ライフサイエンスコーパス: thalamic

1 As thalamic abnormalities occur in some of the earliest sta

2 lume (beta = 0.606, P = .001), together with thalamic activation (beta = -0.410, P = .022), were the

3 To probe monosynaptic thalamic activation of cortical postsynaptic target cell

4 rons is a disynaptic inhibition dependent on thalamic activation of striatal tyrosine hydroxylase int

5 T-signalling, revealing a mechanism by which thalamic activity can help gate retinal input to the SCN

6 We found that dampening mediodorsal thalamic activity caused significant reductions in gamma

7 stence of specific abnormalities in auditory thalamic activity following sound offsets, but not sound

8 macogenetic strategy to decrease mediodorsal thalamic activity in adult male rats and evaluated the c

9 ure as a thermostat that uniformly regulates thalamic activity through negative feedback.

10 larly, photoinhibition of the ALM diminished thalamic activity.

11 g, population auditory cortical responses to thalamic afferent stimulation were studied in brain slic

12 d potentials evoked in the OFC by excitatory thalamic afferent stimulation, and this was prevented by

13 originates from the clustering of ON and OFF thalamic afferents in the visual cortex, we conclude tha

14 the PAF unilaterally, to label cortical and thalamic afferents.

15 ker of neurodegeneration, that (i) secondary thalamic alterations are focally located in specific tha

16 for a scientific commentary on this article.Thalamic alterations have been observed in infarcts init

17 approaches, and the association between PFC-thalamic anatomical connectivity and severity of executi

18 PFC-thalamic anatomical connectivity may be an important tar

19 In addition, thalamic and auditory-midbrain activity was correlated w

20 auditory evoked response data, and show that thalamic and brainstem sources can be correctly estimate

21 lapses) typically attributed to attentional thalamic and frontoparietal circuits, but the underlying

22 a terminalis, substantia innominata, various thalamic and hypothalamic nuclei, pedunculopontine nucle

23 The dorsal pallidum projects to both thalamic and midbrain targets similar to the direct and

24 two projection (cortico-pontine and anterior thalamic), and five bilateral association (inferior fron

25 eveal that throughout postnatal development, thalamic, and entorhinal cortical inputs onto hippocampa

26 h lesions in the right occipito-parietal and thalamic areas integrating visual and somatosensory info

27 c areas, posterior tubercle, prethalamic and thalamic areas, optic tectum, torus semicircularis, mese

28 ceive a projection from rostral parts of the thalamic auditory nucleus ovoidalis and from the nucleus

29 olinergic interneurons are a major target of thalamic axodendritic terminals, we examined the VGLUT2-

30 -2-expressing long-range posteromedial (POm) thalamic axon terminals in cortex and induced CaMPARI co

31 In mammals, thalamic axons are guided internally toward their neocor

32 e neurons in non-mammalian species, in which thalamic axons do not grow internally, raised the possib

33 um imaging to characterize the properties of thalamic axons innervating different layers of mouse aud

34 pholipids and PRG-2 are critical for guiding thalamic axons to their proper cortical targets.

35 a common organizing principle that arranges thalamic axons with similar retinotopy and ON-OFF polari

36 Precise connection of thalamic barreloids with their corresponding cortical ba

37 the highest clustering level in somatomotor, thalamic, basal ganglia, and limbic networks.

38 ivity was correlated with recall, cerebellar-thalamic baseline connectivity was correlated with faste

39 response properties of approximately 28,000 thalamic boutons and approximately 4,000 cortical neuron

40 We identified thalamic boutons by their immunoreactivity for the vesic

41 Furthermore, thalamic boutons in M1 targeted spiny dendrites exclusiv

42 neuropathic pain is associated with altered thalamic burst firing and thalamocortical dysrhythmia.

43 It is proposed that thalamic bursts are an adaptive response to pain that de

44 Optically-induced thalamic bursts attenuated cortical theta and mechanical

45 that dopamine depletion selectively weakens thalamic but not cortical afferents onto these neurons,

46 These findings reveal that embryonic thalamic calcium waves coordinate cortical sensory area

47 This mechanism is mediated by spontaneous thalamic calcium waves that propagate among sensory-moda

48 d the extracellular potential of presynaptic thalamic cells and the intracellular potential of postsy

49 usal system that controls the firing mode of thalamic cells based on attentional demand.

50 annels are integrated at the level of single thalamic cells is not well understood.

51 nductance and background excitation of these thalamic cells must be within specific ranges to exhibit

52 tates; (2) thalamic downstates hyperpolarize thalamic cells, thus triggering spindles; and (3) thalam

53 recording reveals that the cortico-striatal-thalamic circuit is tonically hyperactive in mutants, bu

54 omplexity of psychiatric diseases; by making thalamic circuits accessible to mechanistic dissection;

55 Thalamic circuits are targeted by two major inhibitory s

56 hope that principled targeting of identified thalamic circuits can be uniquely therapeutic.

57 b-network included the regions of the fronto-thalamic circuits in the left hemisphere.

58 mation relay to or between cortical regions, thalamic circuits shift and sustain functional interacti

59 nt electrical synapses in the development of thalamic circuits.

60 hase of augmented suppression involves local thalamic circuits.

61 e core function of the pulvinar, the largest thalamic complex in primates, remains elusive.

62 s of interneurons originates from within the thalamic complex, but we now show that during early post

63 ROI analysis revealed reduced PFC-thalamic connectivity and increased somatomotor-thalamic

64 lamic connectivity and increased somatomotor-thalamic connectivity in both chronic and early-stage ps

65 Reduced PFC-thalamic connectivity in schizophrenia correlated with i

66 istent with models implicating disrupted PFC-thalamic connectivity in the pathophysiology of schizoph

67 During task, patients also exhibited altered thalamic connectivity to PHG and FPCN.

68 d to determine the implications of increased thalamic connectivity with sensory cortex.

69 ntrast to reduced PFC-thalamic connectivity, thalamic connectivity with somatosensory and occipital c

70 early stages of psychosis, includes reduced thalamic connectivity with the executive control network

71 In contrast to reduced PFC-thalamic connectivity, thalamic connectivity with somato

72 (PFC) connectivity and increased somatomotor-thalamic connectivity.

73 ms of cyto- and chemoarchitecture as well as thalamic connectivity.

74 eview, we highlight new findings that refine thalamic contributions to cortical rhythms and suggest t

75 reviously unknown principle in neuroscience; thalamic control of functional cortical connectivity.

76 We report a modulation of the cerebellar-thalamic-cortical and cerebellar-basal ganglia networks

77 Interestingly, whereas thalamic-cortical baseline connectivity was correlated w

78 the PFC cannot be dissociated from its main thalamic counterpart, the mediodorsal thalamus (MD).

79 stigate the safety and efficacy of dual-lead thalamic DBS (one targeting the ventralis intermedius-ve

80 INTERPRETATION: Dual lead thalamic DBS might be a safe and effective option for im

81 These results suggest that thalamic deep brain stimulation in tremor likely exerts

82 maturity, will predict pronounced changes in thalamic development, and thereby cognitive and motor fu

83 cal downstates lead thalamic downstates; (2) thalamic downstates hyperpolarize thalamic cells, thus t

84 ein: (1) convergent cortical downstates lead thalamic downstates; (2) thalamic downstates hyperpolari

85 maging has revealed an intriguing pattern of thalamic dysconnectivity in psychosis characterized by r

86 ical specificity, and clinical correlates of thalamic dysconnectivity in psychosis.

87 drug effects were associated with changes in thalamic FCD in both groups.

88 y that is essential for the establishment of thalamic feedforward inhibition mediated by parvalbumin

89 ss and astrogliosis in the thalamus and less thalamic fiber loss by diffusion tensor imaging (DTI).

90 In developing animals, thalamic fibers do not target layer 4 but instead target

91 neuropathic pain is associated with altered thalamic firing and thalamocortical dysrhythmia.

92 eins and do not project to other paralaminar thalamic forebrain targets, and that a previously undesc

93 arly gamma oscillations, a marker for normal thalamic function at this age.

94 us, but whether this diversity translates to thalamic functions beyond relaying information to or bet

95 isease in the mouse has shown how diminished thalamic gain control can lead to attention deficits.

96 of the remaining ones, triggering changes in thalamic gene expression and cortical area size.

97 The habenular and thalamic Gpr151 systems displayed both shared and distin

98 ine, monosynaptic afferents of habenular and thalamic Gpr151-expressing neuronal populations could be

99 Thalamic growth was associated with cognitive and motor

100 Thalamic growth was in turn related to cognitive and mot

101 Seed-based analyses revealed thalamic hypoconnectivity in psychosis localized to dors

102 subplate, consistent with the known relay of thalamic information to layer 4 by subplate neurons.

103 ent study suggest that proper development of thalamic inhibitory circuitry, neuronal morphology, TRN

104 primary cortical injury and remote secondary thalamic injury, and a single treatment can produce pers

105 by DHC also significantly reduced secondary thalamic injury, as DHC-treated stroke mice exhibited 53

106 that cytochrome-oxidase maps closely mirror thalamic innervation.

107 Amplification of thalamic input by recurrent local circuits is thus likel

108 ortex of the STGr, which received 80% of its thalamic input from multisensory nuclei (primarily media

109 L4 is the principal target for thalamic input in sensory areas, which raises the questi

110 ed, but our data suggest that, as in S1, the thalamic input is amplified by the recurrent excitatory

111 b/4 of the primary cortical areas, where the thalamic input is dominated by the lemniscal projection.

112 r, our results suggest that the feed-forward thalamic input may play a key role in initiating and gui

113 The lack of direct thalamic input to inhibitory neurons in M1 may indicate

114 The functional role of the thalamic input to L4 in M1 has largely been neglected, b

115 minals, we examined the VGLUT2-immunolabeled thalamic input to striatal cholinergic interneurons in h

116 so has the canonical circuit motif of a core thalamic input to the middle cortical layer and that tha

117 ocessing: primary visual cortex (V1) and its thalamic inputs from the dorsal lateral geniculate nucle

118 Both AI and R received nearly 90% of their thalamic inputs from the ventral subdivision of the MGN

119 ral area (PV), and other cortical areas; and thalamic inputs from the ventroposterior lateral nucleus

120 n from untuned thalamic inputs, but very few thalamic inputs have been measured in any mammal.

121 n which a sizeable fraction of the trigemino-thalamic inputs project ipsilaterally rather than contra

122 at irregular state-dependent fluctuations in thalamic inputs shape the susceptibility of cortical pop

123 Recently we showed that sources of thalamic inputs to RFA and RWA are similar, but they are

124 We investigated thalamic inputs to the two populations of striatal neuro

125 Cortical and thalamic inputs were greatest onto VIP+ interneurons and

126 mostly in layer 4, the main target of relay thalamic inputs, and layer 3.

127 t to compute visual orientation from untuned thalamic inputs, but very few thalamic inputs have been

128 escribed with surprisingly limited number of thalamic inputs, consistent with recent experimental fin

129 trastriatal circuitry and their cortical and thalamic inputs.

130 to egocentric position, unlike their driving thalamic inputs.

131 ons may inherit their selectivity from tuned thalamic inputs.

132 nt for intraventricular haemorrhage size and thalamic intracerebral haemorrhage.

133 Thalamic intrinsic currents, therefore, may not be conti

134 a pathway that bypasses V1, and connects the thalamic lateral geniculate nucleus directly with the ex

135 HC-treated stroke mice exhibited 53% smaller thalamic lesion size.

136 Finally, we found that focal thalamic lesions in humans have widespread distal effect

137 Early pain was associated with slower thalamic macrostructural growth, most pronounced in extr

138 tical connections and further improvement of thalamic matching.

139 y thalamus and is accompanied by disruptions thalamic metabolic growth and thalamocortical pathway ma

140 nct oscillations, we developed a biophysical thalamic model to test the hypothesis that generation of

141 motor performance through its projections to thalamic motor relay centers, including the mediodorsal

142 tes early pain was associated with decreased thalamic NAA/Cho and microstructural alterations in thal

143 stly, we applied periodic stimulation to the thalamic network and found that entrainment of thalamic

144 We find, for the first time, that the model thalamic network is capable of independently generating

145 al synapses play a role in the maturation of thalamic networks by studying neurons in mice with and w

146 of adaptation on the information conveyed by thalamic neurons about paired whisker stimuli in male ra

147 lesser extent than white, blue and red; that thalamic neurons are most responsive to blue and least r

148 Here we show that, similar to ALM neurons, thalamic neurons exhibited selective persistent delay ac

149 -unit recording of dura- and light-sensitive thalamic neurons in rats to show that green activates co

150 ll enough to provide localized feedback onto thalamic neurons in the visual pathway.

151 in somatosensory cortex and burst firing in thalamic neurons in vivo.

152 ohypothalamic pathway to show that GABAergic thalamic neurons inhibit retinally-driven activity in th

153 a and by colocalization of the antibody with thalamic neurons involved in mGluR1-mediated pain proces

154 eloping excitatory and inhibitory effects in thalamic neurons of the basal ganglia- and cerebellar-re

155 apid eye-movement (NREM) sleep, cortical and thalamic neurons oscillate every second or so between ON

156 driven retinal pathways, fine-tuned in relay thalamic neurons outside the main visual pathway, and pr

157 m downstream neurons in the caudate and from thalamic neurons projecting to the medial frontal cortex

158 lar pathway comprises a unique population of thalamic neurons that do not contain typical calcium-bin

159 We show that in auditory cortical and thalamic neurons, the weight of each receptive field ele

160 cy increases and/or decreases of activity in thalamic neurons.

161 s for synaptic integration and plasticity in thalamic neurons.SIGNIFICANCE STATEMENT In most neurons,

162 ng evidence suggest that the ventral midline thalamic nuclei (reuniens and rhomboid) might play a sub

163 ion with the pattern of connectivity between thalamic nuclei and cortical areas or deep nuclei), whic

164 rdependent relationship between the anterior thalamic nuclei and retrosplenial cortex, given how dysf

165 alterations are focally located in specific thalamic nuclei depending on the initial infarct locatio

166 remains one of the least explored among the thalamic nuclei despite occupying the most thalamic volu

167 suggests that innervation from PV-containing thalamic nuclei extends across superficial and middle la

168 Appreciating the importance of the anterior thalamic nuclei for memory and attention provides a more

169 The lateral posterior and posterior thalamic nuclei have been implicated in aspects of visua

170 -brain functional connectivity of the visual thalamic nuclei in the various populations of subjects u

171 ateral entorhinal cortex and ventral midline thalamic nuclei of neonatal rats.

172 the development and organization of diverse thalamic nuclei remain largely unknown.

173 y attributed to widespread connectivity from thalamic nuclei such as the pulvinar.

174 nformation to deeper layers of the SC and to thalamic nuclei that modulate visually guided behaviors.

175 layer 6 send a dense feedback projection to thalamic nuclei that provide input to sensory neocortex.

176 the anatomical connection from the anterior thalamic nuclei to retrosplenial cortex, and the involve

177 ions and specifications of connectivity with thalamic nuclei together with upcoming studies of cortic

178 Neurons in first-order thalamic nuclei transmit sensory information from the pe

179 waves that propagate among sensory-modality thalamic nuclei up to the cortex and that provide a mean

180 laterodorsal, anteroventral, and parateanial thalamic nuclei, the fasciculus retroflexus of Meynert,

181 reuniens (Re) is the largest of the midline thalamic nuclei.

182 signals reach the cortex via sense-specific thalamic nuclei.

183 signals reach the cortex via sense-specific thalamic nuclei.

184 ior thalamus (pulvinar) and the medio-dorsal thalamic nuclei.

185 t the mammillo-thalamic tract (MTT)/anterior thalamic nucleus (AN) complex would be critical for reco

186 demonstrated previously that parafascicular thalamic nucleus (PF)-controlled neurons in the posterio

187 Paraventricular thalamic nucleus (PVT) neurons receive hindbrain and hyp

188 Two-photon calcium imaging reveals that a thalamic nucleus and a downstream structure, the habenul

189 dorsal thalamus (MDT) is the major olfactory thalamic nucleus and links the olfactory archicortex wit

190 the SGN/V mainly projected to the posterior thalamic nucleus and the lateral hypothalamus (lateral t

191 from nTTD to the contralateral somatosensory thalamic nucleus dorsalis intermedius ventralis anterior

192 amus (MDT) is a higher-order corticocortical thalamic nucleus involved in cognition and memory.

193 ance statement: Cells in the anterior dorsal thalamic nucleus normally fire in relation to the animal

194 This higher order thalamic nucleus therefore conveys diverse contextual si

195 a projection from nTTD to the contralateral thalamic nucleus uvaeformis, a multi-sensory nucleus con

196 ected at low levels in the lateral posterior thalamic nucleus which received input from areas associa

197 The posterior thalamic nucleus, tertiary gustatory nucleus proper, and

198 T and CbMT and with neurons in the reticular thalamic nucleus.

199 ; parasubthalamic nucleus; ventral posterior thalamic nucleus; area postrema; and nucleus of the soli

200 represents an example of absence epilepsy of thalamic origin.

201 eneration of and transition between distinct thalamic oscillations can be explained as a function of

202 s indicate that generation of these distinct thalamic oscillations is a result of both intrinsic osci

203 alamic network and found that entrainment of thalamic oscillations is highly state-dependent.

204 Thalamic oscillators contribute to both normal rhythms a

205 butions to cortical rhythms and suggest that thalamic oscillators may be subject to both local and gl

206 NE modulation and afferent excitation define thalamic oscillatory states and their response to brain

207 These results identify a thalamic pathway for gravity cues to influence perceptio

208 This suggests the prefrontal-thalamic pathway mediates rapid and goal-driven attentio

209 ssing, information is processed by two major thalamic pathways encoding brightness increments (ON) an

210 Finally, we show that FC involving cortico-thalamic pathways is limited, possibly confounded by the

211 This thalamic process enables rapid coordination of spatially

212 Furthermore, LHb- and thalamic-projecting EP neurons are differentially innerv

213 re is well accounted for by a model assuming thalamic projections to two cortical layer-4 cell popula

214 but interrupting thalamo-cortical or cortico-thalamic projections.

215 sum, left posterior corona radiate/posterior thalamic radiate, right superior longitudinal fasciculus

216 beta = -0.194, pcorrected = 0.025), superior thalamic radiation (beta = -0.224, pcorrected = 0.009) a

217 sions within the interhemispheric tracts and thalamic radiation (P < .05, false discovery rate correc

218 h negative age associations strongest in the thalamic radiation and association fibres.

219 part of internal capsule, and left posterior thalamic radiation.

220 bres (beta = -0.184, pcorrected = 0.010) and thalamic radiations (beta = -0.159, pcorrected = 0.020).

221 tios (DVR) of [(18) F]Nifene in white matter thalamic radiations were approximately 1.6 (anterior) an

222 For example, in rodent, thalamic receptive field structure is markedly diverse,

223 and lesion sites in the right occipital and thalamic region, and the left parietal association area.

224 lateral geniculate nuclei, a number of other thalamic regions contribute to aspects of visual process

225 Left thalamic regions formed a delineated functional communit

226 unctional connectivity was evaluated for two thalamic regions of interest, based on the haemodynamic

227 onal connectivity in left orbitofrontal-both thalamic regions with suicidal ideation in MDD were inve

228 results of this study show the relevance of thalamic regulation of the brain networks involved in co

229 hat the thalamic reticular nucleus regulates thalamic relay activity through focal attentional modula

230 T-type calcium currents in the postsynaptic thalamic relay and reticular cells were dramatically ele

231 use dorsal lateral geniculate nucleus (dLGN; thalamic relay for cortical vision).

232 ral geniculate nucleus (dLGN) is the primary thalamic relay for visual information from the retina to

233 r of retinal ganglion cells innervating each thalamic relay neuron.

234 Thalamic relay neurons have well-characterized dual firi

235 Spike trains from thalamic relay neurons showed highly transient, weak cho

236 llar granule cells, hippocampal neurons, and thalamic relay neurons, 4-PIOL evidently displayed diffe

237 matically reducing GABAergic transmission in thalamic relay neurons.

238 c activity was localized to the higher-order thalamic relays of the medial dorsal nucleus and was sel

239 c activity was localized to the higher-order thalamic relays of the medial dorsal nucleus.

240 Different driver sources reveal two types of thalamic relays: first order relays receive subcortical

241 findings reveal that melanopsin enhances the thalamic representation of scenes containing local corre

242 ck is to control the timing and precision of thalamic responses to incoming visual signals.

243 xt improves predictions of both cortical and thalamic responses to stationary complex sounds.

244 ved in cognition and suggest that changes in thalamic resting-state network connectivity may represen

245 argeted by two major inhibitory systems: the thalamic reticular nucleus (TRN) and extrathalamic inhib

246 Clues for a specific involvement of the thalamic reticular nucleus (TRN) come from its unique ne

247 The thalamic reticular nucleus (TRN) is a unique brain struc

248 backpropagation in thalamocortical (TC) and thalamic reticular nucleus (TRN) neurons remains unknown

249 or more eliminates rebound bursting in model thalamic reticular nucleus (TRN) neurons.

250 mouse Ptchd1 is selectively expressed in the thalamic reticular nucleus (TRN), a group of GABAergic n

251 of plasticity at electrical synapses in the thalamic reticular nucleus - paired burst spiking in cou

252 e effect on evoked responses from inhibitory thalamic reticular nucleus and excitatory tectothalamic

253 In thalamocortical and thalamic reticular nucleus neurons, the site of AP gener

254 The searchlight hypothesis proposes that the thalamic reticular nucleus regulates thalamic relay acti

255 ry projections from the visual sector of the thalamic reticular nucleus to the lateral geniculate nuc

256 mpeting hypotheses about the function of the thalamic reticular nucleus.

257 working memory (SWM) and the ventral midline thalamic reuniens and rhomboid nuclei (Re/Rh) have long

258 Indeed, the model exhibited four distinct thalamic rhythms (delta, sleep spindle, alpha and gamma

259 ween each cortical ROI and its corresponding thalamic ROI was quantified and compared across groups.

260 ulators of [Cl(-)]i Neurons of the reticular thalamic (RT) nucleus express reduced levels of KCC2, in

261 an important Cl(-) extruder in the reticular thalamic (RT) nucleus, despite this nucleus having remar

262 Controlling the magnitude (gain) of thalamic sensory responses is a mechanism of attention,

263 We also show that in vivo manipulation of thalamic signalling adjusts specific features of the hyp

264 Our results suggest a causal role for thalamic sleep spindles in hippocampus-dependent memory

265 mic cells, thus triggering spindles; and (3) thalamic spindles are focally projected back to cortex,

266 In-phase optogenetic suppression of thalamic spindles impaired hippocampus-dependent memory.

267 ion approach in mice, we show here that only thalamic spindles induced in-phase with cortical slow os

268 -REM sleep, permitting the cortex to control thalamic spindling by inducing downstates.

269 12 patients with left thalamic stroke underwent a neuropsychological assessmen

270 The pulvinar nucleus is a large thalamic structure involved in the integration of visual

271 fic circuits connecting the TRN with sensory thalamic structures implement these functions is not kno

272 port an intricate ontogenetic logic of mouse thalamic structures.

273 from human participants, we found that most thalamic subdivisions display network properties that ar

274 In this study, we demonstrate that multiple thalamic subdivisions display network properties that ar

275 op is also presented, which reveals that the thalamic subregions innervated by the basal ganglia pref

276 We examined both thalamic surface morphology using anatomical MRI and tha

277 In M1 and S1, thalamic synapses formed only a small fraction (12.1% an

278 ocular dominance and reduces the density of thalamic synapses in layer 4 of the mouse primary visual

279 ptions regarding the spatial distribution of thalamic synaptic inputs into layer 4, the model predict

280 , delta-frequency optogenetic stimulation of thalamic synaptic terminals of lateral cerebellar projec

281 rior medial division, VPMpc) of mice and the thalamic terminal field was investigated across the cort

282 The density of thalamic terminals along individual cholinergic dendrite

283 n the areal striatal density of axodendritic thalamic terminals on cholinergic neurons was due to the

284 ion memory have postulated that the mammillo-thalamic tract (MTT)/anterior thalamic nucleus (AN) comp

285 genu of the corpus callosum and the anterior thalamic tracts.

286 and 80% (95% CI: 28, 100), respectively, for thalamic tumors; and 65% (95% CI: 51, 78) and 94% (95% C

287 Among tested variables, thalamic volume (beta = 0.606, P = .001), together with

288 Patients had decreased thalamic volume (P < .001).

289 tract-based spatial statistics, and reduced thalamic volume (p < 0.0001), and predicted unfavourable

290 Left thalamic volume at baseline correlated with baseline glo

291 bcortical surface was carried out to measure thalamic volume at both time points.

292 ated with structural neuroplastic changes in thalamic volume in patients with early schizophrenia (ES

293 e thalamic nuclei despite occupying the most thalamic volume in primates, it has long been suspected

294 Reduced thalamic volume is consistently observed in schizophreni

295 monstrate that early pain is associated with thalamic volume loss in the territory of the somatosenso

296 elated to improvements in cognition and left thalamic volume preservation following TCT.

297 inferior frontal gyrus (pars triangularis), thalamic volume, T2 lesion load, and age were used to ex

298 respect to improved cognition and change in thalamic volume.

299 and late pain (skin breaks, Scans 1-2) with thalamic volumes and N-acetylaspartate (NAA)/choline (Ch

300 atory analyses revealed significantly larger thalamic volumes in patients taking lithium compared wit