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1                                                             The possibility that an alternate nucleophilic aryl substitut
2  (VEGF) results in thrombotic microangiopathy, we addressed the possibility that Gsalpha knockout may result in impaired
3 DS, the potential for a differential response to drugs, and the possibility that the wrong targets have been studied.
4 h the biophysical mechanisms of animal magnetoreception and the possibility that weak, low-frequency anthropogenic electr
5 ength of microtubule-centrosome attachments is unknown, and the possibility that mechanical force might regulate centroso
6 ence of bronchial hyperresponsiveness and inflammation, but the possibility that airway nerves are dysfunctional has not
7                                                To determine the possibility that SNs are missed, this feasibility study c
8 annel function in maintaining ASL homeostasis and emphasize the possibility that pirfenidone could be employed as a novel
9                                      This study establishes the possibility that the pattern of circulating miRNAs may id
10                          This observation led us to explore the possibility that resident macrophage/monocyte-like cells
11                                                  We explore the possibility that HIV-1 proteins leverage their multifunct
12 ring WS-linked gain-of-function Cxcr4 mutation, we explored the possibility that the lymphopenia in WS arises from defect
13                                             This allows for the possibility that Ceres experienced modest ice-rock fracti
14 ansporters and their regulators were enriched, highlighting the possibility that these miRNAs could modulate key renal tu
15                                     This finding highlights the possibility that the bioactivity of such CECs may be effe
16                                        Here, we investigate the possibility that mRNA structures facilitate the 3'-end pr
17                                             We investigated the possibility that autophagy and polyploidization might als
18 hose that induce plasticity at chemical synapses, and offer the possibility that calcium-regulated mechanisms may also le
19 at also exhibited allosteric agonist activity, leaving open the possibility that direct activation by allosteric agonism,
20                         To better understand this, we probe the possibility that SOA from limonene+NO3 and beta-pinene+NO
21 can be induced by simple morphological variation, and raise the possibility that changes in swimming pattern may be trigg
22  timescales relevant to visually guided behaviors and raise the possibility that propagating activity patterns serve neur
23 gered by the vesicle docking C2B-PIP2 interaction and raise the possibility that Syt1 rings could pre-form on the synapti
24 ard to the pathogenesis of this enigmatic disease and raise the possibility that therapies specifically directed at these
25                                            These data raise the possibility that pathological aggregation could compromis
26                                            These data raise the possibility that persistent or occult neurologic and lymp
27                                        These findings raise the possibility that targeting NRP1 or its NCD interactors ma
28                             Overall, our observations raise the possibility that local production of astrocyte proteins m
29                                         These results raise the possibility that genetic variants affect behavioral drive
30                                         These results raise the possibility that other TPR domains may be similarly influ
31                                           Our results raise the possibility that septic patients have impaired insulin cl
32  of the NAc in behavioral responses to AMPH, and they raise the possibility that modulation of PV+ interneuron function m
33  distinguish between folded and unfolded EGF repeats raised the possibility that they participate in a quality control pa
34                                   Analysis from TCGA raised the possibility that hyperactive PPAR signaling, either due t
35                                                 This raises the possibility that an epigenetic process might operate duri
36                                                 This raises the possibility that anatomical connectivity between the PFC
37                                                 This raises the possibility that HMBA or related small molecules may be e
38  varies systematically with imposed conditions, this raises the possibility that it may be used as a strain marker in wel
39                    If this event is not unique, this raises the possibility that other evolutionarily new plant metabolit
40                                                 This raises the possibility that, at least for some CS mutations, sulfony
41 isease evolution than was previously understood and raising the possibility that an age-related clonal process of preleuk
42 diated signalling that precedes pathogen encounter, raising the possibility that small molecules targeting SFKs could mod
43 with the amount of adducts formed by endogenous FA, raising the possibility that epigenetic mechanisms may contribute to
44 uroimaging studies in substance-use disorders, thus raising the possibility that neural processing of negative affect may
45  and developing olfactory system of the six species suggest the possibility that specific, non-random components of the d
46                                      This finding suggested the possibility that exogenous viral infections could likewis
47                                          These data support the possibility that public health initiatives targeting prim
48                          Available clinical results support the possibility that effects of LABA/LAMA combinations on hyp
49 that RBK1 directly phosphorylates ROP4 and ROP6, supporting the possibility that RBK1 effects on auxin-responsive cell ex
50 latelets to kill S. aureus directly; and, second, we tested the possibility that platelets enhance macrophage phagocytosi

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