ライフサイエンスコーパス: tetanization

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1 tal control animals at 5, 15 and 60 min post-tetanization.

2 P was observed when oxo-m was present during tetanization.

3 purely postsynaptic protocol, intracellular tetanization.

4 to mechanical stress provoked by repetitive tetanizations.

5 After intracellular tetanization, 32 of 76 regular inputs (42%) showed long-

6 cytochemical studies show that both repeated tetanization and application of forskolin stimulate the

7 ynthase blocked L-LTP induced by three-train tetanization and reduced L-LTP induced by four-train tet

8 es in slices can be induced by intracellular tetanization: bursts of postsynaptic spikes without pres

9 early long-term potentiation induced by weak tetanization can be converted into lasting late long-ter

10 Here we asked if intracellular tetanization can induce long-term plasticity in auditory

11 In contrast, tetanization elicited LTP of monosynaptic inhibitory res

12 Thus, intracellular tetanization had a normalizing effect on synaptic effica

13 rat hippocampal slices by applying repeated tetanization in reduced levels of magnesium.

14 e long-term changes induced by intracellular tetanization involved both pre and postsynaptic mechanis

15 that can be induced by three- or four-train tetanization, lasts >3 hr, and is reduced by inhibitors

16 TP) that can be induced by one- or two-train tetanization, lasts approximately 1 hr, and is cAMP-depe

17 by the cAMP-dependent protein kinase (PKA), tetanization most likely activates a Ca2+-dependent prot

18 L-LTP was induced with three trains of tetanization of 1 s duration at 100 Hz separated by 10-m

19 on of an LTP obtained with a single train of tetanization of 1 s duration at 100 Hz.

20 long-term potentiation by subsequent strong tetanization of a separate input.

21 by prior or subsequent strong high-frequency tetanization of an independent input to a common populat

22 Plasticity that results from tetanization of input fibres does not depend on calcium

23 te myofilament response to Ca2+, assessed by tetanization of intact cells over a range of [Ca2+], was

24 ited PKMzeta activity at various times after tetanization of Schaffer collateral/commissural-CA1 syna

25 king in coupled neurons, and mGluR-dependent tetanization of synaptic input - are separate pathways t

26 uding paired activity in coupled neurons and tetanization of the input to coupled neurons.

27 was reported previously that repeated brief tetanization of the posterior eight nerve can produce lo

28 lation of CaMKII in hippocampal slices after tetanization of the Schaffer collateral pathway.

29 itory interneurons in vitro before and after tetanization of the thalamo-LA pathway, one of the major

30 thalamus and long-term depression following tetanization of this input to the postsubiculum, respect

31 the Mauthner cell, can be evoked by afferent tetanization or local dendritic application of an endoge

32 -AngIV prior to, but not 15 or 30 min after, tetanization prevented stabilization of LTP.

33 When dopamine was applied first, tetanization produced additional potentiation of the mix

34 her NO or cGMP analogs paired with one-train tetanization produced late-phase potentiation, and the c

35 uired for associative learning, but standard tetanization protocols fail to potentiate nuclear cell E

36 Induction of plasticity by intracellular tetanization required a rise of intracellular [Ca(2+)],

37 Tetanization resulted in LTP of the EPSPs elicited in bo

38 Tetanization studies in intact myocytes revealed that 43

39 no group differences, however at 60 min post-tetanization the slopes of the field excitatory postsyna

40 The results indicated that at 5 min post-tetanization there were no differences in field potentia

41 At 15 min post-tetanization there were no group differences, however at

42 ntrol hippocampal slices by L-LTP-generating tetanization were significantly reduced in mutant slices

43 tion and reduced L-LTP induced by four-train tetanization, whereas an inhibitor of PKA was more effec

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