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1 ti-N-methyl-d-aspartate receptor (NMDAR) encephalitis alter the levels of dopamine 1 receptor (D1R) and dopamine 2 recept
2  WTalphaA proteins was determined by an in vitro assay, and the levels of intracellular Ca(2+) uptake and Na, K-ATPase mR
3 (p-value < 0.05, FC > 1.5) in CD patients and controls, and the levels of four differed in patients with active and inact
4  changes about lung function test, smoking history, sex and the levels of D-dimer among two groups.
5 orphogenesis relies on repeated use of dynamic behaviors at the levels of intracellular structures, individual cells, and
6  indicated that FO replacement by dietary AM did not change the levels of most biochemical (ALB, AMY, TP and GLOB), antio
7                       Inhibition of Beclin 1 did not change the levels of several key inflammatory molecules or the numbe
8 pid content was unchanged relative to age-matched controls, the levels of whole SN triglycerides were correlated with inf
9 ues, PDA tissue array and cell lines were used to determine the levels of MPC-1 and KDM5A expression, and their relations
10 d Nkx3.1(+/+) mice were analyzed over one year to determine the levels of Nkx3.1 expression and effects of the mutant pro
11                  Therefore, the use of UNC0638 did diminish the levels of H3K9me2 and H3K9me3 in SCNT-derived blastocysts
12                                                Furthermore, the levels of dACC Glx and dACC GSH positively correlated wit
13                                          On the other hand, the levels of eicosadienoic acid and octadecanoic acid were s
14                                                    However, the levels of lysine glycation in collagen, which is not cons
15              Patients with AH exhibited specific changes in the levels of intermediates of glycolysis/gluconeogenesis, th
16                                                  Changes in the levels of metabolic hormones (glucocorticoids) in respons
17          Quantitative real-time PCR detected an increase in the levels of immune stimulatory inflammatory cytokines, for
18  a 3-fold depletion of ILC3 in the colon and an increase in the levels of NK cells in tonsils and oral lymph nodes.
19 uction in c-Cbl in colonic epithelium is likely to increase the levels of nuclear beta-catenin in the intestinal crypt, a
20 aginal Ag-specific T cell responses whereas CCL28 increased the levels of specific T cells only in the vaginal mucosa.
21 the endoplasmic reticulum (ER) to phagosomes, and increases the levels of peptide-empty MHC-I conformers that can be load
22 nsgenic B301 roots lowers the frequency of HR and increases the levels of successful SG4 parasitism and overexpression de
23 ief from excess levels of non-native proteins by increasing the levels of folding factors removed the feedback inhibition
24                                              Interestingly, the levels of endogenous c-Myc mRNA and protein were augmente
25                                    According to literature, the levels of acrylamide in coffee vary with the percentage o
26                          Serum samples were used to measure the levels of tumor necrosis factor-alpha (TNF-alpha) and cyt
27 e treatment of bovine fibroblasts with UNC0638 did mitigate the levels of H3K9me2.
28 and we demonstrated that ablation of SRSF6 did not modulate the levels of the Httexon1 transcript.
29                                                   Moreover, the levels of fructose and glucose significantly increased du
30                  In the liver of the mice treated with OCA, the levels of cytochrome P450 potentially involved in VPA met
31                     In conclusion, we identified changes of the levels of several proteins and thus of the metabolic path
32                                              In particular, the levels of antigen expression at the cell surface may dete
33 s the potential exposure of ABX to the brain, we quantified the levels of each ABX in the brain by liquid chromatography-
34      Bead-based epitope assay (BBEA) was used to quantitate the levels of epitope-specific (es)IgA, esIgE, esIgD, esIgG(1
35 ransmembrane trafficking protein previously known to reduce the levels of amyloid-beta, which is critical in the pathogen
36 e led to numerous clinical trials that have aimed to reduce the levels of circulating insulin and to abrogate insulin sig
37 e models, in which autophagy upregulation is able to reduce the levels of toxic proteins, ameliorate signs of disease, an
38 g protein 1 (SAMHD1), a dNTP triphosphohydrolase, regulates the levels of cellular dNTPs through their hydrolysis.
39                                               Of six SCFAs, the levels of two differed significantly (p-value < 0.05, FC
40                               Importantly, we observed that the levels of gC are lower in the absence of pUL47.
41                                           Here we show that the levels of Bim, a BH3-only protein, are significantly incr
42 or tau transgene alone and interestingly without changes to the levels of tau expression, phosphorylation or aggregation.
43          The affected levels in development corresponded to the levels of reduction in pathogenicity, suggesting that Ppa
44 bolites associated with increased lifespan, and upregulates the levels of tyrosine-derived neuromediators.
45 id levels were increased 2 weeks after tendon release, when the levels of high-energy phosphates and glycerophospholipids
46 activation of CPC-3 was abolished under conditions in which the levels of charged tRNAs were altered.
47 rization properties of SecA were strongly compromised while the levels of individual SecYEG translocon components, SecA a
48 ly), involved in vitamin E metabolism, were associated with the levels of octadecanedioate and vitamin E metabolites (gam
49             Comparable events are observed clinically, with the levels of cf-mt-DNA elevated in older deceased organ dono
50 2% for H3K27me3) of genes associated with DBSs matched with the levels of gene expression inferred from RNA-seq data; (v)