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1 +10 mV, indicating increased permeability of tetraethylammonium ion.
2 y, at very low and at high concentrations of tetraethylammonium ion.
3 was confirmed by studying simple transfer of tetraethylammonium ion.
4 inopyridine and quinine and insensitivity to tetraethylammonium ions.
5 nt cations including tetramethylammonium and tetraethylammonium ions.
6 potentiation occurred in the presence of the tetraethylammonium ion, a K+-channel blocker.
7                    We propose that 4AP, like tetraethylammonium ion and other quaternary ammonium ion
8 ns, the K+ channel blockers 4-aminopyridine, tetraethylammonium ions and XE991.
9                                          The tetraethylammonium ion fits snugly in the interior of th
10 ation was perturbed by application of either tetraethylammonium ions or the Shaker (Sh)B peptide to t
11  efflux is approximately 0.1 mM), but not by tetraethylammonium ions or verapamil.
12        In the first, we constructed a mutant tetraethylammonium ion-sensitive KCNQ4 subunit and teste
13 ly with KCNQ5 by patch clamp analysis of the tetraethylammonium ion sensitivity of the resulting curr
14 ansport down its diffusion gradient, whereas tetraethylammonium ion substitution for K+ did not affec
15 s are more sensitive to blockade by internal tetraethylammonium ion (TEA) than KvLQT1 channels.
16                                   Unlike the tetraethylammonium ion (TEA), neither JC638.2alpha nor C
17 er currents by using the BK channel blockers tetraethylammonium ions (TEA(+); 1 mM) or iberiotoxin (2
18 e silent arteries by charybdotoxin (CTX) and tetraethylammonium ions (TEA) induced dose-dependent dep
19 with a molecule (diethyl ether) or a cation (tetraethylammonium ion) trapped inside.
20                     The IK(V) was blocked by tetraethylammonium ions with an IC50 of 5.2 mM and was u

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