximately the s

19 increase myoplasmic [Ca2+] during twitch and tetani, but not to reverse the shift in optimum length d

20 ism preventing hyperactivity and undesirable tetani, by preventing subsequent stimuli eliciting actio

21 Such tetani caused a modest AP decrease, to a similar extent

22 cted against lethal infection by Clostridium tetani cells and Bacillus anthracis spores following top

23 ns occur in hippocampus in vitro after brief tetani delivered to afferent pathways.

24 d identification of both C. botulinum and C. tetani demonstrates a sensitivity and specificity simila

25 We found that B. anthracis and C. tetani epitopes were the most promiscuous of the group t

26 pathway entrance in homodimeric Clostridium tetani H(+)-PPase upon ligand binding.

27 ngens, Clostridium botulinum and Clostridium tetani, has shown that no spoVF orthologues exist in the

28 hibited summation, unfused tetani, and fused tetani in a frequency-dependent manner.

29 eatest impact on the response, i.e. the anti-tetani incubation time and the dilution factor of the la

30 Repetitive spaced tetani induce glutamate receptor-dependent stable restru

31 ive immune response against live Clostridium tetani infection in mice can be elicited by an adenoviru

32 n of circulating toxin and elimination of C. tetani infection, control of spasms and convulsions, mai

33 epidemiology, treatment, and prognosis of C tetani infections.

34 Clostridium tetani infects wounds contaminated with dirt, feces, or

35 ) after theta bursts, but not high-frequency tetani, produced a rapid, transient expression of synapt

36 The assay consisted of spiking the anti-tetani sample directly onto the toxoid modified SPE, and

37 o identify any of the 7 C. innocuum and 9 C. tetani strains tested), and 8 of 15 (53.3%) Bacteroides

38 Evidence from tetani suggests the slow force responses result from inc

39 tect a fragment of the neurotoxin gene of C. tetani (TeNT) and was used in conjunction with previousl

40 TeNT) is an exotoxin produced by Clostridium tetani that causes paralytic death to hundreds of thousa

41 ament architecture of Alp12 from Clostridium tetani that is constructed from four protofilaments.

42 itopes of Bacillus anthracis and Clostridium tetani toxins, as these bacteria do not depend on human

43 ute stress, the LTD induction by serotonin + tetani was significantly impaired.

44 Isometric twitches and tetani were recorded from the whole muscles and from a s

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